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70 Cards in this Set

  • Front
  • Back
LIST FLOW CHART TO NERVOUS SYSTEM


NERVOUS SYSTEM


/ \


CNS PNS


/ \


AUTONOMIC SOMATIC


/ \


SYMPATHETIC PARASYMPATHETIC


(GAS) (BREAK)

PERIPHIAL NERVOUS SYSTEM

ALL NERVOUS TISSUE OUTSIDE THE CENTRAL NERVOUS SYSTEM
CENTRAL NERVOUS SYSTEM

CONSISTS OF THE BRAIN AND SPNAL CORD
AUTONOMIC NERVOUS SYSTEM

PART OF THE PERIPHIAL NERVOUS SYSTEM AND CONTROLS CRUCIAL CARDIO, RESP, DIGESTIVE, AND REPRODUCTIVE FUNCTIONS "AUTOMATIC"
SOMATIC NERVOUS SYSTEM

CONTROLS VOLUNTARY "MOTOR" FUNCTIONS
SYMPATHETIC NERVOUS SYSTEM


FIGHT OR FLIGHT RESPONSE CONTROLS STRESS INDUCED FUNCTIONS I.E. HEART BEATING FASTER BREATHING FASTER ECT...


ARISES FROM THORACIC AND LUMBAR REGION OF SPINE


PARASYMPATHETIC NERVOUS SYSTEM

FEED AND BREED OR REST AND DIGEST RESPONCE CONTROLS VEGITATIVE FUNCTIONS I.E. DIGESTION ARISES FROM BRAIN STEM AND SACRAL REGION OF SPINAL CORD (RUN THROUGH THE FACIAL NERVES)
ACETYLCHOLINE (ACh)

USED IN THE PREGANGLIONOC NERVES OF THE PARASYMPATHETIC NERVOUS SYSTEM. SYNAPSES THAT USE IT ARE CALLED CHOLINERGIC
NOREPINEPHERINE (NATURALLY OCCURING)

HORMONE


PRIMARY POSTGANGLIONIC TRANSMITTER IN SYMPATHETIC NERVOUS SYSTEM.


SYNAPSES THAT USE IT ARE CALLED ADREGENIC


CONSTITUTE 20% OF WHAT IS RELEASED BY ADRENAL MEDELLA

α1 RECEPTOR

CAUSE PERIPHIAL VASO CONSTRICTION

α2 RECEPTOR

INHIBIT THE OVERRELEASE OF NOREPINEPHERINE IN THE SYNAPSE

ß1 RECEPTORS

CAUSE AN INCREASE IN HEART RATE, CARDIAC CONTRACTILITY FORCE, AND CARDIAC AUTOMATICITY AND CONDUCTION
ß2 RECEPTORS

CAUSE VASODIALATION AND BRONCHODIALATION
ß3 RECEPTORS
CAUSES BREAKDOWN OF FAT AND GENERATION OF HEAT
SYMPATHOMIMETICS

MEDICATIONS THAT ACT ON THE SYMPATHETIC NERVOUS SYSTEM

SYMPATHOLYTICS

MEDICATIONS THAT INHIBIT SYMPATHETIC NERVOUS SYSTEM FUNCTIONS
PARASYMPATHOLYTIC (ANTICHOLINERGENIC)


MEDICATION THAT BLOCKS ACTIONS OF THE PARASYMPATHETIC NERVOUS SYSTEM


I.E. ATROPINE


PARASYMPATHEMEMIC (CHOLINERGENIC)
MEDICATIONS THAT ACT ON THE ACTIONS OF THE PARASYMPATHETIC NERVOUS SYSTEM
ADRENAL MEDELLA

