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177 Cards in this Set
- Front
- Back
One of the most important sequelae of GCA
|
vision loss
|
|
_____ is considered a prime ohthamlic medical emergency as blindness is preventable
|
GCA
|
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Race GCA is prevalent in
|
WHITE, european
|
|
T/F Men are twice as effected as women in GCA
|
FALSE
opposite is true |
|
What age does GCA usualyl occur?
|
>55 mean presentation is 71
|
|
__________% vision loss occurs in GCA
|
15-20%
about 8.5 days until loss usually occurs |
|
T/F GCA is usually bilateral
|
FALSE
|
|
Most common complaint of patient with GCA
|
headache occipital loe
|
|
Headache and _______ are strong indicators of GCA
|
jaw (50% of patients)
|
|
_% of patients with GCa experiency polymyalgia rhuematica
|
50
fever of unknown orgin, respiratory symptoms and facial pain |
|
fever of unknown orgin, respiratory symptoms and facial pain are traits of what?
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polymyalgia rheumatica
|
|
T/F APD present with GCA
|
FALSE
|
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T/F GCA is a painless loss of vision
|
FALSE
PAINFUL |
|
Optic disc swelling in GCA is due to
|
AION
anterior ischemic optic neuropathy Most common cause of vision loss in GCA |
|
Most common cause of vision loss in GCA
|
AION
|
|
T/F AION is characterized by high erthyrocyte sedimentation rate
|
T
|
|
What test confirm GCA?
|
Erythrocyte Sedimentation Rate and Temopral artery biopsy
|
|
Average ESR for male
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age/2
|
|
ESR average for women
|
(agee+10)/2
|
|
Perferred method of ESR
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Westergren method
|
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T/F A patient with GCA may experience diplopia
|
T
may also experience amarosis fugax |
|
How do you treat GCA?
|
Corticosteriods
reduce symptoms butdo not shorten course of disease daily oral dose 40-80 mg Azathiprine, Methotrexate, and cylcosporine, dapsone have also been used |
|
Intravenous methlypreidnisone is the preferred treat for patients with GCA that have experienced
|
visual loss
|
|
CRAO presents with
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sudden, unilateral, painless loss of vision
|
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T/F CRAO is a painless loss of vivsion
|
T
|
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T/F CRAO tends to be bilateral
|
FALSE
unilateral |
|
A cherry red spot and a pale ONH with mild retinal edema are signs of
|
Central Retinal ARtery Occlusion
|
|
What causes CRAO
|
thrombosis in retobulbar portion of CRA
|
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Restoration of vision not possible if retinal circulation has been completely interrupted for more ______ minutes!!!
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100
|
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To help patient with CRAO, give paitnet
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Topical drops to lower IOP, IV Diamox, IV mannitol, and possible IV steriods
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Patients with hyphema should be considered to have a ______________________ until proven otherwise
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ruptured globe
|
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Low intraocular pressure is known as
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hyptony
|
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MRIs are contraindicated when
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ferromagnetic FB is suspected
|
|
The gold standard for imaging
|
CT`
|
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Degree of damage is directly proportional to the _________of the FB and inversely proportional to its ______-
|
velocity
size |
|
The retaining of copper or lead in eye is known as
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chalocosis
|
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Removing a foreign body with the external electromagnet is known as
|
pars plana virectomy
|
|
A rapid rise in abdominal pressure especialyl against a closed glottis is characterstic of the
|
Valsalva maneuver
|
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What type of hemorrages are present in Valsalva retinopathy?
|
red, doma shaped
vitreous hemorrhage |
|
T/f Valsalva retinoapthy tends to be self limiting
|
T
|
|
T/F An APD is usually present with Diabetic paopllopathy
|
T
see indistinct disc margin with diffuse swelling and elevation with fine telangiesctatic patten of vessels |
|
T/F most casses of idaetic papillopathy occur early 2nd-3rd decade of life
|
T
tend to see in longstanding juvenile onset diabetes |
|
VF defect seen with diabetic papillopathy
|
enlarged blidn spot, central scotoma
|
|
T/F perseptal cellulitis is most common in geriatric populations
|
FALSE
pediatric |
|
Suppurative means
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pus containing
|
|
preseptal suppurative cellulitis is most commonly due to what bacteria?
