Path And Pharm 11

Front 1

Functions of liver

Back 1

Remove toxins/glucose
Make albumin, coagulation factors, & bile

Front 2

Etiology for Hepatitis

Back 2

Hep A-E
Drugs

Front 3

S/S of Hepatitis

Back 3

Jaundice & Constitutional

Front 4

Pathogenic Process for Cirrhosis

Back 4

Necrosis & Repair

Front 5

S/S of Cirrhosis

Back 5

Portal Hypertension->peritoneal edema
Large Spleen
Esophageal Varices
Jaundice

Front 6

Gallstones

Back 6

Cholelithiasis

Front 7

Warts
Pathogenic Processes:
Etiology:

Back 7

Chronic Inflammation + Hyperplasia
HPV

Front 8

Eczema Pathogenesis
and Etiologies and outcomes

Back 8

Type 4 (Cell-Mediated) Chronic Inflammation (T Cytotoxic)

Genetic + Environmental

Resolve or Chronic

Front 9

Psoriasis Pathogenic Processes

Back 9

Type 4 Chronic Inflammation; Hyperplasia

Front 10

Psoriasis S/S

Back 10

Red plaque
silvery scale

Front 11

Hives

Back 11

Urticaria

Front 12

Urticaria pathogenesis, etiology

Back 12

Type I Chronic Inflammation
Allergens

Front 13

Redness; papulovesciular; oozing, and crusting are all S/S of

Back 13

Eczema

Front 14

Red plaque, silvery scales; pinpoint bleededing; extensor/dorsal surfaces

Back 14

Psoriasis

Front 15

S/S of GERD

Back 15

Heartburn

Front 16

Pathogenesis of Celiac

Back 16

Type 2 Chronic & Necrosis

Front 17

We can get skin edema in

Back 17

both the epidermis or dermis

Front 18

Psoriasis: Hyperplasia of ___

Back 18

Keratinocytes

Front 19

Name the 2 skin diseases that have both genetic & environmental etiologies

Back 19

Both Eczema & Psoriasis have

Front 20

Rendess, edematous, plaques; pruritis

Back 20

Urticaria/Hives

Front 21

Pathogenic Responses for Burns

Healing can be...

Back 21

Necrosis, Acute Inflammation, Repair

Re-epithelialize with no scar
Granulation Tissue with scar (fibrosis)

Front 22

2 Benign Neoplasias of the skin

Back 22

Seborrheic Keratoses
Nevus = Mole

Front 23

Seborrheic Keratoses Etiology:
Mutation of
Results in an increase of

Back 23

FGF (fibroblastic growth factor) receptor
keratinocytes

Front 24

Dark, waxy plaques; “stuck on”

Back 24

Seborrheic Keratoses

Front 25

Etiology of Melanoma

Back 25

Genetics/Sun Exposure

Front 26

Etiology of Nevus
Pathogenesis: Mitosis of

Back 26

Genetics +/- Sun Exposure
Melanocytes

Front 27

Basal Cell Carcinoma:
Etiology:
Involves the ___ epidermal cells (karatinocytes)
S/S:

Back 27

Genetics +/- Sun Exposure

deepest

slow growing Pearly Papules

Front 28

Most common human malignancy

Back 28

Basal Cell Carcinoma

Front 29

Squamous Cell Carcinoma:
Etiology: ___

Back 29

Sun Exposure; skin trauma

Front 30

Compare/Contrast Basal Cell vs. Squamous Cell
___ vs. ___ appearance
grows___ vs ___
Both rarely ___
Both involve

Etiologies: Both have ___.
Difference: ___ vs. ___

Back 30

pearly vs scaly
slow vs more rapidly
rarely metastasize
keratinocyte

Sun Exposure
Genetics vs. skin trauma

Front 31

Red, scaly plaque; Sharp borders; +/- ulceration

Back 31

Squamous Cell CA

Front 32

Breast Cancer scientific name

Back 32

Breast Adenocarcinoma

Front 33

Etiology of Breast Cancer

Back 33

Genetics
BRCA Gene 1&2

Front 34

Breast Cancer:
Malignant clone of ___
If a woman is E/P positive, we can use ___ to block the receptors

Back 34

duct epithelial cells
Tamoxafin

Front 35

___—between the endocervix and ectocervix where infection occurs

Back 35

External Os

Front 36

What do you get a papsmear for?

