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41 Cards in this Set
- Front
- Back
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What is Dentinal Sclerosis?
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Protective dentin response. Can be physiologic (aging), or pathological (caries, crystals form..)
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Acute Pulpitis
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High virulent bacteria
Abscess Formation Non-sclerosed dentinal tubules (no reactive-protective dentin response) |
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Chronic Pulpitis
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Lower virulence bacteria
Slow progression thru sclerosed/calcified tubules Fibroblasts present, necrotic pulp |
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Reversible Pulpitis
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Pain with cold, short duration, localized pain.
Removal of stimulus --> heals |
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Irreversible Pulpitis
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Pain with hot, long duration, harder to localize
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What types of necrosis will you see in pulpal necrosis?
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Abscess = liquefactive
If blood supply problem / trauma / hypoxia = Coagulative Widespread bacteria/N-waste = Gangrenous (smelly) |
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Periapical Abscess
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Purulent exudate at apex of nonvital tooth.
Sensitive to percussion (elevated tooth) Tx: incise and drain. A-b's if severe. |
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Parulis
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Un-tx abscess draining into gingiva
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Ludwig's Angina
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Un-tx abscess draining into floor of mouth
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Periapical Granuloma
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Misnomer! Not true granuloma
ASYMPTOMATIC See granulation tissue, no EPT response, radioluscent. Tx: RCT and/or ext |
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Periapical (Radicular) Cyst
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Apex of non-vital tooth
Asymptomatic No EPT response Similar to periapical granuloma...tx is the same..(endo/ext) |
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Phoenix Abscess
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Abscess within periapical granuloma, due to CHANGE IN FLORA
- immune compromised / etc |
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Osteomyelitis
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Inflammation of bone, very destructive.
From fracture / tooth infection Mandible and Men more frequent Lots of predisposing factors... |
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Acute Osteomyelitis
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Rapid spread of periapical abscess
Very painful Histo: no osteocytes. Dead bone (sequestrum) tx: high high dose antibiotics |
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Chronic Osteomyelitis
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From acute or de novo
Pain, but less intense and severe than acute Will see fibrotic tissue mixed into sequestrum |
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Condensing osteitis (Focal sclerosing osteomyelitis)
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Formation of NEW BONE at the apex.
Kids more often Cause: low-grade infection of pulp Tx: endo or exo (?) bone WILL have osteocytes! |
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Tooth Fusion
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Tooth count is one less.
Ex: #2 and 3 merge into one tooth Dx: must count all the teeth |
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Tooth Gemination
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Teeth still fused, but normal tooth count. (ex: two "parts" of a tooth coming together and fusing to form one)
Dx: must count all the teeth |
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Microdontia
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Pegged teeth
Down's syndrome common |
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Mesiodens
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miniature tooth found in midline
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Dens invaginatus (dens in dente)
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tooth in a tooth
lateral most common |
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Dilaceration
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root takes a different direction
Can be developmental OR TRAUMA during dev Difficult to extract |
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Taurodontism
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neck/cervical elongated
"bull teeth" |
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Hypercementosis
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Excess cementum production
Associated with paget's disease. Teeth are okay. |
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Concrescence
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Root fusion between adjacent teeth. If ext one, the other must go also
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Enamel Hypoplasia
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Enamel has little pits in places.
Conditions: mother has virus, early onset chicken pox, measles Chronic: will get tertiary dentin |
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Attrition
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grinding//bruxism on occlusal surface
Chronic: will get tertiary dentin |
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Abrasion
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acid..chewing on weird stuff...
Chronic: will get tertiary dentin **this definition sucks** |
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Erosion
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from acid accumulation. Bulimia.
Chronic: will get tertiary dentin |
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Ectodermal Dysplasia
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Form of hypodontia
Reduced ectoderm. (hair fragile/lack sweat glands) |
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Hereditary Oligodontia
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Form of hypodontia
Heterogenous (multiple causes) Will transmit to offspring no matter what |
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Cleidocranial dysplasia
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Form of hyperdontia
Missing clavicles |
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Familia adenomatous polyposis "FAP"
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Form of hyperdontia
Prone to cancer also. |
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Amelogenesis Imperfecta
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heterogenous (dom/rec/X-link)
Hypoplastic (production defect) Hypomaturation (produce normal, bad mature) Hypocalcified (produce normal, bad calc) AMELX / FAM83H... Teeth do not bond well!!! Will need full-coverage |
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Dentinogenesis Imperfecta
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Type 1: OI assoc.
Type 2: DSPP mutation only Yellow/orange color Bulbous crowns, cerv constriction Obliterated pulp chambers (dentin overgrowth) |
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Dentin Dysplasia Type 1
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DSPP gene
Radicular dentin screwed "Rootless teeth" Pulps obliterated |
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Dentin dysplasia Type 2
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similar to DI
DSPP gene Coronal Dentin |
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Hypophosphatasia
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alkaline phosphatase defect, needed for bone metab
phosphoethanolamine in urine no or little CEMENTUM Large canal/pulp, PDL screwed ALPL / DLX3 gene defects |
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Hyperbilirubinemia / Fluorosis / Tetracycline
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Bilirubin --> green teeth
Fluorosis --> doesn't weaken tooth Tetracycline --> yellow/red |
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Porphyria
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genetic, weird stuff gets incorporated into dev teeth that shouldn't, weakens and browns them
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Regional Odontodysplasia
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1-3 neighboring teeth abnormal
ghost teeth, thin enamel large pulp canal possible bv supply prob during dev if in adult dentition, must have had in primary |
