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43 Cards in this Set
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Important history & PE for AAA |
history: smoking, HTN, HLD, FH of AAA, history of connective tissue disorders PE: tachycardia and hypotension, acute distress, mental status. Inspect mass, check all pulses, look for pallor or other signs of hypotension. Check for popliteal aneurysm. |
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Tests and treatment for AAA |
Tests: U/S or CT unless you think it is ruptured (straight to OR). U/S not as sensitive. Angiogram to show lumen patency, show if iliac or renal is involved. Treatment is surgery for non-ruptured, dilation 5.5-6 cm. Need to know size, location (prerenal, perirenal, infrarenal), CV/P status. Options include endograft (not if close to renal arteries), endovascular repair. Immediate lap. if ruptured, clamp aorta. |
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Management of ruptured vs elective AAA? |
ruptured= surgical emergency, elective you can monitor based on size (surgery after 5 cm). <5 cm 4% risk, 5-6 cm 10%, 6-7 cm 16%, 7-8 cm 30% rupture risk. |
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Complications and result of treatment for AAA? |
large fluid shifts, MI, athroembolism, de-clamping hypotension, ischemic injury, acute renal injury, hemorrhage, mesenteric ischemia from surgery. Exsanguination in OR if ruptured. Long term complications- impotence (due to interruption of hypogastric circulation or nerves on aorta), fistula formation (to duodenum or IVC), graft infection or leak, poor distal perfusion, recurrence. |
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pathophys of AAA |
atherosclerotic plaque develops, tunica media and elastin degrades, aorta become suceptible to high pressures leading to dilation and aneurysm formation |
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questions to ask for acute abdomen? lab/imaging workup? |
?s: FARCOLDER, bowel symptoms, urinary symptoms, GYN if female. labs: bHCG if female, CBC, LFTs, amylase, lipase, bili, BMP, UA. EKG to r/o MI. imaging: upright CXR and abdominal film to look for free air, abdominal u/s, abdominal CT |
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What is direct vs indirect tenderness? Psaos, obturator signs? |
direct = inflammation at the site indirect = peritoneal signs psoas = extend hip and internally rotate obturator = flex at hip and internally rotate |
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DD for each location RUQ |
RUQ: cholecystitis, perforated duodenal ulcer, hepatic abscess RLQ: appendicits, diverticulitis, meckel's LLQ: diverticulitis, volvulus LUQ: splenic rupture, abscess diffuse: SBO, leaking aneurysm, mesenteric ischemia periumbilical: early appendicitis, SBO suprapubic: ecoptic, torsion, abscess, psoas abscess, incarcerated hernia |
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What is included in the risk assessment for anesthesia? What must be done before surgery? |
pulmonary function, COPD, cardiac status, previous reactions to anesthesia. EKG (>40 men 50 women), CXR if >60, CBC, co-ags, BMP, type and cross |
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What is ASA criteria for pre-op risk? |
1: normal healthy patient 2: mid systemic disease but no functional limitations 3: moderate to severe systemic disease with some functional limitations 4: moderate to severe systemic illness that is constant threat to life, incapacitated functionally 5: pt about to die 6: brain dead with organ harvestation |
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What is the management plan for the following conditions? external hemorrhoids perianal abscesses anal carcinoma anal fissure |
EH: sitz bath, nonconstipating analgesics, fiber, excision PA: incision and drainage anal carcinoma: biopsy fissure: sitz bath, fiber, botox, topical anesthetics. nitroglycerin, topical Ca blocker, lateral spinchterotomy |
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What is the pathogenesis of TIA? What is amaurosis fugax? |
atherosclerotic plaque in common carotid, pieces break off and enter into cerebral circulation. Important because they ^ likelihood of stroke. AF is transient blindness due to occlusion of opthalmic artery. |
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Labs and testing to evaluate TIA? Managment? |
duplex ultrasound to detect flow and degree of stenosis MRA detects functional flow and estimates plaque thickness carotid angiogram is gold standard. get carotid endarterectomy if neuro symptoms from stenosis or asymptomatic w/ >70% stenosis. |
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What are the risks of carotid endarterectomy? How can you minimize risk? What is stroke risk postop? |
risk of death is low (1-2%), can injure vagus nerve, cause a hematoma, seizures. use eeg monitoring, aspirin pre-op, complete clot dissection, shunt blood bypass to protect brain from ischemia. stroke risk post-op is 5%. |
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DD of chest pain? Tests to get? |
MI, aortic dissection, PE, pericarditis, esophageal rupture, etc. Get an EKG, cardiac enzymes, angiogram to look for aortic dissection, chest xray. |
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What meds can you use to stabilize pt with MI? |
