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PREECLAMPSIA
10% OF ALL FIRST TIME PREG WOMEN AFFECTED BY THIS SYSTEMIC DISEASE CHARACTERIZED BY PERIPHERAL VASCULAR RESISTANCE AND ORGAN DAMAGE.

25% WITH CHRONIC HTN WILL GET PREECLAMPSIA.

THIS IS A DISEASE F PREGNANCY FROM MILD TO SEVERE CHARACTERIZED BY PROTEINURUA AFTER 20TH GEST WK.

CURE: DELIVERY. BUT WILL HAVE COMPLICATIONS UP TO 5 DAYS POST PARTUM.
PREECLAMPSIA MEDICAL MANAGEMENT
ANTIHYPERTENSIVES:

HYDRALAZINE - VASODILATOR

METHYLDOPA - WORKS ON CNS

LABETOLOL - BETA BLOCKER

ALL ABOVE ARE FIRST LINE DRUGS.

NIFEDAPINE - CALCIUM CHANNEL BLOCKER. 2ND LINE DRUG.
PREECLAMPSIA

MEDICAL MANAGEMENT

MONITOR MOM AND FETUS ______
DRUGS OF CHOICE ARE __________
GOAL IS ______ AND _______
ONLY CURE IS _____
DELIVERY INVOLVES ______ (DRUG)
ONCE DX, WOMAN AND FETUS SHOULD BE MONITORED WEEKLY. ANTIHYPERTENSIVES ARE USED TO CONTROL BP. GOAL --> CONTROL BP AND SEIZURE PREVENTION.

ONLY CURE IS DELIVERY WITH SEVERE PREECLAMPSIA WHEN MATERNAL STATUS IS DETERIORATING.

CARE IN DELIVERY INVOLVES MAG.

DELIVERY CURES, BUT STILL @ RISK FOR COMPLICATIONS INCLUDING ECLAMPSIA UP TO 5 DAYS PP.
CHRONIC HTN
HTN BEFORE ______ GEST. WEEK
PLACES WOMAN AT HIGH RISK FOR DEVELOPING _______
HTN BEFORE CONCEPTION OR BEFORE 20TH GEST WEEK. PLACES WOMAN AT INCREASED RISK FOR DEVELOPING PREECLAMPSIA.
PREECLAMPSIA/ECLAMPSIA
SYSTEMIC DISEASE WITH HTN ACCOMPANIED WITH PROTEINURIA AFTER 20TH GEST WEEK. ECLAMPSIA IS THE CONVULSIVE STAGE.
PREECLAMPSIA SUPERIMPOSED ON CHRONIC HTN
HYPERTENSIVE WOMEN WHO DEVELOP NEW ONSET PROTEINURIA, OR PROTEINURIA BEFORE 20TH WEEK GEST., OR SUDDEN UNCONTROLLED HTN.
GEST HTN
HIGH BP DETECTED FIRST TIME AFTER MID PREGNANCY WITHOUT PROTEINURIA. TYPICALLY DX POST PARTUM. NO CHANGE IN WOMAN. 5-6% OF ALL PREGNANCIES.
RISKS FOR PREECLAMPTIC WOMEN
CEREBRAL EDEM, HEMORRHAGE, STROKE, DIC, PULMONARY EDEMA, CHF, HEPATIC FAILURE, RENAL AND LIVER FAILURE, ABRUPTIO PLACENTA
PREECLAMPSIA:

NEWBORN RISKS
1
2
3
4
5
PREMATURITY
DELIVERY MAY BE INDICATED PRETERM

IUGR r/t DECREASED UTERPLACENTAL PERFUSION

LOW BIRTH WEIGHT

FETAL INTOLERANCE TO LABOR

STILLBIRTH
THESE WILL PUT YOU AT RISK FOR DEVELOPING PREECLAMPSIA
NULLIPARITY - number of times a woman has given birth
AGE < 19 OR > 35
OBESITY
MULTIPLE GESTATION
FAMILY HX OF PREECLAMPSIA

PREEXISTING HTN OR RENAL DISEASE

HX OF PREECLAMPSIA / ECLAMPSIA

DM
PREECLAMPSIA:

ASSESSMENT FINDINGS
CHANGE IN COAGULOPATHIES

LABS: INCREASED LIVER FUNCTION TESTS, DECREASED KIDNEY FUNCTION.

DECREASED GFR = OLIGURIA (S/S OF SERIOUS KIDNEY DAMAGE)

ELEVATED BP: HTN 140/90 OR GREATER

PROTEINURIA OF 1+ OR >

LOW PLATELET COUNT (BELOW 100,00)

SEVERE PERSISTANT HA

DECREASED ALBUMIN = EDEMA

PULMONARY EDEMA

RETINAL ARTERY SPASMS CAUSE CHANGE IN VISION / BLURRED OR DOUBLE

SEIZURES
HTN
140 OR >
---------------
90 OR >


2ND LEADING CAUSE OF DEATH AND CONTRIBUTES TO NEONATAL MORTALITY AND MORBIDITY

MOST COMMON PREGNANCY COMPLICATION .

6-8% OCCURENCE
PREECLAMPSIA

PATHOPHYSIOLOGY
REDUCED ORGAN PERFUSION SECONDARY TO VASOSPASM AND ENDOTHELIAL ACTIVATION.

INCREASE IN BP R/T INCREASE IN PERIPHERAL RESISTANCE

NO KNOWN ETIOLOGY

WOMAN'S UTERUS DOES NO REMODEL ITSELF LIKE IT SHOULD. ARTERIES REMAIN THICK CAUSING DECREASED PLACENTAL PERFUSION.