ENDOCRINE GLAND THAT SITS ATOP THE KIDNEY AND SECRETES EPI AND NOREPI

EPINEPHERINE


SYMPATHETIC AGONIST


VASOCONSTICTOR


↑HR


↑BP


↑CARDIAC CONTRACTILITY FORCE


NOREPINEPHERINE


SYMPATHETIC AGONIST


PERIPHIAL VASO CONSTRICTER


↑BP IN CARDIOGENIC SHOCK AND HYPOTENSIVE EMERGENCIES AS LONG AS IT IS NOT RELATED TO HYPOVOLEMIA



ATRPOINE


Primary agent in organophosphate poisoning




MEDICATION NAMES



CHEMICAL: CHEMICAL MAKEUP OF THE DRUG


BRAND NAME:TRADE NAME, DEVELOPED BY COMPANY IDENTIFIES AS PROPRIATARY PROPERTY USUALY WITH COPYRIGHT SYMBOL


GENERIC: SHORTHAND VERSION OF THE CHEMICAL NAME LESS EXPENSIVE BUT SAME QUALITY

DRUG ADMINISTRATION ROUTES


ENTERNAL:PASSES THROUGH THE G.I. TRACT


PARENTRAL: LIQUID DOES NOT PASS THROUGH THE G.I. TRACT I.V. I.O. I.M. SUB Q


ORAL: THROUGH THE MOUTH


BUCCAL: BETWEEN MOUTH AND CHEEK ON MUCOS MEMBRANES


RECTAL: YOU KNOW WHERE


ETT: ET TUBE


PHARMACOKINETICS

THE STUDY OF HOW A DRUG ENTERS MOVES THROUGH, AND EXITS THE BODY

PHARMACODYNAMICS

THE STUDY OF A DRUGS EFFECTS ON THE BODY

OSMOSIS

A MOLECULE MOVING ACROSS A MEMBRANE

DIFFUSION

MOLOCULES INTERMINGLEING

ACTIVE TRANSPORT
THE MOVEMENT OF A SUBSTANCE WITH THE HELP OF ENERGY
FACILITATED TRANSPORT

THE MOVEMENT OF A SUBSTANCE WITHOUT THE USE OF ENERGY

INFILTRATION




"BLOWING" THE VEIN OR GOING THROUGH THE VEIN AND PERFUSING FLUIDS IN THE TISSUE OUTSIDE THE VEIN

CLOT OBSTRUCTION

CAN BE FORMED IF A SALINE LOCK IS NOT USED AFTER I.V. ACCESS HAS BEEN ACHIEVED, OR A FLUSH HAS NOT BEEN PERFORMED



COLLOID


SOLUTION THAT CONTAINs LARGE PROTINES THAT CANNOT PASS THROUGH THE CAPLIARY MEMBRANE. REMAIN IN THE CIRCULATORY SYSTEM FOR A LONG TIME


I.E. PLASMANATE,DEXTRAN,HESPAN


CRYSTALLOID


PRIMARY PRE HOSPITAL I.V. FLUIDS CONTAIN ELECTROLYTES AND WATER BUT LACK LARGER PROTINES CLASSIFIED BY THEIR NUMBER OF PARTICALS PER UNIT VOLUME.


I.E. LACTATED RINGERS* * BOTH WILL HAVE 2/3 LOSS IN EXTRACELLULAR SPACE WITHIN 1HR


NORMAL SALINE*


5% DEXTROSE IN WATER


ISOTONIC SOLUTION

EQUAL TO BLOOD PLASMA WILL NOT CAUSE SIGNIFICANT ELECTROLYTE OR FLUID SHIFT

HYPERTONIC SOLUTION

HIGHER CONCENTRATION THAN THAT OF THE CELLS WHEN ADMINISTERED THEY WILL CAUSE FLUID TO SHIFT OUT OF THE INTERCELLULAR SPACE INTO THE EXTRACELLULAR SPACE AND WILL DIFFUSE LATER IN REVERSE ORDER

HYPOTONIC SOLUTION

LOWER CONCENTRATION THAN THAT OF THE CELLS. WHEN ADMINISTERED THEY WILL CAUSE FLUID TO SHIFT OUT OF THE EXTRACELLULAR SPACE INTO THE INTERCELLULAR SPACE AND WILL DIFUSE LATER IN REVERSE ORDER



INTRAOSEOUS ROUTE


I.O. SITES ARE IN THE PROXIMAL TIBIA, MEDIAL MALLEOLUS OF THE DISTAL TIBIA, HUMERAL HEAD, AND STERNUM


INDICATED WHEN I.V. ACCESS CANNOT BE OBTAINED


CONTRAINDICATED IN FRACTURE ON SIDE OF ACCESS, OSTEOPOROSIS AND ESTABLISHMENT OF A PERIPHIAL I.V. SITE


INTRADERMAL INJECTION


LESS THAN 1ML


10-15° ANGLE


25-27GUAGE


FOREARM OR UPPER BACK


DO NOT RUB


LOCAL RESPONSE

SUBCUTANEOUS INJECTION


NO MORE THAN 1 ML


45° ANGLE


WHERE YOU CAN EASILY PINCH SKIN UPPER ARMS, THIGHS AND OCCASIONALY ABDOMEN


24-26 GUAGE




INTRAMUSCULAR INJECTION


CAN DELIVER 5.0ML OR MORE


90° ANGLE


DELTIOD MUSCLE 3-4 FINGERBREADTHS BELOW SHOULDER BUTT, THIGH


21-23 GUAGE


OCULAR MEDICATION DELIVERY

APPLY MEDICATION DROPS OR GEL ONTO CONJUNCTIVE SAC NOT ON EYE ITSELF UNLESS SPECIFICALLY INSTRUCTED. TELL PT. TO HOLD EYE SHUT FOR 1-2 MIN.


