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8 Cards in this Set

  • Front
  • Back
adrenal myelolipomas occur in ___ (age group)
- postpubertal adults!
Ectopic adrenal cortex lacks a __
In Cushing's syndrome, the zona ___ and __ are thickened bilaterally
zona fasciculata and zone reticularis
- note: in some cases the hyperplastic adrenal glands may only weigh slightly more than normal
congenital adrenal hyperplasia is due to a partial or complete enzyme deficiencies resulting in a lack of __ and resultant __
- lack of cortisol
- inceased ACTH and cortical hyperplasia
the principle deficiencies seen in congenital adrenal hyperplasia are:
- 21-alpha hydroxylase deficiency (95% cases)

- 11-beta hydroxylase deficiency (3%)

- 17-alpha hydroxylase deficiency
In CAH, the adrenal cortex appears brown due to __
lipofuscin pigment
In males with CAH, bilateral benign testicular or paratesticular masses histologically resembling __ may be produced
- resemble Leydig cell tumors
- these masses are ACTH-sensitive and often regress with steroid therapy
- this finding usually does NOT require surgical excision!!
primary pigmented nodular adrenocortical disease, what the heck is this?
- Essentials, p 661
- aka: microadenomatous hyperplasia
- more common in young women
- S/Sx of hypercortisolism
- osteoporosis is a prominent
- familial cases: part of Carney's complex:
- myxomas (cardiac, cutaneous, and mammary)
- pigmented skin lesios (ephelides, lentigines, blue nevi)
- endocrine overactivity (acromegaly due to pituitary adenoma, precocious puberty due to large cell calcifying Sertoli cell tumor of testes)
- Schwannomas

- multiple pigmented cortical nodules (1-3 mm) in an otherwise atrohic cortex
- glands usually nml size! or smaller

- nodules composed of large granular eosinophilic cells, some with large hyperchromatic nuclei
- lipofuscin pigment

- pigmented cortical adenoma: larger, solitary, 3-6 cm