UTEROPLACENTAL PERFUSION CAN BE DECREASED 50% BEFORE ONSET OF S/S

POOR ORGAN PERFUSION = INCREASED PERIPHERAL RESISTANCE = INCREASED BP = INCREASE IN FAT DEPOSITS ON LIVER = EPIGASTRIC PAIN = LIVER DAMAGE = HELLP SYNDROME = LOW GFR = OLIGURIA = PROTEINURIA

COAGULATION SYSTEM IS ACTIVATED AND THROMBOCYTOPENIA OCCURS = CLOTTING CASCADE = PLATELET COUNT BELOW 100,000
MILD PREECLAMPSIA
SYSTOLIC BP 140-160
DIASTOLIC BP <100
PROTEINURIA-TRACE TO 1+
SERUM CREATININE - NORMAL
SEVERE PREECLAMPSIA
SYSTOLIC - >160
DIASTOLIC 110 OR >
PROTEINURIA - 2+ OR >
HA
VISUAL CHANGES
UPPER ABD PAIN
OLIGURIA
SEIZURES - POSSIBLE
ELEVATED SERUM CREATININE

ELEVATED LIVER ENZYMES

FETAL GROWTH RESTRICTION (R/T DECREASED BLOOD FLOW)

POSSIBLE PULMONARY EDEMA
MAG
CNS DEPRESSANT PROVEN TO DECREASE SEIZURE ACTIVITY WITHOUT LONG TERM EFFECTS TO WOMAN AND FETUS

TOCOLYTIC

PREVENT PRETERM LABOR

STOPS PT FROM CONTRACTING

DECREASED BP SO PT WONT STROKE OUT ON YOU
MAG LOADING DOSE
4-6g DILUTED IN 100 ML IVF OVER 15-20 MIN
MAG RN ACTIONS
PT WILL BE HOT.
AC
NO BRIGHT LIGHTS
EYE PATCHES
NO VISITORS
NO NOISE
MAG ASSESS FOR
HA, VISUAL CHANGES, HYPOREFLEXIA, DTRs AND CLONUS, DOCUMENT DAILY WEIGHT, ASSESS FOR EDEMA, ASSESS FOR FLUID RETENTION
MAG RN INTERVENTIONS
ASSESS VS BEFORE ADMIN
VS Q 5-15 MIN WHEN LOADING
THEN Q 30-60 MIN UNTIL PT STABILIZES

ASSESS FOR DTRs Q 2H

STRICT I&O

ASSESS S/S OF TOXICITY
MAG S/S OF TOXICITY
DECREASED REFLEXES=PENDING RESPIRATORY DEPRESSION

LESS THAN 14 BREATHS PER MINUTE

OLIGURIA = OU < 30 ML/HR

SOB OR RR < 24

CHEST PAIN
EKG CHANGES
ABSENCE OF PATELLAR DEEP TENDON REFLEXES
DECREASED LOC
CARDIAC DYSRYTHMIAS



MAINTAIN SEIZURE PRECAUTIONS, KEEP RESUSCITATION EQUIPMENT BEDSIDE
CONTINUOUS FHR MONITORING
MAG CONTINUOUS INFUSION
MAINTENANCE IS _____g/hr in ______mL IVF

MEASURE SERUM LEVELS AT ____ TO ______ hr

IV INFUSION SHOULD CONTINUE FOR ____hr POST DELIVERY
2g/HR IN 100ML IVF FOR MAINTENANCE.

MEASURE SERUM LEVELS AT 4-6HR . IV INFUSION SHOULD CONTINUE FOR 24 H POST DELIVERY

USE INFUSION PUMP TO MAINTAIN RATE
MAG PT WILL FEEL
HOT
FLUSHED
SEDATED
MAG ANTIDOTE
CALCIUM GLUCONATE OR CALCIUM CHLORIDE

5-10mEq GIVEN IV SLOWLY OVER 5-10 MIN
MAG TOXICITY SUSPECTED
STOP INFUSION
ADMIN ANTIDOTE
PREVENT CARDIAC ARREST
WHICH OF THE FOLLOWING IS AN INDICATION TO TURN OFF MAG IN A WOMAN WITH PREECLAMPSIA?
BP 190/110
N/V
EPIGASTRIC PAIN
RESPIRATORY RATE 13 BREATHS PER MINUTE
RESPIRATORY RATE OF 13 BPM
THE GOAL OF MAG THERAPY IN TREATING PREECLAMPSIA IS TO
PREVENT SEIZURES
MAG EXPECTED SE

RESPIRATORY DISTRESS CAN OCCUR WITH LEVELS > ______ mg/ dL
FLUSHING
DROWSINESS
HA
LETHARGY
N/V

RESPIRATORY DEPRESSION CAN OCCUR WITH SERUM LEVELS ABOVE 9mg/dL
MAG THERAPEUTIC LEVEL
4-7mEq/ L
ECLAMPSIA
TRIGGERED BY:
CEREBRAL -
VASOSPASM
HEMORRHAGE
ISCHEMIA
EDEMA
ECLAMPSIA WARNING SIGNS
SEVERE AND PERSISTANT HA
EPIGASTRIC PAIN
N/V
HYPERREFLEXIA WITH CLONUS
RESTLESSNESS
THESE WILL PUT YOU AT RISK FOR DEVELOPING PLACENTA PREVIA
COCAINE
SMOKER
HTN
DM

ENDOMETRIAL SCARRING:
PREVIOUS PLACENTA PREVIA
PRIOR C-SECTION
ABORTION
MULTIPARITY -(twins triplets...)

IMPEDED ENDOMETRIAL VASCULARIZATION:
ADVANCED MATERNAL AGE
UTERINE ABNORMALITIES SUCH AS FIBROIDS, ENDOMETRITIS

INCREASED PLACENTAL MASS:
LARGE PLACENTA
MULTIPLE GESTATION
WHAT IS PLACENTA PREVIA?

1 OUT OF ______ DELIVERIES

_______ ARE CONTRAINDICATED
1/300 DELIVERIES
OCCURS WHEN PLACENTA ATTACHES TO LOWER UTERINE SEGMENT OF UTERUS, NEAR OR ON CERVICAL OS

BLEEDING OCCURS R/T PLACENTAL SEPARATION FROM INTERNAL CERVICAL OS OR LOWER UTERINE SEGMENT AND THE INABILITY OF THE UTERUS TO CONTRACT AT VESSEL SITES

VAG EXAMS ARE CONTRAINDICATED
PLACENTAL PREVIA CAUSES
MATERNAL BLOOD LOSS WHICH RESULTS IN DECREASED OXYGEN CARRYING CAPACITY THAT DIRECTLY IMPACTS OXYGEN DELIVERY TO MATERNAL ORGANS

PLACENTAL BLOOD FLOW IS DECREASED = DECREASED OXYGEN TO FETUS
PLACENTA PREVIA PUT WOMEN AT RISK FOR:
HEMORRHAGIC / HYPOVOLEMIC SHOCK R/T EXESSIVE BLOOD LOSS

