• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/31

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

31 Cards in this Set

  • Front
  • Back
Pt reports abdominal cramping and diarrhea after he eats wheat products. Biopsy shows enteritis with loss of villi and lymphocytic infiltration of the lamina propria. what is the dx
celiac dz
this dz is characterized by foamy macrophages in the lamina propria
whipples dz
pt presents with sudden severe abdominal pain and tenderness. What are you worried about?
Ischemia/Infarction
pt presents with severe abdominal pain, abdominal distension, vomiting, constipation, and failure to pass flatus. what is the dx
Intestinal obstruction
Most cases of small bowel obstruction are caused by one of four things. What are they?
hernias, adhesions, intussusceptions, and volvulus
pt c/o intermittent attacks of diarrhea, fever, and abdominal pain. Labs show vit B12 deficiency. Bx shows bowel wall thickening and creeping fat. A deep fissure extending into the muscle wall, and noncaseating granuloma is present in the lamina propria
CD
pt presents c/o recurrent bouts of bloody mucoid diarrhea that may last for days, weeks, or months followed by asymptomatic periods. Bx of colon shows marked chronic inflammation of the mucosa with atrophy of colonic glands, moderate submucosal fibrosis, and a normal muscle wall.
There is also presence of knobby pseudopolyps.
Ulcerative colitis.
increased risk of colon cancer
ulcerative colitis
Colonic perforation or toxic megacolon are potential complications of this disease.
UC
congenital absence of Meissner submucosal and Auerbach myenteric plexuses in a segment of GI tract. Peristalsis will not progress past the affected segment and the bowel proximally will dilate and eventually become a megacolon.
Hirschprung disease
This is an autosomal dominant condition associated with the development of 500-1000 colonic polyps. The polyps begin to develop by age 16. Without treatment the risk of cancer is 100% and so prophylactic colectomy is the typical treatment.
familial adenomatous (FAP) polyposis syndrome
FAP syndrome with extra colonic manifestations that can include osteomas and fibromatosis
Gardner syndrome
FAP syndrome with central nervous system tumors.
Turcot syndrome
most common site of colon CA
rectosigmoid colon
pt presents with anemia & blood in the stool, changes in bowel habits & weight loss, malaise, and weakness
Colorectal cancer
Tumor Stage
Tis
intramucosal
Tumor Stage
T1
submucosa
Tumor Stage
T2
Extending into the muscularis propria but not penetrating through
Tumor Stage
T3
Penetrating through the muscularis propria into subserosa
Tumor Stage
T4
Tumor directly invades other organs or structures
On barium enema 65 y/o male with low fiber diet shows outpouchings of the mucosa and submucosa through a gap in the muscularis
Diverticular disease
Sessile
-flat
Polyp
-like a mushroom
Adenoma
- benign
Adenocarcinoma
- malignant
Hyperactive mucosa
-benign non neoplastic polyps
2 types of adenomatous polyps
tubular adenoma and the villous adenoma
these are by far the most common type of adenomatus polyps. These are the classic pedunculated mushroom-shaped adenoma
Tubular adenomas
this type of adenomatous polyp is sessile, large, flat, and villous
Villous adenomas
The highest risk is for CA is in _____________
villous adenomas greater than 4 cm in size.
some ________ secrete copious amounts of protein and potassium-rich mucus and can result in hypoproteinemia and/or hypokalemia.
villous adenomas