Study your flashcards anywhere!

Download the official Cram app for free >

  • Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off

How to study your flashcards.

Right/Left arrow keys: Navigate between flashcards.right arrow keyleft arrow key

Up/Down arrow keys: Flip the card between the front and back.down keyup key

H key: Show hint (3rd side).h key

A key: Read text to speech.a key


Play button


Play button




Click to flip

84 Cards in this Set

  • Front
  • Back
History of Present Illness
-Onset, duration, allergies, toxic exposure, lifestyle and dietary habits, smoking habits
-Symptoms patient is reporting: dyspnea, tachypnea, orthopnea, paroxysmalnoctural dyspnea (PND)
-Cough: productive, non-productive, sputum (1L with cold) production, hemoptysis (assess mouth and gums first)
Cardiopulmonary Symptoms
-Chest discomfort:
*causes: fluid in the lungs, pneumona, pleurisy, esophogeal, astma
-Edema, wght gain (2.2lbs = 1L)
-Dizziness, palpitations
-Fatigue, pallor
-Leg claudication: leg cramps w/activity and goes away when activity is stopped
(arterial insufficiency)
Cardiopulmonary Assessment
-Overall appearance of pt
*vital signs
*circumoral pallor (around mouth and nose)
-Auscultation of the heart and lungs
-*wheezing (constriction of the bronchi), crackles (fluid and air hitting one another), pleural rubs (sand paper)
-Respiratory: rate, depth, rhythm, equal expansion
-Observe for use of accessory muscles to breath
Observe for abnormalities:
*clubbing, barrel chest, sternal retractions, nasal flaring, changes in mental status
*intercostal retracting and diaphram breathing usually seen in kids
-Evaluate the extremeties
*Heart murmurs
Diagnostic Tests
-O2 saturation
-ABGs: arterial blood gasses
-CBC: complete blood count
-PFTs: pulmonary function tests
-Chest x-ray
-Heart catheterization
Upper Airway
-Anything above the larynx
Respiratory Conditions
-Common cold
-Sinusitis (acute or chronic)
*Pain over affected sinuses and headache
*maxillary (eyes and jaw)
*Frontal (forehead)
*Ethmoid (whole face)
-Tx: antibiotics, analgesics, decongestants, surgery
-Causes: deviated septum
*hypertrophy of turbinate bones (scrape away at the turbinates)
-Can be viral (cold) or bacterial or mechanincal (singers, speakers, intubation)
-*Chronic problem due to smoking, vocal abuse, endotracheal intubation, alcohol
-Can become severe enough to close off airway
-Only in young children
-Dry cough
-AKA spasmotic laryngitis
-partial airway obstruction
-Comes on suddenly following a cold (usually in middle of night)
-Barking seal cough
-Temp seldom elevated
-Tx: moist heat to relieve obstruction (shower)
(airway for babies dime size,
nickle for children)
-Inflammation of larynx, trachea, bronchi
-Very serious, complications of a cold
-Only in children < 4
-Thick sputum, high temp, couphing
-Tx: maintain airways, antibiotics, humidified O2
Other Upper Respiratory Problems
-Sore throat > 3 days: should have a throat culture
*can lead to endocarditis if not treated
-Pharyngitis: usually viral, can be bacterial
*group B-hemolytic strept: very common
-Tonsillitis: if not treated can lead to Peritonsillar abscess
-Causative agents:
*bacterial: 1. strept, 2. staph A, 3. pseudomonis (over watering plants)
*viral: CMV
*Fungal (aspergillus)
*Aspiration pneumonia: any dysphagic pt
*mycoplasmic, legionnaire's
-Pneumnocystitis (common w/HIV)
-Symptoms: SOB, extreme fatigue, crackles, wheezing, pleural effusions, increased WBCs
-Mortality increases > 70yoa
-x-ray: black is air, white is fluid
Pneumonia Risk Factors
-Elderly (more chronic illness... anything respiratory, CHF, diabetes, renal failure)
-Chronic medical condition
-Nutritional defects
-Alcoholism/drug abusers
-Neuro problems: impaired gag reflex, dementia
Nursing Management of Pneumonia
-Assess oxygenation status
-Obtain sputum specimen before antibiotics (in the am before breakfast)
-Avoid cough suppressants
-Respiratory therapy for mucolytic agents
-Ample rest periods (space activities)
-Patient teaching
*flu vaccine for those at risk