UNRESOLVED BLEEDING AT TERM CAUSES EXANGUIATIONSIN IN 10 MIN

ANEMIA
POTENTIAL Rh SENSITIZATION AS Rh-neg WOMEN CAN BECOME SENSITIZED DURING ANY ANTEPARTUM BLEEDING EPISODE
PLACENTA PREVIA
NEWBORN RISKS
placenta previa:

UTEROPLACENTAL BLOOD FLOW DISRUPTION CAUSES FETAL STATUS TO DECLINE

FETAL COMPROMISE
BLOOD LOSS, HYPOXIA, ANOXIA, DEATH, FETAL ANEMIA, NEONATAL MORBIDITY R/T PRETERM BIRTH
PLACENTA PREVIA ASSESSMENT FINDINGS

PAINLESS ________
FIRST BLEEDING EPISODE RARELY _________

BLEEDING OCCURS NEAR END OF _______ TRIMESTER OR INTO ________ TRIMESTER
NO ________!!
PAINLESS BLEEDING
BLEEDING NEAR END OF 2ND TRIMESTER OR 3RD TRIMESTER.

FIRST BLEEDING EPISODE RARELY SERIOUS

NO VAG EXAMS!!!!
PLACENTA PREVIA
ABSOLUTELY NO
VAG EXAMS
PLACENTA PREVIA EMERGENCY MEDICAL MANAGEMENT
C SECTION WHEN MOM OR BABY IS COMPROMISED

C SEC PRETTY MUCH WITH ALL WOMEN BECAUSE PLACENTA IS AT OR NEAR CERVIX, AND CERVICAL DILATION CAUSES PLACENTAL HEMORRHAGE

VAG DELIVERY MAY BE ATTEMPTED IS PLACENTA IS LOW LYING AND ONE CAN PROCEED WITH C SECTION IF NECESSARY

BLOOD IS TRANSFUSED AS NEEDED

IF WOMAN AND FETUS ARE STABLE AND BLEEDING STOPS, DC HOME WITH BEDREST, ANTENATAL SURVEILANCE, AND CLOSE PROXIMITY TO HOSPITAL
ABRUPTIO PLACENTA ASSESSMENT
HYPOVOLEMIC SHOCK
HYPOTENSION
OLIGURIA
THREADY PULSE
SHALLOW IRREGULAR BREATHS
PALLOR
ANXIETY
POSSIBLE VAG BLEEDING
SEVERE ABD PAIN
UTERINE CONTRACTIONS
UTERINE TENDERNESS
RIGIDITY WHERE BLOOD IS
HYPERTONUS, INCREASE UTERINE DISTENTION
N/V
DECREASED UO

SHOCK S/S OCCUR WHEN 25-30% BLOOD LOSS HAS OCCURRED

KB TEST IN MOM'S BLOOD BLOOD COULD SHOW FETAL BLOOD RBCs
WHAT IS ABRUPTIO PLACENTA?

CAN BE CAUSED BY WHAT DRUG? _______
CAN BE CAUSED BY COCAINE

SEPARATION OF PLACENTA FROM IMPLANT SITE AFTER 20WKS GEST AND BEFORE DELIVERY

CAN BE PARTIAL OR TOTAL

GRADES 1-3

BLEEDING INTO DECIDUA BASALIS CAUSES HEMORRHAGE AND PLACENTAL SEPARATION
ABRUPTIO PLACENTA:
FETAL ASSESSMENT FINDINGS

_______ OR _________

CHANGE IN _______

LATE _______

DECREASED ____________
TACHYCARDIA OR BRADYCARDIA

CHANGE IN FHR

LATE DECELERATIONS

DECREASED BASELINE
ABRUPTIO PLACENTA ASSESSMENT FROM LECTURE
FOR VAG BLEED, OBSERVE FOR DURATION
AMOUNT
COLOR
CHARACTERISTIC OF BLEED
VS
PAIN
FHR
EMOTIONAL RESPONSE
ABRUPTIO PLACENTA
CLASSIC S/S

SEVERE AND SUDDEN ONSET OF ______ ______
UTERINE ______ AND _________
POSSIBLE ________
SEVERE AND SUDDEN ONSET ABD PAIN
UTERINE CONTRCTIONS
UTERINE TENDERNESS
POSSIBLE VAG BLEEDING

IF BLOOD IS TRAPPED IN CENTER OF PLACENTA, NO BLEEDING SEEN. 10% OF ABRUPTIONS

IS SEPARATION OCCURS AT EDGE OF PLACENTA, BLOOD WILL BE SEEN.
THESE WILL PUT YOU AT RISK FOR DEVELOPING ABRUPTIO PLACENTA
COCAINE USE
PREVIOUS ABRUPTION
HTN DISORDERS OF PREGNANCY
ABD TRAUMA
METH USE
SMOKING
PREMATURE MEMBRANE RUPTURE
UTERINE ANOMALIES/FIBROIDS
A WOMAN WITH ABRUPTIO PLACENTA WILL HAVE THE FOLLOWING RISK FACTORS
HEMORRHAGIC STROKE
DIC
HYPOXIC ORGAN DAMAGE ESP KIDNEYS AND LIVER
POST PARTUM HEMORRHAGE
ABRUPTIO PLACENTA

RISKS FOR THE BABY

OBVIOUS - MOM IS IN SERIOUS CONDITION, BABY IS AT RISK FOR_________

MOM IS BLEEDING, BABY AT RISK FOR
________
________
________ INJURY
FETAL _________
__________
BABY AT RISK FOR PRETERM BIRTH
HYPOXIA
ANOXIA
NUEROLOGICAL INJURY, FETAL DEATH
IUGR
15% RATE OF NEONATAL DEATH
ABRUPTIO PLACENTA EMERGENCY MANAGEMENT

TYPE OF BIRTH_________
MONITOR MOM'S _______ ________
RESTORE ________ ________
MONITOR AND CORRECT _________ ________
C SECTION

MONITORING MATERNAL BLOOD STATUS
RESTORING BLOOD LOSS
MONITORING COAGULATION DEFECTS
CORRECTING COAGULATION DEFECTS
ABRUPTIO PLACENTA:

