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35 Cards in this Set
- Front
- Back
Visceral Pain
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originates from larger interior organs (kidneys, gallbladder, etc)
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Deep Somatic Pain
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blood vessels, joints, tendons, muscles, & bone
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Cutaneous Pain
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skin surface & SQ tissue
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Referred Pain
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felt at a particular site & orginates in another
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Neuropathic Pain
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doesn't adhere to typical, predictable phases in nociceptive pain; most difficult to assess & treat; often perceived long after injury heals
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Acute Pain
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short-term, self limited, lasts the expected recovery time
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Chronic Pain
-malignant or -Nonmalignant |
diagnosed when pain continues for 6+ months
-"intractable" pain, induced by tissue necrosis or stretching of organ by growing tumor (malignant) -pain becomes a disease in itself, e.g. arthritis, low back pain (nonmalignant) |
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Infant considerations
"CRIES" |
at 20 weeks gestation, can feel pain
C-crying R-requires O2 for sat > 95% I-increased vital signs E-expression S-sleepless |
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Gender differences
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Women 2-3 times more likely to experience migraines during childbearing years
More sensitive to pain during premenstrual period Women 6x more likely to have fibromyalgia |
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Nociceptors
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specialized nerve endings, located w/in skin, CT, muscle, thoracic/abd./pelvic viscera, directly stimulated by trauma/injury, indirectly stimulated by chemical mediators released from damaged site; carry pain signal to CNS with A & C fibers
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A fibers
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myelinated, larger in diameter, transmit pain signal rapidly to CNS, localized, short-term, sharp sensations
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C fibers
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unmyelinated, smaller, transmit pain signal slower, diffuse & aching sensations, persist after initial injury
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Interneuron
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fibers synapse, located w/in specific area of cord (substantia gelatinosa)
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Substantia Gelatinosa
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Lamina II-layers of nerve cells divided and numbered; receives sensory input from various areas of body
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Anterolateral Spinothalamic Tract
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pain signal crosses then ascends to brain (AP tract)
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Transduction
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Phase I of nociception; noxious stimuli in periphery, injured tissues release chemicals(prostaglandin, histamine, serotonin, bradykini), these neurotransmitters sent action potential pain message along fibers to spinal cord (dorsal horn), 2nd set of neurotransmitters carry message across synaptic cleft (sub. P, glutamate, ATP)
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Transmission
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Phase II of nociception; impulse moves to brain, opioid receptors able to block pain c exogenous/endogenous opioids, if pain's not blocked-impuls moves to brain to end in brainstem & thalamus, dispersed to higher cortical areas
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Perception
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Phase III of nociception; awareness of pain sensation, limbic system accounts for emotional responses, somatosensory areas characterize sensation
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Modulation
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Phase IV of nociception; pain message inhibited; decending pathways from brain produce 3rd set of neurotransmitters-slow down impulse producing analgesic effect; norepi, neurotensin, GABA & endogenous opioids (endorphins, enkephalins, dynorphins
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Aging Adult Considerations
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pain is not part of normal aging process; somatosensory cortex unaffected by dementia, limbic system affected by dementia-may interpret & report pain message differently
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Initial Pain Assessment Questions
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Where is your pain?
When did it start? What does it feel like? How much pain are you having now?severity scale 0-10 What makes it better or worse? How does your pain limit your ADLs? How do you usually behave when in pain? What does this pain mean to you? |
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Objective data-Joints
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size & contour, measure circumference, check ROM, normally no pain, tenderness, crepitation (audible/palpable crunch)
abnormal-->swelling, inflammation, deformity, diminished ROM, increased pain on palpation, crepitation |
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Objective data- Muscles and Skin
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inspect skin/tissues-color, swelling, masses; test ability to perceive sensation
abnormal-->bruising, lesions, open wounds, atrophy, bulging, analgesia(absent pain sensation), hyperalgesia(increased sensation) |
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Objective data- Abdomen
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observe contour & symmetry, palpate muscle guarding & organ size, note areas of referred pain
abnormal-->swelling, bulging, herniation, inflammation, organ megaly, |
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Acute Pain Behaviors
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guarding, grimacing, vocalizing, agitation, restless, stillness, diaphoresis, change in vitals
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Chronic Pain Behaviors
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Bracing, rubbing, diminished activity, sighing, change in appetitie
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Acute pain response-Cardiac
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tachycardia, elevated BP, increased myocardial O2 demand, increased CO
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Acute pain response-Pulmonary
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hypoventilation, hypoxia, decreased cough, atelectasis
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Acute pain response- GI
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N/V, paralytic illeus
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Acute pain response-Renal
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oliguria ( >25cc/hr), urinary retention
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Acute pain responses- Musculoskeletal
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spasm, joint stiffness
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Acute pain response- endocrine
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increased adrenergic activity
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Acute pain response- CNS
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fear, anxiety, fatigue
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Acute pain response- Immune
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impaired cellular immunity, impaired wound healing
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Poorly controlled chronic pain responses
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depression, isolation, limited mobility/function, confusion, family distress, diminished quality of life
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