Penney--Vascular Syndromes

Front 1

ACA SUPPLIES

Back 1

medial and dorsal aspect of the frontal and parietal lobes
precentral&postcentral gyrus

Front 2

Occlusion of ACA results in

Back 2

CONTRALATERAL PARALYSIS IN THE LEG
OCCLUSION OF BOTH ACA prod bilateral paraylsis in lower limbs

Front 3

ischemia to the postcentral

Back 3

results impaired sensation to the leg

Front 4

MCA SUPPLIES

Back 4

Precentral gyrus, premotor region, somesthetic and auditory areas
parietal area

Front 5

MCA Occlusion

Back 5

DAMAGE TO THE UPPER EXT. AND FACE CONTRALATERALY

Front 6

LENTICULOSTRIATE ART

Back 6

can cause a lacunar stroke

Front 7

PCA SUPPLIES

Back 7

MIDBRAIN, THALAMUS, SUBTHALAMIC NUCLEUS
TEMPORAL AND INF OCCIPITAL LOBES OF THE CORTEX

Front 8

Branches of the PCA

Back 8

ant and post temporal
parieto-occipital

Front 9

Calcarine artery supplies

Back 9

primary visual cortex

Front 10

lesion of the calcarine artery

Back 10

hemianopsia

Front 11

PCA lesion

Back 11

midbrain infarction
unilateral damage to the ventral region causes Weber's

Front 12

Sup Cerebellar artery supplies

Back 12

rostral pons,caudal midbrain, superior cerebellum

Front 13

Lesion of the SCA

Back 13

IPSI LIMB AND GAIT ATAXIA DUE TO DAMAGE TO THE SUP CEREBELLAR PEDUNCLE AND CEREBELLAR EFFERENTS

Front 14

Lesion of the SCA

Back 14

Contra loss of sensation of pain and temp due to damage to the spinothalamic tract

Front 15

Lesion of the SCA

Back 15

Ipsi Horner's syndrome due to damage of descending sympathetic pathways supplying the eye

Front 16

Lesion of the SCA

Back 16

analgesia & thermoanesthesia on the ipsi face due to damage of the nucleus & spinal tract of the trigeminal nerve

Front 17

Pontine paramedian arteries arise from the

Back 17

Basilar artery

Front 18

Pontine Paramedian arteries supply

Back 18

medial portion of the upper and lower pons

Front 19

Pontine Paramdian a occulison

Back 19

contra UMN paralysis dut to involvement of the corticospinal tract

Front 20

Pontine Paramedian a. occlusion

Back 20

ipsi facial weaknes due to damage of dorsal facial nucleus

Front 21

Pontine Paramedian a. occlusion

Back 21

ipsi gaze paresis due to damage to pontine gaze center & abducens nucleus

Front 22

Short Circumferential a

Back 22

branch off of the pontine

Front 23

Short Circumferential a supplies

Back 23

ventrolateral pons
descending sympathetic fibers from the hypothalamus

Front 24

Short Circumferential a supplies

Back 24

portions of the trigeminal and facial nucleus and their fibers

Front 25

Short circumferential a occluisons

Back 25

ipsi horners syndrome
contra hemianesthesia of the body due to damage to the spinothalamic tract

Front 26

Long circumferential a
supplies

Back 26

most of the tegmentum of the rostral & caudal pons

Front 27

Long circum a supplies

Back 27

lateral portions of the midbrain tegmentum

Front 28

long circum a occluison

Back 28

paresis of conjugate eye movements

Front 29

Long circum a occlusion

Back 29

nystagmus & contra hemianesthesia

Front 30

AICA supplies

Back 30

ventral surface of the cerebellum

Front 31

AICA supplies

Back 31

lateral portions of the caudal pons

Front 32

AICA occlusions

Back 32

ipsi los of fascial sensation due to damage to CNV

Front 33

AICA occlusions

Back 33

ipsi horners (due to disruption ofthe descending sympathetic fibers)

Front 34

AICA occlusions

Back 34

contra hemianesthesia due to damage of the spinothalamic tract

Front 35

Anterior spinal supplies

Back 35

medial medulla

Front 36

ASA occlusions

Back 36

medial medullary syndrome 1.contra loss of kinesthesia & discriminative touch (damage to medial lemniscus)
2. UMN paralysis (damage to pyramidal tract)

Front 37

Medial medullary syndrom

Back 37

3. Ipsi paralysis of tongue muscles (tongue deviates to side of lesion-Hypoglossal nucleus

• may affect fibers in CN XII & CST because the two structures are juxtaposed in medulla
• Anatomic basis for inferior or hypoglossal alternating hemiplegia
•Characterized by LMN paralysis of ipsi half of tongue, & contra hemiplegia

