• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/105

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

105 Cards in this Set

  • Front
  • Back
Dr. Graham:
fresh air, exercise, bland diet, abstinence from sex , promote health, 1830
Schleiden and Schwann
developed Cell theory
Virchow
cellular pathology , recommended hygenic measures, defended pasteur, criticized semmelweis. 1848
Darwin
1859
origin of the species
Louis Pasteur
bacteriology, paper on spontaneous generation and vaccinations and hydrophobia
Mendel
hybridization of peas,
paper published in obscure paper, unnoticed for 35 years
lord joseph lister
1845
45% surgical mortality
carbonic acid
criticized from complexity of bandages, franco prussian war
major contributions of semmeilwies
handwashing, didn't get credit till his death when pasteur proved germ theory
rush
copius blood letting, amercian clinician of his time, ablest, calomel and jalop, attended to GW
fist leaches in 1833. then asperin in 1899 then pennicellen in 1948
heroic medicine
blood letting purgatives, emetics , sialogogues
homeopathey
disease could be cured by drugs the smaller the dose, the more effective in stimulating vital force
chiropractic
disease joint oriented nerve interference, innate intelligence
Stills life
born 1828
married 1849, lost two children and wife within one month
helped kansas become a state
lost three children in same someth to encephalitis
osteopathy banner 1874
osteoapthy concepts first formulated
1888 started teaching it when he was 63
established infiermary
graduated first class in 1893
died at age 89, 5,000 D.O's in practice
landmarks in osteopathy
school grows in 1899
1893 first class graduates
founded in 1892 in missouri
cells duty and osteopathy
- cellls duty to maintain themselves
-secondary duty is to contract, secret, syenthesize, phagocytize, and pump
-when stressed, they go inert and stop working
-stressed by inadequate oxeygon, CO2, inadequate sugar, temp changes, p h changes, shearing forces, sub optimal ECF environment
-function in peristalis, ciliary clearance, wound healing GI barrier, maintenance, nt synthesisis, phagocytosisi, proton pumping
causes of somatic dysfunction
-trauma
-ergonomics
-overuse
-birth
-gestation
-visceral pain
-
cause of skeletal dysfunction
-arthrodial dysfunctions that are caused by:
-myofascial dysfunctions
-vascular fluid dysfunctions
-neurologic dysfunctions
TART findings by clinical description associated with Acute or chronic
Acute- causes increased sweating and reduced blood flow to skin and other organs
Chronic- causes skin leasions and thoracic cage damage, cool, stringy appearrance
Tenderness , TART
hyperalgesia:pain in excess of stimulus
-local:
-diffuse: entire limb involved
-position dependent; strain , counterstrain modality of OMM
Asymmetry , TART
-left side , right side not mirrored
-elevated 1st rib
-short leg
-orbits, ear, bone eminences, unlevel
-ASIS,PSIS,ILA, pubes
Reduced range of motion, TART
-rotate more to one side than the other
-can flex but not extend
-joint can glide anterior but not posterior
-encounter restrictive barriers
Tissue texture changes, TART
edema-swelling
-nodules
fashial bands: extra tight strips within a sheet of connective tissue
-acute injury: edema with warm moist red skin, muscle spasm, increased tone
-chronic: skin cool, pale, muscles stringy, thickened , adhered
-imparied lympathic drainage
cause of skeletal dysfunction
-arthrodial dysfunctions, which are caused by myofascial dysfunctions, which are caused by vascular fluid dysfunctions, which are caused by neurologic dysfunctions
Arthrodial dysfunctions
joint function in only one direction, joint locks, motions limited or absent, uneven weight distribution, wearing of cartilage
Myofascial dysfunctions
-spasm, hypertonicty, traction on fascial web, adhesions between adjacent structures, cause and maintain arthrodial dysfunctions
Vascular and lympathic
- arterial; direct compression or vasoconcstriction
venous; direct compression which is common or suction failure
-lympathic; compression on lympathic vessel
-ECF; stasis, can contribute to myofascial dysfunctions and tissue texture changes
Nuerologic
- hyperaglesia
-vasoconstriction
-hypersecretion
-disordered GI
-uterine peristalsis
-referred pain at adjacent innervations
-contributes to dysfuntctions of myofascia
five motion barriers
anatomic-bones restrict motion
physiologic-as far as patient can move it
elastic-between physiologic and anatomic
pathologic-restiction of motion associated with patho changes such as ligaments, foreign bodies, ect

restrictive-a fucntional limit that abnomrally dminishes the normal physolgic range, imposed by somatic dysfunction
OMM techniques and abbreviations
-dir
-ind
-st
-art
-lar/blt
-me
-cs
-hvla