*pneumovax shot
Pneumonia Classification
-community acquired (CAP)
-Hospital acquired
-pneumonia in the immunocompromised host
-aspiration pneumonia
Dx of Pneumonia
-History (especially of recent resp. tract infection)
-chest x-ray
-blood culture (bacteremia occurs frequently)
-sputum examination
-Nose bleed
*trauma, chronic infection, violent sneezing, blowing, picking
*blood thinners
*low platelet count
-Tx: apply pressure on nose, ice packs, FORWARD tilt of head, nasal packing
*drugs: neosanerphrine, silver nitrate
*surgery: caterize
Deviated Septum
-Submucosal resection
-Nasal septoplasty
*both usually done as an out patient procedure
Nasal Fractures
ORIF (open reduction internal fixation) ASAP becuase of swelling makes landmarks hard to see
-Nursing considerations: *watch for covert and overt bleeding
*maintain airway
*appetite problems
Vocal Cord Polyps and Nodules
-Interference with voice production
-Usually develops because of voice abuse, smoking, allergies
*surgical stripping (bronchoscopy)
*watch for bleeding and airway
*Lung Abscesses*
-Encapsulated infections
*TB, pulmonary embolus, CA, COPD, open trauma
-Tx: bronchoscopy to drain it
-Hard to treat d/t being encapsulated
-High doses of antibacterial (antibiotics)
Pleural Effusions
-Accumulation of fluid in the pleural space
-Causes: fluid overload, chest CA, CABG, TB, pneumonia, thoracotmy: open up of the chest (open heart surgery)
-Common Terms:
*hemothorax: bloody fluid
*Chylothorax: Chyle is usually a milky fluid comprised of lymph drainage
*Empyema: accumulation of pus in the pleural space.
Pleural Effusions Symptoms
-Absent or decreased breath sounds
-Assymetrical chest expansion
-Dullness with percussion
-3rd spacing fluid (can't be removed easily)
Pleural Effusions Treatment
-Thoracentis (complication includes pneumothorax)
*Positioning pt
*done at the bedside
*stat x-ray after
*reassure pt that they should feel much better after procedure
-Inflammation of the pleura
-Symptoms: pleural friction rub, pain on inspiration, gaurded breathing
-Often follows resp. infection or occurs where chest tubes were inserted
-Tx: NSAIDs or steroids
Chronic Obstructive Pulmonary Disease (COPD)
-Broad term used to describe conditions with a chronic obstruction to EXPIRATORY airflow
-Chronic Bronchitis
*saO2 often clouds status of pt (need ABGs for accurate assessment)
-Air remains trapped in the lungs
-CO2 is usually the driving mechanism for breathing
-D/t cigarette smoking
*which causes connective tissue damage to the alveoli
*the alveoli overinflate and lose their elasticity then aren't able to deflate enough to expel the air
-Other causes: chronic infections, asthma, dust, age
*occurs more in men and those with reduced income and lack of education
Treatment for Emphysema
-Purpose is to improve the quality of life
-Steroids (anti-inflammatory)
-Antibiotic prophylaxis
O2 therapy for Empysema
-Because of the high levels of CO2 in the blood over a long period of time, the primary stimulus to breath is from a reduction in O2 rather than an increase in CO2 in normal patients
Effects of Smoking
-Increases blood pressure and heart rate (vasoconstriction of vessels)
-Carbon monoxide damages arterial wall encouraging fatty build up of cholesterol
-It increases the adhesiveness of platelets
-Increases the thickness of blood
-Damage of alveoli (butterfly to brocolli)
-Causes CA, heart disease, and lung disease
How To Quit
-Cold turkey (with help): best results
*Bupropion (zyban) and nicotine patch: taken 7-12 weeks is highly effective
-Nicotine fading: not effective just inhale more
-Nicoting patch or gum: s/e similar to smoking
-AKA airway reactive disease
-Airways react to allergens, exposure to irritants, exercise
*cause increase mucus production which causes a cough
*Bronchi narrow, dyspnea occurs
*Wheezing can occur with bronchospasm
-Not all asthmatics wheeze
-Secretions form with inflammation
Asthma Etiology
-Infections: asthma exacerbates an infection in the lungs
-Family hx
-Temperature changes: bronchospasms
Treatment of Asthma
