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196 Cards in this Set
- Front
- Back
|
DMARDS vs. NSAIDS for RA
|
Start pt. with DMARDs and NSAIDs d/c NSAIDs when DMARDs begin to take effect. Close monitoring required with DMARDs because of toxicity.
|
|
|
colchicine and gout
|
|
|
|
allopurinol and gout
|
|
|
|
see #1
|
|
|
|
calcium interactions
|
|
|
|
how to take a bisphosphonate
|
|
|
|
goal of therapy for osteoporosis
|
|
|
|
montelukast
|
|
|
|
how to use metered dose inhalers (MDI)
|
|
|
|
glucocorticoids & asthma
|
|
|
|
see #10
|
|
|
|
MOA of expectorants
|
|
|
|
combination cold medicines
|
|
|
|
cromolyn
|
|
|
|
use of decongestants for colds
|
|
|
|
disulfiram reaction and PUD meds
|
|
|
|
omeprazole
|
|
|
|
sucralfate
|
|
|
|
misoprostal
|
|
|
|
laxative of choice for abdominal cramping and nausea
|
|
|
|
MOA of various laxatives
|
|
|
|
adverse effects of different types of laxatives
|
|
|
|
lactulose
|
|
|
|
treatment for emetogenic chemotherapy
|
|
|
|
diphenoxylate
|
|
|
|
therapeutic use of sulfasalazine
|
|
|
|
adverse effects of antiemetics
|
|
|
|
definitions from the 1st antibiotics slide (Chap 82)
|
|
|
|
mechanisms of antimicrobial resistance
|
|
|
|
inappropriate use of antibiotics
|
|
|
|
understanding CDC's 12 steps
|
|
|
|
factors contributing to antibiotic resistance
|
|
|
|
|
"Start pt. with DMARDs and NSAIDs d/c NSAIDs when DMARDs begin to take effect. Close monitoring required with DMARDs because of toxicity.
|
|
|
|
"anti-inflammatory agent used only for Gout.
|
|
|
|
"in the initial months of treatment you may see an increase in acute gouty attacks (prevent this with low doses of colchicine). D/C immediately if rash develops because it may be a sign of hypersensitivity syndrome. Allopurinol inhibits drug metabolizing hepatic enzymes so decrease doses of warfarin, mercaptopurine and azathioprine (chemotherapy). Pts. on ampicillin may develop a rash (d/C immediately)- Reduces blood levels of uric acid by inhibiting xanthine oxidase
|
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|
"Ca interactions:
|
|
|
|
"
|
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|
|
"he objective of osteoporosis treatment is to ""reduce the occurrence of fractures"" p.860. (which is why we use drugs to decrease bone resorption or promote bone formation)To reduce bone resorption and increase bone formation
|
|
|
|
" Montelukast: lukotriene receptor blocker
|
|
|
|
" - dosing is accomplished with 1 or 2 puffs
|
|
|
|
" suppresses inflammation & promote synthesis of brochial beta2 receptors and in turn increase their response to beta2 agonists
|
|
|
|
|
|
|
|
loosen phlegm, example Guaifenesin
|
|
|
|
"Usually contain two of more of the following:
|
|
|
|
"OTC in some forms- safest of all anti-asthma medications
|
|
|
|
"Decongestants: activate alpha1 adrengic receptors on nasal blood vessels, causing vasodilation and therefore shrinkage of swollen membranes followed by nasal drainage.
|
|
|
|
"
|
|
|
|
Prilosec, PPI irreversibly inhibits gastric secretion, only dosed once daily 15-30 minutes before meal. Short half life. Short term therapy for ulcers and GERD, d/t irreversible characteristic tx should be limited to 4-8 weeks. Prodrug, active from inside parietal cells in the stomach. Adverse effects: HA, GI (nausea and vomitting), and possibly cancer (carcinoid tumor)
|
|
|
|
"protective barrier up to 6 hrs, adheres to ulcer.