ASA COX inhibitor Bblocker slow heart rate via Beta1 Calcium channel blocker work in two ways impair muscle contractility or vasodilation ACEi diuresis by inhibiting angiotensin I → II Diuretic remove extra fluid via kidneys to decrease preload Nitrates vasodilation |
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What info would the following tests give you in setting of an MI? ECG, stress test, echo, cardiac catheterization |
ECG ST elevation or depression, inverted T waves, Q waves Stress test myocardial response when demand is increased Echo localize dyskinetic wall segments, valvular dysfunction, EF Cardiac catheterization coronary anatomy and sites of lesions |
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What are the indications for bypass surgery vs angioplasty? |
presence of left main disease (blockage of left main coronary), three vessels disease needs bypass. The simpler the problem, the more amenable it is to angioplasty and stenting. |
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What are the symptoms of PVD? What tests should you order? |
Claudication, rest pain, gangrene, poor wound healing, edema that is reduced with elevation. Order doppler study to study flow, duplex u/s ABI, arteriography to delineate arteries using dye. MRA, PVR. |
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What is dx test of choice for DVT? Treatment? |
Use u/s. Duplex combines u/s with doppler to visualize and detect flow. Contrast venography rarely used anymore. Treat with IV heparin to prevent further dvts. Follow PTT, need long term anti-coag with warfarin. |
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Findings of actue arterial occlusion? Treatment? Conesquences post-op? Long term? |
Pain, Pallor, Paresthesias, Pulselessness, Pokilothermia, Paralysis. Time is key, need to revascularize within 6 hrs. Give heparin right away, go to the OR. Don't do angio or TPA, will only delay. Balloon catheter embolectomy is procedure of choice. Watch out for compartment syndrome, may need fasciotomy. Long term needs, chronic anti-coags. |
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Findings in claudication? #1 location? |
Ischemic pain in legs, often relieved by rest. Exam for pulses, bruits, skin for ulcerations, nervous system for motor and sensory function. Hair loss, dependent rubor, loss of sensation, etc suggest chronic nature. superficial femoral is most common location of occlusion. |
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What to do after findings of arterial occlusion on physical exam? How do you manage claudication long term without surgery? |
Calculate the ankle-brachial index by measuring systolic arterial pressure in legs and arm with a Doppler device. Get PVR/pulse volume recordings to localize the level of the occlusion. ABI < 1.0 suggests disease, <0.3 is severe. Surgery depends on lifestyle of pt. Most pts don't get it. Exercise, lifestyle changes, followup is key. |
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How is aortoiliac occlusive disease managed differently than less severe PVD? |
More progressive, surgery more likely. Suggested by absent/weak femoral pulses. Don't wait for rest pain or ulceration to develop. Options afterwards are lifestyle modifications, percutaneous transluminal angioplasty for single short segment stenosis. If bilateral loss, may need an aortobifemoral bypass graft. |
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How is ulceration in a pt with claudication managed differently? |
Need to revascularize, get surgery. Angiogram is needed to assess anatomy, ulcer will likely heal with debridement and wound care after revascularization. Use grafts to bypass obstructions, balloon dilations, stents. |
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What is the clinical picture of SBO? What is the workup and management? |
Nausea, crampy pain, distenstion, no BMs, tympanitic, mildly tender. Get an upright PA and lateral CXR and flat/upright abdominal x-ray. Will see multiple air fluid levels, no air in colon or rectum. See hypochloremic hypokalemic metabolic alkalosis with paradoxical aciduria. Correct w/NS+K IVF, NG tube, serial labs and x-rays. |
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What findings would warrant surgical exploration in SBO? |
localized tenderness, very high WBC count, high temp, metabolic acidosis (suggests ischemia or necrotic bowel), or if non-surgical management fails. |
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PE and history important questions for SBO? |
surgical history, orthostatic vitals, assessment of the groin, digital rectal exam, sigmoidoscopy. |
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What are the risk factors for colon cancer? How do we screen, evaluate? What tests are needed before surgery? Treatment post-surgery? |
Age >50, personal history of polyps or cancer, FH, colorectal syndromes, UC or Crohns disease. Screen with colonoscopy at 50 or earlier if FH before 50. Evaluate with colonoscopy. CXR, CT abd/pelvis, LFTs r/o metastasis CEA establish baseline CBC, coags 5FU based chemotherapy if >stage 2 disease. |
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What is done pre-op for biliary colic, cholecystitis, choledocolithiasis? |
BC: NPO, IVF, IV analgesia, can schedule surgery electively AC: NPO, IVF, IV analgesia, IV abx. Need to do cholecystectomy within 48 hrs. Choledocolithiasis: ERCP to remove stones, then cholecystectomy once stable. |