|
Staph auerues
Strep pyogenes |
|
T/F anaerobes and polymicrobial infections are common causes of preseptal suppurative cellulitis
|
FALSE
LEAST likely causes |
|
Pediatric nonsuppurative preseptal cellulitis most likely due to
|
Hemophlus influenza and Strep Pneumoniae
|
|
Normal position of the globe finding allows us to differentiate preseptal cellulitis from
|
orbital cellulitis
|
|
T/F VA should be unaffected in preseptal cellulitis
|
T
|
|
An infection within theorbit, posterior to the orbital septum is know nas
|
orbital cellulitis
|
|
Most common cause of orbital cellulitis
|
extension of infection form NASAL sinuses
> 90% of cases |
|
Post-traumatic and post surgical orbital cellulitis is usually due to
|
Staph aureus
|
|
T/F Pain upon eye movement is a feature of preseptal cellulitis
|
FALSE
orbital |
|
T/F Diplopia is present in orbital cellulitis
|
T
|
|
Rhinorrhea is a symptom of
|
sinusitis (leakage of CSF into nasal cavity)
implies orbital cellulitis |
|
T/F Orbital cellulitis patients have diminished sensation in CN V1
|
T
also see reduced color vision poptosis |
|
If signs of orbital cellulitis occur BILATERALLY, consider the possibility that the patient has
|
cavernous sinus thrombosis
|
|
Cavernous sinus thrombosis involves pareisis of what CN?
|
3,4, and 6, as well as decreased V sensory function
dilated sluggish pupil present |
|
An accumulation of purulent material between the periobital and the bony walls of the orbit is known as
|
Subperiosteal abcess
|
|
Subperiosteal abcess is usually due to
|
sinuisits, especially ethmoidal
|
|
What can differentiate Subperiosteal abscess from oribtal cellulitis?
|
CT scan
Ultrasound |
|
T/F Angle Closure Glaucoma tends to occur more often in women, and more often in hyperopic patients
|
T
|
|
T/F Angle Closure Glaucoma is a common occurance in caucasians
|
FALSE
higher rate in East Asia, Alaska, Greenland |
|
An AC < _____ mm is a high risk for angle closure
|
2
Refer for laser peripheral iridotomy 2.1-2.5 mm AC depth should be reffere dto glaucoma specialist |
|
irido-lenticular touch cuasing an obstruction of aqueous circulation in midperipheral region of lens is known as
|
pupillary block
|
|
If pupillary block is associated with lax peripheral iris tissue, iris ___ may result, which leads to obstruction of angle structures and closure of trabeculum
|
bombe
|
|
Type S puppillary block starts at _____ and does NOT occlude trabecular structures
|
schwalbe's line
a more bening form of closure |
|
T/F Type S puppilary block is a more benign form of closure
|
T
does NOT occlude trabecular structures |
|
Type ___ pupillary block produces a continuous zone of touch between iris and trabeculum
|
B
Associated with PAS (peripheral anterior synechiae) |
|
This type of puppillary block is associated with peripheral anterior synechiae
|
B
|
|
Angle closure due to physical crowding of angle by superfluous iris tissue following pupillary dilation is known as
|
Plateau Iris
Due to anterior insertion of iris producing type B obstruction of TM |
|
Pupillary block due to tumescence and enlargement of lengs, forcing iris and ciliary body into anterior chamber is known as
|
Phacomorphic glaucoma
|
|
Malignant glaucoma occurs when
|
forces posterior to lens (vitreous pressure) pushes the iris diaphragm forward. SEe contact between lens equator and ciliary body
CANNOT benefit from iridotomy |
|
T/F Malignant glaucoma is treated with iridotomy
|
FALSE
does not help Treat with hyperosmotics, atropine (indefinitely), b-blockers, CAI, Chandler's procedure, etc |
|
Symptoms:
Pain photophobia halo blur abdominal pain vomiting RED EYE Partial mydrasis EXTREMELY high IOP Likely diagnosis? |
Acute angle closure- stage 1
sudden extreme IOP elevation > 50 if pupils are small and no mydriasis exists, it is probably only uveitis |
|
Immediate treatment of Acute angle clusre
|
Iridotomy
B-blocker+ alpha 2 adrenergic+ topical CAI Oral CAIs Analgesic and anti0emetic topcial steriod SUPINE position 1 hour followed by miotic |
|
T/F Miotics do not work on iris sphincter when IOP > 40
|
T
due to ischemia and paralysis of sphincter this is why you do not give a patient a miotic for the first hour with acute angle closure |
|
Stage 2 of acute angle closure, expect to see IOP
|
< 35 or by 25% of presentiong IOP
if not, give 50% glyerol or 20% mannitol |
|
Idiopathic inflammaiton involving the orbita apex is known as
|
Oribtal apex syndrome
|
|
T/F Orbital apex syndrome involves pain behind globe, + APD, and marked restriction of ocular motlity
|
T
|
|
Tolosa-Hunt syndrome is a specific form of apical idiopathic inflammation involving the superior orbital fissure and the
|
CAVERNOUS syinus
rapid progression acute onset |
|
If a patient has pain behind globe, multiple cranial nerve involvement (KEY DIAGNOSIS), eyelid edema and congunctival chemosis unilaterally with an APD it is probably what syndrome?
|
Tolosa Hunt
|
|
Monofixation snydrome is tested with
|
4 pd base out.
Patient does not maintain binocular vision so there is no fusion of eyes. Patient has a central suppression scotoma |
|
Giant paving stone papillae are often seen with this kind of conjunctivitis
|
vernal
|
|
treatment for vernal conjunctivitis
|
prednisolone acetate qid
erythromycin tid lodoxamide qid scopolamine bid artifical tears coolr compress |
|
Traumatic optic neuropathy occurs in approximately __% of head trauma cases
|
3%
|
|
What is the most common cause of optic nerve trauma?
|
motor vehicle and bike accidents
|
|
Direct optic nerve trauma is usually caused by
|
falling onto a pointed object
|
|
T/F Optic Nerve Trauma always has an APD present
|
T
|
|
The complete disinversion of the entire optic nerve is known as ____ and there is a total APD present, as well as widesprea retinal infarction
|
Optic nerve avulsion.
Follows a diffuse impact on globe or injury from a blunt instrument. occurs when there is partial or complete tearing of the optic nerve from the globe at the level of the lamina cribrosa. |
|
T/F Anterior optic neuropathies occur more frequently that posterior indirect optic neuropathies
|
FALSE
Posterior indirect occurs more often than either direct or indirect anterior optic neuropathies -damage typically due to fracture of sphenoid which damages intracanicular section of the ON |
|
T?F young males tend to have ore posterior indirect optic neuropathies
|
T
follows blow to face, forehead or temporal |
|
T/F Fundus of patient with posterior indirect optic neuropathies appear normal
|
T
optic atrophy supervenes only after weeks have past |
|
Choice of imaging orbits and head in trauma cases
|
CT
|
|
International Optic Nerve Traumma study found that
|
no clear enefit was found for either corticosteriod therapy or optic canal decompresion surgery
|
|
If patient has an aneurysm in the pshenoid sinus, patient will have ____ and loss of vision
|
Epistaxis (nosebleed)
|
|
The ABC's of orbital injuries
|
Priority:
establish an AIRWAY, control for BLEEDING, restore CIRCULATION |
|
During an orbital injury, if any reduction in eye movement is present, this test should be performed to distinguish entrapemtn and muscle contusion from paresis
|
Forced duction
|
|
Emphysema and crepitus could suggest what type of fracture?
