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61 Cards in this Set

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Nurse tells parents about post-op feeding following pyloromyotomy. Parents understand when state they will:
1. Avoid bubbling baby after feeding to prevent vomiting.
2. Rock baby to sleep after feeding to keep baby calm
3. Slowly increase the volume offered accoridnt to the MD orders
4. Maintain the baby on antiemetics to prevent vomiting
3. Slowly increase the volume offered accoridnt to the MD orders
GERD compared to GER
GERD is more serious and consists of:
1. poor wt. gain --> failure to thrive
2. Esophagitis
3. Respiratory sx (secondary to aspiration)
4. Consequences: Damage to esophageal mucosa & Pneumonia or asthma r/t aspiration
Parents want to know why child who just had cleft palate sx needs restraints. Nurse says:
1. This device is frequently used post-op to pretect the IV site in small children
2. The restraints will help us maintain proper body alignment
3. Elbow restraints are used post-op to keep kid's hands away from sx site
4. Restraints help maintain the child's NPO status
3. Elbow restraints are used post-op to keep kid's hands away from sx site
When do most reflux issues occur?
After eating when stomach is dilated.
Name symptoms of GER
1. Vary from occasional emesis to frequent vomiting & failure to thrive
2. Frequent hungry & irritable
3. Frequent upper respiratory tract infections. Or: stridor (inspiratory); Wheezing (expiratory), recurrent cough
How is GER diagnosed?
1. History (?)
2. Upper GI (barium swallow or contrast medium)
3. pH probe is BEST --> Let baby eat and if pH is down (acid) = GER; Eat and no change = not GER
Which s/s does nurse recognize as indication of moderate dehydration in prechooler?
1. sunken fontanel
2. Diaphoresis
3. Dry mucous membranes
4. Decreased urine specific gravity
3. Dry mucous membranes
What meds treat GER and how do they work?
1. Antacids --> Neutralize refluxed material and decrease resultant discomfort, but not often used in infants and children due to aluminum content
2. H2 (histamine) blockers --> Pepcid, Rantac --> Decrease gastric acid production
3. Proton pump inhibitor --> Prilosec --> Inhibit gastric acid secretion to reveal symptoms and heal irritated tissue
4. Reglan (only in book, not notes) --> Increase esophageal motility and tone
Nursing assessment of infant suspected of having necrotizing enterocolitis would be:
1. pH of the stomach contents
2. Neurological status q 2 hr
3. Rectal temp q 2 hr
4. Abdominal girth q 4 hr
4. Abdominal girth q 4 hr
S/S of pyloric stenosis
* Occur between 2-8 wks after birth (peak is 3-5 wks)
* Symptoms manifest as hypertrophy (organ enlargement) worsen
* projectile vomit
* Hungry
* Fails to gain wt
* Decreased # stools
* Eventually, dehydration and electrolyte imbalances
What type of acid-base imbalance would an infant with pyloric stenosis be at risk for?
MIX!!

* Metabolic alkalosis from vomiting

* Metabolic acidosis from lack of fluid --> producing lactic acid
You are caring for 4 m.o. infant with GERD admitted for cough, cyanosis and upper resp infection. Which statement by the dad would indicate need for further teaching?

A. I should feed baby 6 oz q 2 hrs
B. I need to burp baby q 2 oz that he eats
C. I will give baby Zantac to decrease stomach acidity
D. Baby will likely outgrow this disease by 1 y.o.
A!!

At 4 months old, the baby's stomach can hold 150-200 mL, which is about 5-6 oz. Don't want to totally fill up.
Would the chloride levels in an infant with pyloric stenosis be low or high?
LOW cos HCl in vomit. (Cl = chloride)
What is sx called to treat pyloric stenosis? Describe it. What are pre-op & post-op nursing duties?
PRE-OP:
* IV, give fluids
* correct electrolyte imbalances
* keep NPO

SX = pyloromyotomy
* ligating pyloric sphincter
* usually fast & laparoscopically
* performed as soon as rehdration and electrolyte balance is restored

POST-OP
* Pain control
* feeding protocols vary by MD: start small and increase; may start pedialyte
* sponge baths
Mom asks: Why does vomit of child with pyloric stenosis look different than other children's vomit? Nurse explains b/c no bile in vomit b/c:
1. The GI system is still immature in newborns and infants
2. Obstruction is above the bile duct
3. Emesis is from passive regurgitation
4. Bile duct is obstructed
2. Obstruction is above the bile duct
Nursing management for cleft lip, palate
* Most infants able to breast feed or bottle feed with modifications
* Fed upright with frequent burping
* Watch for attachment problems with parents
** Assess for weight gain --> will take them longer to eat**
Infant returns from initial surgery for Hirschsprung's disease. Which routine post-op intervention will nurse NOT do:
1. Maintain child NPO until bowel sounds return
2. Monitor rectal temp q 4 hrs
3. Reunite parents asap
4. Assess sx site q 2 hrs
2. Monitor rectal temp q 4 hrs
Complications of cleft palate
* speech development
* high risk for ear infection b/c Eustachian tubes don't ventilate well (pressure not normal)
* dental and orthodontic problems (if palate pulled together, might be crowded)
What symptoms apply to dx of moderate dehydration in a 4 month old?
1. Elevated HR
2. Urine specific gravity of 1.038
3. Wt gain
4. Polyuria
5. Slow cap refill
1. Elevated HR
2. Urine specific gravity of 1.038
5. Slow cap refill

