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113 Cards in this Set

  • Front
  • Back
Chief Complaint & Caution
Headline sentence containing patient identification & data, followed by main reason for visit

Add note if source and reliability are questionable
History of present illness & dimensions of symptoms
Describe each presenting complaint & include dimesions of symptoms such as: LQCSATADA

Location - Where? Does it move
Quality - What does it feel or look like.
Chronology - onset, duration, frequency, evolution
Severity - Does it interrupt activities or sleep
Aggravating and alleviating factors
Treatment - Self treated?
Associated symptoms
Disability & Adaptation - How it has affected you & those around you. How you managed.
Attributions - What do you think caused it, what has it meant to you.
Past Medical history
MPOMAHSETDH

Medical conditions or illnesses
Past hospitalizations
Operations or injuries
Medications
Allergies
Habits - Smoking, Drugs
Sexual Hx - Birth control
Exposures
Travel
Diet
Health maintenance - Regular eye, dental, physicals
Social History
BWRFLRSM

Background
Work
Residence
Family/relationships
Leisure
Religion
Satisfaction/stress
Medical costs
Family History
Parents
Siblings
Children
General - Health problems that run in the family
Dental History
PFLLFP

Past perio treatment
Family history
Last dental visit
Last scaling
Frequency of recall
Problems with treatments?

Home care - Toothbrush, floss, rinse
Three parts of perio probe
Handle Shank Working end
Probing depth vs Attachment level
Probing depth - Gingival margin to deepest point reached by probe

Attachment level - Distance from fixed point like CEJ to deepest point. Not affected by position of gingival margin
Uses of PD probe(5)
Estimate pocket depth & attachment loss
Quantify plaque
Quantify gingival inflammation, recession & width
Approximate configuration of bony defect
Relationship of pocket depth to mucogingival junction
Factors affecting probing
Morphology of root
Inflammation
Depth of pocket
Obstructions

Force
Angulation of probe
Probe width
Shape of probe tip

Local factors
Prior treatments
Reproducibility
Repeated probing
Nabor's probe
Instrument used to detect layout and extent of furcations
Modified Hamp Index
Degrees

1: <3mm horizontal loss of attachment

2: >3mm horizontal loss of attachment but does not encompass total width of furcation

3: Horizontal through and through destruction of PD tissue

4: Visible through and through furcation
Glickman Classification
Grade

I - Incipient or early when pocket is suprabony primarily affects soft tissue. No radio

II - Has horizontal component with communicating furcas. May have vertical bone loss. Maybe radio

III - Through and through with definite radio

IV - Clinically visible furcation
Vertical classification
Subclass
A: 1-3mm vertical depth
B: 4-6mm
C: >7mm

Class A: Destruction up to 1/3 interradicular height
Class B: 2/3
Class C: Into apical 1/3
Automatic constant Force probe
Precision of 0.1mm with a range of 10mm. Sterilizable with constant probing force & digital readout
Tooth mobility
Miller classification

0: No mobility
1: Perceptible mobility
2: <1mm facial-lingually but no apical movement
3: >1mm apical as well as lateral movement
Causes of tooth mobility (6)
Attachment loss
Occlusal trauma
Spread of inflammation
PD surgery (Transient)
Pregnancy
Pathology in Jaws
Fremitus
Palpable with visible movement of tooth when subjected to occlusal forces
Normal height of crestal bone in relationship to CEJ
1.5-2mm apical
Fenestration vs Dehiscence
Window vs complete opening with massive root exposure
Exhostosis
Benign growth of bone on top of bone
Osseous Defects
Suprabony - Horizontal bone loss with 0 walls

Infrabony - Vertical bone loss with 1,2, or 3 walls which affects only 1 tooth. Craters are 2 walls & affects 2 adjacent root surfaces similarl.

Interradicular defects - With furcation involvement
Interradicular defects classifications
Horizontal classification - I,II,III,IV
I - Less than 3mm horizontal attachment loss
II - More than 3mm but not through and through
III - Through and through
IV - Clinically visible furcation

Vertical classification Subclasses A,B,C
A - Vertical bone loss of 3mm or less
B - 4-6mm
C- 7mm or more
Topography of Osseous defects
Soft tissue pockets - Pseudo/gingival, suprabony & infrabony

Anatomic osseous defects - Horizontal vs vertical loss

Remaining osseous walls
Hemiseptum
Osseous defect with only proximal wall remaining
When interproximal bone is more apical than bone on radicular surface
Reverse Architecture
Moat
Circumferential bone loss with four remaining osseous walls
Radiograph uses (7)
Detects:
Boneloss
Widened PDL space
Crown to root ratio
Shape of root to length of root trunk
Root proximity
Implant evaluation
Pathologies
Radiographs in diagnosis of PD
- Not a substitute but only supplement to clinical examination

