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13 Cards in this Set
- Front
- Back
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Marker of endothelial injury and activation in acute respiratory distress syndrome (ARDS)
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Endothelian and von Willebrand
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Morphology of acute lung injury (ALI)
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Diffuse alveolar damage with alveolar walls becoming lined with waxy hyaline membranes
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Chronic bronchitis
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Bronchus; mucus gland hyperplasia and hypersecretion; cough and sputum production
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Bronchiectasis
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Bronchus; airway dilation and scarring; persistent infections; cough, purulent SMELLY sputum and fever. Permanent dilation of bronchi and bronchioles caused by destruction of the muscle and elastic tissue
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Small-airway disease/ bronchiolitis
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Bronchiole; inflammatory scarring/obliteration; cough and dyspnea
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Emphysema
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Acinus, irreversible airspace enlargement and wall destruction; dyspnea
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Panacinar emphysema
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Uniformly enlarged from bronchiole to terminal alveoli, unlike centriacinar it is found in lower lungs, associated with alpha1 antitrypsin deficiency
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Why do alveolar walls become damaged?
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Protease-antiprotease mechanism, aided and abetted by imbalance of oxidants and antioxidants
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Interstitial emphysema
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Entrance of air into the connective tissue stroma of the lung, mediastinum, or subcutaneous tissue
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Reid Index
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Normally .4, indicates size of mucous gland, increased in bronchitis
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Atopic v non-atopic asthma
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Atopic is allergy induced
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Morphology of asthma
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Overdistended lunges with small areas of atelectasis. Curshmann spirals (mucus plugs). Charcot-Leyden crystals
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Associated with primary ciliary dyskinesia and Kartagner syndrome
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Bronchiectasis
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