-cr/bmt
direct
can be on any tissue type
indirect
can be on any tissue type
soft tissue
soft tissue; passive, usually direct, muscle fascia
articulary
passive, direct, joint artic, low velocity, mod amplitude
ligament art release
pasive or active, indirect or direct
target tissue is ligaments and joints
muscle energy
active, direct, tissue target is muscle joint
counterstrain
active direct, muscle joint
high velocity
pasive, direct, joint, high velocity, low amplitude
ost cran feild
direct or indirect, all tissues targetd
myofascial relase
pasive, direct or indirect, mucsle, fascia, joints
still
passive, direct and indirect, targets joints, direct and indirect release
visceral
passive or add active, direct or indirect, organs fascia
chapmans
passive, lymph, pelvic treatment
lympathic
passive, or add active, lympth, no barrier
inhibitory
INH, passive, direct, neuromuscular junctions, no barriers
4 basic osteopathic tenets
-body is a unit
-structure and function are reciprically interrelated
-the body possesses self regulatory and self healing mechanissm and is capable of health maintenance
-rational treatment is based on above principles
OMM corrollaries
-movement of body fluid is essential
-nervous system plays crucial role
-somatic components of disease are manifestations and factors
-when normal adaptability is disrupted, disease may ensue
OMM prescription basis
-awarenss of OMM
-history and physical exam
-relevant testing results
-accurate diagnosis
-appropriate treatment goals
-skilled application of treatment modalities
-delivery of treatment
-reevaluation and revistiing
sinuses
frontal, maxillary, ethmoidal, sphenoidal
upper respiratory systems
-ehtmoid, sphenoid sinus
-maxillary sinus
-nasal airway
-pharyengeal airway
-larnyx
-
lympathic tissue organization
-lympatic duct
-thoracic duct
-cisterna chyli
-thoracic duct inlet
clincial infectous symptoms manifest:
-host defense breakdown leading to increased local tissue immunity and reaction
-increased local interstitial lymphatic fluid leukocyts
-increased CNS sympathetic efferent tone back to initially infected inflamed target tissue
anatomaic structure of lympathic system
-lymph fluid
-collecting ducts
-lymph tissue
lymphoid organs
-tonsils
-thymus
-appendix
-peyers patches
-bone marrow
-spleen
-lymph node
tonsils
ring of lymphoid tissue
-palatine tonsil with lymph tissue
-soft palate is heavily infiltrated with lymphocytes
-build immunity early in life
Thymus
-maturation of T lymphocytes
-does not filter lymph
-reaches greatest size at two
spleen
largest mass of lymphoid tissue
-found in upper left quadrant
-connects with vascular circulation, cleans blood
-destroys damaged or deformed RBC and make IG
Peyers patches
-clusters of follicles in intestines
-b cell proliferation
-major source of antibody production
appendix
-off of large intestine
-peyers patches
bone marrow
-red; rbc,wbc,platelets
-yellow; fat
lymph node
only tissue that filters lymph
-400-450
connected to lymphatic vessels, but not circulatory system
lymphatic organs excluding lymph nodes
-connected by circulatory systems
-not lymphatic vessels
-not located along the course of lymph ducts and do directly function in filtration of lymph
lymphocytes
-T cells from thymus; directly attack and manage immune response
-B cells from bone marrow; make antibodies that immobilize antigens until they can be destroyed
Filter lymph
-macrophages destroy and remove microrganisms and debris that enters the lymph
protection via lymphatic system
-activates immune response
compostiion of lymph
water, proteins, lipids, carbs, enzymes, urea, minerals, hormones, dissolved gases, cells, toxins, bacteria, body wast, cellular debris
lymphatic vesels
-interconnected cappilaries, collecting vessels, lymph nodes, trunks and ducts
-travel along veins or arteries
-FORMS A ONE WAY SYSTEM THAT ENCOURAGES FLOW TOWARD THE HEART
Lymphatic system is NOT in:
-brain
-spinal cord
-epidermis
-endomysium
-bones, teeth, bone marrow
microstructure of lymph
-formed by endothelial cells loosley joined and overlapping
-collagen filaments anchor the cells to surrounding trissues so that increase in interstitial fluid volume opens the mini valves
-prevents vessels from collapsing
-right lymphatic duct
-drain lymph from right upper arm, right side of head, and thorax
-formed by merger in the right jugular trunk, subclavian trunk, and right transverse cervical trunks
thoracic duct
-recieves lymph from the rest of the body
-arises anterior to the first two lumbar vertebrae as a cisternachyli; this collects lymph from two large lumbar trunks that drain the lower limbs and from the interstinal trunk that drains digestive organs
-empties into venous circulation at the junciton of the internal jugular vein and the subclavian vein on left side of body
