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77 Cards in this Set

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What is an isometric muscle contraction?

No change in length during contraction

What is an isotonic muscle contraction?

A change in length of muscle during contraction

What is an eccentric muscle contraction?

The muscle that is lengthening (antagonist)

What is a concentric muscle contraction?

The muscle that is shortening (agonist)

What is flexion?

Decrease in joint angle. (bending)

What is extension?

Increase in joint angle (straightening)

What is abduction?

Movement away from the midline

What is adduction?

Movement towards the midline

What is dorsi-flexion?

Decrease in angle of ankle joint - (naughty toes - toes towards shin)

What is plantar-flexion?

Increase in angle of ankle joint (pointing toes)

Name locations of hinge joint.

Ankle, knee, elbow

Name locations of ball and socket joint

Hip joint, shoulder joint

Other types of joint.

Radio-ulna, pivot, gliding, saddle

Name the four rotator cuff muscles


Supraspinatus


Infraspinatus


Teres Minor


Subscapularis

SITS


Give three structural characteristics of a slow twitch muscle.

Small or red



Many mitochondria



High density of myoglobin



High density of capillaries



Low glycogen stores, low PC stores


Give two functional characteristics of a slow twitch muscle.


Resistant to fatigue


Slow speed of contraction


Low force produced during contraction

Give three structural characteristics of a fast twitch muscle.


White


Few Mitrochondria


Low density of myoglobin


Low density of capillaries


High glycogen stores, high PC Stores

Give two functional characteristics of a fast twitch fibre.

Fatigue quickly


High speed of contraction


Large force produced


How might the proportion of muscle fibres determine the success of a performer?

People with a mix of muscle fibre types may perform successfully in both aerobic and anaerobic activity or team games



People with high proportion of slow twitch or Type 1 most likely to perform successfully in endurance activities



People with high proportion of fasttwitch or Type 2 fibres most likely to perform successfully in anaerobic or explosive events


What is the effect of a warm up on the skeletal muscle? (Give 4)

Less risk of injury



Increased Muscle temperature



Increased Elasticity



Increased Flexibility



Greater Speed of muscular contraction



Greater Force of muscular contraction



Improves Performance in power based activities


What is the effect of a warm-up on the vascular system? (Give 4)

Increased Blood flow due to vascular shunt mechanism



Vasomotor control centre redistributes blood from organs to the working muscles



Vasoconstriction Of arterioles or pre-capillary sphincters decrease blood flow to organs



Vasodilation Of arterioles or pre-capillary sphincters increase blood flow to working muscles



Increased Venous return



Skeletal Muscle pump squeezes veins forcing blood back towards the heart



Pocket Valves in veins ensure one way blood flow



Respiratory Pump pulls blood up towards the heart



Smooth Muscle in veins contracts



Increased Venous return increases stroke volume (Starling’s Law)



This all leads to increased oxygen delivery toworking muscles


What is the effect of a cool-down on the skeletal system? (Give 3)

Decreased Risk of Delayed Onset of Muscle Soreness



Which is swelling that cause’s pain



Experienced 24-72 hours after exercise



Due to microscopic tears in the muscle fibres

What is the effect of a cool-down on the vascular system?

Heart rate gradually decrease



Increased Enzyme activity involved in breakdown of lactic acid



Maintains Blood flow



Skeletal Muscle pump remain active which prevents blood pooling



Vascular shunt mechanism remains active


Give 3 physiological factors that affect joint stability.

Strength of ligaments



Type of joint



Size or strength of muscles



Strength of tendons



Injury To connective tissue, amount of weight supported by joint


How can physical activity help joint stability?

Ligaments become stronger or more elastic



Muscle strength increases, muscle hypertrophy



Increase in number of muscle fibres



Tendons become stronger



Increased thickness of (articular) cartilage


What is Osteoarthritis?

Osteoarthritis is a degenerative joint disease



Caused by a loss of articular cartilage



In osteoarthritis cartilage is destroyed quicker than it is replaced



Friction between the ends of bones causes’ pain, swelling,



Bone spurs can be formed where friction occurs



Osteoarthritis commonly affects weight bearing joints, it commonly affects hips or knee's


How can P.A develop osteoarthritis?

An injury to a joint or damage to growth plate can cause onset of Osteoarthritis.