Norm USG= 1.005 - 1.035
Recurrence rates of intussusception
Air enema treatment = 15%

Surgery treatment = 7.5%
6 week old with hx of frequent vomiting after feedings and failure to gain wt. DX of gastroesophageal reflux is made. While planning discharge teaching to parents, nurse should include instructions to:
1. Dilute the formula
2. Delay burping to prevent vomiting
3. Change from milk-based formula to soy-based formula
4. Position the infant at a 30- o 45-degree angle after feedings
4. Position the infant at a 30- o 45-degree angle after feedings
How do you know when intussusception treatment has been successful?
Passage of normal brown stool (vs. the hallmark "currant jelly stool" of intussusception)
How is Hirschsprung's disease diagnosed?
X-ray with contrast
-- or --
rectal biopsy to look for ganglion cells (most reliable)
Treatment of Hirschsprung's disease
* Sx removal of the affected bowel soon after diagnosis -- reattach the healthy areas
* Colostomy vs. end-to-end anastomosis
* Most have return of bowel function and can become continent
Nursing management of Hirschsprung's patient before and after surgery
PRE-OP:
* Normal saline enemas
* NPO

POST-OP:
*Typically stay for one night unless colostomy is done (then might stay for 2 to allow for teaching)
* Pain control
*** Monitor abdominal circumference ***
* If colostomy present, what color should it be? Beefy red
True or False:
Pain relief from appendicitis is a good sign.
False. Likely perforation.
If perforation occurs, what are the complications?
* Infections
* Adhesions -- scar tissue in peritoneum
* Abscesses
Describe appendicitis pain
* Most intense at McBurney's point: halfway between the anterior superior iliac crest & umbilicus

* Rebound tenderness -- press in at McBurney's OK, but when let go it hurts
14 y.o. boy brought to ED with dx of rule out appendicitis. Complaining of RLQ pain. Nurse's most appropriate action to assist in managing his pain would be:
1. insert a rectal tube
2. Apply an ice bag
3. Apply a heating pad
4. Administer an intravenous antispasmodic agent
2. Apply an ice bag
Dx and Tx for appendicitis
DX: CT with contrast (best)

TX: Appendectomy (open or laparoscopic)
-- pt on abx for 24 hours and then go to OR (if not perfed)
Who is most likely to get appendicitis?
* Adolescent males
* Rare before 2 y.o.
* Most common emergency abdominal sx for kids
A 3 month old has gastroesophageal reflux but doing well otherwise. What can mom do to decrease reflux?
1. D/C breast feeding asap
2. Increase frequency of feeding and keep them small
3. Place baby in prone position with head flat
4. Place infant in car seat after feeding
2. Increase frequency of feeding and keep them small
What can happen if Celiac Disease is untreated?
(In order of events)
1. Body can't digest gluten
2. Results in buildup of glutamine (metabolite)
3. Glutamine is toxic to mucosal cells of intestine
4. Affects absorption of nutrients b/c cells damaged
5. If untreated, CA can develop (r/t toxic metabolite)
S/S of Celiac disease?
EARLY SIGNS:
* Steatorrhea (r/t fat malabsorption)
* Abdominal pain
* These will often begin at age 6 months - 2 y.o., when foods with gluten are first fed to kid

LATER SIGNS:
* Failure to thrive
* Malnutrition
* Osteoporosis
While gathering data on 16 month old, nurse gathers data. Which is related to dx of hirschsprung's disease?
1. Bile-stained vomit
2. Decreased urine output
3. Poor wt gain since birth
4. Intermittent sharp pain
5. Alternating constipation and diarrhea
1. Bile-stained vomit

3. Poor wt gain since birth

5. Alternating constipation and diarrhea
DX Celiac disease?
*** Duodenal biopsy looking for mucosal damage --> no villi ***
* analysis of fecal fat content
TX if Celiac disease -- when does body start returning to "normal"
* SX improve within days-weeks
* Intestinal villi return to normal in abut 6 months
* Catch-up growth occurs over the next 1-2 yrs
S/S of TEF?
* Maternal hx of polyhydramnios b/c baby can't swallow amniotic fluid.
* Increased salivation & drooling b/c no place for saliva to go
* With first feeding, formula comes up thru nose. --> Cyanosis, Choking, Coughing are classic signs
After delivery of baby with omphalocele, the nurse would do what?
1. Weigh baby
2. Insert orogastric tube
3. Call the blood band for 2 units of blood
4. Cover the sac with moistened sterile gauze
4. Cover the sac with moistened sterile gauze
DX of TEF?
* Try to pass an NG tube and get X-ray --> then hook up to suction and collect secretions.
* NG tube will be coiled in blind pouch
TX of TEF?
* IV fluids cos NPO
* abx b/c risk of aspiration
* sx correction immediately (not compatible with life)
Post-op care for TEF baby?
* Will have an NG tube to suction --> do not manipulate b/c there is a suture line that could be disrupted

