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110 Cards in this Set

  • Front
  • Back
Peripheral Arterial Disease
Arterial Ulcers
Buerger's Disease
Raynaud's Disease
Acute Arterial Occlusion
are all what type of peripheral vascular diseases?
Arterial Disorders
Thrombophlebitis
Chronic venous insufficiency
Venous ulcers
Pulmonary emboli
are all what type of peripheral vascular diseases?
Venous Disoorders
What are the 8 P's of Vascular Assessment?
Pain
Pallor (color)
Pulse
Parasthesis
Paralysis
Puffiness
Pinkies (cap. refill)
Poikilothermia (Temp)
In the three P's, where will you assess pain?
In the extremity.
In the three P's what colors can the pallor be?
Pale, bright red, bluish, brownish
In the three P's, where will you take a pulse?
Dorsalis Pedis and Posterior Tibial
In the three P's, what do you do if you can't find a pulse?
Get a dopler if you cannot find a pulse.
In the three P's, what would indicate parasthesis?
Do they have numbness or tingling in the affected area?
In the three P's, what would indicate paralysis?
Can they move the affected area?
In the three P's, what would be a sign of puffiness?
Edema or not.
In the three P's, what would you look for in pinkies?
Capillary refill - less than three seconds is normal.
In the three P's, what would you look for in poikilothermia (temp)?
Is the affected are cool, warm?
If the affected area is cool in assessing poikilothermia (temp), what would you do next?
Feel up the leg until you feel warmth. This is probably the spot where it is occluded.
Predominently in males and in the legs, seen in the 6th to 8th decades, commoncirculatory problem which narrowed arteries reduce blood flow to your limbs, may be completely asymptomatic, symptoms usually occur when blockages are greater than or equal to 60%, caused by atherosclerosis or acute thrombus, if you have PAD you are at increased risk for CVA and MI?
Definition of Peripheral Arterial Disease
Arteries become narrowed and blood flow decreases in?
Artherosclerosis
What builds up collateral circulation?
Exercise
Intermittent claudication, leg numbness or weakness, cool lower extremities, sores on feet and legs that don't heal, hair loss of feet and legs, change in toenails, diminished or absent pedal or post tib pulses, rest pain, when les are elevated pale and cyanotic, deep red rubor when legs are dependent,
are symptoms of?
PAD
What is the most common symptom of PAD?
Intermittent claudication
Occurs when a muscle is not getting enough oxygen, then it is exercised?
Intermittent claudication
What kind of change in the toenails are we looking for as a symptom of PAD?
Thick and ridged toenails.
In PAD, this is described as pain worse than a toothache and excrutiating when they put their foot up. It usually starts in the big toe then spreads?
Rest pain
Smoking
Obesity
Diabetes
HTN
Hyperlipidemia
are what kind of risk factors related to PAD?
Treatable or modifiable
Increasing age
Family hx
increased levels of CRP and homocysteine
are what kind of risk factors related to PAD?
Not modifiable
Physical exam (8 P's)
Ankle brachial index (ABI)
Ultrasound
MRA
Arteriogram
Blood tests
are what for PAD?
Diagnosis and Tests
What is the normal resting ankle-brachial index?
1 or 1.1
This means that your blood pressure at your ankle is the same or greater than the pressure at your arm, and suggest that you do not have significant narrowing or blokage of blood flow?
A normal resting ankle-brachial index of 1 or 1.1.
A resting ankle-brachial index of less than 1 is?
Abnormal
If the ABI is <0.95?
Significant narrowing
If the ABI is <0.8?
Intermittent claudication with exercise.
If the ABI is <0.4?
Symptoms may occur when at rest.
If the ABI is <0.25?
Severe limb-threatening PAD.
Pre Procedure - H&P and pt education, labs, IV and IVF
Intra Procedure - Conscious sedation
Post Procedure - Line removal, lying flat, increase po fluids
What procedure is this?
Arteriogram (femoral)
STOP SMOKING
Lifestyle modification to reduce risk factors
Exercise to increase collateral circulation
Medications,
are what for PAD?
Collaborative Treatment
Medications used in Collaborative Treatment in PAD

Pletal, Trental?
Medications used in Collaborative Treatment in PAD

Heorrheologic
Medications used in Collaborative Treatment in PAD

Statins?
Medications used in Collaborative Treatment in PAD

Cholesterol Lowering Agents
Medications used in Collaborative Treatment in PAD

CA channel blockers, ACE inhibitors, Beta blockers?
Medications used in Collaborative Treatment in PAD