|
|
|
|
"
|
|
|
|
milk of magnesia
|
|
|
|
"1)bulk forming: following ingestion (are nondigestible, nonabsorbable) swell in water to form a viscous solution- softening fecal mass and incr its bulk (volume further enlarged by bacteria)
|
|
|
|
"bulk forming: esophageal obstruction, and intestinal obstruction of not taken with H2O
|
|
|
|
Lactulose: osmotic laxative, also used to reduce blood amonia levels for portal htn, hepatic encephalopathy
|
|
|
|
"want to decrease N/V so we would use:
|
|
|
|
"antidiarrheal
|
|
|
|
"1)effective against acute episodes of mild to moderate ulcerative colitis
|
|
|
|
"1) serotonin receptor antagonists: headache, diarrhea, dizziness, weak, tired, constipation
|
|
|
|
"chemo: suppress imm sys, kills proliferating cells even healthy ones
|
|
|
|
"1) microbes may elaborate drug-metabolizing enzymes: produce enzymes which inactivates antibiotics
|
|
|
|
Untreatable infection, fever of unknown origin, improper dosage, lack identification of organism, and omission of surgical drainage.
|
|
|
|
"Prevent infection
|
|
|
|
using antibiotics promotes the emergence of drug-resistant microbes
|
|
|
DMARDS vs. NSAIDS for RA
|
"Start pt. with DMARDs and NSAIDs d/c NSAIDs when DMARDs begin to take effect. Close monitoring required with DMARDs because of toxicity.
|
|
|
colchicine and gout
|
"anti-inflammatory agent used only for Gout.
|
|
|
allopurinol and gout
|
"in the initial months of treatment you may see an increase in acute gouty attacks (prevent this with low doses of colchicine). D/C immediately if rash develops because it may be a sign of hypersensitivity syndrome. Allopurinol inhibits drug metabolizing hepatic enzymes so decrease doses of warfarin, mercaptopurine and azathioprine (chemotherapy). Pts. on ampicillin may develop a rash (d/C immediately)- Reduces blood levels of uric acid by inhibiting xanthine oxidase
|
|
|
see #1
|
|
|
|
calcium interactions
|
"Ca interactions:
|
|
|
how to take a bisphosphonate
|
"
|
|
|
goal of therapy for osteoporosis
|
"he objective of osteoporosis treatment is to ""reduce the occurrence of fractures"" p.860. (which is why we use drugs to decrease bone resorption or promote bone formation)To reduce bone resorption and increase bone formation
|
|
|
montelukast
|
" Montelukast: lukotriene receptor blocker
|
|
|
how to use metered dose inhalers (MDI)
|
" - dosing is accomplished with 1 or 2 puffs
|
|
|
glucocorticoids & asthma
|
" suppresses inflammation & promote synthesis of brochial beta2 receptors and in turn increase their response to beta2 agonists
|
|
|
see #10
|
|
|
|
MOA of expectorants
|
loosen phlegm, example Guaifenesin
|
|
|
combination cold medicines
|
"Usually contain two of more of the following:
|
|
|
cromolyn
|
"OTC in some forms- safest of all anti-asthma medications
|
|
|
use of decongestants for colds
|
"Decongestants: activate alpha1 adrengic receptors on nasal blood vessels, causing vasodilation and therefore shrinkage of swollen membranes followed by nasal drainage.
|
|
|
disulfiram reaction and PUD meds
|
"
|
|
|
omeprazole
|
Prilosec, PPI irreversibly inhibits gastric secretion, only dosed once daily 15-30 minutes before meal. Short half life. Short term therapy for ulcers and GERD, d/t irreversible characteristic tx should be limited to 4-8 weeks. Prodrug, active from inside parietal cells in the stomach. Adverse effects: HA, GI (nausea and vomitting), and possibly cancer (carcinoid tumor)
|
|
|
sucralfate
|
"protective barrier up to 6 hrs, adheres to ulcer.
