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45 Cards in this Set

  • Front
  • Back

Hypodontia

partial absence of teeth


most common: 3rd molar


2nd mandibular premolar


lateral maxillary incisor


males - mandibular lateral incisor


females - 2nd maxillary premolar

Anodontia

Complete absence of teeth

Hypodontia syndromes

Ectodermal dysplasia


Chondroectodermal dysplasia

Ectodermal dysplasia

X-linked recessive - common in males


Hair, sweat glands or teeth fail to develop


Anodontia - teeth that do erupt are cone-shaped structure


salivary gland hypoplasia - xerostomia


Chondroectodermal dysplasia

Ellis van Creveld syndrome


AR


mesodermal tissues also affected - limbs, polydactyl and congenital heart defects


teeth similar to ectodermal dysplasia


fusion of upper lip to maxillary ridge occurs

Hyperdontia

extra teeth


most common: mesiodens


4th maxillary molar (disto or paramolar)


familial tendency


Hyperdontia syndromes

Gardner's syndrome


Cleidocranial dysplasia

Gardner's syndrome

AD


polyps of colon


osteomas of facial bone


epidermoid cysts and desmoids


multiple unerupted and supernumerary teeth

Cleidocranial dysplasia

AD


affects bones of shoulders and craniofacial bones


delayed loss of deciduous teeth


numerous unerupted and supernumerary teeth

Microdontia

generalized - pituitary dwarfism


localized - maxillary peg lateral, maxillary 3rd molar

Macrodontia

generalized - pituitary gigantism or hemifacial hypertrophy


localized - rare, fusion can be misleading

Taurodontism

enlarged pulp chamber, rudimentary development of the roots


familial


associated with Klinefelter's syndrome or amelogenesis imperfecta


neanderthal skulls

Cusp of Carabelli

lingual of max first molar


occasionally on buccal surface of molars and premolars

Dens evaginatus

between buccal and lingual cusps of molars and premolars


more prevalent in inuit

Talon cusp

cingulum of max or manx permanent incisor

Enamel pearl

furcation area of molar


1-9%


Asian prevalence

Cervical enamel extension

triangular extension of enamel from the buccal CEJ to the bifurcation of molars


associated with furcation involvement or development of an inflammatory cyst (buccal bifurcation cyst)


8-33%


Asian prevalence

Gemination

single tooth bud invaginated and gives rise to 2 incompletely divided teeth


single root and canal

Fusion

fusion of 2 tooth buds to give rise to a single tooth


root canals may be separate or fused


fusion may occur with a supernumerary tooth

Concresence

fusion of the cementum of 2 teeth


occurs after root formation complete

Dilaceration

sharp bend in the root of the tooth


result of trauma in root formation

Dens in dente

dens invaginatus


invagination of enamel and dentin into the crown


usually affects lateral incisors


Hypercementosis

occlusal trauma, unopposed teeth or inflammation

Hypoplasia

quantitative


can be affected by trauma or therapeutic radiation

hypocalcification

qualitative

Turner's tooth

hypoplastic tooth developed from an abscessed primary tooth

Fluorosis

fluoride levels above 1 ppm


white spots or mottled with a chalky or opaque, brown/black pigmentation


may have flaking/fracturing

Congenital syphilis

notched central incisors


peg shaped laterals


mulberry molars


Hutchinson triad - blindness, deafness and dental anomalies

Hypoplasia

febrile childhood infections


rickets, hypocalcemia, hyperparathyroidism, vitamin D deficiency


birth injuries


nutritional inadequacies


Enamel formation

1. Production of an enamel matrix (enameloid)


2. Mineralization of enamel matrix


3. Maturation into harder enamel

Amelogenesis Imperfecta

group of hereditary diseases


defective formation, calcification or maturation of enamel


can affect unerupted teeth, anterior open bite, pulpal calcifications, interradicular dentinal dysplasia, root and crown resorption, cementum deposition, truncated roots and association with taurodontism

Hypoplastic AI

quantitative defect


inadequate deposition of enamel matrix


pitted


yellow teeth


vertical stripes (in females with dominant X type)

Hypocalcified AI

qualitative defect


inadequate mineralization of enamel matrix


soft brownish enamel


Hypomature AI

Enamel matrix begins to mineralize but there is a defect in the maturation of crystal structure


enamel is softer than normal


brown pigmentation


white patches


snow capped appearance

Hypomature/Hypoplastic AI with taurodontism

thin enamel


enamel which does develop is hypomature


yellow brown teeth with pits

Treatment of hypoplasia

veneers or crowns


overdentures or dentures


surgical removal or unerupted teeth


genetic counselling

Dentin

neuroectomesenchyme product


odontoblasts


interglobular dentin - poorly calcified, disorganized or absent tubules can result


Hereditary opalescent dentin DI type 2

AD


1/8000 common


primary and early developed teeth, similar severity in both dentitions


low variability of gene expression


severity will correlate within a family


bulbous crowns, opalescent brownish blue hue


cervical constriction


severe and early attrition (weak DEJ)


pulp chamber obliterated


in rare cases shell teeth, thin dentin with enlarged pulps

Dentinogenesis imperfecta associated with osteogenesis imperfecta DI type1

OI - defective bone formation causing fragile bones, congenita and tarda forms


AD or AR


DI is not associated with all 4 types of OI


DI in 50% of OI patients


DI type 1 = primary dentition

Treatment of DI

Crowns


dentures/overdentures


usually end up with dentures/implants

Radicular dentin dysplasia (dentin dysplasia type 1)

Rootless teeth


1/100,000 AD


both dentitions


crowns amber/bluish


roots truncated, multiple periapical radiolucencies


mobile and misaligned teeth


chevron shaped pulp chamber from obliteration


islands of dentin and osteodentin surrounded by primary dentin - boulders in a stream


anatomical orientation not defective dentin quality

Coronal dentin dysplasia (dentin dysplasia type 2)

rare AD trait


both dentitions


pirmary teeth have opalescent amber translucency


do not undergo attrition


pulp chamber obliterated upon eruption


normal coronal dentin with an abrupt change to amorphous atypical dentin


permanent have normal colour with thistle shaped pulp chambers

Pulpal dysplasia

extended pulp chambers with multiple pulp stones


connected to coronal dentin dysplasia

Clinical management

Type 1 - meticulous homecare


Type 2 - permanent does not require special management

Regional odontodysplasia

ghost teeth


usually maxillary incisors


failure of eruption or delayed eruption


enamel and dentin are thin and malformed


ghost like appearance radiographically


possible trauma or infection


unerupted teeth should be removed


less effected teeth should be maintained if possible with stainless steel crowns or composite restorations