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48 Cards in this Set

  • Front
  • Back
4 Types of Vomiting
Spitting up
Regurgitation
Vomiting
Projectile vomiting
5 Causes of vomiting in the infant and child.
Improper feeding techniques
Infection
Dietary problems
Motion sickness
Psychological causes (unpleasant odors, sights, and fright)
Obstruction of GI tract
Where is the vomiting center located
reticular core of the medulla
Signs and symptoms of vomiting.
Can happen at any time
Forceful, often projectile
Smells very sour, appears curdle, yellow, bloody
Child may cry, hurts, pain, before & after
Continues until the stomach empties, then dry heaves
Amount is a full stomach content
Diagnostic procedures that may be done to determine an organic cause of vomiting.
CBC
Electrolytes
BUN
urine tests
ABG's
blood culture
glucose
X-Rays
The main aims of treatment for the infant and child who has been vomiting.
Position to prevent aspiration
Rinse mouth/brush teeth after vomiting
Begin small amount of ORT; 5-10 ml q 1-5 minutes
More liberal fluids once vomiting stops; progression as tolerate to regular food
Antiemetics
Define gastroesophageal reflux.
relaxation or incompetence of lower esophageal sphincter, permitting reflux of gastric contents into the esophagus
GERD etiology.
Unknown; common in premature infants, children with neurological impairment and following some kinds of esophageal surgery
Signs and symptoms of GERD
vomiting
weight loss/ FTT
increased appetite
respiratory problems-asthma
recurrent otitis media
bleeding
Diagnostic tests used for GERD.
Barium swallow
radionuclide
intra-esophageal pH probe monitoring
Medical/Surgical management of GERD.
thickened feedings with cereal
small volume feedings
slow feedings
burp often during feedings
upright, prone when awake
supine when asleep (30 degree angle)
tucker sling
Formulate a plan of care for the infant and child with GERD.
Alteration in nutrition: less than body requirements r/t vomiting

Fluid volume deficit r/t vomiting

Knowledge deficit of home care r/t lack of prior experiences (CPR training)
Define diarrhea.
increase number of stools, a decrease in their consistency with an increase fluid content and a tendency for the stools to be greenish in color
Diarrhea etiology.
primary cause is bacterial, viral, parasites and fungus invasion of the GI tract
Lab exams used in diagnosing diarrhea.
CBC
CMP
x-rays for bowel abnormalities
stool culture
guaiac test/ hemoccult
stool pH (litmus paper or dipstick reagent paper)
reducing substances
Pathophysiology of diarrhea.
prostaglandins are released, causing cramping and increase peristalsis
Methods of treatment of diarrhea.
ORT
regular diet as soon as rehydrated
lactobaccilus supplementation
no OTC antidiarrheal drugs
skin care
prevention
How is the vomiting center stimulated to cause vomiting.
The center receives stimuli from:
- Higher cortical center
-Chemosensitive trigger zone
-Reflux excitement
Higher cortical center
caused by psychologic disorders due to unpleasant sights, odors, and frights
Chemosensitive trigger zone
stimuli from chemical and drug toxins, infections, radiation, IICP, and inner ear infections
Reflex excitement
caused by vagal and sympathetic nerves resulting from GI disturbances and viscera
Signs and symptoms of regurgitation.
Occurs with feeding
Little force, runs out of mouth
Looks like what just went; formula, breast milk
Not painful
Occurs once per feedings
Amount (1-2 teaspoons)
GERD medications
Reglan
Tagamet
Zantac
Pepcid
Prilosec
Differences between the child and the adult in their need for fluid balance
Fluid balance comprises a greater fraction of infant's total weight; reaches adult % at 2 years
Normal fluid requirement for pediatric patient:
100mL/kg/day
1000mL + 50mL/kg/day (each additional kg >10kg)
1500mL + 20mL/kg/day (each additional kg > 20kg)
Dehydration definition
occurs whenever the total output of fluid exceeds the total intake.
Examine the need for water balance in infant and children
Infants excrete 1/2 of their ECF; adults excrete only 1/6

Child less than 2 years don't concentrate urine; immature kidneys

increase respiratory rate; increase water loss , evaporation

if an infant & small child doesn't eat or drink, he loses 8% of weight/day

greater metabolic rate, more waste to excrete, requires more water
3 Types of Dehydration
Isonatremic dehydration

Hypernatremic dehydration

Hyponatremic dehydration
Isonatremic dehydration
most common; water and electrolytes lost in equal portions

Na stays WNL
Isonatremic dehydration treatment
rapidly over 24 hours
1/2 in 1st 8 hours
1/2 over the next 16 hours
Hypernatremic dehydration
Na level > 150
water is lost in greater proportion than electrolytes
Hypernatremic dehydration treatment
slowly over 48 hours ( water and some Na 1/4 NS)

reduce Na no more than 12 mEq/L each 12 hours
Hyponatremic Dehydration
Na level < 135

disproportional high losses of electrolytes relative to fluid lost
Hyponatremic Dehydration treatment
rapidly over 24 hours

1/2 in 1st 8 hours

1/2 over the next 16 hours
Cleft lip
unilateral or bilateral failure of maxillary process to merge with medial nasal elevation
Cleft palate
failure of mesodermal masses of the lateral palatine process to meet.

can occur in area of nasal septum, lateral or medial palates or uvula
Cleft Lip/Palate pathophysiology
during the 7-8 week of fetal life, processes that form the upper lip fuse

the palate fuses at 7-12 weeks
discuss surgical correction of cleft lip
Cheiloplasty surgery
what determine when cleft lip may be repaired
done at 2-3 days of age to 4 weeks

free of oral, respiratory or systemic infections
what determine when cleft palate may be repaired
6-24 months, usually by 1 year

degree of deformity

size of child

width of oropharynx
complications of surgery for both defects
ear infection
difficult feeding, breathing, hearing
tube feeding until treatment
when does tracheoesophageal fistula occur
when there is failure in separation of primitive esophageal and tracheal tubes before the
8th week of gestation

h/o polyhydramnios
fistula
incomplete fusion of the lateral wall of the foregut leads to incomplete closure of the laryngotracheal tube
atresia
deficient growth of entodermal cells of the dorsal walls
3 major types of tracheoesophageal atresia/fistula
Type A – Esophageal atresia: consists of a blind pouch at each end with no communication to the trachea.

Type C – Esophageal atresia, distal TE fistula: proximal esophageal segments terminates in a blind pouch and the distal segment is connected to the trachea or bronchus at or near bifurcation

Type E – Trachea-esophageal fistula: normal trachea and esophagus are connected by a common fistula
S/S of esophageal stresia
drooling
feedings will come back up
abdominal distension
esophageal fistula
vomiting
abdominal distension
choking
coughing
death if untreated
Surgical correction for TEF
may need temporary esophagostomy and gastrostomy