|
|
|
misoprostal
|
"
|
|
|
laxative of choice for abdominal cramping and nausea
|
milk of magnesia
|
|
|
MOA of various laxatives
|
"1)bulk forming: following ingestion (are nondigestible, nonabsorbable) swell in water to form a viscous solution- softening fecal mass and incr its bulk (volume further enlarged by bacteria)
|
|
|
adverse effects of different types of laxatives
|
"bulk forming: esophageal obstruction, and intestinal obstruction of not taken with H2O
|
|
|
lactulose
|
Lactulose: osmotic laxative, also used to reduce blood amonia levels for portal htn, hepatic encephalopathy
|
|
|
treatment for emetogenic chemotherapy
|
"want to decrease N/V so we would use:
|
|
|
diphenoxylate
|
"antidiarrheal
|
|
|
therapeutic use of sulfasalazine
|
"1)effective against acute episodes of mild to moderate ulcerative colitis
|
|
|
adverse effects of antiemetics
|
"1) serotonin receptor antagonists: headache, diarrhea, dizziness, weak, tired, constipation
|
|
|
definitions from the 1st antibiotics slide (Chap 82)
|
"chemo: suppress imm sys, kills proliferating cells even healthy ones
|
|
|
mechanisms of antimicrobial resistance
|
"1) microbes may elaborate drug-metabolizing enzymes: produce enzymes which inactivates antibiotics
|
|
|
inappropriate use of antibiotics
|
Untreatable infection, fever of unknown origin, improper dosage, lack identification of organism, and omission of surgical drainage.
|
|
|
understanding CDC's 12 steps
|
"Prevent infection
|
|
|
factors contributing to antibiotic resistance
|
using antibiotics promotes the emergence of drug-resistant microbes
|
|
|
tetracyclines - bind with Ca and prevent absorption
|
interacts with absorption of antibiotics making it so that neither the Ca nor the A/B are absorbed
|
|
|
oxalic acid - found in coffee and legumes
|
suppresses Ca absorption
|
|
|
phytic acid - found in OJ, bran and whole grains
|
prevents Ca absorption"
|
|
|
Adverse effects
|
rebound congestion, with prolonged use, effectiveness can wear off, CNS excitation, restlessness, irritability, anxiety, insomnia, and can be hazardous for heart patients especially d/t the cardiovascular vasoconstriction. These drugs are subject to abuse and therefore sometimes will be behind the counter at the pharmacy.
|
|
|
DMARDS vs. NSAIDS for RA
|
"Start pt. with DMARDs and NSAIDs d/c NSAIDs when DMARDs begin to take effect. Close monitoring required with DMARDs because of toxicity.
|
|
|
colchicine and gout
|
"anti-inflammatory agent used only for Gout.
|
|
|
allopurinol and gout
|
"in the initial months of treatment you may see an increase in acute gouty attacks (prevent this with low doses of colchicine). D/C immediately if rash develops because it may be a sign of hypersensitivity syndrome. Allopurinol inhibits drug metabolizing hepatic enzymes so decrease doses of warfarin, mercaptopurine and azathioprine (chemotherapy). Pts. on ampicillin may develop a rash (d/C immediately)- Reduces blood levels of uric acid by inhibiting xanthine oxidase
|
|
|
see #1
|
|
|
|
calcium interactions
|
"Ca interactions:
|
|
|
how to take a bisphosphonate
|
"
|
|
|
goal of therapy for osteoporosis
|
"he objective of osteoporosis treatment is to reduce the occurrence of fractures p.860. (which is why we use drugs to decrease bone resorption or promote bone formation)To reduce bone resorption and increase bone formation
|
|
|
montelukast
|
" Montelukast: lukotriene receptor blocker
|
|
|
how to use metered dose inhalers (MDI)
|
" - dosing is accomplished with 1 or 2 puffs
|
|
|
glucocorticoids & asthma
|
" suppresses inflammation & promote synthesis of brochial beta2 receptors and in turn increase their response to beta2 agonists
|
|
|
see #10
|
|
|
|
MOA of expectorants
|
loosen phlegm, example Guaifenesin
|
|
|
combination cold medicines
|
"Usually contain two of more of the following:
|
|
|
cromolyn
|
"OTC in some forms- safest of all anti-asthma medications
|
|
|
use of decongestants for colds
|
"Decongestants: activate alpha1 adrengic receptors on nasal blood vessels, causing vasodilation and therefore shrinkage of swollen membranes followed by nasal drainage.
