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35 Cards in this Set

  • Front
  • Back
Visceral Pain
originates from larger interior organs (kidneys, gallbladder, etc)
Deep Somatic Pain
blood vessels, joints, tendons, muscles, & bone
Cutaneous Pain
skin surface & SQ tissue
Referred Pain
felt at a particular site & orginates in another
Neuropathic Pain
doesn't adhere to typical, predictable phases in nociceptive pain; most difficult to assess & treat; often perceived long after injury heals
Acute Pain
short-term, self limited, lasts the expected recovery time
Chronic Pain
-malignant or
-Nonmalignant
diagnosed when pain continues for 6+ months
-"intractable" pain, induced by tissue necrosis or stretching of organ by growing tumor (malignant)
-pain becomes a disease in itself, e.g. arthritis, low back pain (nonmalignant)
Infant considerations
"CRIES"
at 20 weeks gestation, can feel pain
C-crying
R-requires O2 for sat > 95%
I-increased vital signs
E-expression
S-sleepless
Gender differences
Women 2-3 times more likely to experience migraines during childbearing years
More sensitive to pain during premenstrual period
Women 6x more likely to have fibromyalgia
Nociceptors
specialized nerve endings, located w/in skin, CT, muscle, thoracic/abd./pelvic viscera, directly stimulated by trauma/injury, indirectly stimulated by chemical mediators released from damaged site; carry pain signal to CNS with A & C fibers
A fibers
myelinated, larger in diameter, transmit pain signal rapidly to CNS, localized, short-term, sharp sensations
C fibers
unmyelinated, smaller, transmit pain signal slower, diffuse & aching sensations, persist after initial injury
Interneuron
fibers synapse, located w/in specific area of cord (substantia gelatinosa)
Substantia Gelatinosa
Lamina II-layers of nerve cells divided and numbered; receives sensory input from various areas of body
Anterolateral Spinothalamic Tract
pain signal crosses then ascends to brain (AP tract)
Transduction
Phase I of nociception; noxious stimuli in periphery, injured tissues release chemicals(prostaglandin, histamine, serotonin, bradykini), these neurotransmitters sent action potential pain message along fibers to spinal cord (dorsal horn), 2nd set of neurotransmitters carry message across synaptic cleft (sub. P, glutamate, ATP)
Transmission
Phase II of nociception; impulse moves to brain, opioid receptors able to block pain c exogenous/endogenous opioids, if pain's not blocked-impuls moves to brain to end in brainstem & thalamus, dispersed to higher cortical areas
Perception
Phase III of nociception; awareness of pain sensation, limbic system accounts for emotional responses, somatosensory areas characterize sensation
Modulation
Phase IV of nociception; pain message inhibited; decending pathways from brain produce 3rd set of neurotransmitters-slow down impulse producing analgesic effect; norepi, neurotensin, GABA & endogenous opioids (endorphins, enkephalins, dynorphins
Aging Adult Considerations
pain is not part of normal aging process; somatosensory cortex unaffected by dementia, limbic system affected by dementia-may interpret & report pain message differently
Initial Pain Assessment Questions
Where is your pain?
When did it start?
What does it feel like?
How much pain are you having now?severity scale 0-10
What makes it better or worse?
How does your pain limit your ADLs?
How do you usually behave when in pain?
What does this pain mean to you?
Objective data-Joints
size & contour, measure circumference, check ROM, normally no pain, tenderness, crepitation (audible/palpable crunch)
abnormal-->swelling, inflammation, deformity, diminished ROM, increased pain on palpation, crepitation
Objective data- Muscles and Skin
inspect skin/tissues-color, swelling, masses; test ability to perceive sensation
abnormal-->bruising, lesions, open wounds, atrophy, bulging, analgesia(absent pain sensation), hyperalgesia(increased sensation)
Objective data- Abdomen
observe contour & symmetry, palpate muscle guarding & organ size, note areas of referred pain
abnormal-->swelling, bulging, herniation, inflammation, organ megaly,
Acute Pain Behaviors
guarding, grimacing, vocalizing, agitation, restless, stillness, diaphoresis, change in vitals
Chronic Pain Behaviors
Bracing, rubbing, diminished activity, sighing, change in appetitie
Acute pain response-Cardiac
tachycardia, elevated BP, increased myocardial O2 demand, increased CO
Acute pain response-Pulmonary
hypoventilation, hypoxia, decreased cough, atelectasis
Acute pain response- GI
N/V, paralytic illeus
Acute pain response-Renal
oliguria ( >25cc/hr), urinary retention
Acute pain responses- Musculoskeletal
spasm, joint stiffness
Acute pain response- endocrine
increased adrenergic activity
Acute pain response- CNS
fear, anxiety, fatigue
Acute pain response- Immune
impaired cellular immunity, impaired wound healing
Poorly controlled chronic pain responses
depression, isolation, limited mobility/function, confusion, family distress, diminished quality of life