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129 Cards in this Set

  • Front
  • Back
what can cause Bronchopulmonary Dysplasia?

3 phases:

What does this do to lungs?
iatrogenic cause, increased time on ventillator, RDS prolonged

1. excessive 02 pressure
2. inflammatory
3. fibroproliferation phase

scarring
emphysema, alveoli dont multiple, capillaries inflam and distroy
Name 2 intrinsic pulmonary diseases
Congenital heart disease
resp. distress syndrome
list some causes of RDS
decrease surfactant
acidosis
atelectasis
compliance of lungs is ____ in infants.
increased
3 associated conditions with chronic respiratory failure
CNS disorders like hypoxia, apnea..

congenital airway abnormality

intrinsic muscle disease (md or botulism)
what pulmonary structure is developed by 16th - 24th week?

when can respiration occur?

when does ant lung pouch, tach and esoph seperate?
bronch epithe, mucus, supportive cartiledge, capillaries with air blood interface.

24th week

24-26 days post conception
Alveoli increase in numbers until....
8 years old and then continue to grow in size and complexity
what position has increased arterial 02 tension?

what position was decreased arterial tension?
prone (usually contraindicated)


supine (this is a bad thing)
whats the best way to enhance infant ventillation an circulation?
early ambulation
3 segmental lung expansion techniques
sensory feedback
sidely and rocking
pressure on opp lobe
normal bp in infant (1 month)

6 months =
85/46

95/53
General PT interventions for cardiac defects
- assess neuro
- edu precautions
- early mob after surgery to prevent complictions
Hypoplastic Left sided heart syndrome =

TX =
malformation of valvue, decreased blood flow

norwood procedure - staged, so right side does all the work. left side does nothing.
Pulmonary stenosis is reistance to-
BF from R ventricle
what condition gives mixed blood to the body?
overriding aorta
palliative repair of tetralogy of fallot =
anastomosis from subclavian A to pulm A

corrective repair would be to close VSD
Describe transposition of great arteries =
pulm off LV and Aorta off RV
What is tricuspid atresia?
congenital lack of tricuspid vlavue (ra --> rv ) decreased BF
List 4 defects of tetrology of fallot

when should you get surgery?

How can PT releieve cyanosis?
VSD
overriding aorta (straddle)
Pulm stenosis
RV hypertrophy

-turbulance-

pulm signs like nail bed clubiing

squatt to increase systemic reistance
patent =

atresia =
opened

closed
what is coarticulation of aorta?
constriction of aorta
Basic mechanism of neonatal cynotic defects-
r --> L shunting de oxy blood bypass lungs to the body (15- 30% decreased 02)
List 5 neonatal Cyonotic Defects =
tetralogy of fallot
transpiosition of great artieries
triscuspid atresia
pulm atresia
hypoplastic L sided heart synd.
Timig of VSD surgery depends on?
tolerance to defect
resp distress
wt gain
signs of resp distres =
diaphoresis
fatigue with feeding
dec endurance
What is patent ductus arteriosis?


results in?
opened hole shunting L --> R from aorta to pulm A (caused from dialated with hypoxia at birth, commen with primi)

CHF from inc work, bounding pulse
Ventrical Septal defect =


prognosis is worse if?
L --> R shunt during systole (when R vent contracts) ?????????????????



hole is not in muscle????
What is atrial septal defect?

TX =
foramen ovale opened
( L --> R)

suergey B/W/ 4-6 years to prevent heart failure in adulthood.
what 3 things drain into the fetal IVC?
placenta blood
LE blood
Liver blood
List the route fetal blood takes inside the heart
right atrium
foramen ovale
left atrium
left ventricle
aorta
what fetal shunt communicates between descending aorta and pulm A?
ductus arteriorosus
what fetal shunt allowed blood to bypass the body?
ductus arteriorosus
what does the ductus venonosus do?
liver and IVC connection
what fetal shunt allows blood to bypass lungs?
foramen ovale
(located between atria)
Primary general ped pt

Seconday-
strength
rom
MS integ
soft tissue mobs
________________

sensory re edu
practice skills in different evnir
Cause of congenital heart defects-
85% unknown (genes and environment combo)

10% associated with other probs like: DS, trisomy 18, turners, vacterl, marfans, maternal DM
what pt modality is contraindicated in OI?

what is good?

parental edu
PROM

positioning, inc sensory motor deve, splinting fx, mobility traniing

safe handling
loos clothes
roll off diaper
enrich environment
List some manafestatinos of OI
lax joints, weak muscles, Osteoper, blue sclera, hernias, easy bruise, bad teeth, disproportions, inc sweat
Medical management of osteogensis impoerfects
maybe diet, resp care, reserach and intramedullary rods
PT for non ambulating MD patients
WC fit
foot wear
energy conservation
a/prom
resp
problem solve
----- = inhereted disorder of connective tissues
OI (collegen synthesis doesnt go beyond reticular stage- no organization)
medicla management with mild MD
corticosteriods to enhance dystrophin expression and protect against fiber damage)
MD progression into teen looks like
resp involvement, heart probs and cog issues.