functions of lymphatic system
-interstitial trash and fluid collection
-conserves plasma protein
-gaurdian of health
-nutrient absorber
manifestations of clinical conditions due to poor lymphatic flow
-impairment of diaphragm and restricted thoracic cage motion (asthma, emphysema, bronchitis, acute respiratory illnes)
-impariment of skeletal muscular system;(prlonged inactivity especially in supine position, leads to somatic dysfunctions)
-hyersympathetic stimulation; stress
-alteration of anatomy; lymph node removed, edema
-impaired fluid dynamics
-
clinical importance of lympathic flow
-balanced system
-increased resorption of fluids
-increased circualtion and respirations
-decreased proteins interstitium
five contraindictions to OMM treatment of lymphatic system
-fracture
-central line
-infection, sepsis
-malignancy
why use soft tissue treatment
-screenign exmination for finding TART
-reduce muscle hypertonicity
-stretch and increase elascitity
-improve circulation
-increase venous and lymphatic drainage
-stimulate stretch reflex hypotonic muslces
-promote patient relaxation
-reduce patient guarding
-potentiate the effect of other omm techniques
-improve physician patient relationship
contraindictions to use soft tissue
-sprain or strain
-fracture or dislocation
-neurologic or vascular compromise
-osteoperosis and osteopenia
-malignancy
-infection
7 basic manual techniques for soft tissue
- inhibition
-stretching
-kneading
-petrissage/skin rolling
-deep friction/deep muscle injuries
-tapotement/striking muscle ot increase its tone and atrerial perfusion
-effelurage/ stroking movement
what things to be aware of when doing manipulation
-physician comfortable
-patient comfortable
-physician minimize energy expindenture
-force should not create pain
-push pull muscle away from bone
-dont irritate patient skin
-use leverage
-apply pressure with finger pads
-counterforce with transverse motion
-perpendicular stretch can be applied
-compression for deeper tissues
-monitor patient responses
-listen with hands
-be curious
-become eclectic in style
muscle spindle reflex
-stabilize and protect muscle
-senstive to changes in length
Golgi reflex
-stabilize and protect
-senstive to changes in muscle tension caused by passive stretch or contraction
muscle stretch reflex
-contraction in response to stretching within muslce, autonomic muscle regulation
Sinus Drainage
-frontal sinus duct
-nasolacrimal duct
-anterior ethmoid drainage
-maxillary sinus duct
-posterior ethmoid drainage
-spehnoidal sinus drainage
Trigeminal VI
-supraorbital nerve exiting from supraorbital forament
Trigeminal V2
-infraorbital nerve exiting from infraorbital foramen
CNX
-heart, lung, stomach, liver, pancreas
-vagus nerve
CNIX
-lacrimal salivary glands
CNVII
-lacrimal salivary glands
cervical sympathetic chaing ganglion
-internal carotid artery
-superior cervical ganglion
CNIII
eye
lymphatic treatment
-decompress throracic and lympathic ducts at venous sites
-facilitate cervical lymphatic flow in a proximal to distal fashion employing effurage
-facilitate thoracic excursion through rib raising on throacic pump
-facilitate normalization of peripheral and CNS balance
-remove or reduce cervical segmental and fascial sympathetic facilitiaton by doing suboccipital release
-perform sympahtetic inhibitions to supra an dinvraorbital nerves at their sites
Osteopathy vs. allopathy
-osteophaty offers a concise medical philosophy that can be clinically applied to any contemporary practice, and is articualted through the four principles
osteopathy vs allopathy
-offers distinctive integrative diagnositic and treatment modality that is OMM
direct treaments of OMM
soft tissue
ARt
HVLA
ME
MFR
osteopathy in cranial feild
Indirect
CS
MFR
OCF
osteopathy vs. allpathy
OMM strives to find and facilitate patients health instead of focusing on disease
Facitlitation-
the muscoskeletal system allows us to act out our unique human experience. Requires supply and maintenance systems,CV,resp,GI. The communication systems connect the consumers with the
-when a problem develops in the body’s communicating system function will be compromised.
-facilitation represents a serious problem with the communicating systems
-1)The maintenance of a pool of neurons in a state of partial or subthreshold excitation, in this state, less afferent stimulation is required to trigger the discharge of impulses
2)May be due to: sustained increase in afferent input, aberrant patterns of afferent input, changes within the affected neurons themselves or their chemical environment. When muscles are tight, good blood can’t get in and bad blood can’t get out, muscles are stagnant, bad PH. Affects the neuronal transport systems. Stasis is bad.
