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47 Cards in this Set

  • Front
  • Back
q.d. (qd or QD)
once a day
BID
two times a day
TID
three times a day
q.i.d
means 4 times a day
Indications for positive pressure ventilation
1. Acute exacerbation of COPD

2. status asthmaticus

3 Hypoxemia (CF, Pneumonia, CHF), Neuromuscular pt (GB, ALS, MG)

5 DNR

6 Facilitate weaning

7 Chronic respiratory failure

8 Nocturnal hypoventilation, desaturation.

9. Acute cardiogenic pulmonary edema

10. community acquired PNA

YOU MUST BE SPONTANEOUSLY BREATHING TO USE NIPPV
In acute asthmatic crisis, patients present with
hypercarbia and hypoxemia
IPAP
Inspiratory level
increased pressure for boost on inhalation
increased ventilation when raised
Aids ventilation (PaCO2)
Reduces WOB
EPAP
Expiratory level (FRC)
Same as PEEP / CPAP while exhaling
Aids oxygenation (PaO2)
Increases FRC
Keeps lungs at a baseline pressure
The difference between IPAP and EPAP is

Increased PS =
PSV (pressure support ventilation)

Increased ventilation (PCO2)
NIPPV - BiLevel
Treatment Goals
CHF
Cardiogenic pulmonary edema
PEEP/CPAP/EPAP
elevates the baseline airway pressure.
Keeping the baseline pressure aids in oxygenation by recruiting alveoli increasing FRC
Decreases WOB
Alveoli splinting = Pulmonary Edema clearing
Decreases preload to heart by positive thoracic pressures
IPAP
is pressure for tidal volume enhancement

aids in reduction of work of breathing as well as ventilation of PCO2
BPAP Contraindications
Hemodynamic instability
ICP > 20 mm HG
Patient with Hypoventilation (absolute, must be spontaneously breathing)
Hazards and complications of CPAP
Barotrauma
Hypoventilation PcO2 (must be spontaneously breathing
Gastric distention
Vomiting and aspiration
Hazards of NPPV
leaks
nasal, sinus or patient discomfort
gastric insufflation
eye irritation
barotrauma
aspiration pneumonia
mucous plugging
hypoxemia
pulmonary toilet
If a patient is determined to have thick secretions and he is having trouble expectorating (spitting up), and this is deemed to be causing respiratory distress, than any effort necessary is done to break up secretions so the patient can spit them up. This usually includes bronchodilator therapy, mucolytic (mucomyst), and chest physiotherapy, cough and deep breathing and incentive spirometry.
LAM
LAM, or lymphangioleiomyomatosis (lim-FAN-je-o-LI-o-MI-o-ma-TO-sis), is a rare lung disease that mostly affects women of childbearing age.

In LAM, abnormal, muscle-like cells begin to grow out of control in certain organs or tissues, especially the lungs, lymph nodes, and kidneys.

Over time, these LAM cells can destroy the normal lung tissue. As a result, air can’t move freely in and out of the lungs. In some cases, this means the lungs can’t supply the body’s other organs with enough oxygen.
Report Expectations
Age
Sex
DX
Hx
Therapy
BS
02 Therapy / Sat (last)
Cough
Code Status
ETOH
stands for ethanol. The acronym is commonly used in the description of a patient who displays symptoms of over consumption or even withdrawal form alcohol beverages.
Eosinophilic pneumonia (EP)
a disease in which a certain type of white blood cell called an eosinophil accumulates in the lung. These cells cause disruption of the normal air spaces (alveoli) where oxygen is extracted from the atmosphere. Several different kinds of eosinophilic pneumonia exist and can occur in any age group. The most common symptoms include cough, fever, difficulty breathing, and sweating at night. EP is diagnosed by a combination of characteristic symptoms, findings on a physical examination by a health provider, and the results of blood tests and x-rays. Prognosis is excellent once most EP is recognized and treatment with corticosteroids is begun.

When a cause can not be found, the EP is labeled "idiopathic." Idiopathic EP can be divided into "acute eosinophilic pneumonia" (AEP) and "chronic eosinophilic pneumonia" (CEP) depending on the symptoms a person is experiencing.

Individuals with CEP are often diagnosed with asthma before CEP is finally recognized.

The common characteristic among different causes of EP is eosinophil overreaction or dysfunction in the lung.