death by 25
onset of MD

signs in first 2-5 years

when do they loose ability to walk
2-6 years


late walking, clumsy

7-12
Discribe progression of treatment for torticollis
Neonate: manual prom/mfr, control posture, massage, visual stim

3 month: cont above, soft brace, helmet

later: surgical length, maybe botox
____ - x linked genetic disease with spont or inherited deletion of a portion of X chomosome
MD
Theories of cause for congenital muscular torticollis


secondary complications

prog
abn vascular pattern
muscle rupture
heredity
crowding, intrauterine compartment
compression inj
_____________


worsened scoliosis
plagio ceph
sensory motore development
hip displasia
______________

50-70%^ spont recover
better with early detection
prog = 3.2 months
CMT is more commen in what gender?
males (r > L )

head tilts towards and rotates away
List medical management of con limb def
stump care
phantom limb pain
general conditions
prosthetics
VACTERTL syndrom =
non random assoc of birth defects with unknown genetic cause
when during development is embryo most susceptible to teratogens cause limb def?

examples:
3-7 wk

ex) thalimide, BCP, irradiation, VACTERL
in development, when do limb buds appear?
3-4th week, then differnciate by 7th.

(prox-> distal and ue > Le)
list come commen causes of Cong Limb Def (mmost commen type is conge, not aqired)
- embryonic formation failure
- duplication
- over/under growoth
- consecutive band theroaty
- mengiococal defect
- terogenic facotirs
What muscle biopsy findigs will you see with AMC?
fibrous fatty tissue
AMC commen resting posture =
flexed and dislocated hip
club foot
IR shoulder
flexed wrists with up
AMC mobility patterns

describe times surgical manegem,ent
early rolling
later transfer

clubfeet- when pull to stand
knees - ambulate
wrist- last resort
List associated characteristics with AMC

AMC is classified based on-

describe big difference between the 2 maj
scoliosis
dimpling over skin with 2 joints
hemangiomas
absent finger creases
congenital heart disease
facial abnomalities
report rbs
abn hernias

resting posture

1- ext knee, fex elbows
2- flexed knee, ext elbow
Pathophys theories of AMC
hyperthermia of fetus from materal fever

prenatal viral inf

vascular comp

uterine fibroid tumer

septuim in uterus

reductino of amniotic fluid

musltiple births
Clinical Manafestations of AMC
- severe joint contractures
- dec muscle dev
- amyloplaisia
- normal cog and speech
- dec body size
AMC =
arthrogyroposis multiplex congenita

non progressive stiffness with contracture in 2 or more joints and weakness or absence of muslce develo assoc with neurgenic and myoplastic disorders
mechanisms of contractures in AMC

order of prevalance of body parts effected
lack of fetal movement

foot
wrist
knee
elbow
shoulder
negatives for harrington rod
dec sag plane correction
inc flatten lumbar lordosis
positives for Luque rods

negatives
- inc bone quality
- prevents loss of lumbar lordosis

_____________

inc risk of neu probs due to inc flex
Myelogram
tests for CNS abnormality
Cobb method =
measures scoliosis of x ray
treatment of A. i. S (scoliosis) depends on
skeletal maturity
growth potential
curve mag
what scoliosis side compressed disc causing degeneration

what side casuses dec lung capacity
concave

VEX
(lung cap is increased on cave side)
what type of curve persists when you laterally bend?
structural
Commen A i S onset-

theories
10-18 years (lateral curvature of spine)

- insuf ligaments
- assym weakness
- unequal fiber types
- fam abnorm
- genetic?
Progression of Post of PT of Erbs Palsy
0-4 weeks: immob

4-6 weeks:
scar massage
PROM
immob
active transfer
motor learning

after 6 weeks:
strengthen
discontonue splint
dec mvt sunbstitutions
what test detects changes in self perception?
comp - canadian occupational perforamnce measure
pre surg assessment for erbs palsy

what muscles are commenly transfered?

which are commenly released?
social-cog
current funct: joint integ , length and strength of muscule transfer

lats and teres maj

pec maj
capsule
subscap
most mobile joint
shoulder
How can IGF-1 help erbs?
(insulin growth factor)
improves muscle region after injury

may help preserve degeneration
what position do you want to splint erbs arm?

list neuro rec techniques
supinate
ext rot
wrist ext

(to allow weight bearing)
____________

forced use with shaping
place and hold
3 surgery options for neurotmesis
neurolysis
nerve grafting
neurotization
whats a good functional assessment test for erbs with ages 4-8
mallet

and fyi these people favor late rec over early surgery
Long term erbs deficits
cortical chanes in developing NS
- change in neuro presentation
- habitual patterns
- may lead to disuse
List some interventions for ERBS
prom, splint, tape, NMES, surgery, position, neuro re-edu
toricollis secondary to erbs can lead to ...
.visual neglect
muscle tightness
plag
dec sensorimotor
dec body awareness
whats a good developmental test for infants?
AIMS
3 fuinctional impairments to look for in a 3 month year old