Antihypertensives
Medications used in Collaborative Treatment in PAD

Plavix, ASA
Medications used in Collaborative Treatment in PAD

Antiplatelets
Ineffective peripheral tissue perfusion
Chronic pain
Activity Intolerance
Riskk for impaired skin integrity
are Nursing Diagnosis for?
PAD
Exercise for collateral circulation
Keep legs and feet dependent
Keep legs and feet warm (no direct heat, wool socks, etc.)
are intervention for what nursing diagnosis related to PAD?
Interventions for the Nursing diagnosis of, Ineffective peripheral tissue perfusion.
Pharmacologic (analgesia as ordered)
Nonpharmacologic (stress reduction techniques, alternative therapy)
are intervention for what nursing diagnosis related to PAD?
Interventions for the Nursing diagnosis of Chronic Pain.
Pace ativities
Gradual increase in duration and intensity of exercise
Rest with dependent extremities if claudication develops,
are intervention for what nursing diagnosis related to PAD?
Interventions for the Nursing diagnosis of Activity Intolerance.
Assess feet and legs daily
Keep legs and feet clean and dry
bad cradle and float heels,
are intervention for what nursing diagnosis related to PAD?
Interventions for the Nursing diagnosis of Risk for impaired skin integrity.
Done for progressive severe symptoms, tissue ischemia, symptoms affecting quality of life, or gangrenous and pregangrenous wounds?
Revascularization
Angioplasty and Stent placement
Artherectomy
Endarderectomy Surgery
Bypass Surgery (Femoral-femoral bypass, femoral-popliteal bypass, femoral-tibial bypass, axillo-femoral bypass)
Amputation (last resort, BKA - below knee amputation, AKA - above knee amputation)
are part of?
Revascularization
Check ACT if < 150 may pull line if no closure device used
Keep affected leg straight and HOB flat for 2-6 hours as ordered
Monitor femoral site for bleeding or heatoma
Assess pedal puls, VS and sit q15min x4, then q30min x4, then q1h x4
Educate patient on what to do if bleeding occurs
Provide plavix as ordered and educate patient,
are what part of Stent Procedure?
Post Stent Procedure
What should you educate the patient to do if bleeding occurs after a stent procedure?
Apply pressure and get help.
If the patient needs to cough or sneeze after a stent procedure, what should they do?
Apply pressure at the site.
What does the effectiveness of an endarderectomy depend on?
Particular area of blockage
Extent of arterial blockage
What two types of bypass grafting surgery are used in PAD?
Aortal bypass grafting surgery and Femoral bypass grafting surgery
Keep SBP <160
Neurovascular checks q1h for 1st 4h then q2h (use 8 P's)
Assess dressing for drainage with NV checks
TCDB q1WA and IS q2WA
Early ambulation, OOB for all meals, IVF and give meds as ordered, stict I/O's, pain control,
are postoperative care for?
Endarderectomy, Femoral Popliteal Bypass, and Bypass Grafting Surgeries
Distal to area of diminished blood supply, wound margins are even, can be deep or superficial, wound beds may be pale and have dry necrotic eschar and exposed tendons, usually don't drain much, surrounding tissues shine - light- hairless, usually very painful - pain reduced by dependent positiona dn worsened with elevation?
Arterial ulcers
Also known as Thromboangitis obliterans (TAO)
Rare d/o characterized by inflammation and spasms of small and medium arteris and veins
Leads to blockages of arms and legs causing clots to form and ischemia of limbs
Occurs in males <40 who smoke, may have gentic link?
Beurger's Disease
Episodic vasospactic disorder of small cutaneous arteries usually in fingers/toes
Occurs in young women - freq. associated with collagen disease
Precipitated by exposure to cold, emotional upset, smoking and caffeine, Characterized by white, blue, red color changes
Treatment not required-self limiting?
Raynaud's Disease
Develops quickly and abruptly, caused by thrombus, embolism, or trauma
More common in patients with Afib, MI, Prosthetic valves, Cardiomyopathy, Infective endocarditis
Thrombus lodges at sites of bifurcation and atherosclerotic blockage
Blood flow diminished or absent distal to thrombus
Symptoms depend on size and location of thrombus?
Acute Arterial Blockage
Embolus
Thrombosis
Trauma?
Type of acute arterial blockage
Severity depends on if there is collateral flow, absent distal pulses, sever pain in extremity, pale, cyanotic, or mottled extremity, may have line of demarcation, cold or cool to touch extremity, parasthesia or paralysis of extremity, must restore flow to prevent tissue death?