|
paranasal sinus
|
|
Anosmia is the loss of this and can be an indication of a dural tear in the roof of the ethmoid sinus or cribiform plate
|
smell
|
|
The most common type of conventional radiography used for assessing orbital flor and rim is
|
Water's conventional radiography
|
|
What kind of images are taken with a CT
|
3 mm axial and coronal images through the sella
Coronal images are crucial!! |
|
Approximate 1/3 to 1/2 of orbital blowout fractures are associated with fractures of
|
lamina papyracae
|
|
T/F Medial wall is thinner than the orbital floor and is a common site of blowout fractures
|
FALSE
it is NOT a common site of blowout fractures because it has added support of ethmoid |
|
Zygomatic fractures are also known as _____ fractures because of the dislocation of the zygoma from frontal bone, arch, and maxilla
|
tripod or tripartite
Do surgery if patient cannot open mouth or there is a lot of facial asymmetry |
|
Patients with blow-out fractures are put on a cours eof antibiotics to prevent
|
orbital cellulitis
|
|
T/F whited eye vlowout fractures are common in children who have marked motility restriction up and down, lack enopthalmous and have minimal soft tissue signs of trauma
|
T must to surgery
|
|
T/F MRI is senstitive to calcifiation
|
FALSE
lacks fine cortical bone details |
|
T/F CT is cheaper than MRI
|
T
has higher spatial rsultion and GREAT bone imaging |
|
Treatment for scleritis
|
topcial steriods and cycloplegic, NSAIDS
FU 6 weeks |
|
A chronic problem where the cornea continuously gets injured. The desmosome and hemidesmosome junctions thus are not repairing correctly. This is known as
|
Recurrent corneal erosion is the ANSWER in the test. they should be repairring but because they dont, and you get RECURRENT CORNEAL EROSION.
|
|
What do you do when a patient comes in after getting something in their eye?
|
THIS IS ON THE TEST: IDENTIFY THE POISON/CHEMICAL AND USE COPIOUS IRRIGATION
o Identify the problem, then irrigate. o Some dusts cannot be washed away and must be dusted off away. -Photochemicals CAN injury eyeball. |
|
or any corneal or anterior segment injuries base management includes
|
Antibiotics and AntiInflammatory --> 4th generation quinolone antibiotic, CYCLOPLEGIA, STERIOD FOR 1-2 WEEK, Bandage contact lens, motrin
|
|
CONTACT LENS ASSOCIATED ACUTE RED EYE CAN COMe FROM:
|
TEST QUESTION All of the above with CLAARE.
Hypoxia, mechanical, toxic are ALL POSSIBLE MECHANISMS. |
|
Injury due to burn is known as
|
Test: ESCHAR KERATOPATHY
BURN question=THE ANSWER IS ESCHAR!!!! USE BASE MANAGEMENT AND ALSO NEED TO GET RID OF DEAD TISSUE via DEBRIDEMENT. |
|
Test: Rust ring needs to be removed when foreign body is removed.
Ihe instrument of choice |
is a 27.5 inch needle in a half gauge syringe
|
|
A PENETRATING INJURY leading to a D SHAPED PUPIL IS DUE TO
|
BLUNT TRAUMA
see a PEAKED PUPIL |
|
Approximately _% of injuries occur in workplace, mostly construction sites
|
50
|
|
The leading cause of blindess in young adults is due to
|
trauma
|
|
T/F Standardization Classification of ocular trauma is used to asses mechanical, chemical, electrical, and thermal ocular injuries
|
FALSE
NO chemical, electrical or thermal injuries are assessed using this system |
|
An open globe, full thickness wound caused by a blunt object is known as
|
a Rupture gloe
|
|
A perforating injury involves _____ full thickness wounds (entrance and exit) of eye wall
|
two
both wounds are caused by same agent |
|
A closed globe injury of the eye wall or bulbar conj that is caused by a sharp object whose wound is at the impact site is known as a ____ laceration
|
lamellar
|
|
The most prognostic basis of final visual outcome with an eye injury is
|
VA!!