8month- 1 year?
prone propping
prehension
transitional mvt

wt bearing with transitions
bimanual functioning
interview questions about erbs HX
vaccuum
breech
clav fx
birth duration
degree of initial arm mvt
What muscles are commenly compensatory mm for erbs?
delts and pec maj
window into infants muscle activation
reflexes such as ANTR, placing reaction, moro, hand grasp, parachute response
Horners Syndrome

S/S:
cerival gang chain injury (symp)

ptosis (dropped eye)
miosis (constric pupil)
anhydrosis (dec sweat)
posture for shoulder dystocia
waiters tip ( AD IR)
prognosis if muscle is denervated fir iver 18-24 months
irreversible motor endplate degeneration and muscle fibrosis
natural hx of erbs

prognostic factors:
better in 2 months

but if inc to 3-6 months may need tendon transfer
over 6 then micro surg
______________

total envirnment
elevated hemidiaphram (phrenic)
winged scap (LTN)
horners
What nerve injury type is reversible

which isnt and req surg?
neuropraxia

neuroptmesis
regeneration AFTER wallarian deg =

what infuences this rate

axons cannot regrow if
1mm/day, 1 inch per month

-scar
- connection site mismatch


endoneural tube is distroyed
nerve plexus avulsion

can this be corrected with surg?
pregang tear, disrupting rootles
seperating motor cell body from axon (sensory cell body remains connected)

NO
nerve plexus rupture =
post gang tear M/S variable with intact

POSSIBLY corrected with surgery
perinatal risks for brachial plexis birth palsy

inc birth weight of

2 types of palsy
fetal macrosonia
rolonged labor
mutli preg
breech
prev palsy hx
gestational dm


over 4500g


avulsion and rupture
List nerve roots

erbs

klumpes
c5-c6 (extneded erbs goes to 7)

c8-ti
adaptation heirarchy
- least intrusive to more
- schedule
- adaptive materials
- provide assistance
good test for ambulation deficits
BOT
good test for functional activities
PEDI
___ = statement of childs present level of edu perforamnce`
IEP
for school based PT, child must fit into ....
catagory set by the state
good test for motor skill restrictions
PDSM 2
5 developmental domains
cog
physical
communication
social
adaptive
child link =
primary service coordination and assessent and treatment in everything
IFSP -

IEP =
individualized family service plan


individualized edication plan
IDEA =

discribe three parts
individual with disabilities edication act

A = general
B= IEp and 18-21
C = 0-3 EI
who recieved EI
disability
developmental delays at risk in anyof the 5 domains
3 stages of praxis
idea / goal
plan
executeion
E stim used for
strength
muscle facilitation
cp
praxis =
organization of sensory input and exceution of a respoinse to it
____ = rapid scoliosis with sb lesion under t12
tethered cord syndrome
Discribe 3 principals of sensory integration
1. adaptive responses facilitae the integration of sensory stim

2. people seek out sesory motor exp that have organizational effect

3. kids dont passivly absorb, they select most useful
inner drive is generated by
limbic system
clinical manafestations of tethered cord syndrome-
change giat
LE hypertonis
change urology
rapid scoliosis
assym mmt
lbp
ass pain
Why do SB ambulators, tend to loose ambulation by adolesence into adulthood?
change body proportions
decrease neuro status
MSK probs
bowel and bladder issues
List commen SB infant impairments (spina bifida)
hip dislocation
low muscle tone
dec prom (hip and ankle contractures)
verbal performance requires-
fine motor, speed, memorym R and L hemisphere integration
Who has cocktail personality possibly
SB
List some causes of SB UE dyscoordination
cerebellar ataxia

motor cortex/pry damage from hydroceph

delayed develop

arnol chiari
do SB have sensory deficits?
yes skipped dermatomes
A person with SB at L5 would be limited in walking as an adult due to

expected level of function for L1-L3
weight gain

most WC
S/S of Arnold Chiari
strider with insp
sleep/cry apnea
gerd
vocal cord paral
dysphag
bronch aspir
hydro


(a deformation of cerebellum, medulla and c-spine and hernation, causing block of csf in 4th vent)
S/S shunt infection with kids with SB
change in gait
fever
scoliosis
dec grip
inc HA
dec activity
dec school performance
weight changes
inc incontinence
irritability
S/S SB occulta
inc UTI and LBP
inc subacut fat and hirsutism
dimples
skin pig changes
Spinda bifida etiology
maybe dec folic acid, but no one knows- commen in cletic nations
list three ventillator weaning stratigies
1. spont breath trial with cpap
2. PSV- pt triggered breathes
3. simv- breathes are vent or spont
major goal for infants on ventillation
prevent secondary complications
Atrial septal defect results in:
R sided elnargement
asymptomatic
creshendo/decrecendo

need surg to prevent heart failure in adults
ventral septal defect results in:
pulm A enlrges with stenosis
Left sided enlargmement
CHF from Inc work

TIMING of surgery depends on
- tolerance to defect
- resp distress
- wt gain
what is a patent ductus arterioris

results in
l --> R shunt (aorta to pulm A )


CHF from inc work and bounding pulse