* ABX
* IV fluids --> small feedings
* Might have a G-tube
What are two abdominal wall defects & what (generally) causes them?
1. Gastroschisis
2. Omphalocele

Caused by problem at 11 wks gestation. Before 11 weeks, the intestines are located outside of the abdomen. But at 11 wks, they move inside.
Nurse has completed discharge teaching for celiac disease. Teaching successful when mom says child must comply with gluten-free diet:
1. Throughout life
2. Until the child has achieved all major developmental milestones
3. Only until all sx are resolved
4. Until the child has reached adolescence
1. Throughout life
What is gastroschisis?
* Intestines herniated thru abdominal wall (usually to the right)
* Small intestine only (usually)
* NOT usually associated with other congenital issues
* No membrane covers bowel
* Umbilical cord intact
* More common in infants of moms who smoke
* 1 : 10,000 births
What is omphalocele?
* Intra-abdominal contents herniated thru umbilical opening
* Covered by a translucent sac (peritoneal tissue)
* Sac may also contain bowel, gall bladder, internal genitalia, bladder, spleen and pancreas
* Often associated with other congenital defects
* 1 : 5,000 births
DX omphalocele?
prenatal US
TX omphalocele?
* Sac protected after birth by keeping it moist
* Temp regulation (hypothermia likely)
* IV fluid replacement
* SX repair with "silo" (like a ziploc baggie to stay hydrated).
* Repair usually staged 7-10 days to prevent intra-abdominal pressure
* Could take wks for bowel function to return. TPN for nutrition
* NPO
Nurse teaches child about celiac disease. Child understands when he chooses which meal?
1. Beef and barley soup, rice cakes & celery
2. Ham and cheese sandwich with lettuce and tomato on rye toast
3. Beef patty on a hamburger bun and home fries
4. Baked chicken, green beans & slice cornbread
4. Baked chicken, green beans & slice cornbread
Which defect -- gastroschisis or omphalocele -- would have more problems with fluid management?
Gastroschisis b/c not covered by peritoneal tissue
Child with celiac should not eat: (select all that apply)
1. Rice
2. Wheat
3. Oats
4. Barley
5. Corn
2. Wheat
3. Oats
4. Barley
What is necrotizing enterocolitis? (NEC)
* Inflammatory disease of he intestinal tract where the bowel mucosa die
What groups most often get NEC?
* premature infants
* 4-13% of preemies, with a 40% mortality
Pathology of necrotizing enterocolitis?
1. Bacterial infection (immune system is not developed in preemies)
2. Edema and inflammation cause vascular compromise
3. Bowel mucosa (not just lumen) dies
4. Bacteria invade necrotic tissue --> gas causes abdominal distention
5. Bowel may perforate --> SEPSIS
10 yr old dx with "rule out appendicitis." Boy states, "It doesn't hurt anymore." Nurse suspects that:
1. Boy is afraid of going to sx
2. Boy is having prob expressing pain
3. Appendix has ruptured
4. Boy trying to get attention
3. Appendix has ruptured
S/S of NEC
* feeding intolerance
* vomiting
* bloody diarrhea (due to breakdown of bowel mucosa)
* abdominal distention
DX necrotizing enterocolitis?
**** X-ray = gold standard ***
Medical prevention & treatment of NEC?
PREVENT:
* give breast milk (antibodies)

TREATMENT:
* NPO
* NG tube to decompress bowel
* IV fluids / TPN, lipids
* abx to prevent, treat sepsis
* serial abdominal circumference checks
* serial x-rays to detect perforation (until can get to OR)
* Immediate sx if perforation
After necrotizing enterocolitis
* short bowel syndrome -- may need bowel transplant (!)

* Cholestasis (disruption of bile flow) --> complication of being on TPN; can be prevented with ursodiol (med that prevent gallstone formation)
An 18 month old infant w/ hx of cleft lip and palate admitted for palate sx. Why should toothbrush not be used after sx?
1. Toothbrush frightening to kid
2. Kid no longer has deciduous teeth
3. Suture line could be interrupted
4. Child will be NPO
3. Suture line could be interrupted
What is Esophageal Atresia and Tracheoesopageal Fistula? (TEF)
Esophagus is not a continuous tube.
-- Ends in a blind pouch = EA
-- And is connected to the trachea = TEF