- Earliest signs of PD must be detected clinically while radiograph is helpful in detecting existing disease
Lamina Dura
Thin radiopaque border adjacent to PDL
Presence of widened PDL space
Combined with fremitus could indicate occlusal trauma
Crown to Root ratio
Extra-alveolar to intraalveolar ratio of tooth not anatomic crown to root ratio

1:1 ratio is limit for favorable prognosis
Chronic & Aggressive Periodontitis classifications
Extent: Localized ≤30% vs Generalized >30%

Severity
Slight: 1-2mm or 0-25% bone loss
Moderate: 3-4mm or 25-50% bone loss
Severe: 5mm or >50% bone loss
Medications that induce overgrowth
Dilantin
Cyclosporin
Ca++ blockers
Factors determining PD diagnosis (5)
- Age of onset
- Rate of progression
- Presence or absence of clinically detectable inflammation
- Extent and pattern of attachment loss
- Signs & symptoms like pain, ulceration, plaque, calculus
Theories of PD disease progression
Continuous paradigm - Localized sites show progressive loss of attachment over time

Random Burst - Activity is random with multiple bursts & extent varies with site

Asynchronous multiple burst - Several sites exhibit bursts over finite period then prolonged inactivity. Other sites have no activity.
Etiology of PD
Plaque & Host response
Tooth accumulated materials
Acquired pellicle
Food particles
Plaque - Biofilm
Calculus
Materia alba
Mediators that determine events in Periodontitis
Proinflammatory/Destructive
- IL-1B
- IL-8
- TNF-a
- Prostaglandin E (PGE)
- Matrix Metalloproteinases (MMP)

Anti-inflammatory/protective
- IL-1ra
- IL - 4
- IL - 10
- Transforming growthfactor B (TGF-B)
- Tissue inhibitor of matrixmetalloproteinases (TIMPs)
Risk Factor vs indicator
Risk Factor - Environmental, behavioral or biologic factor that directly increases probability of disease occuring. If removed, reduces that probability.

Risk Indicator - Probable contributing factor that has not yet been confirmed.
Risk factors associated with PD disease
Diabetes
Smoking
Red Complex - Troponema denticola, Porphyromonas gingivalis, Bacteroides forsythus
Risk indicators associated with PD
Aging, Gender, genetic predispositions, systemic illnesses, stress, calculus, nutrition, home care
Local contributing factors to PD disease (6)
Anatomical factors
Iatrogenic factors
Calculus
Trauma
Chemical injuries
Caries
Prognosis & when determined
Prediction to the progress, course, outcome of disease, & results of treatment

Determined after the diagnosis & before treatment plan
Criteria for successful prognosis (3)
- Recognize, control or eliminate etiological factors
- Ability to correct damage
- Patient's attitude
Factors affecting overall prognosis (5)
Age
Medical status
Risk factors: Smoking, Diabetes
Rate of progression
Patient cooperation & compliance
Factors involving individual prognosis (6)
Percentage bone loss
Probing depth
Mobility
Distribution & type of bone loss - Revealed at time of surgery
Root anatomy - More surface area better for PDL. Root concavities & palatal grooves
Crown to root ratio - 2:1 is good
Furcation involvement factors (5)
- With class III furcation, the most difficult problem & reason for failure is caries
- Maxillary 1st premolar with furcation involvement has poorest Px.
- Mandibular more favorable than maxillary
- Mandibular First molar more favorable than second due to external oblique ridge
- Maxillary molars with buccal & mesial furca more favorable than molars with distal furca due to access
Characteristics of Hopeless teeth (4)
Mobility of Class 3
Bone loss beyond apex
Vertical fractures
Non-restorability due to extensive caries
McGuire & Nunn's system for prognosis
Good - Control of etiological factors & adequate PD support. Easy to maintain by patient

Fair - 25% attachment loss and/or class I furcation involvement

Poor - 50% attachment loss with Class II furcations.

Questionable - Greater than 50% attachment loss resulting in poor crown to root ratio. Mobility of 2 or greater with class II to III furcations. Close to root

Hopeless - Inadequate attachment to maintain tooth. Extraction
Objectives of Periodontal Care (5)
Remove etiologic factor
Maintainable pocket depths
Create maintainable gingival & Osseous architecture
Restore function & Esthetics
Maintenance by patient & doctor
Advantages to shallow Pocket depths (4)
Easier to clean
Less bleeding
Less anaerobes
Less possibility of disease progression
Treatment plans
Initial preparation/therapy:
- Control inflammation
- Remove Accretions
- Control Etx
- Substitute program of homecare
Through - Oral hygiene instruction, Scaling & root planing, Operative, extraction of hopeless teeth