Manifestations of acute arterial blockage
Usually apparent by the signs and symptoms
8 P's of vascular assessment
Arteriogram is used to determine the size, location of occlusion, and for?
Diagnosis of Acute Arterial Blockage
Anticoagulant therapy with IV Heparin
Immediate Embolectomy (if no done within 4-6h can have renal failure, compartment syndrome, or ARDS
Thromboendarterectomy with grafting
Intra-arterial infusion of thrombolytic agent (-r-tPA, streptokinase, or urokinase - bleeding complications - monitor labs)
Oral anticoagulation,
Treatment for?
Acute arterial blockage
Also called venous thrombus
Most common disorder of the veins
Formation of a clot in association with inflammation of the vein
Superficial or deep?
Thrombophlebitis
Venous Stasis
Increased coagulability of blood
Vessel damage
this is called?
Virchow's Triad
Vessel Trauma leads to platelets going to injured site and clot forms with platelets and fibrin this leads to RBC trapped in fibrin network and clot grows and leads to inflammatory response triggered causing pain, redness and swelling at the area of the thrombus. Pieces my break loose and travel through circulation (emboli). Fibroblasts invade thrombus which scars the vein and destroys valves (valve damage is permanent)?
Pathophysiology of acute arterial blockage
Dysfunctional valves
Inactive muscles (obesity, CHF, no regular exercise, immobility, preganancy, recent post-partum, atrial fibrillation, orthopedic injuries?
Venous stasis
Hematologic disorders
Cancers
Systemic Infections
Estrogen based oral contraceptives
Hormone replacement
Smoking?
Hypercoagulability states
Damaged endothelium has decreased fibrinolytic properties predisposing development of thrombus (Presence of IV cath longer than 48 hours, IV therapy with high dose antibiotics, K+, chemotherapy or hypertonic solutions, contaminated IV equipment?
Endothelium Damage
Primarily caused by IV and IVF
Palpable firm subcutaneous cordlike vein
Surrounding area tender, reddened and warm
Mild temp elevation
Treatment (warm moist compress, antinflammatories)?
Superficial Thrombophlebitis
Usually asymptomatic, usually in calfs, dull aching pain with ambulation, unilateral leg edema, warm, reddened skin, calf tenderness, +Homan's sign (unreliable), lower extremity edema/cyanosis, upper extremity neck, back, face edema, and cyanosis?
Deep Vein Thrombosis
Pulmonary emboli
Chronic venous insufficiency (valvular destruction, retrograde flow of blood, venous stasis, arterial circulation impaired)?
Complications of DVT
Coagulation studies
Non-invasive venous studies (venous doppler, duplex scanning, plethysmography-done with sono)
Invasive (venogram-causing DVT, Lung scan-VQ scan, Pulmonary arteriogram-gold standard of PE)
Diagnostic tests for DVT
Elevation
Warm moist heat
Compression stockings
Mild oral analgesics
Anticoagulation therapy,
are what type of treatments for DVT?
Conservative
Venous thrombectomy
Vena cava filter (Greenfield) - if the pt can't tolerate oral coag. med's,
are what type of treatments for DVT?
Surgical
Drugs that inhibit certain clotting factors (prevent clot formation, prevent extension of a preformed clot, does not dissolve clot)
All anticoagulants work in the clotting cascade but do so at different points,
these are in reference to?
Anticoagulant therapy
Turns off 3 main activating factors (activated II, X, IX)
Turns off the coagulation pathway and prevents clots from forming
Monitor for Heparin induced thrombocytopenia
Check PTT q6h until stable while on heparin gtt
Protamine is the antidote?
Heparin
Works similar to heparin
More specific for activated factor X than for activated II
Much more predictable anticoagulation response
Frequent lab monitoring is not required
Lovenox (given SC once daily)
Low Melecular Weight Heparins
Inhibits clotting factors II, VII, IX, X
Rely heavily on vitamin K for synthesis
Interferes with the proper production of vitamin K
End result is dysfuntional slotting and prevention of clot formation
Teach patients to eat consistent amount of green leafy vegetables daily
Vitamin K is antidote
Must have blood drawn to monitor levels?
Coumadin
Bleeding
Risk increases with increased dosage
Increased in pts taking ASA, plavix, or other drugs that impair platelet function
Teach pts that bleeding should not occur and to notify the MD and get levels checked if bleeding gums, very heavy menstrual flow, etc.
Discontinue therapy
Reversal agents?
Anticoagulation Side Effects
ASA
Dipyridamole
Plavix and Ticlid
Trental
Aggrenox?
Antiplatelet Agents
Home care -