-Inhaler for pt to use in respiratory distress
-Rescue inhaler: beta agonist
*albuterol (prevention), alupent, maxair, tornalate, brethaire,
-Epinephrine: SQ (potent pulmonary dilator)
-Steroid use: not a rescue medication
*prednisone, medrol, prelone, pediapred orally, IVP (solumedrol), inhaled, syrup
*with inhaled steroids: rinse mouth after use to avoid infection from candida albicans
*types: vanceril, azmacort, flovent
-NSAIDs: intal, tilade
-Leukotriene Inhibitors: accolate, singulair
Other COPD
-Chronic Bronchitis
*inflammation of the bronchi
*excessive mucus at least 3 months out of the year
*pockets in main stem bronchus where mucus and bacteria love to flourish
Sleep Apnea
-Characterized by excessive daytime sleepiness, increased incidence with obesity, snoring
-Usually upper airway obstruction of soft palate and tongue
-C-PAP machine to control: continuous positive airway pressure
-Sleep study
-Monitor EEG, O2, as it can be related to pulmonary heart issues, Ears, nose, throat problems
Restrictive Lung Disease
-AKA stiff lung
-No cure
-Tx with O2 only
-permanent damage to the lung tissue
-Examples: coal miner's lung (silicosis), asbestosis
-AKA: arterialsclerotic heart disease (ASHD), Coronary artery disease (CAD), Peripheral vascular disease (PVD), renal artery disease, aortic aneurysm...
-Hardening of the arteries
-Minor risk factors:
*obesity, exercise, diabetes
-Major: cholesterol, smoking, high blood pressure
-Male higher than women until menopause
-Family hx
Cholesterol Management
-First obtain fasting lipid panel
*total cholesterol
*HDL (high density lipoprotein)
*LDL (low density lipoprotein)
-Homocysteine levels
-C-reactive protein levels < 1.0mg/L
*any inflammation in the body will cause this
Cholesterol Management
-Nutritional Education: mono/polyunsaturated fats
*avoid red meat and dairy
-Medical Therapy:
*mevacor, pravachol, zocor, lipitor, lescol, crestor, zeita: statins inhibit cholesterol production (liver panel fx should be done)
*Lopid: decreases triglycerides
*Folic Acid: decreases Homocysteine
*Anti-oxidants: nyacin (causes more liver damage than statins), vitamin B
Diagnostic Tests for ASHD
-Angiogram: infection of Dye into an artery (femoral)
-Thallium treadmill
-Doppler Study: carotid artery disease
-Echocardiogram: structure of the heart (resting and with exercise)
-12 lead EKG
-TEE: Transesophogeal echocardiogram
Angiogram Complications
-Allergic reaction to the dye
-Occlusion (most common): check distal pusles
Peripheral Vascular Disease
-Any form of artery disease not pertaining to the heart
-Arterial vascular insufficiency
-A form of ASHD which includes all vascular structures:
*ateries, veins, arterioles, venules
-PVD Assessment:
*skin changes
Chronic Arterial Insufficiency
-Pulses: decreased or absent
-Color: pale, dusky, red on depression
-Temp: cool
-Edema: absent/mild
*atrophic/loss of hair over foot
*toe nails thickened and rigid
*Ulceration: if present involves toes or points of trauma
*gangrene may develop
Chronic Venous Insufficiency
-Pulses: normal (may be difficult to feel d/t edema)
-Color: normal or cyanotic
-Temp: normal
-Edema: present
-Skin: brown pigmentation around ankles, stasis dermatitis
-Ulceration: does not develop
-Gangrene: does not develop
Assessing Edema
-Go up the bone to determine edema score
PVD Symptoms and Nursing Dx
-Intermittent claudication pain
-Burning pain
-leg cramps
-deformed nails, hair changes
-decreased cap. refill
-RN Dx: altered tissue profusion, activity intolerance, sleep pattern disturbance, pain
PVD Medical Management
-Drug therapy
-Surgical manangement
*aorto-illiac bypass
*aorto-bifemoral bypass
*endarterectomy: cleaning of the carotids (c/p STROKE)
-Neurogenic Pain:
*tricyclic antidepressants (Elavil: amitriptyline, Pamelor: nortriptyline)
*antisiezure: neurontin, tegretol
Acute Arterial Occlusion
-Usually thrombosis or embolus (always an emergency)
-Symptoms: acute ischemia, pain, cool to touch, discolored
-Tx: anticoagulation and/or surgical embolectomy
-Out-pouching of vessels