Reevaluation or Presurgical evaluation
- OH status
- PD condition
- Mobility
- Need for PD surgery
- Occlusal adjustment
- Splinting

Periodontal surgery: Sextant analysis
-Specific area & Type of surgery required
- Implants surgery

Post surgical evaluation
Fixed/Removable prosthodontics
PD maintenence
Treatment plan Presentation
- Describe pathogenesis & clinical findings
- Encourage questions
- Discuss length of treatment & costs
- Obtain informed consent
Goal of PD therapy
To preserve dentition in a state of health, comfort & function with appropriate esthetics
Clinical Parameters used to judge success of PD therapy (4)
- Reduction in bleeding upon probing.
- Reduction in probing depth & regain attachment
- Occlusal stability
- Aesthetic satisfaction
Biofilm
An accumulation of microbial cella within a matrix to optomize use of available nutrients.
Effects of plaque removal
Retard calculus formation
Resolve gingival inflammation
Plaque reorganizes supra and subgingivally within 24hrs.
Oral hygiene in Ex
Can be considered a contributing factor or risk indicator
Oral hygiene instructure
Establish need in patient's mind
Motivate
Instruct
Reinfore
Oral Hygiene Evaluation
Question current practices
Allow pt to demonstrate
Examine dentition for plaque, calculus, stain
Toothbrush Bristle characteristics (3)
-Rounded ends
-Bristle hardness is propotional to diameter squared & inversely proportional to length squared.
- Made natural from hogs or artificial nylon
Dentrifice components
Abrasives such as silicon, oxides, & Granular polyvinyl chlorides
Water
Soap or detergent
Flavoring
Therapeutic agents such as flourides
Dentrifice abrasion precautions
Powder is 5x more abrasive than paste & can be a problem for pts with root exposure.

Dentin abraded 25x faster than enamel & 35x for cementum.
Triclosan & pyrophosphate
Triclosan - Anti-plaque
Crest - Anti-calculus
Potassium nitrate & peroxide
Potassium nitrate - Desensitizing agent

Peroxide - Whitening agent
Dentrifice abrasives
Phosphates
- Dicalcium phosphate dihydrate
- Calcium pyrophosphate to reduce calcium ion availability & increase flouride ion

Carbonates
- Sodium bicarbonate - least abrasive
- Calcium carbonate (Chalk)

Silicas - Cleans and thickens toothpaste
Potential side effects of Tartar control toothpaste
Perioral rash & bronchospasm
Stannous flouride
Antibacterial flouride good for plaque/gingivitis, caries, & sensitivity. However, requires stabilization
Sodium Hexametaphosphate
Advanced anticalculus and whitening agent

Higher molecular weight & greater tooth coverage

Strong attraction to calcium hydroxyapetite
Mechanism of tooth sensitivity
Dentinal tubules contain fluids from pulp that can transmit pain
- Cold fluids shrinks
- Air evaporates fluid
- Sweets cause osmotic movement

Mechanoreceptors of pulpal nerves also attach to odontoblasts & protrude into the tubule
Causes of sensitivity (6)
Acids from foods
Abrasions
Restorative procedure
Fracture
Scaling/Root planing
Bleaching agents
Ways of reducing sensitivity (4)
Plugging or sealing tubules
Saliva for remineralization
Burnishing
Ions - such as Flouride, potassium nitrate, oxalate, Calcium phosphate
Key of toothbrushing
Thoroughness & manipulative skills of brusher
Indications for interdental aids
Size of interdental space
Tooth misalignment
Presence of furcation
Orthodontic appliances
Presence of fixed prosthesis
Gingival massage
May produce epithelial thickening, keratinization & increased circulation but it is questionable of any of this will protect against microbes & other local irritants.
Oral irrigation Devices advantages & disadvantages
May remove debris around ortho & pros
May disrupt plaque
Can be used to deliver chemotherapeutics

- However, may cause transient bacteremia following irrigation
Plaque Score technique
Use water soluble dye like Erythrosine

Record stainable plaque on all four surfaces of six preselected teeth
0 - No plaque
1 - film of plaque at free gingival margin
2 - Moderate accumulation within gingival crevice visible to naked eye
3 - Abundance of soft matter within gingival crevice

Average score for tooth and mouth
Effects of medications on plaque
Patients taking Anti-depressants & Anti-hypertensives had more plaque
Epidemiology & Objectives (5)
Study of distribution & determinants of diseases & application to control health problems