|
|
|
disulfiram reaction and PUD meds
|
"
|
|
|
omeprazole
|
Prilosec, PPI irreversibly inhibits gastric secretion, only dosed once daily 15-30 minutes before meal. Short half life. Short term therapy for ulcers and GERD, d/t irreversible characteristic tx should be limited to 4-8 weeks. Prodrug, active from inside parietal cells in the stomach. Adverse effects: HA, GI (nausea and vomitting), and possibly cancer (carcinoid tumor)
|
|
|
sucralfate
|
"protective barrier up to 6 hrs, adheres to ulcer.
|
|
|
misoprostal
|
"
|
|
|
laxative of choice for abdominal cramping and nausea
|
milk of magnesia
|
|
|
MOA of various laxatives
|
"1)bulk forming: following ingestion (are nondigestible, nonabsorbable) swell in water to form a viscous solution- softening fecal mass and incr its bulk (volume further enlarged by bacteria)
|
|
|
adverse effects of different types of laxatives
|
"bulk forming: esophageal obstruction, and intestinal obstruction of not taken with H2O
|
|
|
lactulose
|
Lactulose: osmotic laxative, also used to reduce blood amonia levels for portal htn, hepatic encephalopathy
|
|
|
treatment for emetogenic chemotherapy
|
"want to decrease N/V so we would use:
|
|
|
diphenoxylate
|
"antidiarrheal
|
|
|
therapeutic use of sulfasalazine
|
"1)effective against acute episodes of mild to moderate ulcerative colitis
|
|
|
adverse effects of antiemetics
|
"1) serotonin receptor antagonists: headache, diarrhea, dizziness, weak, tired, constipation
|
|
|
definitions from the 1st antibiotics slide (Chap 82)
|
"chemo: suppress imm sys, kills proliferating cells even healthy ones
|
|
|
mechanisms of antimicrobial resistance
|
"1) microbes may elaborate drug-metabolizing enzymes: produce enzymes which inactivates antibiotics
|
|
|
inappropriate use of antibiotics
|
Untreatable infection, fever of unknown origin, improper dosage, lack identification of organism, and omission of surgical drainage.
|
|
|
understanding CDC's 12 steps
|
"Prevent infection
|
|
|
factors contributing to antibiotic resistance
|
using antibiotics promotes the emergence of drug-resistant microbes
|
|
|
DMARDS vs. NSAIDS for RA
|
"Start pt. with DMARDs and NSAIDs d/c NSAIDs when DMARDs begin to take effect. Close monitoring required with DMARDs because of toxicity.
|
|
|
colchicine and gout
|
"anti inflammatory agent used only for Gout.
