Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
166 Cards in this Set
- Front
- Back
DJD order
|
index DIP, great toe MTP, knee, index PIP, thumb MCP (yours), LS joint, cervicals
|
|
if not osteoarthritis, Mr. Mennel, then what?
|
joint dysfunction
|
|
Strains become a chronic problem because they heal with
|
inelastic scar tissue
|
|
study of forces and their effects
|
mechanics
|
|
application of mechanical laws to living things
|
biomechanics
|
|
bones from _______, joints from ______
|
levers, hinges
|
|
concept of levers/bones and joints/hinges evolved from
|
Industrial Rev
|
|
continuous changes in the position of an object
|
motion
|
|
human joints have ____ planes of motion
|
6 (3 rotation, 3 translation)
|
|
rotation is ______ around an axis
|
tilt (cartesian)
|
|
translation is
|
glide and slide
|
|
what is most common path of motion?
|
curvilinear - rotation and translation together
|
|
planes
|
sagittal plane: F/E
coronal plane: R/L flexion Transverse or Horizontal plane: R/L axial rotation |
|
what plane is axial rotation
|
transverse or horizontal
|
|
geometry of motion
|
kinematics
|
|
study of visual appearance of motion without accounting for Force
|
kinematics (pretty)
|
|
movement of center of mass includes flight paths, velocities, accelerations, displacements
|
kinematics
|
|
relationship between force and changes it makes in body motion
|
kinetics = force!
|
|
6 Kinematic forces
|
neutral
compression distraction shear lateral bending, torque/counter rotation |
|
which kinematic force is a combination of motion?
|
lateral bending (rotation, lateral flexion)
|
|
Mass x ? =force
|
angular velocity
|
|
MVA at 100,000 deg. per sec is equivalent to ? mph?
|
1400mph
|
|
muscle power is determined by cross section, length/tension ratio, _______ and __________ type.
|
fatigue, fiber
|
|
shortening, creates acceleration, power generation, begins with flexion
|
concentric contraction
|
|
lengthening contraction, deceleration, power absorbed, opens joint
|
Eccentric (evolving, lengthening, opening, power absorbing, slowing down - eccentric circles of thought)
|
|
no change in muscle length but stabilizes (no waver)
|
Isometric contraction
|
|
strongest of all human contractions but also where injuries occur
|
Eccentric (ever widening, lengthening, opening, power absorbing, slowing down eccentric circles of thought are the strongest and therefore an injury is prone to injury)
|
|
what kind of energy enhances force production by 20%
|
Elastic plastic (stretch reflex)
|
|
elastic energy is the free lunch - it's free energy with a ratio of muscle to tendon stretch at
|
20:1
|
|
Great Index finger race demonstrates
|
activation of stretch/elastic plastic reflex
|
|
javelin throwing would be what stretch reflex?
|
rotary! torque! elastic winding and unwinding that is typical of NCAA Div I female basketball player injuries
|
|
3 components of a lever
|
fulcrum (joint), effort (muscle), resistance (weight)
|
|
joint muscle weight is?
|
fulcrum effort resistance
|
|
where is the fulcrum of a 1st class lever
|
between the resistance and effort (weight and muscle) Balanced resistance on one end with weight on other
TRAP ON SKULL |
|
example 1st class lever
|
trapezius on skull
|
|
where is resistance on a 2nd class lever?
|
resistance is between the fulcrum and lever, like a wheel barrow (like a pry bar)
GASTROC contraction |
|
least efficient lever
|
3rd class where there is muscular effort between the weight and fulcrum
BRACHIALIS ON RADIUS |
|
allows for fluid, balanced movements such as gait
|
Dynamic stabilization
|
|
when walking, muscles that allow dynamic stabilization?
|
gluteus medius
ADDuctors (weak glut medius is Trendelenburg gait - compensatory swing) |
|
foot splay in a runner is due to
|
weak dynamic stabilizers (glut med and adductors)
|
|
what maintains transverse stable hip?
|
adductors opposing aBductors
|
|
if the adductors are dominant in hip, what results?
|
lateral tilt, pathologic pronation, coxa varus, genu valgus
|
|
knee hyperextension is called
|
genu recurvatum
|
|
hyperextension of knee is sign of ______________ tracking patella
|
patella
|
|
lateral tracking patella due to knee
|
hyperextension
|
|
how does the ankle move as ankle mortise?