Grades the injury |
|
4 variables of ocular trauma the system is based on
|
Type of injury
Grade of injury (VA) Presence of RAPD Zone of injury |
|
T/F While Using VA's to grade eye injuries, a grade of 4/200 to light perpection is the most severe
|
T
|
|
A VA of 20/50-20/100 following an eye injury implies that the injury is
|
moderate
|
|
A VA of 19/100-5/200 following an eye injury implies that the injury is
|
severe
|
|
The zone of injury with an open gloe injury is defined by the location of the most ________ full thickness aspect of the globe opening
|
POSTERIOR
|
|
A zone 1 open globe injury is limited to
|
the cornea or corneoscleral limbus
|
|
This zone of an open globe injury is limited to the anterior 5 mm of the sclera
|
2
|
|
Zone 3 of this type of injury extends the full thickness into the sclera more than 5 mm posterior to the limbus
|
OPEN globe
|
|
T/F You do not describe a closed globe injury as penetrating, perforating or ruptured globe
|
T
no full thickness opening of the globe has occured |
|
Closed gloe injuries involve blunt force resulting in ________ injury, while sharp forces result in ____ injuries
|
contusion
lamellar-lacerating |
|
Because closed globe injuies do not involve full thickness wounds, zones of injury are based on
|
the tissues injuried
|
|
Zone 1 closed globe injuries are limited to
|
bulbar conj, sclera, or cornea
aka corneal abrasions, conj hemes, intracorneal FB |
|
This zone for closed globe injuries involes structures in anterior seg up to and including the lens, zonules, and pars plicata of ciliary body
|
2
|
|
Zone 3 of this type of injury inclues plars plana, choriods, retina, vitreous, or optic nerve
|
closed globe
|
|
When media opacity precludes the assessment of posterior structures following an eye injury, use a _______ to determine zone of injury
|
B scan ultrasonography
|
|
Damage caused by direct contact with eyeball by any blunt object is known as a
|
contusion injury
|
|
T/F a contusion injury refers to a blow NOT directly striking the eye, but more commonly, by a strike to the head
|
FALSE
Concussion injury! |
|
Vossius' ring is a sign of
|
iris change during ocular trauma
a mild concussion can cause scattering of IPE, leaves an imprint on lens capsul |
|
Iris changes during ocular trauma include
|
Voissius ring
Acute anterior uveitis Irdiodialysis (tear at iris root) Iridoschisis (splitting of itis) Sphincter tears |
|
When erythroytes settle into the anterior chamber, a _____ is formed.
Suspension of erythrocytes in aq is known as |
hyphema
microscopic hyphema |
|
T/F Re-bleeding is more severe than the initial hemorrhage
|
T
|
|
T/F Blacks and those with higher IOPs are more likely to experience seciary henirrhages
|
T
|
|
Initially IOP raises due to bleeding into a limited space and then pressure falls to subnormal levels during what traumatic event?
|
Traumatic hyphema
IOP often remains low for several days due to shutdown of ciliary body function |
|
T/F microhyphema is visible without a slit lamp
|
FALSE
use 3 mm wide beam |
|
Grade 4 hyphema
|
total clotted hyphema
|
|
When blood fills 1/2 of aq chamber its what grade?