Back 36

Cervical Squamous Cell Carcinoma

Front 37

Cervical Squamous Cell Carcinoma:
Inactivates ___ --> results in malignant clone of ___

Back 37

Tumor Suppressor Genes
squamous cell

Front 38

Leiomyoma common term
Etiology:
Pathogenesis

Back 38

Fibroids
Unknown
Benign cloning of the myometrium

Front 39

Leiomyoma S/S for larger tumors
Diagnosed by

Back 39

Bleeding outside normal periods
Pregnancy difficulties

Ultrasound

Front 40

PID stands for
Etiology

Outcomes

Back 40

Pelvic Inflammatory Disease

STD; Neisseria Gonorrheae; chlamydia

Ectopic Pregnancy

Front 41

Pathogenesis for Fallopian Tubes

Back 41

Acute Inflammation ->Necrosis->Repair->Infertility due to obstruction

Front 42

Pelvic Floor Disorders: Etiology

Back 42

Any disease in pelvis; pregnancy/delivery

Front 43

Practicing control of the pelvic organs

Organs peaking through pelvic muscles

Back 43

kegels

prolapse

Front 44

Birth control Pills are composed of both ___

Back 44

Estrogen & Progesterone

Front 45

Female oral contraceptives:
Act to inhibit ___ and impair___.
Name some methods

Back 45

ovulation
uterine lining build-up
Pills; transdermal; vaginal ring

Front 46

Progesterone Only Contraceptives:
mini BCP: only ___ effective
12 week injection: ___
5 year implant: ___

Back 46

98%
Depo-provera
Nonplant

Front 47

List 2 benefits and a drawback to Postmenopause replacement therapy

Back 47

Reduces hot flashes
Maintains bone density

Increases Cardiovascular disease (for MI)

Front 48

Inflammation of prostate, epididymsis, testis

Back 48

Prostatitis
Epididymitis
Orchitis

Front 49

Why would men take estrogen?

Back 49

Prostatic Diseases

Front 50

BPH stands for ___.
What do we not need in this defn?

Etiology?

S/S

Back 50

Benign Prostatic Hyperplasia
Benign

Idiopathic

Lateral Prostatic Enlargement

Front 51

Benign Prostatic Hyperplasia pathogenesis:
Increase in the number of cells from___ in lateral regions.

Back 51

circumscribed nodules

Front 52

Urinary obstruction; dribbling; frequent peeing; difficult starting are all S/S of

Back 52

Benign prostatic Hyperplasia

Front 53

Cancer of the prostate is called ___

Etiology:

Typically occurs with

Back 53

Prostatic Adenocarcinoma (since Prostate is a gland)

Unkonwn

BPH = benign prostate hyperplasia

Front 54

Lateral Prostatic Enlargement: ___
Posterior Prostatic Enlargement: ___

Back 54

BPH
Prostatic Adenocarcinoma

Front 55

How can we test for Prostatic Adenocarcinoma?

Back 55

Check for the increase Prostate Specific Antigen (PSA)

Front 56

Testosterone and derivatives = ___ = ___

Back 56

Androgens = Anabolic steroids

Front 57

Anabolic = ___
Steroids = ___ (included cortisone, aldosterone, TESTOSTERONE, estrogen, progesterone)

Back 57

to build up
group of hormones derived from cholesterol

Front 58

Anabolic steroids promote protein ___, ___ growth, and increase in ___ production

Back 58

synthesis
bone
RBC

Front 59

Adverse effects of Anabolic Steroids
Bone:
Mood:
Skin:
Infection:
Reproductivity:

Back 59

Stunts growth
Aggressive/Depression
Acne
HIV
testicular atrophy/dec. sperm count