|
dorsiflexion, plantar flexion
|
|
subtalar biomechanics (think of sailboat on water pictures)
|
neutral, dorsi/plantar flex, med/lat tilt, aB/aDDuction,
DEAb PIAd |
|
DEAb
|
Subtalar motion of Dorsiflexion, Eversion, Abduction
|
|
PIAd
|
subtalar motion of Pronation Inversion Adduction
|
|
the ability of a joint to separate or gap
|
joint play/gapping
|
|
the small elastic springiness at the end of passive ROM (at the elastic barrier)
|
End Feel
|
|
in addition to joint play (gap) and end feel (spring), there is a 3rd motion which includes both joint play and end feel?
|
Accessory motion
|
|
total joint movement includes the voluntary _____ plus or minus ?
|
ROM +/- joint play /end feel
|
|
Voluntary movement depends on
|
joint play (gap) & end feel (spring)
|
|
gted.
|
pain!!!
|
|
when specific joint play (gap)/end feel (spring) is restored by manipulation, the pain abates. Test (________) + Impulse (__________)
|
diagnosis + treatment is same as Test + Impulse
|
|
Joint play/end feel can only be restored by
|
a DYNAMIC THRUST given in correct direction (not by a drug)
|
|
what does normal muscle movement depend on?
|
normal joint movement
|
|
what do the repetitive constellation pattern of MFTP's tell you?
|
the joint is not free to move so neither are the muscles that move it, hence trigger points
|
|
You can't restore muscles if the __________ does not have restored motion.
|
joint (concomitant soft tissue component with all subluxations and why you should stretch)
|
|
Negative cascade can cause joint degeneration, but is started by
|
impaired muscle fcn
|
|
how does Mennel define joint dysfunction?
|
as the loss of joint play (gap) or end feel (spring)
|
|
How is the degree of joint dysfunction determined upon clinical exam or xray/video flouroscopy?
|
by comparing the contralateral joint!
|
|
The Mennel Restriction Theory requires the examiner to do 2 things:
|
Test joint play/end feel + treat with Impulse of dynamic thrust in correct direction
|
|
firm but giving end feel, where resistance builds with lengthening like stretching a piece of leather
|
CAPSULAR leather
|
|
firmer than capsular end feel, like knee extension
|
LIGamentous knee
|
|
what two end feels (spring) are basically the same?
|
Capsular and Ligamentous (leather firm but giving resistance builds with length)
|
|
giving, squeezing, painless as in full elbow or knee flexion
|
soft tissue approximation end feel
|
|
hard, non-giving abrupt
Elbow extension |
bony end feel
|
|
firm but giving, builds with elongation, not as stiff as capsular
Hip flexion |
Muscular end feel
|
|
guarded with pain flexion or muscle contraction. Cannot be assessed due to swelling or pain
|
Muscle spasm end feel
- protective splinting is abnormal |
|
a bouncy, springy Tigger like quality
|
Inter-articular end feel
-abnormal is meniscal tear or joint mice |
|
normal end feel resistance is MISSING, and not met at a normal stopping point. Joint will have odd degree of give or deformation.
|
EMPTY end feel
-abnormal is joint injury leading to hypomobility or instability |
|
When does the tibia internally rotate faster than the femur?
|
with EXCESSIVE SUBTALAR JOINT PRONATION, causing dramatic twisting of knee and surrounding soft tissues
|
|
How to re-supinate the foot
|
tighten the Plantar fascia
|
|
failure to re-supinate the foot (get your arch back) causes
|
strains the plantar fascia
|
|
longest PROprioreceptive pathway in the body
|
the foot!
|
|
how many bones in the foot? arches?
|
26 , 4
|
|
regions of foot
|
forefoot, midfoot, hindfoot
|
|
role of the foot is as a mobile __________, rigid +++++.
|
adapter, lever
|
|
the foot has 4 multiaxial movements
|
subtalar/talo-navicular/pronation (DEAb)/supination (PIAd)
|
|
what muscle supports the the medial longitudinal arch?
|
tibialis anterior and posterior
|
|
keystone of medial longitudinal arch
|
head of TALUS
|
|
keystone of Lateral longitudinal arch (and Dr. Ebbets favorite)
|
CUBOID
|
|
keystone of Tarsal Transverse Arch
|
think: TARSAL transverse is cuboid + cuneiforms 1/2/3 so Keystone is 2nd cuneiform and Cornerstone is cuboid
|
|
how is the controversial anterior transverse arch supported?
