Pathology Heart, Kidney, Lungs

Front 1

Back 1

Boxcar nuclei indicative of LV hypertrophy

Front 2

Tet spells

Back 2

Tetralogy of Fallot; relieved by squatting to improve O2 circulation. Children suddenly develop deep blue skin, nails and lips after crying, feeding, having a bowel movement, or kicking his or her legs upon awakening

Front 3

1) ASD a/w ___________

2) PDA a/w ___________

3) Mitral valve prolapse a/w __________

4) Hypertrophic cardiomyopathy a/w __________

Back 3

1) Down's Syndrome

2) Turner Syndrome

3) Marfan's Snydrome

4) Freidrich's ataxia

Front 4

Back 4

Patent Ductus Arteriosus (PDA), specifically the adult form, where the obstruction is proximal to the aorta. Collateral circulation to the internal thoracic arteries causes rib notching.

Front 5

Progression of MI

A)

B)

C)

D)

E)

Back 5

A) Coagulative necrosis with wavy fibers (removes nucleus) - 1 day old

B) PMN infiltrate; risk for fibrinous pericarditis - few days to 1 week old

C) Removal of necrotic debris by macrophages; risk for ventricular wall rupture and cardiac tamponade - 1 week old

D) Granulation tissue characterized by loose CT and many capillaries -- few weeks old

E) Healed infarct with dense collagenous scar; risk of aneurysm or Dressler Syndrome - 1 month old

Front 6

Complication from MI

Back 6

Recent expansion anteroapical infarct with wall stretching and thinning and mural thrombus; may lead to system emboli to brain.

Front 7

Complication from MI

Back 7

Apical LV aneurysm which will likely lead to CHF

Front 8

LV hypertrophy due to calcific aortic stenosis

1) Congenital relationship?

2) _____________ murmur

3) Abnormal heart sound?

Back 8

1) A/w bicuspid aortic valve

2) Crescendo-decrescendo mid-systolic ejection murmur

3) Paradoxically split S2

Front 9

Vegetations a/w heart diseases

1)

2)

3)

4)

Back 9

1) Rheumatic heart disease (along line of closure of affected valve)

2) Infective endocarditis (large, irregular, destructive masses extending from valve leaflets to adjacent structures)

3) Nonbacterial thrombotic endocarditis (small'ish non-destructive vexations along line of closure)

4)Libman-Sacks endocarditis (a/w SLE) are seen on either side of the affected valve

Front 10

In RF, vegetations alone line of closure of affected valve is called ________. Myocarditis with focal inflammatory lesions are called _______. Modified myocytes that look like "caterpillar cells" are called _________.

Back 10

Verrucae; Aschoff bodies; Anitschkow cells.

Front 11

Infective endocarditis. Describe clinical signs seen.

LL, LR

Back 11

LL: nailbed splinter hemorrhades

LR: Janeway lesions

+ "Roth spots" which are retinal hemorrhages

+ Painful fingertip nodules which are Osler nodes

Front 12

1) Subacute endocarditis a/w what bug?

2) Acute endocarditis a/w what bug?

3) IV drug users are susceptible to pathology in ______ valve with _______ (what bug?).

Back 12

1) Strep viridans on previously myxomatous MV

2) Staph aureus

3) Tricuspid; staph aureus

Front 13

_____________ is associated with primary tumor of the small intestine that secretes 5-HT. Classic sx include facial flushing, cramps, diarrhea, asthma-like bronchoconstriction.

Back 13

Carcinoid heart disease

Front 14

_____________ myopathy causes a banana-shaped ventricular lumen and possible sudden death in young athletes.

Back 14

Hypertrophic cardomyopathy with asymmetric septal hypertrophy.