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What are the causes of obstructive jaundice? How would you work it up? |
Gallstones, tumor, stricture of the common bile duct (prior biliary surgery). Bilirubin, LFTs, Alk phos, amylase, lipase Abdominal US look at CBD Abdominal CT ERCP if mass can be visualized allows for brushings for cytology Upper GI endoscopy and endoscopic US assessment of pancreatic head , allows for assessment of tumor, local metastasis, nodes |
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Important history and physical for acute groin pain? |
FARCOLDER, worse when you wake up? Bowel habits, change when bearing down, diet, nausea, vomiting, fevers, pain, past surgeries. Examine lying down and standing, first look for any bulges or masses in groin, any visible skin changes then palpate the area and ask the patient to cough and/or bear down feel for any masses or bulges and if they are reducible |
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What should be done prior to intervention for hernias? What is the appropriate intervention? What are risks of surgery? |
CBC, CT imaging. All hernias need surgery eventually, need surgery emergently if incarcerated. bleeding, infection, recurrence, nerve damage/entrapment of ilioinguinal nerve |
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Important history and physical for breast disease? |
Ask about risk factors for breast disease, nipple discharges, changes in size/shape/symmetry of the breast. Last time of mammogram, family history of breast disease. Exam: inspect with arms at sides, with arms raised color, symmetry, size, shape, contour, dimpling, erythema, edema, thickening of skin with porous appearance. Palpate breast while patient supine, palpate all 4 quadrants and nippleareolar complex for discharge, palpate axilla along lateral border of anterior and posterior axillary fold, medial and lateral wall of axilla and apex of axilla. |
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What diagnostic test would you order for breast disease? |
under 28 ultrasound (mammogram no use because breast is too dense in this age group) over 28 ultrasound and mammogram Both followed by FNA or core needle aspiration depending on findings of US or mammogram |
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What types of malginancy in the breast? Rx for invasive carcinoma? |
DCIS, invasive ductal carcinoma, invasive lobar carcinoma, malignant phyllodes. lumpectomy + radiation = mastectomy, need for sentinel lymph node dissection and possible axillary dissection, possible hormone or chemotherapy. |
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How do you stage breast cancer? What are considered important pathological aspects of the histology? |
Stage I tumor is <2cm without metastasis or nodes look at margins if did a lumpectomy to make sure got all of the cancer, look at sentinel lymph node if positive have to take all lymph nodes in axilla, look for Estrogen Receptor status and if HER2 positive will dictate treatment |
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What are the common causes of hypercalcemia?Differentiate between primary, secondary and tertiary hyperparathyroidism. |
in outpatient setting parathyroid adenoma in inpatient setting cancer primary hyperparathyroidism increased production of PTH due to hyperplasia or tumor of re Pparathyroid gland secondary hyperparathyroidism increased release of PTH in response to low calcium (hypocalcemia) often seen with renal failure tertiary hyperparathyroidism increased release of PTH after long standing secondary hyperparathyroidism and results in hypercalemia |
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Pre and peri-op mgmt of parathyroid disease?
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pre: need to rule out cancer with bone mets, check phosphorus, PTH, ca again, lytes, renal function. Can do sestamibi scan to localize the adenoma, makes procedure less invasive, U/S, CT MRI. rule out MEN 1 or 2 solitary Adenoma surgical removal of adenoma (usually 1 parathyroid) Multiple gland hyperplasia remove three glands or all 4 with implantation of 30g parathyroid tissue to retain function |
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What are important parts of hx in neck mass? How would you investigate? |
Radiation. Hypo/hyperthroidism: Temp intolerance, weight change FHx Voice/airway changes, Dysphagia, Dyspnea Physical TSH w/ US -If normal or high → FNA -If suppressed → radioisotope scan Hot → radioablation Cold/warm → FNA |
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What extra investigations do you need for medullary carcinoma of the thyroid? |
Calcitonin levels MEN 2: urine metanephrines, PTH/Ca2+ Thyroidectomy. Lateral neck dissection for palpable nodes. 80% at 10 years. Monitoring with serum calcitonin.
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What extra investigations do you need for follicular carcinoma of the thyroid? |
Follicular neoplasms must have tissue biopsy, not just cytology, for proper diagnosis. Treatment involves thyroid lobectomy, which is therapeutic as well as diagnostic. I131 radioablation, which is used for adjuvant therapy following surgery |
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What are the 2 common causes of hyperthyroid? Rx? |
Grave’s = radioablation Toxic multinodular goiter = excision |