IF MATERNAL STATUS IS STABLE AND FETUS IS IMMATURE...
HOSPITALIZATION
CLOSE MONITORING OF FHR AND STATUS
CORTICOSTEROIDS MAY BE GIVEN TO ACCELERATE FETAL LUNG MATURITY
ABRUPTIO PLACENTE NURSING ACTIONS
MONITOR FOR VAG BLEEDING
ASSESS ABD PAIN
PALPATE UTERUS FOR CONTRACTIONS, TENDERNESS, HYPERTONUS, INCREASED UTERINE DISTENTION
MANAGE N/V
ASSESS FOR DECREASED UO
MONITOR MOM FOR DECREASED BP AND TACHYCARDIA
MAINTAIN IV ACCESS WITH LARGE BORE NEEDLE FOR EMERGENCY BLOOD TRANSFUSIONS
ADMIN 02 VIA FM @ 8-10L
ASSESS FHR FOR BASELINE CHANGES
MONITOR LAB FINDINGS AND CBC AND CLOTTING CASCADE
PROVIDE EMOTIONAL SUPPORT
PROVIDE INFORMATION TO MOTHER AND FAMILY REGARDING TX PLAN AND INFANT STATUS
DISTURBED SLEEP PATTERNS DURING PREGNANCY
INSOMNIA LASTS THROUGHOUT PREGNANCY.
IMPLEMENT SLEEP HYGIENE MEASURES LIKE REGULAR BEDTIME, RELAXING ACTIVITIES BEFORE BEDTIME
COMFORTABLE SLEEP ENVIRONMENT WITH BODY PILLOW, EXTRA PILLOWS, TEACH BREATHING EXERCISES AND RELAXATION TECHNIQUES, EFFLEURAGE IS A MASSAGE TECHNIQUE VERY LIGHT TOUCH WITH 2 FINGERS IN 2 REPETITIVE CIRCULAR PATTERNS OVER GRAVID OF ABD., WARM BATH, WARM BEVERAGE PRE BEDTIME.
CHOLELITHIASIS IN PREGNANCY
GALLSTONES: PROGESTERONE INDUCED RELAXATION OF SMOOTH MUSCLE RESULTS IN DISTENTION OF GALL BLADDER AND SOWS EMPTYING OF BILE. BILE STASIS AD ELVATED CHOLESTEROL LEVELS CAUSE GALLSTONES. 8% OF ALL PREGOS WILL GET GALL STONES.
CBC
RBC INCREASED BY 30%
RBC VOLUME INCREASE BY 17-30%
WBC INCREASSE
Hg and Hct DECREASED R/T IRON DEF ANEMIA THAT MOTHER IS MOST LIKELY DEVELOPING DUE TO INCREASED NEED FOR IRON FROM THE FETUS
INCREASED FIBRINOGEN AND INCREASED PLASMA FIBRIN = HYPERCOAGULATION.
BLOOD VOLUME DURING PREGNANCY
BV INCREASES 40-50% OR BY 1500 ML
GOOD WEIGHT GAIN DURING PREGNANCY
25-35lbs
PURPOSE OF PRECONCEPTION CARE IS
TO IMPROVE PERINATAL OUTCOMES
PRESUMPTIVE SIGNS OF PREGNANCY ARE PHYSIOLOGIC CHANGES FELT/PERCEIVED ONLY BY _______
PHYSIOLOGIC CHANGES PERCIEVED BY WOMAN
PHYSIOLOGIC CHANGES IN PREGNANCY
ARE PROTECTIVE OF THE WOMAN AND FETUS
INTIMATE PARTNER VIOLENCE
CROSS ALL LEVELS AND NUMBER 1 COD
RHOGAM WOULD BE ADMIN AT 28 WEEKS GEST TO WHICH WOMAN?
BLOOD TYPE 0-
BLOOD VOLUME INCREASES DURING PREGNANCY BY
1500ML
10 WEEKS GESTATION WITH N/V TELL HER TO
EAT SMALL FREQUENT MEALS
PRESUMPTIVE SIGNS OF PREGO
PERCEIVED BY THE WOMAN HERSELF.
INCREASED URINATION
INCREASED URINATION FREQUENCY
AMENORRHEA, N/V, BREAST CHANGES, FATIGUE AND QUICKENING - movement of fetus in utero
HARMFUL EFFECTS OF SMOKING
nicotine causes _________
smoking causes _______ and _________
NICOTINE CAUSES REDUCED UTERINE BLOOD FLOW

SMOKING CAUSES LBW AND PREMATURITY

PHYSIOLOGICAL EFFECTS OF SMOKING ARE A RESULT OF TRANSIENT INTRAUTERINE HYPOXEMIA.

CIGARETTE SMOKE HAS MANY CHEMICALS LIKE NICOTINE AND CARBON MONOXIDE. THESE CAUSE ADVERSE PREGNANCY OUTCOMES LIKE LBW AND PREMATURITY.

CARBON MONOXIDE BINDS TO HEMOGLOBIN REDUCING O2 CARRYING CAPACITY OF THE BLOOD.
HARMFUL EFFECTS OF SUBSTANCE ABUSE
ALCOHOL


________ AND _______ BIRTH DEFECTS
_____% WILL DRINK WHILE PREGNANT
MOST COMMON _____
__________ BORN A YEAR WITH FASD
13% WILL DRINK WHILE PREGO.
MOST COMMON TERATOGEN
PASSES TO FETUS EASILY
BABY'S LIVER PROCESSES IT SLOWER, SO BABY ALCOHOL LEVELS REMAIN ELEVATED LONGER.
PHYSICAL AND MENTAL BIRTH DEFECTS
FETAL ALCOHOL SPECTRUM DISORDERS (FASD) DESCRIBES EFFECTS OF ALCOHOL EXPOSURE PRIOR TO BIRTH.
40,000 BABIES/YR BORN WITH FASD.
HARMFUL EFFECTS OF SUBSTANCE ABUSE:
ILLICIT DRUGS

COCAINE AND HEROIN

COCAINE CAUSES 2 THINGS
5.2% WILL USE DRUGS
MJ
COCAINE
AMPHETAMINES
HEROIN
ECSTACY

COCAINE CAUSES VASOCONSTRICTION THAT CAN IMPACT UTERUS AN PLACENTA CAUSING ABRUPTION OF PLACENTA OR PRETERM BIRTH

HEROIN - METHADONE TX IS RECOMMENDED TO PREVENT STILLBIRTH
ILLICIT DRUGS CAN CAUSE THE FOLLOWING FOR THE BABY

6 THINGS
STILLBIRTH
LBW
PRETERM BIRTH
IUGR
NEONATAL WITHDRAW SYNDROME
SIDS
ILLICT DRUG USE CAN CAUSE THE FOLLOWING FOR THE MOTHER