|
3
|
|
Grade 2 hyphema
|
blood fills 1/3-1/2 chamber
|
|
Grade 1 hyphema has this much blood in chamber
|
< 1/3 of chamber
|
|
Duration of uncomplicated hyphema is
|
5-6 days
pt presents with acute pain, reduced VA elevated IOP |
|
Meds for hyphema
|
atropine TIB
prednisolone qid-q2h if no corneal defects topical beta blocker or Alphagan P, if still high IOP give system CAI ad possibly IV mannitol inpatient meds:aminocaprioc with an antiemetic AVOID asprins or nsaids |
|
T/F ciliary Body trauam shows an increase in IOP
|
FALSE
decrease aq production occurs and leads to hypotony |
|
When the ciliary body detaches from the sclera, it is known as
|
cyclodialysis
|
|
What type of glaucoma is gonio CONTRAINDICATED fors in the first few day?
|
angle recession glaucoma. 71-86% ave hyphema and should no do gonio for the first few days to do change of bleeding into anterior chamber
|
|
The most common cause of unilteral cataracts in youth is
|
traumatic cataracs
|
|
T/F The further back in the lens capsule the contusion cataract is located, the earlier the time of inury
|
T
|
|
The name of a traumatic cataract=
|
Rosette
|
|
What is the primary cause of retinal dettachment?
|
A PVD
|
|
A DIRECT rupture of the choroid is known as ______- and is a tear anteriorly at the site of injury oriented parallel to _______
|
COUP
ora serrata |
|
T/F A Coup choroidal rupture occurs posteriorly away from the site of injury generally crescent in shape and concentrated with the optic nerve head
|
FALSE
Contrecoup!! |
|
3-4 weeks after a choriodal rupture expect to see
|
choriodal neovascualrization
treatwith photocoagulation |
|
A simultaneous brak in the choriod and retina resulting from a high velocity missile penetrating the orbit but not the globe is known as
|
Chorioretinitis Sclopeteria
concussion type, nonpenetration 2 areas of injury: area adjacent to path of missile and macular area |
|
A macula hole is what color?
VA is usually around |
orange to red orange due to exposure of choriocapillaris
20/100-20/400 |
|
Commotio Retinae is a transient _____ or opacification at the level of deep sensory retina after blunt trauma
|
whitening! The blood vessels should still be visible over whitening because damage is DEEP. In CRAO, blood vessels are obscured
|
|
T/F Blood vessels are visible over whitening of retina that occurs during commotio retinae
|
T
The blood vessels should still be visible over whitening because damage is DEEP. In CRAO, blood vessels are obscured http://www.opt.pacificu.edu/ce/catalog/10310-SD/Trauma%20Pictures/Commotio%20Retinae.jpg versus http://eyelearn.med.utoronto.ca/ClinicalSkills/images/Fundo12.jpg |
|
Retinal dialysis occurs _____% during traumatic retinal breaks
|
53%
root to root break from otra to ora |
|
T/F Rhegmatogenous retinal detachment is the most common type of retinal detachment.
|
T
t occurs when a break (tear or hole) in the retina allows fluid from the vitreous humor to enter the potential space beneath the retina. This causes the retina to separate from the layer beneath, known as the retinal pigment epithelium (RPE). |
|
This type of retinal detachment is most common in the advanced stages of diabetic retinopathy,
|
Tractional retinal detachment occurs when fibrous membranes (essentially scarring processes) in the vitreous humor and retina produce mechanical traction on the retina, literally pulling the retina from the underlying layer, the retinal pigment epithelium (RPE).
|
|
_______retinal detachment occurs with conditions that disturb the blood-retinal barrier, i.e., conditions that allow the build-up of fluid beneath the retina.
|
Exudative
|
|
3 Treatments of Retinal Detachments
|
Scleral buckling
Pneumatic retinpexy Vitrectomy |
|
Shaffer's sign
|
PIGMENT in vitreous. Retinal detachment occurs as proliferation changes have begun
|
|
After laser surgery what color are the spots initially?
|
white, as they have not healed, then it turns into black spots.
|
|
Treatment of Hydrops
|
Lower IOP via CAIS
fit 1 week later with RGP PK may be indicated |