• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/30

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

30 Cards in this Set

  • Front
  • Back
What is the initial treatment for pediatric GI bleeding?
Gastric lavage
Acid suppression
Supportive care
What is the difference between functional and pathological GER in children?
Pathological is caused by relaxation of the LES, associated with more severe symptoms.
Functional is associated with mild regurgitation that improves with time.
What are symptoms of pathological GER in children?
Forceful emesis after meals.
Irritability
Respiratory problems
Laryngeal symptoms
Food averson
Sandifer's syndroe
What is Sandifer's syndrome? How does it relate to GER?
Spasmodic torsional dystonia with arching of the back. Occurs because arching helps lengthen the esophagus, increase LES pressure and prevents aspiration.
How is pathologic GER treated? (feeding habits, drugs, surgery)
Hold infant upright 30 min after feeding.
Thicken feedings.
Hypoallergenic formula
H2 blocker, PPI, prokinetics, cholinergic agonist.
Surgical- Nissen fundoplication
What are future complications of pathologic GER?
Esophageal strictures
Barrett's esophagus
Poor weight gain
Failure to thrive
What are the three growth curve abnormalities that can indicate failure to thrive?
Drop off in height, weight, or head circumference.
Why is hospitalization used for failure to thrive children?
Feeding to supplement.
Observe parent-child interactions
Monitor complications
Investigations- CBC, lead, UA
What pediatric populations most often have chronic abdominal pain? What is the pathophysiology?
Girls age 9-10
Visceral abdominal pain associated with c-type pain fibers.
What are typical symptoms of chronic abdominal pain in children? HOw does Apley's law apply?
Central nonradiating pain
Nausea, anorexia
The further the pain from the umbilicus, the more likely to have pathology.
How is chronic abdominal pain treated in children?
PPI or H2 blocker
Stool softeners
Reduced lactose diet
Regular follow up visits
How is enterobiasis transmitted? How does it present? How is it diagnosed and treated?
Fecal-oral autoinnoculation in children 5-10 year old.
Presents as perianal pruritis
Diagnosed by presence of eggs on scotch tape test.
Treated with mebendazole.
What are the four types of congenital diaphragmatic hernias?
Bochdalek- posterolateral
Morgagni- anterior
Central
Hiatal
Why is it important not to bag infants with a diophragmatic hernia?
Intubate, instead of bagging, because air can enter the esophagus and into the abdomen.
What are risk factors for necrotizing enterocolitis?
Prematurity
Presence of patent ductus arteriosus
What factors contribute to necrotiing enterocolitis?
Abnormal intestinal flora
Intestinal ischemia/ reperfusion
Intestinal mucosa immaturity/ dysfunction
What are radiologic findings of necrotizing enterocolitis?
Pneumotosis intestinalis- gas within the submucosa
Pneumoperitoneum
Dilated loops of small bowel
How is necrotizing enterocolitis prevented?
Breast milk
Avoid broad-spec antibiotics
Increase enteral feeds
Probiotics
What are symptoms of short bowel syndrome involving the jejunum? Ileum?
Jejunum- nutrient malabsorption
Ileum- loss of water and electrolytes, B12 and bile salt loss. Diarrhea
How is short bowel syndrome treated acutely and managed?
Acute- nutritional support, correct fluid/electrolyte losses.
Adaptation- increase amounts PO and decrease parenteral feeds.
Maintenance- manage nutrient deficiencies, fiber, antidiarrheals.
When does pyloric stenosis present? How does it present? What lab abnormality is found?
Presents at 2-4 weeks.
Projectile emesis after feeding, palpable "olive", visible peristalsis
Hypokalemic hypochloremic alkalosis present due to loss of gastric secretions.
What is duodenal atresia? What is the presentation? What sign is seen on xray?
Failure to recanalize lumen during development.
Presents as bilious vomiting, distended abdomen, maternal polyhydramnios, jaundice.
Double bubble sign seen on xray
What are Ladd's bands? How are they involved with malrotation?
Malrotation- failure to fully rotate in development.
Ladd's bands are obstructing bands of peritoneum left across the duodenum
How does malrotation present in infants? What sign is seen on xray? How is it treated?
Bilious emesis, acute obstruction
Recurrent abdominal pain
Diagnosed with double bubble or corkscrew sign of KUB.
Treat with removal of the volvulus.
What syndromes are associated with intussusception? What can cause it?
Cystic fibrosis
Henoch Schoenlein purpura
Polyposis syndrome
Can be caused by Peyer's patches, Meckel's, polyps, etc.
What are symptoms of intussusception?
Severe, colicky pain.
Vomiting
Currant jelly stool
Sausage shaped mass often in RLQ
Abdominal distension
What are the typical xray findings for insussusception? How is it treated?
Coiled spring sign
Treated with gastrograffin/barium/ air enema
What are symptoms of Hirschsprung disease? How is it diagnosed?
Failure to pass meconium, abdominal distension, no rectal tone.
Xray: dilated proximal colon and microcolon distally.
Rectal biopsy: hypoganglionic area
What is the classic presentation of appendicitis? How does it change with perforation?
Malaise, anorexia, colicky RLQ pain, nausea, vomiting.
After perforation, transient relief of pain followed by diarrhea, dehydration, sepsis, SBO symptoms
What are physical exam findings for appendicitis?
Rebound tenderness
Guarding over McBurney's point
Rovsing's sign- pain in RLQ on palpation of LLQ
Obturator sign- pain on rotation of R. hip
Iliopsoas sign- pain on extansion of R. hip