***DONT WORRY ABOUT THIS ONE SO MUCH***
PRETERM LABOR
PPROM
POOR WT GAIN
POOR NUTRITIONAL STATUS
PLACENTAL ABNORMALITIES:
PLACENTA PREVIA, ABRUPTIO PLACENTA
SYMPTOMS OF 1ST TRIMESTER
FATIGUE
MORNING SICKNESS
EAT SMALL FREQUENT MEALS LOW IN FAT AND EASY TO DIGEST
TENDER SWOLLEN BREASTS
INCREASED UO ESP AT NOC
DIZZINESS
HEARTBURN
CONSTIPATION
FOOD CRAVINGS
FOOD AVERSIONS
IUGR
A DECREASED RATE OF FETAL GROWTHE USUALLY DUE TO A DECREASE IN CELL PRODUCTION RELATED TO CHRONIC MALNUTRITION

2 TYPES:
1) SYMMETRIC AND ASYMETRIC

WOMAN MAY LOOK OR MEASURE SMALL FOR GEST AGE
SYMMETRIC IUGR
GENERALIZED PROPORTIONAL REDUCTION IN THE SIZE OF ALL STRUCTURES AND ORGANS EXCEPT FOR BRAIN AND HEART.
CAUSED BY TERATOGENS, CONGENITAL INFECTIONS, GENETIC PROBLEMS.
ASYMMETRIC IUGR

CAN BE CAUSED BY MATERNAL OR PLACENTAL CONDITIONS THAT IMPEDE BLOOD FLOW

CAN BE CAUSED BY 3 THINGS. 2 ARE PLACENTA RELATED 1 IS MOTHER RELATED.
DISPROPORTIONAL REDUCTION IN ORGANS AND STRUCTURES.
CAUSED BY MATERNAL OR PLACENTAL CONDITIONS THAT OCCUR LATER IN PREGNANCY AND IMPEDE PLACENTAL BLOOD FLOW.

CAN BE CAUSED BY PREECLAMPSIA, PLACENTAL INFARCTS, SEVERE MATERNAL MALNUTRITION
NAEGELE'S RULE
STANDARD FORMULA FOR DETERMINING EDD BASED ON LMP.

FIRST DAY OF LAST MENSTRUAL PERIOD -3 MONTHS + 7 DAYS

LMP APRIL 27
- 3 MONTHS
-------------------------
JANUARY 27
+ 7 DAYS
------------------------
FEB 3
2 FACTORS INFLUENCE NAEGELE'S RULE
REGULARITY OF WOMAN'S MENSES

LENGTH OF WOMANS MENSES
MOST WOMEN GIVE BIRTH WITHIN THE TIME PERIOD OF
3 WEEKS BEFORE TO 2 WEEKS AFTER EDD
WINDOW FOR TERM GEST IS
5 WEEKS FROM 37 WEEKS TO 42 WEEKS GEST.
NORMAL 3RD TRIMESTER FINDINGS
NORMAL GROWTH AND DEVELOPMENT OF FETUS
BACK PAIN
BIG BOOBS
REALLY TIRED
BRAXTON HICKS CONTRACTIONS
DISCHARGE
FREQUENT URINATION
HEARTBURN
CONSTIPATION
SOB
VARICOSE VEINS/SPIDER VEINS
HEMORRHOIDS
39 WEEK PRIMIGRAVIDA
FIRST TIME PREGNANCY
IS CONSIDERED FULL TERM
NUTRITIONAL COUNSELING FOR HIGH PRIORITY PATIENTS
EAT FRESH FRUITS, VEGGIES, LEAN PROTEIN, LOW FAT DAIRY, WHOLE GRAINS, SMALL AMOUNTS OF HEALTHY FATS.
3RD TRIMESTER
FOCUSED ASSESSMENT INCLUDES ALL ASPECTS OF 2ND TRIMESTER AND A PELVIC EXAM TO ID CERVICAL CHANGES.
3RD TRIMESTER ASSESSMENT OF FETAL WELL-BEING
AUSCULTATION OF FHR
RECORD WOMAN'S ASSESSMENT OF KICK COUNTS
LEOPOLDS TO DETERMINE FETAL POSITION

GROUP B STREP SCREENING

1 HR GLUCOSE TEST AT 24-28 WEEKS

CBC
3RD TRIMESTER FETAL GROWTH
____ TO ___ INCHES LOING

___ TO _____ LBS

HAS _____ FAT

HAS ESTABLISHED _____ AND _____ CYCLES
17-20" LONG
6-8 LBS
SUB Q FAT
ESTABLISHED SLEEP AND ACTIVITY CYCLES
UNDERWEIGHT
BMI < 19.8 28-40 LB
PERFECT WEGITH
BMI 19.8- 26 25-35 LBS WEIGHT GAIN
OVER WEIGHT
BMI 26.1 - 29 15-25 LB WEIGHT GAIN
OBESE
BMI > 29 GREATER THAN 15 LB WEIGHT GAIN
PRENATAL NUTRITIONAL RECOMMENDATIONS
FRUITS
2 CUPS

AVOID RAW UNPASTEURIZED JUICES
PRENATAL NUTRITIONAL RECOMMENDATIONS
VEGGIES
2 1/2 CUPS

3 CUPS DURING 2ND AND 3RD TRIMESTERS

VEGGIES THAT ARE DARK AND HAVE RICH COLORS ARE MORE NUTRITIOUS

AVOID RAW SPROUTS
PRENATAL NUTRITIONAL RECOMMENDATIONS
LOW FAT/NON FAT DAIRY PRODUCTS
3 SERVINGS

1 1/2 OZ OF CHEESE, 1 CUP MILK OR YOGURT

AVOID RAW AND UNPASTUERIZED PRODUCTS

AVOID SOFT CHEESES
PRENATAL NUTRITIONAL RECOMMENDATIONS
MEATS
POULTRY
FISH, EGGS, DRY BEANS
NUTS
3 (2 OZ) SERVINGS

SELECT LEAN AND LOW FAT SOURCES

LIMIT FISH AND SHELLFISH TO 12 OZ / WEEK AND AVOID LARE FISH WITH HIGH MERCURY CONTENT (SWORDFISH, TILEFISH, SHARK, KING MACERAL)