*abdominal aorta most common
*ASHD main cause
*2.5cm normal
*5cm palpable
*blow out at 6cm
Raynaud's Disease
-Spasms of the arteries
-Leads to ischemia
-Usually fingers and toes
-Medical therapy
-Pt education
-Avoid cold climates and smoking
-Tx: calcium channel blockers
*diltiazem, procardia XL
-Superficial vs deep vein thrombosis (DVT) (leads to pulmonary embolism)
-Venous stasis (bedrest, hypovolemia, tissue injury, a-fib)
-Hypercoagulability of blood
*smoking, birth control, hormone replacement
Risk factors: post-op, irregular heart beat, infection in general
-Thrombus: stay put
-Embolus: travels
*thrombus: keep clot where it is
DVT Symptoms
-Unilateral: redness, pain, edema, +Homan's sign, inflammation
-Diagnostic Tests: veinogram, ultrasounds
*prevention is ideal: early ambulation, pneumatic stockings, prophylactic anticoagulation
*antigoagulatoin for a clot
Normal Clotting Cycle
Platelets-thromboplastin-prothrombin-fibrinogen-fibrin-formed elements (wbcs, rbcs) = clot
Anticoagulation Therapy
-Coumadin: works by blocking the formation of prothrombin
-Heparin & Lovenox: work by preventing the conversion of fibrinogen to fibrin
-None of these drugs dissolve the clot, just keeps them from getting bigger
-Advantages: *consistant results across all patients
*fast acting
*reverses in seconds
-Disadvantages: *kill pt if given to much IV or SC
*Illness called Heparin Induced Thrombocytopenia
-Test Used: PTT (partial thromboplastin time): measured in seconds (1.5-2.5 secs longer than normal)
-Reversal drug: protamine sulfate
-Complications: hematoma, hemorrhage, HIT
-Administer: *subcut. same as lovenox
*IV: needs to be on pump, to RNs to be present during changes in programming (critical drip)
-Advantages: oral, inexpensive
-Disadvantages: inconsistant across pts, 2-5 days to reach therapeutic blood levels, pts are often on 2 blood thinners
-Test Used: PT (protime): measured in seconds (1.3-2.0 times the normal, INR of 1.5-2.5)
-Reversal drug: vitamin K
-Complications: hemorrhage pregnancy deformities, long metabolic time (in system long after it has been d/c)
-Administer: oral: ask why is pt getting this, and what is their PT, given at 1800 so the MD can be notified of the daily INR
-Advantages: consistant results
-Disadvantages: Subcut. admin
-Lab work: don't need daily labs, Platelet count
-How to admin:
-Complications: hemorrhage
Thrombolytic Therapy
-Actually dissolves the clot
-Streptokinase, Urokinase (aslo used to declot a central line), Activase, TPA, Repro
*all IV critical drip infusions
*heparin is given after thrombolytics
-Given for:
*acute stroke
*acute coronary syndrome(Acute myocardial ischemia)
-TPA most common: tissue plasmainogen activator
DVT: surgical management
-Home care education on coumadin:
*s/s of bleeding (black tary stool)
*outpatient visits
*Don't change dose or skip, or double-up on a dose
*Call MD if forgot dose
*Informing other MDs on coumadin
*don't get pregnant
*Dietary considerations (don't change diet)
*talk with MD before taking any supplements
Pulmonary Emboli
-Obstruction of blood flow to a pulmonary artery causing necrosis (life-threatening event)
-Most commonly from a DVT
*pleural friction rub
*chest pain
-Risk factors:
*DVT, veinastatsis, hypercoagulability
-Tx: anticoagulation therapy (coumadin for rest of lifes)
-Surgical Intervention:
*vena cava umbrella (interruption)
-1 in 4 develop HTN
-#1 reason people go to MDs and for prescription drugs
-Mostly without symptoms
-95% unknown cause
-5%: pheochromocytoma 240/140, tumor on the adrenal gland, 24h urine test for catecholamine) or renal artery stenosis (BP can't be controlled)
-Pregnancy induced, white coat hypertension
-Women with toxemia increased risk for HTN later in life
-Systolic BP influenced by emotions
Factors Which Influence HTN
-Family hx
-Ethnicity: increased in blacks (usually requires multiple drug therapy)
-Gender: men get it at slightly earlier age
-Personality type A
-Alcohol (especially with excessive drinking)
S/S of HTN
-retinal changes
-arteriol narrowing
