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18 Cards in this Set
- Front
- Back
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WHAT DOES CHRONOTROPIC EFFECT DEAL WITH
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heart rate
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what does ionotropic effect deal with
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heart contraction
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what is abnormal sinus rhythym (sinus tachycardia)
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heart beat > 100bpm therefore faster heart rate
causes: over active SA node(^SNS, ^body temp) |
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what is the difference in EKG of normal vs sinus tachycardia
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intervals have shortened therefore faster heart rate
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what is sinus bradycardia
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slow heart rate
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what is the difference in EKG of normal vs sinus bradycardia
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EKG is normal but invervals are longer
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what is sinus arthymia
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breathing alters the heart rate by altering vagal tone
inspirtation(inhaling) - decrease vagal tone - increases heart rate expiration (exhaling) - decreases vagal tone - increases heart rate |
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how is the EKG of someone with sinus arrythmia
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EKG will look normal but intervals are longer
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what is a first degree block
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AV node still working but slow conduction through AV node (signal will stay longer in the AV node)
SEEN IN EKG W/ PR INTERVAL BEING MORE THAN .2 MS (NORMAL IS .16MS) NOT LIFE THREATENING |
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what is a second degree block
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inconsistency of sending signals (AV NODE WILL SEND SOME SIGNALS BUT NOT ALL)
for a particular amount of impulses the AV NODE gets it only sends a fraction of them, IE the atria contracts 2 times but the ventricle only contracts once |
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what happens in a 3rd degree block
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AV NOT FUCNTIONS (not sending impulses to ventricle)
-bundle of hiss/purkingee fibers can contract ventricles AS A RESULT ATRIA AND VENTRICLE CONTRACT INDEPENDENTLY (atria = 100bpm, ventricle = 45bpm) these people have artificial pace maker on right ventricle |
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what is PREMATURE VENTRICULAR CONTRACTION
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action potential generated in ectopic foci in the ventricle (AP in multiple areas)
FEELS LIKE YOU SKIPPED A HEART BEAT OCCURS NORMALLY and leads to VENTRICULAR TACHYCARDIA MULTIPLE QRST regions |
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what leads to ventricular tachycardia
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premature ventricular contraction
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what is the most serious arythmia
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ventricular fibrilation
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what happens in ventricular fibrillation
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instead of only the SA node conducting the AP multiple parts of the heart conduct an AP but it is not coordinated
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what causes re-entry phenomenon
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increased pathway around the ventricles (dilated heart)
decreased velocity of conduction (ischemia, increase K, electrical stimulation) shortened refractory period of muscles (epinephrine, electrical stimulation) |
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what does electroshock defibrillation do
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all the cells depolarize at the same time therefore all of them undergo refractory period at the same time allowing cells to rest at the same time (SA NODE takes over as pacemaker)
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what happens in atrial flutter
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signal travels as a single large wave
rapid contraction 4:1 artial:ventricle |