|
metatarsal pad, since it is the 5 metatarsal heads of the ball of the foot
|
|
Windlass Effect
|
dorsiflexion of great toe leads to elevation of talo/navicular/cuneiform complex (especialy Navicular). Makes foot a rigid lever during heel strike and fixes plantar fascitis in. From Shoes.`
|
|
Describe the CLOSED KINETIC CHAIN of Lower Extremity (7)
|
1-pathologic pronation, 2-IR of tibia & femur, 3-lower femur, 4-eccentric psoas & piriformis stretch, 5-post rot of Ilia (PI), 6-nutation of sacrum w ER of L5tp, 7-reactive scoliosis
|
|
first step in closed kinetic chain of LE
|
1- pathologic pronation
|
|
2nd step in closed kinetic chain of LE
|
2- IR of tibia and femur
|
|
3rd step in closed kinetic chain of LE
|
3- lowering of femur
|
|
4th step in closed kinetic chain of LE
|
4- eccentric STRETCH of psoas and piriformis
|
|
5th step in closed kinetic chain of LE
|
5- posterior rotation of ilia (PI Ilium)
|
|
6th step in closed kinetic chain of LE
|
6- nutation of sacrum (if ilia went post, then sacrum nutates) with external rotation of L5 t.p.
stars- facet imbrication at L3-L4 |
|
7th final step of closed kinetic chain of LE
|
7- reactive scoliosis
|
|
congenital foot condition with loss of arch, DEAb, and calcaneal valgus
|
Pes Planus (flat foot)
|
|
flat foot is also called
|
pes planus
|
|
foot condition of PIN POINT heel pain, greater in morning. Patient will sleep with foot plantar flexed.
|
Plantar Fasciitis
|
|
deformation of the plantar calcaneal navicular ligament (PCN lig) + may come from walking on HARD LEVEL FLOORS
|
Plastic DeFLOORmation
deformation |
|
Where is the calcaneonavicular ligament and when does it suffer?
|
on medial foot plantar surface, suffers from walking on hard level floors
Plastic deFLOORmation |
|
pathologic pronation and crush of RETINACULUM - associated foot condition is claw toe
|
tarsal tunnel syndrome (Tom Dick & Harry)
|
|
plantar flexed metatarsal head, PIP and DIP with dorsiflexed MTP (the one spiking up to the roof to hit the shoe). TTS shuts off innervation to proximal phalanx.
|
Claw Toe (hammer toe) is plantar, dorsi, then plantar flexed with TSS shut-off
|
|
posterior tibialis is the cause, weak feet, do the foot drills to cure
|
shin splints
|
|
what controls mid-foot pronation?
|
eccentric tibialis posterior contraction
|
|
Why is tibialis posterior responsible for shin spints?
|
numerous insertions, Weak T.P. leads to "too fast" eccentric/lengthening which leads to shin splints
|
|
proximal lesion leading to a more distal compression. ART nerve tracts. More common in Upper Extremity.
|
Double Crush syndrome (Kim)
|
|
which Achilles tendon is more frequently ruptured and why?
|
LEFT - due to right side dominance and people pushing off with their left leg
|
|
Achilles tendon injury affects who and what side?
|
men, left (Calliet, '80) because men weigh more and create more force
|
|
where does a rupture of the Achilles occur?
|
2-4 cm proximal to calcaneus because of frequent crimping (Bowstring effect) and insufficient blood supply
|
|
Poor biomechanics due to ______________________ expose the kinetic chain to forces such as abnormal shearing, bending or torque.
|
structural imbalance
|
|
Bowstring effect
|
superior aspect of calcaneus moves lateral to medial to lateral to medial, etc. so Achilles tendon is moving back and forth in the coronal plane
|
|
gait abnormality can be 3:
|
structural change to bone length or shape, pathological change of soft tissue to articular structures, neuromuscular control of gait
|
|
two causes of abnormal gait:
|
pain and proprioceptive impairment
|
|
pain causing abnormal gait compromises _______ movers and patient uses __________ movers as compensation.
|
primary crash, secondary called in
|
|
proprioreceptive impairment causes gait abnormalities by a combination of factors, such as poor ______, _______ weakness/atrophy, _______ injury.
|
rehab, muscular, recurrent
|
|
2 phases of gait
|
stance & swing
|
|
Stance & ________ are the 2 phases of GAIT
|
Swing
|
|
Which is more, stance or swing? How much is it, percentage-wise?
|
Stance 65%
|
|
3 phases of Stance
|
Double support
Single support Double support again |
|
5 components of Stance
|
Heel strike
Flat foot MID-stance Terminal stance Push off! |
|
Heel strike is
|
supination
|
|
Flat foot is
|
pronation
|
|
Mid-stance is
|
pronated, still, from flat foot
|
|
Terminal stance is
|
supinated
|
|
Push off! is
|
supination
|
|
H, ff, MID, T, Po!
|
He'll flat foot mid-day til pissed off!
S P P S S |
|
Stance components: (65%)
He'll flat-foot mid-day til pissed off! |
Heel strike, Flat foot, Mid-stance, Terminal stance, Push off!