O.D. RIGHT


O.S.LEFT


O.U. BOTH

AURAL MEDICATION DELIVERY


EAR


ADMINISTER DROPS INTO EAR CANAL, HAVE PT LAY ON SIDE FOR 10 MIN

SLUDGE


SALVATION, LACRIMATION, URINATION, DIAPHORESIS, GASTROINTESTINAL UPSET, EMESIS




ORGANOPHOSPHATE POISIONING CAUSES SLOWED HEART RATE CYANOSIS VOMITING AND DIARRHEA

ORGANPHOSPHATE POISIONING TREATMENT

ATROPINE, 2PAM (PRALIDOXIME), BENZODIAZEPINES (DIAZEPAM)

AXON



USUALLY A LONG PROCESS WHICH OFTEN PROJECTS TO DISTANT REGIONS OF THE NERVOUS SYSTEM.


AWAY FROM THE CELL BODY


DENDRITES

SHORT BRANCH LIKE PROCESS WHICH BRINGS SIGNALS TO THE CELL BODY
CELL BODY

CELL

MYELIN SHEATH

FATTY WHITE SUBSTANCE THAT SURROUNDS THE AXON SERVING AS AN ELECTRICAL INSULATOR THAT SPEEDS NERVE IMPULSES TO MUSCLES AND OTHER EFFECTORS

DRUG SOURCES (4 TYPES)


ANIMAL:INSULIN, OXYTOCIN


PLANT:MORPHINE, ATROPINE


MINERAL:SODIUM BICARB, CALCIUM CHLORIDE


SYNTHETIC:ADENOSINE,DIAZEPAM,HEP. B VACC.

CROSS TOLERENCE
TOLERENCE FOR A DRUG THAT DEVELOPS AFTER ADMINISTRATION OF A DIFFERENT DRUG I.E. MORPHINE AND OPIODS
POTENTIATION

ENHANCEMENT OF ONE AGENT BY ANOTHER SO THE COMBINED EFFECT IS GREATER THAN THAT ONE AGENT ALONE.


ACTION POTENTIAL



THE CHANGE IN ELECTRICAL POTENTIAL ASSOCIATED WITH THE PASSAGE OF AN IMPULSE ALONG THE MEMBRANE OF A MUSCLE/NERVE CELL

PROPAGATE




TO CAUSE AN ORGANISM TO MULTIPLY BY ANY PROCESS FROM PARENT STOCK. (TO PRODUCE A NEW PLANT)


DEPOLARIZATION
REDUCTION OF A MEMBRANES RESTING POTENTIAL BECOMING LESS NEGATIVE (IONS FLOWING INTO A CELL)
REPOLAROZATION

REFERS TO THE CHANGE IN MEMBRANE POTENTIAL THAT RETURNS TO NEGATIVE VALUE AFTER DEPOLARIZATION



SYNTHISIS



PRODUCTION OF CHEMICAL COMPOUNDS BY COMBINING PARTS OF AN ELEMENT FROM A WHOLE

TRANSMISSION

TO PASS ALONG, OR THE ACT OF TRANSMITTING

ENDOGENOUS

GROWING OR ORIGINATING FROM AN ORGANISIM

PRE-SYNAPTIC

CELL THAT TRANSMITTS



POST-SYNAPTIC

CELL THAT RECEIVES


SYNAPSE




A STRUCTURE THAT PERMITS A NEURON TO PASS A TRANSMISSION TO ANOTHER


DOPAMINERGIC
↑ DOPAMINE RELATED ACTIVITY IN THE BRAIN

METHYLPHENIDATE

(RITALIN) USED FOR ADHD

DILANTIN

TREATMENT OF GRAND MAL SIEZURES

MORPHINE SULPHATE

SEDATION BY BINDING TO OPIATE RECEPTORS. NALOXONE ↓ BP, SYNCOPE, TACHY/BRADYCARDIA

TRICYCLIC ANTIDEPRESSANTS


ANTIDEPRESSANTS THAT CAUSE SIDE EFFECTS


EXTRA PYRAMIDAL SYMPTOMS




MUSCLE TREMORS AND PARKINSINISIM LIKE EFFECTS


MAOI


MAOIS BLOCK THE BREAKDOWN OF ENZYME THAT METABOLIZES MONOAMINE INTO INACTIVE METABOLITES


AVOID RED WINE, AGED CHEESE



XANEX



BENZODIAZEPINE;ROMAZICON IS THE ANTAGONIST TO IT. AVOID ITS USE IF SEIZURES EXIST