• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/30

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

30 Cards in this Set

  • Front
  • Back
what is Depolarization
Electrical stumulus to cells causes an influx of sodum and calcium into the cells. The two contradctile proteins, actin and myosin use their chemical energy to slide past one another.
what is Repolarization
When calcium and sodium are pumped back into storage after depolarization and the cell is ready to start process over.
what is Automaticity
initiateing impluses in heart
what is excitability
hearst ablility to respond to impulses
what is conductivity
hearts ability to transmit impulses
what is conractility
hear muscle response
electrical stages of the hear
1. Stumulus
2. depolerization
3. repolarization
4. refractory period
what is rhe refractory period
period of time during the repolariztion where the cell is resistant to futher stimulation.
what dose the refractory period prevent
premature activation and sustained activation
conduction system of the heart 6 parts
SA node
intaatrial pathways
AV node
Bundle of HIS
bundle brances
purkinje fibers
P- Wave
attrial contracts-depolarization
PR- interval
aria to av nodel- little bit of time.
QRS
ventricals contracting- depolarization
what are arrhythmias
Abnormal heart beats result form irregularities in automaticity, excitablility, conductivity, contractility.
Types of arrhythmias are defined by
Site of orighin and mechanism of conduction
mechanisms of conduction
Tachycarida, Bradycardia, flutter, fivrillation, premature, heart block
Site of origin of arrhytmias
Sinus, atria, AV(nodal junction), ventricles
Things that enhance automaticity
Lack of oxygen
Chemical toxicity
stretch on fibers
Heart block stages
1degree messages slower
2 degree sometimes messages get through sometimes not
3degree not getting any messages
what do antiarrhythmics do
they depress automaticity, lowing conduction rates, increasing refractoriness to premature stimulation. accomplished by altering movement of one or more ions across the hear membranes.
Indications for Antiarrhytmics
Antiarrhythmics are selected according to type of abnormal rhythm determined by EKG
Contraindications of antiarrhythmics
there are no containdications to anti arrhytmics when needed to stop a fatal arrhythmia.
Actions/ classes of Antiarrhythics: class I
membranepstabalizing agents, work on the fast sodium channels.
Actions/ classes of antiarrhythmics: class I A
by blocking rapid sodium paths, delays repolarization.
Actions/ classes of antiarrhythmics: I B
Block sodium, but accelerate repolarization
Action/ class of antiarrythmics: I C
Massive blocking of sodium without much effecto on repolarization
Action/ class of antiarrythmics: II
beta blockers, block B receptors in the SNS
Action/ class of antiarrhythmics: III
prolong repolarization
Action/class of antiarrhytmics: IV
inhibit the slow calcium channels
Quinidine ( Quinidex)
Subclass: IA
Acion blocks sodium pathways to cause delayed repolarization.

Indications: atrial arrhythmias for long term use

Side effects Av block, hypotension, decrased cardiac output, widened QRS, low platelets, GI upset, cinchonism