REHEAT LEFTOVERS AND READY TO EAT FOODS

MAY REHEAT OR AVOID DELI MEATS
VEGANS/ VEGITARIANS
MONITOR MICRO NUTRIENTS LIKE B12, CALCIUM, IRON, ZINC, CALORIES
PRENATAL NUTRITIONAL RECOMMENDATIONS
GRAINS
6 (1 OZ) SERVINGS

7 OZ SECOND TRIMESTER

8 OZ 3RD

SELECT WHOLE GRAIN PRODUCTS FOR AT LEAST 1/2 OF THE SERVINGS
HIGH VIT C FOODS
ORANGES
OJ
KIWI
BROCCOLI
PAPAYA
GRAPEFRUIT
TOMATOES
CELERY
SPINACH
PINEAPPLE
CRANBERRIES
RASBERRIES
STRAWBERRIES
YOU ARE PREGNANT. CONGRATULATIONS?
BEHIND THIS CARD ARE _____ SIGNS AND SYMPTOMS RELATED TO BEING PREGNANT.
OBJECTIVE S/S OF PREGNANCY THAT CAN BE ATTRIBUTED TO FETUS

AUSCULTATION OF FETAL HEART BY 10-12 WEEKS

OBSERVATION AND PALPATION OF FETAL MOVEMENT BY EXAMINER

SONOGRAPHIC VISUALIZATION OF FETUS

CARDIAC MOVEMENT NOTED AROUND WEEK 4-8 VIA SONOGRAPHIC VISUALIZATION
NUTRITION
FRESH FRUITS
VEGGIES
LEAN PROTEIN
LOW FAT/NON FAT DAIRY
NO SOFT CHEESES
WHOLE GRAINS
SMALL AMOUNTS OF HEALTHY FATS
PRENATAL VIT
FOLIC ACID
CALCIUM
MAG
VIT D
IRON
PRESUMPTIVE SIGNS OF PREGNANCY
SUBJECTIVE SIGNS LIKE PHYSIOLOGIC CHANGES PERCEIVED BY THE WOMAN HERSELF

AMENORRHEA - NO PERIOD
N/V FROM WEEK 2 TO 12

BIG TENDER TINGLING BOOBS

INCREASED BREAST VASCULARITY

FATIGUE

INCREASED UO

QUICKENING
FUNDAL HEGHTS

MID PREGNANCY
FUNDUS REACHES UMBILICUS
UTERUS BEFORE PREGO
SMALL PEAR
3" BY 2" BY 1"
DRINK
8-10 GLASSES H20 / DAY
RECOMMENDED WEIGHT GAIN LESS THAN ____ LBS DURING 1ST TRIMESTER
5 LBS
FUNDAL HEIGHTS

END OF PREGNANCY / 36 WEEKS
FUNDUS REACHES XIPHOID PROCESS
FUNDAL HEIGHT

2ND TRIMESTER

(WEEKS 18-30 / 2ND AND 3RD TRIMESTERS)
FUNDAL HEIGHT SHOULD EQUAL WEEKS OF GESTATION (IN CENTIMETER, GIVE OR TAKE 2CM)
FUNDAL HEIGHTS

20-22 WEEKS
UMBILICUS
FUNDAL HEIGHTS

16 WEEKS
HALFWAY BETWEEN SYMPHYSIS PUBIS AND UMBILICUS
28 WEEK PRENATAL VISIT
FIRST TRIMESTER
28 WEEK PRENATAL VISIT

INITIAL VISIT:

RISK ASSESSMENT
AGE, GRAVIDA/PARA, ADDRESS, RACE/ETHNICITY, RELIGION, MARITAL/ FAMILY STATUS, OCCUPATION, EDUCATION


USED TO DETERMINE RISKS BASED ON SOCIODEMOGRAPHIC CHARACTERISTICS
28 WEEKS INITIAL VISIT
HEALTH AND RISK ASSESSMENT

PREGO HX

COMPLETE PHYSICAL AND PELVIC EXAM

EDD WILL BE DETERMINED

NUTRITION ASSESSMENT WITH 24HR DIET RECALL

PSYCHOSOCIAL ASSESSMENT

ASSESSMENT FOR PARTNER VIOLENCE
28 WEEKS INITIAL VISIT PROCEDURE/ LABS
WBC
CBC
RH
BLOOD TYPE
ANTIBODY SCREEN
Hg, Hct
PLATELET COUNT
RPR, VDRL - SYPHILIS
HIV / HEP B SCREEN
GENETIC COUNSELING BASED ON FAMILY HX
RUBELLA TITER
PPD
UA
URINE CULTURE SENSITIVITY
PAP SMEAR
GONORRHEA AND CHLAMYDIA CULTURES
ULTRASOUND INTRAVAGINAL
28 WEEKS PRENATAL VISIT (STUDY GUIDE)
HX AND PHYSICAL ASSESSMENT

CHART REVIEW, INTERVAL HX, FOCUSED PHYSICAL ASSESSMENT, VS, GLUCOSE DIPSTICK, ALBUMINM KETONES, WEIGHT, FUNDAL HEIGHT, FHR, FETAL MOVEMENT, LEOPOLDS, EDEMA

STERILE VAG EXAM / PELVIC EXAM

NUTRITIONAL FOLLOW UP

RHOGAM GIVEN FIRST AT 28 WEEKS TO RH - WOMEN

1 HR GLUCOSE TEST (24-28 WEEKS)

TEST FOR GRAM + STREP AND PREPARE FOR POSSIBLE TX
ASSESSING FOR DOMESTIC VIOLENCE
ACTUAL OR THREATENED VIOLENCE
HOMICIDE IS THE KILLER!
HOMICIDE COMMITTED BY PARTNER

ASSESS FOR:
WITHIN LAST YR HAVE YOU BEEN HIT, SLAPPED, KICKED OR HURT? SINCE YOU HAVE BECOME PREGO?
WITHIN LAST YR HAS SOMEONE RAPED YOU?
SUPINE HYPOTENSION
HYPOTENSIVE CONDITION THAT OCCURS WHEN MID TO LATE PREGO WOMAN LIES ON HER BACK. UTERUS COMPRESSES INFERIOR VENA CAVA CAUSING DECREASE IN CO AND BP CAUSING DIZZY ANS FAINTNESS. TEACH WOMAN TO LIE ON HER SIDE AND RISE SLOWLY.
GRAVIDA
# OF TIMES A WOMAN HAS BEEN PREGO INCLUDING CURRENT PREGNANCY
PARA
ANY BIRTH THAT OCCURED AFTER 20 WEEKS GEST.
REGARDLESS DEAD OR ALIVE