Front 38

Posterior spinal a supplies

Back 38

fasciculus gracilis & cuneatus
-spinal trigeminal nuclei

Front 39

Posterior spinal artery supplies

Back 39

dorsal and caudal inf cerebellar peduncle
-solitary tract and dorsal motor nucleus of X

Front 40

Posterior spinal artery occlusions

Back 40

loss in conscious proprioception of the ipsi body due to damage of nucleus cuneatus and gracilis

Front 41

PICA arises from the

Back 41

vertebral artery

Front 42

PICA occlusion

Back 42

Wallenberg's syndrome

Front 43

PICA supplies

Back 43

lateral medulla
-spinothalamic tract
-dorsal&ventral spinocerebellar tracts

Front 44

PICA supplies

Back 44

-descending sympathetic fibers
-descending tact of CN V nucleus

Front 45

PICA supplies

Back 45

nucleus ambiguus, portions of the vestibular nucleus

Front 46

Clopidogrel (plavix)

Back 46

block ADP formation & prevent or attenuate platelet aggregation

Front 47

Asprin

Back 47

causes and irreversible acetylation of the enzyme cyclooxygenase and inhibits thromboxane synthesis in platelets

Front 48

reduction of thromboxane synthesis in platelets

Back 48

results in reduced release of this prostaglandin leading to prevention or reduction of platelet aggregation

Front 49

Tirofiban (Aggrastat)

Back 49

blocks GP IIb/IIIa receptors on the platelets and prevents their aggregation

Front 50

GP receptors

Back 50

located on platelet membrane, bind fibrinogen and other molecules and promote clot formation

Front 51

Thrombolytic drugs

Back 51

lyse blood clots and have had success in reversing neurological deficits in patients with stroke if given within a critical time window of onset of symptoms

Front 52

Wallenburg syndrome

Back 52

caused by damage to the PICA

Front 53

Wallenburg clinical manifestations

Back 53

dysphagia, dysarthria
analgesia & thermoanesthesia on the ipsi side of the face due to damage to nucleus and spinal tract of trigeminal nerve

Front 54

Wallenburg clinical manifestations

Back 54

contra loss of P &T due to damage to the spinothalamic tract

Front 55

Wallenburg clinical manifestations

Back 55

vertigo,nausea, vomiting, nystagmus due to damage to portions of the vestibular nuclei

Front 56

Wallenburg clinical manifestations

Back 56

ipsi horners syndrome

Front 57

Wallenburg clinical manifestations

Back 57

ipsi gait & limb ataxia due to damage to cerebellum/inf cerebellar peduncle

Front 58

Horners syndrome caused by

Back 58

SCA, AICA, PICA, short circum

Front 59

Horners symptoms

Back 59

miosis, ptosi, anhydrosis, conjuctival congestion

Front 60

Benedikt syndrom

Back 60

lesion of the ventral and tegmental midbrain

Front 61

Benedikt syndrome caused by

Back 61

damage to ventral and tegmental midbrain

Front 62

Benedikt syndrome charact

Back 62

ipsi oculumotor paresis due to damage to the oculolomotor nucleus

Front 63

Benedikt syndrome charact

Back 63

contra limb ataxia due to damage to the red nucleus and sup cerebellar ped

Front 64

Parinaud syndrome

Back 64

AKA aqueduct of sylvius or midbrain syndrome

Front 65

Parinaud syndrome caused by

Back 65

dorsal midbrain lesion
most common: pineal region tumors and hydrocephalus

Front 66

Perinaud syndrome charact.

Back 66

impaired upward vertical gaze and loss of pupillary light reflex due to damage to pretectal area
cause setting sun

Front 67

Claude syndrome caused by

Back 67

unilateral damage to the tegmental region of the midbrain

Front 68

Clinical manif. of Claude syndrome

Back 68

ipsi oculomotor paresis due to damage to the oculomotor nucleus and CONTRA ataxia & tremor due to damage to the sup cerebellar peduncle

Front 69

Weber's syndrome causes

Back 69

unilateral damage to the ventral midbrain

Front 70

Weber's syndrome

Back 70

involves the PCA

Front 71

Weber's syndrome charact.

Back 71

IPSI PARESIS OF ADDUCTION AND VERTICAL GAZE

Front 72

Weber's syndrome charact.

Back 72

PUPILLARY DIALATION DUE TO DAMAGE TO THE OCULOMOTOR NERVE

Front 73

Weber's syndrome charact.

Back 73

PARESIS OF CONTRA FACE, ARM, AND LEG DUE TO DAMAGE OF THE CORTICOBULBAR AND CORTICOSPINAL TRACTS