Lack of physical activity or increased body weight can cause onset of Osteoarthritis



Activity with large forces travelling through the joints or contact sport can cause Osteoarthritis



Repetitive actions or overuse can cause Osteoarthritis


How can P.A prevent or protect against osteoarthritis?

Increase thickness of cartilage



Thicker cartilage secretes more synovial fluid thatnourishes joint



Increase joint stability - strengthen ligaments



Strengthens surrounding muscles or increases muscle tone or lower body weight

How can high impact sports help prevent Osteoporosis?

Stronger bones - increase in peak bone density



Reduced risk of osteoporosis



Osteoporosis is the weakening of bones, making bones more prone to fractures



Weight bearing activities are best to improve bone health.

What is Linear Motion?

a body moveswith all parts moving at the same velocity in the same direction


What is angular Motion?


a body or part of a body moves in a circle or part of a circle around a fixed point.


What is general motion?


there is a combination of linear and angular motion.


What does Newton's 1st law state?

a body will remain in a state of uniform motion or at rest unless an (external) force acts upon it.



What does Newton's 2nd Law state?


the acceleration or rate of change of momentum of an object is proportional to the force (and takes place in the direction in which the force acts.)


What does Newton's 3rd Law state?


for every action there is an equal and opposite reaction.


How can changes in the COM affect performance?

Lower the centre ofmass the more stable



low CofM performer can resist external forces



Line of gravity within base of support creates a stable position



Line of gravity moving away from centre of base of support reduces balance



Line of gravity outside base of support creates an unstable position



A wide base of support: allows greater movement of centre of mass giving better stability



By moving the centre of mass outside line of action of force a performer can create angular motion.



By moving the centre of mass inside line of action of force a performer can create linear motion



By raising the centre of mass or gravity at take off a body can remain in the air longer or gain more height


How is 02 transported in the blood?

Combines with haemoglobin, forming oxyhaemoglobin. Dissolved in blood plasma



What is blood pressure?

The Pressure exerted by blood against the walls of a blood vessel (artery)

Give a typical value for blood pressure.

120/70mmHg

What is heart rate?

Beats per minute


What is Stroke volume?


Amount of blood ejected from the heart (ventricle) in one beat. (60– 90ml during rest)

What is cardiac output?

Hr x SV (amount of blood ejected from the heart in one minute)


Describe changes in stroke volume from rest to maximal exercise.

Stroke volume increaseswith exercise intensity



SV reaches a maximum value during sub-maximal exercise



Then SV decreases slightly during maximal exercise



SV decreases at very high exercise intensity



Maximal stroke volume =120–200ml



SV decreases because HR is so high there is not enough time for the ventricles to fill completely during diastole.


Identify mechanisms that maintain venous return during exercise.

Skeletal muscle pump



Pocket valves



Respiratory muscle pump



Smooth muscle



Gravity from above the heart


What is Starlings Law?

More blood returning to the right atrium of the heart



Increased stretch of the heart wall during ventricular diastole



Causing greater force of contraction during ventricular systole



As SV = EDV-ESV, stroke volume increases,



Cardiac output = heart rate x stroke volume



Therefore cardiac output increases



More blood returning to the right atrium or heart directly stimulates the SA node which increasesheart rate



Describe intrinsic control mechanisms that cause Q to increase during exercise.

Increased venous return



The right atrium stretches, more blood enters ventricles causing them to stretch further



This increases the strength of contraction of ventricles



This increases stroke volume



Body temperature increases which increases heart rate



Cardiac Output increases as cardiac output = SV x HR


Describe the conduction system that controls the cardiac cycle.

1. Atria fill with blood during atrial diastole



2. Pressure builds in the atria, blood travels passively intothe ventricles during ventricular diastole, SA node sends an impulse



3. Impulse spreads across atria causing atrial systole (contraction of both atria)



4. This causes the remaining blood in the atria to be pushed into the ventricles



5. Impulse reaches AV node



6. Impulse continues down the bundle of His



7. Impulse distributed to the purkinje fibres



8. This causes ventricular systole (contraction of both ventricles) from the bottom upwards.


What are the adaptations of the CV system caused through exercise?