WITHOUT REGARD TO # OF FETUSES (TWINS IS 1 BIRTH, 1 PARA)
ABORTION
PREGNANCY THAT ENDED BEFORE 20 WEEKS GEST WHETHER NATURAL OR MEDICAL
NULLIGRAVIDA
WOMAN WHO HAS NEVER GIVEN BIRTH

OR

NEVER BEEN PREGNANT
PRIMIGRAVIDA
WOMAN WHO IS PREGNANT FOR FIRST TIME
MULTIGRAVIDA
NOT PREGNANT FOR FIRST TIME. THIS IS AT LEAST HER SECOND BIRTH
IPV SCREENING
ACTUAL OR THREATENED VIOLENCE
PSYCHOLOGICAL
EMOTIONAL
PHYSICAL
SEXUAL

16% OR 1/6 WOMEN REPORT ABUSE DURING PREGNANCY
IPV SCREENING

3 TOOLS
WHEN IS THE LAST TIME U WERE HIT SLAPPED OR HURT OR KICKED?
ABCDE'S
ALONE
BELIEF
CONFIDENTIALITY
DOCUMENTATION
EDUCATION
SAFETY
ATTACHMENT
New born family attachment
An important goal during the fourth stage
Promoted by allowing early contact with newborn
Encouragement of eye contact and touch
Allowing time to hold the newborn
Positive material bonding behaviors include making eye contact
Touching and talking to baby
And other positive behaviors such as smiling and cuddling the newborn
This is the best time to institute breastfeeding
The newborn may remain in labor and deleivery room with the family for all of the immediate recovery period.
terbutaline
DECREASES THE RISK OF PRETERM BIRTH BY PROLONGING PREGNANCY

DELAYS DELIVERY FOR 3 DAYS

GIVEN IV OR SQ

MAX DOSE 0.08mg/min

MAX DOSE SQ 0.25 mg Q3-4hrs
TERBUTALINE

MATERNAL SE
TACHYCARDIA
CARDIAC ARRYTHMIA
MYOCARDIAL ISCHEMIA
PULMONARY EDEMA

INCREASED MATERNAL GLUCOSE AND HYPOKALEMIA
TERBUTALINE RN CONSIDERATIONS

MONITOR _____ AND _____
MONITOR ____ FOR FLUID OVERLOAD
WATCH FOR ___________ ________
HOLD MEDICATION IF MOM'S HR > ______
WATCH MOM'S ______ AND _____ (COMMON SIDE EFFECT OF THIS MEDICATION)
MONITOR FHR, UCs,
MONITOR I&O FOR FLUID OVERLOAD
WATCH FOR PULMONARY EDEMA
MONITOR MOM'S HR AND HOLD IF HR > 120
WATCH MOM'S GLUCOSE AND POTASSIUM LEVELS
TERBUTALINE IS A _________

CONTRAINDICATED FOR MOM'S WITH _______ DISEASE
BETA ADRENERGIC AGONIST THAT SUPPRESSES UTERINE ACTIVITY

CONTRAINDICATED FOR MOM'S WITH CARDIAC DISEASE
BETA AGONIST TOCOLYTICS
TERBUTALINE AND RITODRINE

SAME EVERYTHING BOTH ARE THE SAME IF YOU KNOW ONE YOU KNOW BOTH
GEST DM
ANY DEGREE OF GLUCOSE INTOLERANCE WITH ONSET OF PREGO. APPLIES WHETHER GDM IS CONTROLLED WITH DIET AND EXERCISE OR INSULIN AS WELL

7% OF PREGNANCIES GET GDM

200,000 / YR
WHY DO SOME WOMEN GET GDM?
PREGNANCY IS A CONDITION CHARACTERIZED BY PROGRESSIVE INSULIN RESISTANCE THAT BEGINS MID PREGO AND PROGRESSES THROUGHOUT PREGO.
2 MAIN CONTRIBUTORS TO GDM / INSULIN RESISTANCE
INCREASED MATERNAL FAT/ADIPOSITY
INSULIN DESENSITIZING HORMONES MADE BY PLACENTA
GDM : PLACENTA
PLACENTA PRODUCES HCS, CORTISOL, ESTROGEN, AND PROGESTERONE.

HCS STIM PANCREAS TO SECRETE INSULIN IN THE FETUS AND REDUCES UPTAKE OF GLUCOSE IN THE MOM. AS PLACENTA GETS BIGGER, HCS IS MORE EFFICIENT AND CAUSES A PROGRESSIVE INSULIN RESISTANT STATE.
ROUTINE SCREENING FOR GDM DURING WEEKS ___ TO ____
24 TO 28
1 HR NON FASTING ORAL GLUCOSE TOLERANCE TEST +
130 OR 140
+ ORAL GLUCOSE TEST
DO A 3 HR GLUCOSE TOLERANCE TEST TESTING EACH HR AFTER WOMAN HAS INGESTED 100 GRAMS OF GLUCOSE
GDM RISKS FOR WOMAN
HYPOGLYCEMIA AND DKA
DEVELOPMENT OF NON GESTATIONAL DIABETES
GDM RISKS FOR THE BABY

HYPO ______ ______ AND ________

________ IS THE MOST COMMON MORBIDITY

MACROSOMIA PLACES BABY AT RISK FOR ______ ________ INJURY

AT RISK FOR ________ AND _______

AT RISK FOR ___________ DISTRESS SYNDROME



CONGENITAL DEFECTS (5) CONGENITAL DEFECTS ARE ONLY FOR BABIES WHOSE MOMS HAD DM BEFORE PREGNANCY. NOT FOR GESTATIONAL DM.
HYPO MAG, HYPO CALCEMIA AND HYPO GLYCEMIA A FEW HOURS AFTER BIRTH