Chronic eosinophilic pneumonia was first described by Carrington[8] in 1969, and it is also known as Carrington syndrome. Prior to that, eosinophilic pneumonia was a well described pathologic entity usually associated with medication or parasite exposures. Acute eosinophilic pneumonia was first described in 1989
The most common causes of obstructive lung disease are:
Chronic obstructive pulmonary disease (COPD), which includes emphysema and chronic bronchitis
Asthma
Bronchiectasis
Cystic fibrosis
Obstructive lung disease makes it harder to breathe, especially during increased activity or exertion. As the rate of breathing increases, there is less time to breathe all the air out before the next inhalation.
Restrictive Lung Disease
People with restrictive lung disease cannot fully fill their lungs with air. Their lungs are restricted from fully expanding.

Restrictive lung disease most often results from a condition causing stiffness in the lungs themselves. In other cases, stiffness of the chest wall, weak muscles, or damaged nerves may cause the restriction in lung expansion.

Some conditions causing restrictive lung disease are:

Interstitial lung disease, such as idiopathic pulmonary fibrosis
Sarcoidosis, an autoimmune disease
Obesity, including obesity hypoventilation syndrome
Scoliosis
Neuromuscular disease, such as muscular dystrophy or amyotrophic lateral sclerosis (ALS)
what causes respiratory acidosis?
insufficient alveolar ventilation
pulmonary disease, CNS depression, drugs causing respiratory depression
What causes respiratory alkalosis?
Alveolar hyperventilation
Stress, emotional upset, hypoxia, fever, CNS trauma
what causes metabolic acidosis?
Lactic acidosis, ketoacidosis (diabetes), renal failure, diarrhea
what causes metabolic alkalosis
Hypokalemia (most common cause), low chloride, diuretics, corticosteroids, vomiting, nasogastric tube
Duoneb
SVN:
Ipratropium Bromide 0.5 mg
and
Albuterol 2.5mg, qid
Advair
Fluticasone propionate/salmeterol
Spiriva
Tiotropium Bromide
Tobramycin
TOBI
Pulmicort Respules
Budesonide
How many RTs on staff at Palomar?

How many RTs on staff at Pomerado?
7-10


2 minimum
Vent settings to note
Rate
Tidal volume
FIO2
PEEP
Suctioning
90% preparation
10% execution
side effect - Vagal stimulation (potential brady cardia)
continuous pulse ox
suction pressure
80-120 (adult)
60-80 neonate
Hyperoxygenate first
suction 2 times max, O2 in between
cough?
pause
suction
oxygen dissociation curve
Shifts to the right: Low affinity for oxygen Releases oxygen more easily
High CO2
High H+ (low pH)
High temperature
High 2,3, DPG (product of anaerobic glycolysis
Shifts to the left: high affinity for oxygen - Holds on to oxygen more tightly
Fetal hemoglobin
Low CO2
AFB x 3
Also known as: TB culture and sensitivity; Mycobacterial smear and culture
Formal name: Acid-fast bacillus smear and culture and sensitivity
Related tests: TB Screening Tests; Bacterial Wound Culture; Susceptibility Testing; Mycobacteria tuberculosis nucleic acid amplification test; TB NAAT; Body Fluid Analysis; Sputum Culture; Adenosine Deaminase


http://labtestsonline.org/understanding/analytes/afb-culture/tab/test
Ipap
Ventilation
epap
oxygenation
ezpap
5-15 liters per minute

goal: 20 cm H20
splint cough
Pillow splinting is done either seated upright or lying down, holding a pillow over the incision site -- whether abdominal, chest or on either side. The best technique is to wrap both arms or hands as fully as possible across the pillow and press firmly. A slow, deep breath in produces less discomfort in this position. You can cough at the top of this deep breath, pressing firmly as you do so. Splinting is also used at least four times an hour to make it easier to take a series of slow, relaxed deep breaths in and out to keep your lungs expanded, mucus-free and healthy.
MDI components
Medication

Propellant

Surfactant
Albuterol
onset: 15 mins

Peak: 30-60 mins

Duration: 5-8 hours
tension pneumothorax
laceration of lung

air forced into pleural space
Hyponatremia
is an electrolyte disturbance in which the sodium ion concentration in the plasma is lower than normal.
MDI administration
tight seal on MP

slow deep breath in

hold for 5 seconds
Signs and Symptoms of Pneumothorax
sudden, sharp pain
shortness of breath
unilateral chest wall rise
increased heart rate
increased respiratory rate
IPPB reaches pressure limit much sooner than before
What if Pt has an order for Duoneb and Spiriva?
Consult nurse regarding double dosing same family of medicines:

Iptratropium bromide (Duoneb)
and
Tiotropium bromide (Spiriva)