Cardiac hypertrophy - increase in strength of the heart



Resulting in increased stroke volume



Decreased resting heart rate



Bradycardia or resting heart rate below 60bpm



Reduction in blood pressure



Capillarisation at alveoli and muscle cell allows for greater gaseous exchange during external or internal respiration



Better vasoconstriction or vasodilation results in increased efficiencyof vascular shunt mechanism



Increased blood volume



More red blood cells



Increased oxygen carrying capacity in the blood - steeper diffusion gradient of oxygen between the blood and the muscles



Increased VO2 max



Delayed OBLA


What is CHD?

CHD is any condition that is detrimental to the efficiency of the cardiovascular system


What are risk factors for CHD?

Hypertension or high blood pressure (systolic bp above140mmHg and diastolic bp above 90mmHg)



Smoking



Poor diet



stress



Diabetes


What is Arteriosclerosis:

A condition where the walls of the coronary arteries become thicker or less elastic



This prevents vasoconstriction and vasodilation of arterioles



Less efficient vascular shunt mechanism



What is Atherosclerosis:


Fatty deposits build up on the arterial walls



Fatty plaque forms in the arterial walls of the heart



Narrowing of the lumen in the coronary arteries so restricted flow of blood to heart muscle



Increased Likelihood of blood clots



Leading to high blood pressure (hypertension),heart attack, angina or arteriosclerosis


What is a heart attack or myocardial infarction?

A sudden and severe restriction or complete blockage of oxygen supply to the myocardium (heart tissue)



Will usually cause permanent damage to the heart wall


What is angina?

A pain in the chest caused by the partial blockage of acoronary artery



Causes a lack of oxygen to the myocardium


How can aerobic activity prevent CHD?

Cardiac hypertrophy resulting decreased resting heart rate



Helps prevent fatty deposits forming in arteries so helps prevent atherosclerosis



Helps prevent arteriosclerosis, maintains elasticity of arteries



Helps prevent heart attack or angina



Helps prevent blood clots forming as reduced blood viscosity



Reduced body weight



Reduce blood pressure



reduced blood cholesterol



reduced LDL cholesterol



LDL cholesterol is high in blood lipids that build up on the walls of coronary arteries causes atherosclerosis and arteriosclerosis



increased HDL cholesterol



HDL cholesterol is low in blood lipids



It will remove LDL cholesterol from the walls of the coronary arteries



This reduces the risk of atherosclerosis and arteriosclerosis



This in turn reduces the risk of angina or heart attack



less chance of fatty deposits building up on the walls of the coronary arteries and restricting the flow of oxygen to the myocardium


Why can activity be dangerous for old, untrained or obese?

Blood pressure will increase to dangerous levels



Increased risk of heart attack



Increased risk of chest pain due to angina



Increased stress placed on the cardiovascular system


What are the neural factors that affect heart rate during activity?

Chemoreceptors detect decreases in O2 or increases in CO2 or Lactic acid



Proprioreceptors detect increase in movement



Baroreceptors detect increases in blood pressure



Thermoreceptors detect increase in blood temperature



Messages are sent to the cardiac control centre (CCC) in themedulla oblongata



SA node stimulated via the accelerator nerve



The sympathetic nervous system increases heart rate



increases cardiac output



Q = SV x HR / cardiac output = stroke volume x heart rate


What are the neural factors that affect heart rate during recovery?

Chemoreceptors detect increases in the O2 or decreases co2 or Lactic acid



Proprioreceptors detect reduction in movement



Baroreceptors detect decreases in blood pressure



Messages are sent to the SA node via the vagus nerve



The parasympathetic nervous system decreases heart rate.


What is the vascular shunt mechanism?

Chemoreceptors detect increase in (pp) CO2



Proprioreceptors detect movement



Baroreceptors detect increasein pressure



Information sent to the vasomotor control centre (VCC) in the medulla oblongata


How does the VCC use the sympathetic Nervous System?

Decrease nerve impulses to the arterioles or pre-capillary sphincters leading to the muscles



Vasodilate the arterioles leading to the muscles



vasodilate the pre-capillary sphincters leading to the muscles



Increase nerve impulses to the arterioles or pre-capillary sphincters leading to the organs



Vasoconstrict arterioles leading to the organs



Vasoconstrict the pre-capillary sphincters leading to the organs


How do neural factors affect the mechanics of breathing?