|
|
|
allopurinol and gout
|
"in the initial months of treatment you may see an increase in acute gouty attacks (prevent this with low doses of colchicine). D/C immediately if rash develops because it may be a sign of hypersensitivity syndrome. Allopurinol inhibits drug metabolizing hepatic enzymes so decrease doses of warfarin, mercaptopurine and azathioprine (chemotherapy). Pts. on ampicillin may develop a rash (d/C immediately) Reduces blood levels of uric acid by inhibiting xanthine oxidase
|
|
|
see #1
|
|
|
|
calcium interactions
|
"Ca interactions:
|
|
|
how to take a bisphosphonate
|
"
|
|
|
goal of therapy for osteoporosis
|
"he objective of osteoporosis treatment is to reduce the occurrence of fractures p.860. (which is why we use drugs to decrease bone resorption or promote bone formation)To reduce bone resorption and increase bone formation
|
|
|
montelukast
|
" Montelukast: lukotriene receptor blocker
|
|
|
how to use metered dose inhalers (MDI)
|
" dosing is accomplished with 1 or 2 puffs
|
|
|
glucocorticoids & asthma
|
" suppresses inflammation & promote synthesis of brochial beta2 receptors and in turn increase their response to beta2 agonists
|
|
|
see #10
|
|
|
|
MOA of expectorants
|
loosen phlegm, example Guaifenesin
|
|
|
combination cold medicines
|
"Usually contain two of more of the following:
|
|
|
cromolyn
|
"OTC in some forms safest of all anti asthma medications
|
|
|
use of decongestants for colds
|
"Decongestants: activate alpha1 adrengic receptors on nasal blood vessels, causing vasodilation and therefore shrinkage of swollen membranes followed by nasal drainage.
|
|
|
disulfiram reaction and PUD meds
|
"
|
|
|
omeprazole
|
Prilosec, PPI irreversibly inhibits gastric secretion, only dosed once daily 15 30 minutes before meal. Short half life. Short term therapy for ulcers and GERD, d/t irreversible characteristic tx should be limited to 4 8 weeks. Prodrug, active from inside parietal cells in the stomach. Adverse effects: HA, GI (nausea and vomitting), and possibly cancer (carcinoid tumor)
|
|
|
sucralfate
|
"protective barrier up to 6 hrs, adheres to ulcer.
|
|
|
misoprostal
|
"
|
|
|
laxative of choice for abdominal cramping and nausea
|
milk of magnesia
|
|
|
MOA of various laxatives
|
"1)bulk forming: following ingestion (are nondigestible, nonabsorbable) swell in water to form a viscous solution softening fecal mass and incr its bulk (volume further enlarged by bacteria)
|
|
|
adverse effects of different types of laxatives
|
"bulk forming: esophageal obstruction, and intestinal obstruction of not taken with H2O
|
|
|
lactulose
|
Lactulose: osmotic laxative, also used to reduce blood amonia levels for portal htn, hepatic encephalopathy
|
|
|
treatment for emetogenic chemotherapy
|
"want to decrease N/V so we would use:
|
|
|
diphenoxylate
|
"antidiarrheal
|
|
|
therapeutic use of sulfasalazine
|
"1)effective against acute episodes of mild to moderate ulcerative colitis
|
|
|
adverse effects of antiemetics
|
"1) serotonin receptor antagonists: headache, diarrhea, dizziness, weak, tired, constipation
|
|
|
definitions from the 1st antibiotics slide (Chap 82)
|
"chemo: suppress imm sys, kills proliferating cells even healthy ones
|
|
|
mechanisms of antimicrobial resistance
|
"1) microbes may elaborate drug metabolizing enzymes: produce enzymes which inactivates antibiotics
|
|
|
inappropriate use of antibiotics
|
Untreatable infection, fever of unknown origin, improper dosage, lack identification of organism, and omission of surgical drainage.
|
|
|
understanding CDC's 12 steps
|
"Prevent infection
|
|
|
factors contributing to antibiotic resistance
|
using antibiotics promotes the emergence of drug resistant microbes
|
|
|
DMARDS vs. NSAIDS for RA
|
"Start pt. with DMARDs and NSAIDs d/c NSAIDs when DMARDs begin to take effect. Close monitoring required with DMARDs because of toxicity.
|
|
|
colchicine and gout
|
"anti inflammatory agent used only for Gout.