Management of HTN
-Modify risk factors
-dietary considerations:
*modify salt intake (2g normal)
*don't eat: milk, cheese, breads, canned soups
Medical Management of HTN
-Beta Blockers: end in olol
-Alpha 1 blockers: mine
-Calcium channel blockers: pine
-ACE inhibitors: pril
-Angiotension II receptor blockers: artan
-Adrenergic inhibitors:
Heart Failure
-The inability of the heart to pump enough blood to meet tissue needs
-Wide range of severity: mild to cardiogenic shock (80% mortality)
-Causes: CAD, myocardial infarction, cardiomyopathy, valvular or congenital disease, HTN
Pathophysiology of CHF
-Problem with muscle contractility
-Muscle tone (illness where muscle becomes stiff and unfunctioning)
-Fluid volume overload
Left-Sided Heart Failure
-Most common
-D/t impaired ejection of blood from left ventricle
-Blood backs up into lungs
*can back up into right side of heart
*pulmonary congestion (Crackles, listen at bases)
*repiratory distress
*PND (paroxysimal nocturnal dyspnea)
*wght gain
*increased heart rate
*right sided heart failure symptoms
-Pt dx w/CHF needs to be weighed daily
Right-Sided Heart Failure
-Due to impaired ejection of blood from the right ventricle
-Often due to left-sided HF or pulmonary disease
-Backs up into systemic circulation
-S/S: *peripheral edema
*distended neck veins (JVD)
*Liver enlargement
*wght gain
*increased heart failure
*lethargy, fatigue, activity intolerance
*pedal edema while standing
*generalized edema while lying down
Cor pulmonale
-an alteration in the structure and function of the right ventricle caused by a primary disorder of the respiratory system.
Goal of Medical Management
-Blood test to dx: BNP (type B natiuretic Peptide)
-To restore balance between oxygen supply and demand by:
*improving heart contractility
*reducing heart rate
*improving activity tolerance
*reducing afterload (blood pressure)
-Done using:
*antihypertensive drugs: so the heart can pump the blood out against less pressure
Treatment of CHF
-Optimize oxygen of pt
-Vasodilation drugs:
*nitrate therapy
-Inotropic Drugs: to improve the contractility of the heart
*Digoxin, dopamine, dobutamine)
-Drugs to reduce HR:
*Beta blockers (Cardioselective)
-Drugs to reduce B/P:
-Drugs to reduce blood volume
Non-Pharmological Approach to CHF
*oral intake of fluids
*IV fluids
*sodium and alcohol intake
-Treat underlying problem
*prolonged traveling
*extreme weather
Nursing Interventions for CHF
-Assess Resp. status often
-Keep HOB elevated
-Avoid stenuous activity
-Daily wght
-Assess for postural hypotension
-Urinal close by (diuretics)
-Discharge teaching: palpations of heart, extra ankle edema, sleeping with more pillows
Digitalis Toxicity
-Anorexia (usually first sign)
-Blurred vision (halo/rings around lights)
-EKG changes, arrhythmias
-Abdominal pain
-Normal dig serum level:
-Tx: d/c or reduce dose
-Any abnormality of the heart muscle:
*dilated (congestive)
Dilated Cardiomyopathy
-drugs from chemotherapy
-Medical management same as CHF
Hypertropic Cardiomyopathy
-heart is contracting too much
Restrictive Cardiomyopathy
-Makes the heart stiff
Rheumatic Heart Disease
-pt's who have untreated strept
-Mitral stenosis main problem
Anemias and other abnormalities
*reduced # of RBCs d/t bone marrow malfunction
*iron deficiency: RBCs have decreased level of HGB, caused by bleeding, elderly fixed income, pregnancy, side effects of meds: black stool, constipation, must be z-tracked in IM, allergy rx
-Excessive destruction or loss:
*hemolytic anemia: antibodies developed against RBCs, jaundice, tx corticosteroids
*sickle cell anemia:
Common disorders of WBCs
infectious mononucleosis
Normal Lab Values
-Hematoglobin: m(13.5-17.5 g/dl)
f(11.5-15.5 g/dl)
-Hematocrit: m(44-52%) f(39-47%)
-Albumin: (3.5-5.5 g/dl)
*Sodium: 136-145
*Potassium: 3.5-5.2
*Chloride: 96-106
-Glucose: 70-110 mg/dl
-Total Cholesterol: <200 mg/dl
*HDL: 35-85 mg/dl
*LDL: <130 mg/dl
-BUN: 8-28 mg/dl
-Calcium: 8.5-10.5 mg.dl
-Creatine: 0.6-1.2 mg/dl
-Uric Acid: 2.5-8 mg/dl
-Magnesium: 1.8-3.0 mg/dl
-Phosphorus: 2.3-4.7 mg/dl