Chemoreceptors detect decrease in O2 or increase in CO2 or lactic acid



Proprioceptors detect movement



Baroreceptors detect increase in pressure



Themoreceptors detect increase in blood temperature



Messages are sent to the respiratory control centre (RCC) in the medulla oblongata



which stimulates the inspiratory centre



expiratory centre stimulated by baroreceptors or stretch receptors


What are the mechanics of breathing during active inspiration?

Increased stimulation of external intercostals via intercostal nerve



Increased stimulation of the diaphragm via phrenic nerve



External intercostal muscles (EIM) and diaphragm contract harderor more (than at rest)



Sternocleidomastoid (SCM) or scalenes or pectoralis minor contract



Ribs move up and out morethan at rest - volume or area of thoracic cavity increases more than at rest.



More air rushes into lungs, increased depth of breathing than at rest


What are the mechanics of breathing during active expiration?

Expiration becomes active rather than passive



Stimulation Of expiratory muscles. Additional muscles are used: internal intercostals, rectus abdominus



Ribs move down and in more than at rest.



More air forced out of the lungs, increased rate of breathing from rest



increases minute ventilation



Minute Ventilation = Tidal Volume x breath frequency


Describe gaseous exchange of 02 at the aveoli.

Partial pressure in the alveoli is the same at rest as during exercise



Partial pressure in the capillaries is lower during exercise than at rest



Diffusion Gradient is steeper during exercise than at rest



More oxygen moves from the alveoli to the blood during exercise than at rest



Hb is fully saturated during exercise but not at rest


Describe carbon dioxide external respiration

Gases move from high partial pressure to low partial pressure



CO2 diffuses from the blood to the alveoli



There is a high partial pressure in the blood



There is a low partial pressure in the alveoli



There is a diffusion gradient between the alveoli and the blood


Describe oxygen external respiration

Oxygen diffuses from the alveoli to the blood - oxygen diffuses down the diffusion gradient



There is a high partial pressure of oxygen in the alveoli



During exercise muscles use more oxygen



So there is a lower partial pressure of oxygen in the blood



There is a steeper diffusion gradient of oxygen



More oxygen diffuses from the alveoli to the blood


Describe the process of internal respiration During exercise

More oxygen is available for diffusion into the muscle cell



Dissociation Curve shifts right meaning a greater dissociation of O2 from haemoglobin



Increase in the temperature of the blood



Reduces affinity of oxygen to haemoglobin



More oxygen being used in the muscle cell



Decrease in the partial pressure of oxygen in the muscle



Increased Diffusion or concentration gradient (of O2)



More Carbon Dioxide or Lactic Acid in blood



Increased acidity/ decrease in pH of the blood / Bohr Effect


Describe the effect of carbon monoxide on the transport of 02 in the blood

Less efficient gas exchange or diffusion



Haemoglobin has a higher affinity for carbon monoxide than O2 - less oxygen combines with haemoglobin



The partial pressure of oxygen in the blood decreases



Less oxygen is carried or transported in the blood



Less O2 delivered to muscles


Describe the effects of altitude on the respiratory system.

Decrease in atmospheric pressure (air thinner)



Causes increase in breath frequency



Causes an increase in water loss



Decrease in pressure of oxygen in atmospheric air compared to sea level(less oxygen in the air compared to sea level)



Decrease in efficiency of the respiratory system



Decrease in ppO2 in the alveoli



Reduced O2 diffusion gradient at the alveoli



Less O2 diffuses into the blood - decreased gaseous exchange betweenalveoli and blood



Less O2 combines with haemoglobin (haemoglobin is less saturated at lungs)



Less oxygen is transported in the blood - less oxygen is transported to the working muscles



Decrease in efficiency of internal respiration



Reduced O2 diffusion gradient atthe muscle



Decrease in O2 dissociation from haemoglobin to myoglobin



Less O2 diffuses into the muscle cell



Increase in chemoreceptor stimulation



Chemoreceptors detect lower O2 level



Information sent to respiratory control centre (RCC)



Inspiratory centre and expiratory centre stimulated



Leading to increased depth and rate of breathing



Increased risk of altitude sickness or dizziness or nausea or vomiting



Air is dryer



Increased risk of dehydration


Describe the effects on performance whilst performing at altitude.