Objectives
- Identify cause & risk factors
- Identify extent of problem
- Study history & prognosis
- Evaluate existing & new preventative & therapeutic measures
- Provide foundation to develop public policy & regulations
Incidence
Number of new cases that occur during a specific time / Number of persons at risk of developing the disease during that time
Prevalence
Proportion of population that has the disease
Case fatality rates
Number of individuals dying during specified period of time after disease onset / number with the disease
Criteria for causality (4)
Strength of association
Biological plausibility
Dose-response effect
Temporality
Effects of smoking on PD
Vasoconstriction to cause ischemia
Direct damage to macrophages & fibroblasts to delay wound healing
Less bleeding due to tissue keratinization
Scaling & rationale
Instrumentation of crown and root to remove plaque, calculus and stains

Calculus is a mechanical irritant, provides surface area for plaque & Interferes with plaque removal
Root planing
Treatment procedure designed to remove cementum or surface dentin that is impregnated with calculus or contaminated with toxins or microbes. Some unavoidable soft tissue removal occurs.
Goals & objectives of scaling & root planing
Decrease bacterial load
Create a smooth hard cementum surface free of calculus & toxins
Produce tooth surface that is biocompatible with sulcular epithelium
Decrease inflammation, pocket depths & gain attachment
Endotoxin characteristics
Can penetrate up to 12 microns beneath cemental surface & attracts inflammatory cells, cytokines, stimulates bone resorption etc.
Root modification
Scaling & root planing may create a smear layer of irregular dentin surface & rinsing will not remove it. Less endotoxin

Cervical cementum may be removed in 1-4 strokes
Pathogenic bacteria in scaling & root planing
Effective against motile rods & spirochetes

Ineffective against invasive Porphyromonas gingivalis & Actinobacillis Actinomycetemcomitans
Scaling & root planing factors to consider (4)
Severity of PD disease
Inflammation, bleeding, suppuration
Tissue contour, tone & architecture
Reduced pocket depths after scaling/root planing due to, & time
Recession of gingiva
Gain in attachment level
Inability to probe a pocket due to close adaptation of gingiva to tooth

Healing in 4-6 weeks & 9-12 months for maturation of periodontium
Advantages of calculus removal prior to surgery
Decreased inflammation to make tissue easier to incise & reflect
Decrease amount of time flap needs to be open
Possibly eliminate surgery altogether
Critical probing depth
2.9mm scaling
4.2mm surgery
5.5mm osseous surgery
Patient operator position 2&4
Patient:
Supine with feet leveled during maxillary
Back of chair raised 30-40 degrees for mandibular

Operator:
Height of chair should be at elbow height
Right handed between 8-1
Left between 11-4
Feet flat & back straight
Ergodynamics
Hips slightly higher than knees by 5-15 edegrees
Position knees under patient
Feet firmly on floor
Anatomy of an instrument
Terminal shank between blade & first bend

Functional shank extend from working end to the shank bend closest to handle.

Working end - face, cutting edge, back, toe
Instrument handle factors
Weight: Hollow handle increases tactile transfer & decreased muscle fatigue

Diameter: Larger handle maximizes control & reduces fatigue

Serration: Smooth handle decreases control & increases fatigue
Balanced instrument
Working end centered on line running through long axis of handle
The Sickle
Used for supragingival calculus for anterior teeth

Anterior sickle scaler - blade & shank in same plane

Posterior sickle scaler - angulated shank
The hoe
Similar action to chisel with pull stroke.

Sharp corners & limited application with subgingival calculus
The chisel
Push stroke for mandibular anterior proximal
The File
Miniature series of hoes on a single blade
The Curette uses, shape & strokes
For supra and subgingival scaling & root planing.

Spoon shaped

Push/pull strokes
Gracey Curettes
Specific

1/2,3/4,5/6 - Anterior teeth
7/8,9/10 - Facial & lingual of posterior teeth
11/12 - Mesial of posterior
13/14 - Distal of posterior
Columbias
More universal

2R2L for anterior
4R4L for posterior
13/14 used for anything
Gracey vs Universal curette
Gracey
Facial surface is 60-70 degrees to shank
Site specific
One cutting edge

Universal
Facial surface is 90 degrees to shank
Non-site specific
Two cutting edges
Instrument activation
Adapt blade to tooth surface
Close blade
Insert blade to base of pocket
Opel blade to establish cutting edge
lock fingers on curette
Apply fulcrum pressure & lateral pressure on tooth
Move instrument apical and coronal push pull
Fulcrums(5)
Built-up fulcrum
Split fulcrum
Cross arch fulcrum
Opposite arch fulcrom
Extraoral fulcrum
Polishing
To remove supragingival plaque & stain with prophy paste.

Can remove up to 5 microns of enamel

Ribbed cup causes more heat
Prophy jet
Very effective in removing stain
Uses sodium bicarbonate
However, can abrade epithelium
Root conditioning
Citric acid
Tetracycline
Ethylenediaminetetraacetic Acid (EDTA)

May help to remove smear layer caused by scaling/root planing