S.P.P.S.S. Supinated/Pronated |
|
Swing phase
|
35% - leg is not in contact with ground
|
|
Along with Pain and Proprioreceptive Impairment, what are more causes of abnormal gait?
DIMPP |
Muscular weakness
Impaired control Deformity (Pain and Proprio) |
|
muscular weakness causes abnormal gait by
|
not enough strength to lift body so muscle substitution (2nd mover recruit), ie develops from crutches or a limp
|
|
Impaired control of the body causes abnormal gait - what is damaged?
|
CNS
can't control timing and intensity MS, Cerebral palsy, brain/sc injury |
|
He'll flat-foot Mid-day til pissed off!
|
Heel strike S
Flat foot P MID-stance P Terminal stance S Push off! S |
|
why is push off of stance (65%) considered supinated?
|
Windlass effect of great toe dorsiflexed - rigid lever position
|
|
Normal gait is all the things you imagine (brisk, smooth, purposeful) and?
|
Narrow-based, straight/unwavering, arms swing, trunk sways, heels almost touch in passing, thighs don't touch, 180 turns are smooth, stopping is abrupt/unwavering/sure
|
|
Pain and Proprio, Muscle weak, Impaired CNS and __________ cause abnormal gait.
|
Deformity
|
|
decreased ROM, tissue spasm, contracture and restriction can all cause abnormal gait due to ?
|
deformity
|
|
When is Single support
|
2,5,6
|
|
When is pronation during gait?
|
2,5,6 (any time there is single support)
|
|
During heel strike, foot is supinated but because it is on the way to pronation, what effect is created?
|
Bowstring of Achilles and Internal rotation of tibia (double support)
|
|
During flat foot (pronation/single support), what happens to the swing leg?
|
loses ground contact (single support and internal rotation)
|
|
At Mid-stance (MID-day), the foot is in pronation leading to supination. We know the foot is in pronation at mid-stance because
|
there's only one foot on the ground/single support and all prior single supports are pronated
|
|
During Terminal stance, we begin ___________ and foot supination so we essentially go from double support to single support.
|
toe off = double support to single support/supination
|
|
PUSH OFF! is heel off, and foot supination at
|
toe off.
|
|
STEP FLAWS (acronym)
|
Spastic/Timid/Extrapyramidal/Paretic/Footdrop/Limp/Apraxia-ataxia/Waddling/Sensory
|
|
Step flaws -diminished height and length
MS, Scissors gait, CP |
Spastic
|
|
sTep flaws - multiple sensory defects
. Visual, neuropathic, vestibular, fear of falling |
Timid
|
|
stEp flaws - festinating (parkinsonism), short stepped, arms hang, glue footed
|
Extrapyramidal
|
|
steP flaws - partial or incomplete paralysis,
steppage gait (toe strike), CVA, asymmetrical length and height |
Paretic
|
|
step Flaws - high stepped
peripheral neuropathy, diabetes, pernicious anemia, often irreversible |
Foot drop
|
|
step fLaws - pain limiting factor, cause?
|
Limp
|
|
step flAws - wide based
lurching forward, veering, erratic, cerebellar dysfunction |
Apraxia/Ataxia
|
|
step flaWs - hip disease
stiff hip or knees, muscular dystrophy |
Waddling
|
|
step flawS - impairment of visual, vestibular, or nervous system
DJD, weakness, high stepped |
Sensory
|
|
postures in action
|
dynamic stabilization
|
|
if one cannot maintain posture, then there is a ___________ breakdown. With this comes decreased performance.
|
Technique
|
|
dynamic stabilizers of LE
|
Posterior Tibialis
Adductors Glut Medius Abdominal obliques |
|
PGAA
|
Posterior tibialis
Gluteus medius Abdominal obliques Adductors (dynamic stabilizers) |
|
inability to make purposeful movements
|
apraxia
|
|
drop of contralateral hip due to weak gluteus medius (a dynamic stabilizer of PGAA) and ipsilateral weakness
|
Trendelenburg sign
|
|
primary glut medius fibers
|
anterior (hip ABduction and Internal R)
|
|
secondary glut medius fibers
|
posterior (hip ABduction and EXTERnal rotation)
|
|
fatigue at limits of speed endurance leads to
|
foot splay due to glut medius fatigue (primary anterior Internal rotator fibers crash and posterior secondary external rotators take over)
|
|
controls the velocity of posterior foot pronation for a smooth eccentric contraction
|
posterior tibialis
|
|
most commonly affected foot joint of OA
|
great toe
|
|
what nerve provides special sensation between the 1st and 2nd toes
|
deep peroneal/fibular nerve
|
|
antagonist of posterior tibialis
|
peroneus brevis (completes the "stirrup" around the ankle)
|