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26 Cards in this Set

  • Front
  • Back
Normal intervals and segments:
PR
QRS
QT
PR: <0.2 s
QRS: <0.8 s
QT: <0.4 s
isoletric is normal in
PR and ST (flat line)
chest leads:
V1
V2
4th ICS either side of sternum
chest leads: V4
5th ICS MCL
chest leads: V3
between V2&V4
chest leads:V6
5th ICS, mitral area
chest leads: V5
5th ICS between V4&V6
bradycardia
tachycardia
bradycardia: <60
tachycardia >100
Large box intervals to count on EKG
300-150-75-60-50
what does regularly irregular mean?
RR intervals of different length, but overall pattern is present
Ex of Regular Regular EKG
Normal Sinus Rhythm
Sinus Bradycardia
Sinus Tachycardia
Regularly irregular EKG
Sinus Arrhythmia
premature atrial beat
foci overrides SA node (ex: adrenaline) Pwave different, irregular rate
what is the EKG sawtooth?
Atrial flutter, strong ectopic focus
Irregularly irregular, and pt. needs to be on coumadin is...
atrial fibrillation
Hypokalemia causes this..
low O2
much wider QRS
Premature ventricular contractions
When must you defibrilate?
Ventricular Tachycardia
no normal contractions, blood isn't circulating to organs
death
ventricular fibrillation
Conduction abnormalities (4)
1st degree atrioventricular block
2nd degree atrioventricular block, type 1
2nd degree atrioventricular block, type 2
3rd degree atrioventricular block
1st degree atrioventricular bloc
PR interval is > 0.2
impulse in AV node is delayed, longer than normal pause before ventricular stimulation
prolongued PR interval, longer than one large square
2nd degree atrioventricular block, type 1
Mobitz type I Wenckebach
progressively longer PR duration until no PR
going, going, gone...
2nd degree atrioventricular block, type 2
Mobitz type II
consistent normal PR, but then punctual Pwave with no QRS

ex- 2P:1QRS
or
3P:1QRS
3rd degree block, or complete
no relationship between P waves and QRS complex
AV node completely blocked
ventricular fibrilation + 3rd degree block =
not living
sympathetic
norep elicits excitatory response from B1 andrenergic receptors in <3
increase SA node pacing
increases force of myocardial contraction
constricts arteries = increase BP
parasympathetic
ACH activates cholinergic receptors to produce inhibitory effect
decrease SA node pacing
decrease force of contraction
dialates arteries = BP decrease