Aerobic performance deteriorates



Cannot Train at the intensity or for as long as possible at sea level



Reversibility Will occur



VO2 max is reduced



Increase In lactic acid - slower removal of lactic acid



Early Fatigue or OBLA


E.g. cyclistsin the Tour de France



Low intensity exercise less affected E.g.mountain walking



Some anaerobic are unaffected



Some Anaerobic performances benefit from lower air resistance E.g. throwing events: discus or javelin will travel further



In some anaerobic activities performance deteriorates



Decreased Tolerance to or buffering of lactic acid



Increased Levels of lactic acid inhibits enzyme action E.g. 200m or 400m or 800m


What are the positive effects of training on the respiratory system.

After 4-6 weeks altitude training increases efficiency of respiratory system (acclimatisation occurs)



Increased number or surface area of alveoli



Increased capillary density at alveoli or muscles



Increased capacity for diffusion at alveoli or muscles



Increased release of EPO



Increased haemoglobin or red blood cell



Increased oxygen carrying capacity of blood



Increased strength of respiratory muscles e.g. diaphragm orintercostals or scalenes



Increased Lung volumes or capacity or depth of breathing or tidal volume


What is Asthma?

reduces the amount of oxygen getting in to the lungs



It is a reversible narrowing or constricting of respiratory airways (asthma makes it hard to breathe)



Causing coughing, breathlessness, wheezing, mucus production, chest tightness


What are the causes of Asthma?

Asthma is most commonly caused byinflammation of the bronchus



It is usually brought on by triggers



Allergens e.g. exhaust fumes, pollen, hair and dust



Exercise induced asthma (EIA)



Drying of airways increased by more breathing exercise



More likely with high intensity exercise



More likely when exercising on cold days


E.g. Winter sports – cooler air tends to bedrier


E.g. Water sports, swimming– due to chlorine


What are the physiological effects of asthma on people performing in endurance activities.

Can cause unconsciousness- can be dangerous



Can limit athletic performance due to reduced function of respiratory system



Less oxygen is supplied to the muscles



Specially limits aerobic athletes who are reliant on oxygen uptake



Tidal volume reduced



Efficiency of gaseous exchange at the alveoli and the muscle reduced



Causing increased levels of lactic acid to be produced -early onset of fatigue or OBLA


What methods are available to help control Asthma

Medical inhalers


Blue inhalers e.g. ‘Ventolin’etc. Used during exerciseto relieve symptoms, Used before exercise to relax airways (dilate bronchi)



Brown, beige, white, red, orange e.g. ‘Becotide’ / Symbicort etc


Used daily, To reduce inflammation in airways, Inhaled before exercise as preventative measure to improve lung function



Inspiratory muscle Training


Use of respiratory equipment to develop strength of respiratory muscles


Forced inspiration and expiration exercises e.g. ‘Powerbreathe’


Use twice a day, 30 breaths, Maximal inspiration and maximal expiration



Breathing Control or breathing exercises



Diet


Increased intake of vitamins or fresh fruit or vegetables


Drink lots of water (to avoid dehydration)


Increased intake of fish oils


Reduced intake of salt


Caffeine acts as a bronchodilator, caffeine now off banned IOC list


What are the effects of smoking?

Smoking Can cause:



Decreased Elasticity of respiratory structures, damage to or irritation of respiratory structures



Narrowing of airways or respiratory pathways



Tar in lungs



Coughing,shortness of breath and wheezing



Increased Likelihood of asthma attack or developing asthma



Frequent lung infections


What are the effects of smoking on performance?

Reduction in performance of endurance athlete (endurance athletes find it harder to compete)



Decreased lung volume or capacity - decreased volume of air or oxygen reaching alveoli


E.g. tidal volume, minute ventilation and vital capacity



Reduction insurface area for gaseous exchange or diffusion



Cigarette smoke contains carbon monoxide



Haemoglobin has ahigher affinity for carbon monoxide than oxygen



Haemoglobin Combines with carbon monoxide instead of oxygen during external respiration



Reduced saturation of oxygen with haemoglobin at lungs, therefore reduced ppO2 in blood



increased levels of carbon monoxide in blood



decreased levels of oxygen in blood



Less oxygen delivered to working muscles



Decreased Diffusion or concentration gradient of oxygen



increased diffusion distance for gaseous exchange due to tar build up