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11 Cards in this Set

  • Front
  • Back
CN V- Trigeminal
General, Oral Periph (unilateral, bilateral), Speech Findings
rarely affected individually

oral periph
-unilateral LMN - jaw deviates to weak side
-bilateral LMN - jaw droops open
-could notice drooling

Speech findings
-little effect
CN VII- Facial
General
affected individually fairly often

- Tumors or infections of VII
- Acoustic neuromas (also affect CN VIII)
- Bell’s Palsy
- unilateral facial nerve paralysis affecting upper and lower facial muscles
CN VII- Facial (2)
Oral Periph (unilateral, bilateral)
oral periph

-unilateral LMN
half of face (upper and lower) hypotonic and weak
nasolabial fold flattened
asymmetrical facial movements

-bilateral LMNs - much less common
symmetrical muscle weakness
drooling
synkinesis
- abnormal contractions of weak muscles near normally contracting muscles
- faulty reinnervation after recovery from LMN lesion
CN VII- Facial (3)
Speech Findings (Bilateral & Unilateral)
speech findings
-bilateral - poor labial closure - labial sounds
-unilateral - artic distortion (primarily labial, bilabial) - frication rather than plosion
CN IX- Glossopharyngeal
General, Oral Periph (unilateral), Speech
rarely damaged in isolation

oral periph findings
- unilateral
asymmetric gag reflex
**absent gag reflex means nothing

speech - little effect
CN X- Vagus
Lesions, Etiologies
intramedullary lesions - bilateral effects
extramedullary lesions - unilateral effects
extracranial lesions - unilateral effects to restricted portions of CN X because of branching

etiologies
-aneurysms of the aorta, internal carotid
-surgery: open heart surgery or carotid endarderectomy that cuts CN X
CN X- Vagus
Oral Periph (unilateral, side), Bilateral CN X
oral periph findings
unilateral
-asymmetric velum elevation pulls toward strong side
-diminished gag reflex on affected side

bilateral CN X
-minimal velar movement
-absent gag
-weak cough and coup
- inhalatory stridor if VFs paralyzed toward midline
indirect laryngoscopy
- multiple X branches: abductor paralysis of vfs
- recurrent laryngeal branch: paramedian positioning of vfs
because cricothyroid contributes to adduction
CN X- Vagus
Speech Findings (Bilateral)
speech findings
- pronounced effects on phonation, resonance

bilateral
hypernasality, nasal emission, imprecise plosives, ***breathy phonation****
facial grimace possible
CN X- Vagus
Speech Findings (Unilateral Sup. & Recurrent Laryngeal Branches)
speech findings
-unilateral
sup and recurrent laryngeal branches
breathy/aphonic, reduced loudness & pitch, diplophonia, pitch breaks

sup lar branch alone
breathiness and pitch alterations possible

recurrent lar lesions
Unilateral - breathy-hoarse, decreased loudness, diplophonia
Bilateral - inhalatory stridor - airway compromise, little effect on phonation (VFs at midline)
Bilateral high lesion – breathy, aphonia

during connected speech - incr breaths/min - air wastage

Important to refer for laryngoscopic exam

**Don't Memorize whole slide, shows you that you can have subsets of dysarthria depending on which branch of nerve is damaged
CN XII- Hypoglossal
Etiologies, Oral Periph (unilateral & bilateral)
etiologies
- neck surgery, tumors, trauma

oral periph findings

-unilateral
atrophy on weakened side
fasciculations
deviates to weak side on protrusion
tongue movements diminished

-bilateral
symmetrical weakness - little ROM
poor saliva management
CN XII- Hypoglossal
Speech Findings
speech findings

imprecise articulation for lingual phonemes

unilateral lesions: can compensate - only distortions

bilateral lesions - severe artic problems
altered resonance because of tongue carriage