ted hose (on in morning before legs dependent, off hs)
stop smoking, exercise (daily walks and leg exercises)
avoid constrictive clothing, garters, girdles
avoid contraceptives
Coumadin therapy
get PT levels monitored as ordered
notify MD of bleeding
diet (consistent greens)?
Discharge teaching for DVT prevention
Most commonly caused by DVT
Other causes are trauma, varicose veins, or unknown etiology
Develops due to incompetent valves?
Chronic Venous Insufficiency
Lower leg edema, worse with standing, leg pain and itching that increases with standing, brown, brawny pimentation of LE and foot, stasis dermatitis (causes the itching), thick hard fibrous SC tissues (lipodermasclerosis), normal pulses, ulcerations (superificial, uneven edges, over medial and anterior ankle)?
Manifestations of Chronic Venous Insufficiency
Elevate legs while resting and during sleep
Avoid prolonged sitting or standing
Do not cross your legs
Avoid tight contrictive clothgarters and girdles
Wear TED hose, SCD's, AVI's, keep skin clean, soft, and dry
Walk frequently to prevent venous stasis?
Nursing Care and Treatment of Chronic Venous Insufficiency
Gauze with calamine lotion, glycerin, and zinc oxide
Hardens and place Ace wrap over (left on 1-2 wks)
Unna boot, treatment for CVI
Unna Boot
Dressing changes as ordered for venous ulcers (boric acid, saline wet to dry, topical steroid, antifungal cream, antibiotic cream)
Surgery as last resort for chronic non healing wounds?
Treatments for CVI
Caused by lack of blood flow, pallor on elevation, rubor on dependency, no edema, rest pain, intermittent claudiacation, cool skin, decreased or absent pulse, severe pain if acute, ulcers pale bed, ulcers on toes, feet, shins, even wound edges?
Manifestations of arterial disorders
Caused by incompetent valves or DVT. brown, brawny discoloration, cyanosis on dependency, significant edema, aching pain, stasis dermatitis, normal skin temperature, normal pulse, ulcers pink bed, superficial ulcers, ulcers on ankles, uneven wound edges?
Manifestations of venous disorders
Blood flow obstruction in the pulmonary vascular system by an embolus?
Definition of Pulmonary Embolism
Most arise from deep vein thrombi
May dislodge spontaneously or by mechanical force
Medical emergency?
Pulmonary Embolism
Thrombi in circulation
Obstructing pulmonary arteries
Inflammation
Constrict bronchi
Hemorrhage, infarction, or infiltrates
Increased workload on heart leads to right sided heart failure?
Pathophysiology of Pulmonary Embolism
Depends on size of the emboli and size and number of blood vessels occluded (usually happen abruptly over minutes, dyspnea and tachypnea, tachycardia, cough, chest pain or pleuritic pain, anxiety and impending doom, low grade temp, crackles, hemoptysis, confusion, delirium, or decreaased LOC, syncope, cyanosis?
Pulmonary Embolism symptoms
D-Dimer
CT chest with contrast, V/Q lung scan
Venous studies
ABG
Pulmonary angiography
EKG to r/o cardiac?
Diagnostics in PE
Anticoagulation with heparin bolus and gtt
Start coumadin concurrently as it takes about 4 days to be therapeutic
Monitor for anticoagulation complications
Maintain resp status
Prevention is key (early and frequent ambulation, leg exercises, SCD's, TED hose, AVI boots, no pillows under knees?
Treatment for PE
Trauma
Congenital abnormalities
Peripheral vascular disease (Major cause)
Diabetes Mellitus
Reconstructive
Improve quality of life,
are references to?
Amputations
AKA - above knee amputation
BKA - below knee amputation
these are the class of?
amputations
Open
Closed are ________ of amputations.
Types
ROM exercises: pre-op and post-op
Elevate first 24 hours
Proper body alignment, BKA stump in extended position
Monitor stump/incision
Compression dressing to fit prosthesis
Pain management
No weight bearing for two weeks?
Post op management for amputations
Hemorrhage
Infection
Delayed Healing
Phantom Pain
Contractures
are complications of?
Amputations
Pain
Disturbed body image
Impaired physical mobility
Ineffective tissue perfusion
Grieving
are nursing diagnosis for?
Amputations
INR is used to evaluate this med?
Coumadin
Seen in amputees?
Phantom pain
Vessel damage?
Thrombophlebitis
Superficial wound?
Venous Ulcer
Inferior Vena Cava Interruption?
Greenfield filter
Piece of clot?
Emboli
Diagnostic test?
Arteriogram
Pale bed?
Arterial Ulcer
Hypercoagulability?
Smoking
Brownish discoloration?
Hemoriterosis
Treatment for CVI?
Compression
Used in the diagnosis of DVT/PE?
D-Dimer
Factor X assay used to evaluate this med?
Heparin
Immobility?
Stasis