|
|
|
allopurinol and gout
|
"in the initial months of treatment you may see an increase in acute gouty attacks (prevent this with low doses of colchicine). D/C immediately if rash develops because it may be a sign of hypersensitivity syndrome. Allopurinol inhibits drug metabolizing hepatic enzymes so decrease doses of warfarin, mercaptopurine and azathioprine (chemotherapy). Pts. on ampicillin may develop a rash (d/C immediately) Reduces blood levels of uric acid by inhibiting xanthine oxidase
|
|
|
see #1
|
|
|
|
calcium interactions
|
"Ca interactions:
|
|
|
how to take a bisphosphonate
|
"
|
|
|
goal of therapy for osteoporosis
|
"he objective of osteoporosis treatment is to reduce the occurrence of fractures p.860. (which is why we use drugs to decrease bone resorption or promote bone formation)To reduce bone resorption and increase bone formation
|
|
|
montelukast
|
" Montelukast: lukotriene receptor blocker
|
|
|
how to use metered dose inhalers (MDI)
|
" dosing is accomplished with 1 or 2 puffs
|
|
|
glucocorticoids & asthma
|
" suppresses inflammation & promote synthesis of brochial beta2 receptors and in turn increase their response to beta2 agonists
|
|
|
see #10
|
|
|
|
MOA of expectorants
|
loosen phlegm, example Guaifenesin
|
|
|
combination cold medicines
|
"Usually contain two of more of the following:
|
|
|
cromolyn
|
"OTC in some forms safest of all anti asthma medications
|
|
|
use of decongestants for colds
|
"Decongestants: activate alpha1 adrengic receptors on nasal blood vessels, causing vasodilation and therefore shrinkage of swollen membranes followed by nasal drainage.
|
|
|
disulfiram reaction and PUD meds
|
"
|
|
|
omeprazole
|
Prilosec, PPI irreversibly inhibits gastric secretion, only dosed once daily 15 30 minutes before meal. Short half life. Short term therapy for ulcers and GERD, d/t irreversible characteristic tx should be limited to 4 8 weeks. Prodrug, active from inside parietal cells in the stomach. Adverse effects: HA, GI (nausea and vomitting), and possibly cancer (carcinoid tumor)
|
|
|
sucralfate
|
"protective barrier up to 6 hrs, adheres to ulcer.
|
|
|
misoprostal
|
"
|
|
|
laxative of choice for abdominal cramping and nausea
|
milk of magnesia
|
|
|
MOA of various laxatives
|
"1)bulk forming: following ingestion (are nondigestible, nonabsorbable) swell in water to form a viscous solution softening fecal mass and incr its bulk (volume further enlarged by bacteria)
|
|
|
adverse effects of different types of laxatives
|
"bulk forming: esophageal obstruction, and intestinal obstruction of not taken with H2O
|
|
|
lactulose
|
Lactulose: osmotic laxative, also used to reduce blood amonia levels for portal htn, hepatic encephalopathy
|
|
|
treatment for emetogenic chemotherapy
|
"want to decrease N/V so we would use:
|
|
|
diphenoxylate
|
"antidiarrheal
|
|
|
therapeutic use of sulfasalazine
|
"1)effective against acute episodes of mild to moderate ulcerative colitis
|
|
|
adverse effects of antiemetics
|
"1) serotonin receptor antagonists: headache, diarrhea, dizziness, weak, tired, constipation
|
|
|
definitions from the 1st antibiotics slide (Chap 82)
|
"chemo: suppress imm sys, kills proliferating cells even healthy ones
|
|
|
mechanisms of antimicrobial resistance
|
"1) microbes may elaborate drug metabolizing enzymes: produce enzymes which inactivates antibiotics
|
|
|
inappropriate use of antibiotics
|
Untreatable infection, fever of unknown origin, improper dosage, lack identification of organism, and omission of surgical drainage.
|
|
|
understanding CDC's 12 steps
|
"Prevent infection
|
|
|
factors contributing to antibiotic resistance
|
using antibiotics promotes the emergence of drug resistant microbes
|