MACROSOMIA IS THE MOST COMMON MORBIDITY

MACROSOMIA PLACES BABY AT RISK FOR BIRTH INJURY -BRACHIAL PLEXUS INJURY

IUGR AND ASPHYXIA

RESPIRATORY DISTRESS SYNDROME
POLYCYTHEMIA
HYPERBILIRUBINEMIA
PREMATURITY

CONGENITAL DEFECTS (HEART, SKELETAL, NEUROLOGICAL, GU, GI)

CARDIOMYOPATHY
STILL BIRTH
MORE ON GDM
WOMEN WITH DEFICIENT INSULIN SECRETORY CAPACITY DEVELOP GDM. MOTHER'S INSULIN DOES NOT CROSS PLACENTA. BABY IS EXPOSED TO MOTHER'S HYPERGLYCEMIA. BABY'S PANCREAS WORKS OVERTIME TO MAKE INSULIN

MORE INSULIN IN BABY MAKES BABY GROW HUGE (MACROSOMIA)
PREGO WOMEN KEEP YO DAMN HbA1C
below 6%
GDM RN ACTIONS
TEST GLUCOSE 4X DAY
1 FASTING AND 3 TWO HR POSTRANDIAL CHECKS A DAY

SUGGESTED GLUCOSE CONTROL FASTING < 95 BEFORE MEALS OR > 140 AT 1 HR AFTER MEALS AND LESS THAN 120 AT 2 HR AFTER MEAL

TEACH TO MONITOR KETONURIA Q AM

TEACH S/S OF DKA, HYPO AND HYPERGLYCEMIA
GDM LECTURE
NO CHEWING GUM OR ANYTHING WHEN GETTING TESTED

BLURRED VISION COMMON PROBLEM AND INCREASES STROKE RISK FOR MOM

WEIGH WITHOUT SHOES AND ASSESS FEET
BIG BABIES
MACROSOMIC BABIES

USUALLY > 4000 GRAMS
USUALLY HAVE DM BY 2 OR 3 YRS OLD

GIVE D10/SUGAR WATER IF HYPOGLYCEMIC AT BIRTH

MAY HAVE CONGENITAL ANOMALIES LIKE HIGH DROPS AND ENLARGED HEART
PIH LABS
ELEVATED SERUM CREATININE 72 mg/dL

Hct LEVELS < 35

LOW PLATELET COUNT 100,000 OR <

ELEVATED LIVER ENZYMES (AST > 41)
GDM MEDS
DIET
EXERCISE
INSULIN (40% OF THE TIME)
CARDIOVASCULAR DISEASE
DURING PREGO CAN BE CATEGORIZE AS CONGENITAL, ACQUIRED, RHEUMATIC, OR ISCHEMIC
CARDIAC DISEASE

RISKS FOR THE BABY
IF MOM'S CIRCULATION IS IMPAIRED - IUGR

FETAL HYPOXIA = CNS DAMAGE

DEATH
CLASS II CARDIAC DISEASE
SLIGHT LIMITATION IN PHYSICAL ACTIVITY

ASYMPTOMATIC

MATERNAL MORTALITY IS < 1%

PLASMA VOLUME INCREASES BY 40-50% WHEN PREGO

CO INCREASES BY 40-50% BY TERM

HR INCREASE BY 20-30% AND STROKE VOLUME INCREASE BY 11-32%

ALL THESE CAN EXACERBATE CARDIAC DISEASE
PT ASSESSMENT
DETERMINE EDD

PRESUMPTIVE AND PROBABLE SIGNS OF PREGNANCY

POSITIVE SIGNS OF PREGNANCY

HYPEREMESIS GRAVIDARUM - FREQUENCY, AMOUNT, CHARACTER OF VOMIT

I&O, SKIN TURGOR, MEMBRANES, PSYCHOSOCIAL ASSESSMENT, FETAL STATUS
Rh STUFF
IF MOM IS Rh (-), NEED TO KNOW DAD'S Rh. IF DAD IS ALSO Rh (-) NO Rh PROBLEM WILL OCCUR. IF DAD IS Rh (+) PROBLEM CAN OCCUR. NEED TO ASSESS MOM FOR ANYTHING IN HER HX THAT MAY HAVE CAUSED SENSITIZATION
AFP SCREENING
alpha fetoprotein

WHEN IS MOM'S BLOOD DRAWN?
WHEN IS AFP PRODUCED IN GESTATION?
WHAT DOES AFP SCREEN FOR? (4 THINGS)
WHAT DOES A LOW AFP LEVEL MEAN?
AFP is produced early in gestation. mom's blood is drawn between 16 and 18 wks. AFP screens for developmental defects like NTD's (neuro tube), anencephaly, omphalocele, gastroschisis. a low level of AFP is a red flag for down syndrome and requires further testing.
AMNIOCENTESIS

USUALLY DONE FOR GENETIC TESTING BETWEEN ____ AND ___ WEEKS GESTATION.

ASSESSES 3 THINGS
assesses lung maturity, hemolytic disease, intrauterine infection. ultrasonography is used to guide needle. usually done for genetic testing between 14-20 wks. less than 1% fetal loss rate if done after 15 weeks gest. increases to 2% - 5% if done when baby is < 15 wks gest.
KICK COUNT
mothers assessment of fetal well-being based on movement. Used to see if baby is getting enough oxygen. to be done once a da for 2 hrs. mom should sit and place both hands on belly to feel movement. 10 kicks in 2 hrs is good.

not 10 kicks?? drink OJ or eat a snack, try again. Call Dr if no change. usually first indicator that something is wrong with baby.
QUAD SCREEN
triple screen or quad screen at 15-20 wks gest screens for neural tube defects and trisomy 21. NOT DIAGNOSTIC. if (+) NEED AMNIOCENTESIS TO DX AND VERIFY.

triple marker screen AFP, hCG, estriol, AND inhibin A (to make it a QUAD) all used to increase detection for trisomy 21. 80% efficient. =)
LOW MATERNAL SERUM ALPHA FETOPROTEIN (MSAFP) AND UNCONJUGATED ESTRIOL =
abnormality
hCG and inhibin A levels are twice as high when ______ is present
trisomy 21
low estriol =
NTD's (neural tube defects)
L/S RATIO RESULTS
lecithin and sphingomyelin are 2 phospholipids that are detected in amniotic fluid.

the ratio between the 2 provide info on surfactant levels.

L/S > 2:1 in a NON DIABETIC WOMAN = lungs are mature! =)

L/S ratio 3:1 in DIABETIC WOMAN = lungs are mature