3)Once established it can be sustained by normal CNS activity; even if the original injury has healed, or the insult has been removed. Involved in creating a memory in CNS.
:inflammation is facilitation
Somatic Dysfunction
- impaired or altered function of related components of the body framework.
Nociceptors / PAN’s / Nociception
PAN-
small or lightly unmyelenated
-distributed throughout tissues of body
-Only tissues that are not innervated: articular and hyaline cartilage, nucleus pulposus and spinal discs, central nervous system parenchyma//KNOW THIS
-activated by by mechanical , thermal, and chemical energy
-repitive firing
NOCICEPTION
the physical action of exciting the small caliber primary afferent nerve endings
-nociceptors(PANS) send impulses into the spinal gray matter where they synapse
-PANs are involved in somatic dysfunction and facilitation and allostasis
-even if nociception input does not reach the conscious level as pain it can still facilitate spinal pathways
Allostasis / Allostatic Load
-the process of how the body responds to stressful situations; physical, psychosocial, acute, and chronic
-when stressed/threatened in the neuroendocrine immune system responds
-rapidly releasing the three C’s: catelcholamines, cortisol, cytokines
-long term acitivation of this wears you out: increased BP, obesity, increased acivity of fibrinogenic system, increased atherosclerosis
-Allostatic Load:adaptive condition
summation of effects of exposure to stress
-accumulation of damage from prolonged altered chemical state
-failure to restore homeostasis
-the price paid for chronic exposure to stress mediated neuroendocrine adaptations
-markers: systolic blood pressure, diastolic bp,waist to hip ratio, HDL cholesterol, total cholesterol HDL ratio, urinary cortisol, epinephrine and norepinephrine levels, DHEA
-somatc dysfunction triggers the arousal system of the brainstem, nociceptors
-SD is a noxious stimulus to the ANS whether perceived consciously or not
-the arousal system of the brainstem is coupled to the allostatic response
. The following reflexes
-If a segmental tart is visceral origin, there is : no edema, more elastic end feel at the barrier of ROM, 2 or more vertebral segments involved , linkage phenomenon, vertebra and its associated rib have same findings with passive motion testing
- If it is somatic in origin: there is edema, firmer end feel at the barrier of ROM, and may be limited to one vertebral segment.
a. Somato-somatic (S-S)
a. Somato-somatic (S-S)
-produces a reflex response in a segmentally related somatic structure
b. Viscero-visceral (V-V)
-vesceral sensory input produces a reflex response in a segmentally related visceral structure
c. Somato-visceral (S-V)
- produces a reflex response in a segmentally related visceral structure: the word that is first is where the signal starts from, and the second word is where the signal goes too. : input from somatic stimuli, send signal via afferent limb of the reflex into the spinal chord, synapses into interneurons on spinal cord, causes activity in a visceral organ through the sympathetic or parasympathetic efferent motor neurons./ex/ somatic dysfunction in upper mid thoracic spin exacerbating asthma
d. Viscero-somatic (V-S)
- produces a reflex response in a segmentally related somatic structure: input stimulus from a visceral structure. Send signal via afferent nerve of the reflex into the spinal chord. Synapses onto interneurons in spinal chord, activates sympathetic outflow back to visceral structures, and via motor neurons to skeletal muscle/ example asthma causing tart in upper mid thoracic facilitated segments
. Describe the relationship between, Somatic Dysfunction, Nociception, Facilitation, and Allostasis
-
8. Describe the relationship between Nociception, Facilitation, Allostasis, Homeostasis, and health & disease
-
9. Describe the physiology of how OMM can impact Nociception, Facilitation & Allostatic load
-correct the biomechanical insults to nerves and nerve cells that lead to disturbances in excitation, conduction, and trophic function
-alter the propioceptive and other discharges from somatic tissues to restore balanced intelligible reliable patterns of sensory feedback to the spinal chord
-soften or silence the somatic input to the feedback loops initiated elsewhere to arrest or retardation of aberrant impulses.
-OMM is directed toward the HOST: balancing of sympathetic and parasympathetic tone in order to enhance the self regulatory mechanisms of the body
-changes CNS by decreasing afferent load
-Vagus nerve, CNX. Parasympathetic innervations to many viscera
-know the viscerosomatic reflex levels for sympathetic and parasympathetic innervations of:
Lung parenchyma
Heart
Kidneys and adrenals
READING ASSIGNMENT:
Attached article by Beau Branyon
Other Answers for objectives can be found in:
Ward, Foundations for Osteopathic Medicine, 2nd ed., pp137-145, 150-153