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118 Cards in this Set

  • Front
  • Back
5 Types of incontinence
1. Stress
2. Urge
3. Overflow
4. Reflex
5. Functional
4 Types of Urinary Retention
1. Obstruction
2. Detrusor Muscle
3. Neurogenic
4. Voluntary
Stress Incontinence
PATHOPHYSIOLOGY/DEFINITION
Pelvic muscle relaxation
weak urethra
related to multiple pregnancies, <estrogen
prostate surgery

S/S
loss of small amounts of urine
sneezing, coughing, lifting

NURSING ED
Kegal's
Avoid irritating foods: caffeine, NutraSweet, citrus juice
Urge Incontinence
PATHOPHYSIOLOGY/DEFINITION
<bladder capacity
bladder irritation
CNS disorders

S/S
Can't reach toilet in time after urge felt.

NURSING ED
Anticholinergics
Kegal's
Toileting schedule; goal 300cc w/ each void
Diurectics early in the day
Overflow Incontinence
PATHOPHYSIOLOGY/DEFINITION
urinary retention

S/S
Dribbling urine

NURSING ED
check for residual urine
cholinergics
toileting schedule
Reflex Incontinence
PATHOPHYSIOLOGY/DEFINITION
spinal cord injury
hypereflexia of detrusor muscle

S/S
involuntary loss of moderate amt of urine
no warning
Functional Incontinence
PATHOPHYSIOLOGY/DEFINITION
Self care deficit interferes with ability to respond

S/S
Inability to get to toilet on time

NURSING ED
Toileting schedule
reduce delay in toileting
answer lights quickly
Obstruction Retention
PATHOPHYSIOLOGY
BPH
Infection/trauma
scarring

S/S
overflow incontinence
firm distended badder often displaced to the side.

NURSING ED
may require surgery
difficult to cath
coude tip
Detrusor muscle retention
PATHOPHYSIOLOGY
Surgery (commone post-op complication)
Medications (anitcholinergics)

NURSING ED
Don't allow 8hrs w/o void
bladder scan
straight cath
run water, hands in water,
AMBULATE
cholinergic meds (Urecholine)
Neurogenic Retention
PATHOPHYSIOLOGY
Spinal cord injury
diabetes

NURSING ED
Trigger points
crede
valsalva +manual pressure
teach self cath
Voluntary Retention
PATHOPHYSIOLOGY
Voluntarily hold urine past urge to void
teachers, nurses

NURSING ED
Go when you need too!!!!
Creatinine Clearance
90-130
specific gravity of urine
1.005-1.030
BUN
5-20: slightly higher-up to 23 for people over 60
serum creatinine
0.5-1.2
calcium
9-11
phosphorus
1.7-2.6
magnesium
1.5-2.5
normal WBC,
and name for high and low
related to
4,500-10,000
low- leukopenia
high-leukocytosis
fight infection
normal platelet level
name for high and low
related to
150,000-400,000
low- thrombocytopenia
high-thrombocytosis
clotting
RBC
name for high and low

hemoglobin level
hematocrit level
low- anemia
high- polycythemia
hemoglobin-12-18
hematocrit-36-54%
potassium level
3.5-5.3
sodium level
135-145
albumin level
physical sign albumin level is low
3.5-5.0
edema
chloride level
95-105
Therapeutic Dig level
4 nusing interventions for high dig level
0.5-2

1. monitor potassium levels
2. monitor for signs of toxicity (headache, nausea...)
3. monitor BP and HR
4. apical pulse
total cholesterol
<200
LDL cholesterol
<100
HDL choesterol
>60
triglycerides
<150
Therapeutic INR
concern high INR.. order?
concern low INR. order?
2-3
high- blood isn't clotting fast enough, bleeding risk. order- decr coumadin dose.
low- blood clotting too fast, risk for clots, order- incr coumadin dose.
normal pH
7.35-7.45
Normal CO2
35-45
Normal HCO3
22-26
Lab that reflects severity of pt's HF
BNP
2 lab values that reflect damage to heart muscle after heart attack.
CKMB and troponins
List 3 signs of cystitis and 5 patient education points related to prevention.
cystitis- inflammation of the bladder from infection or obstruction.

s/s- dysuria, urgency, nocturia, pyuria, hematuria, suprapubic discomfort, tired, anorexic. elderly confused and incontinent.

Pt Teaching points.
1. encourage fluids 2-2.5 quarts/day
2. void when urge is felt. empty bladder every 3-4 hours
3. wipe front to back
4. females void before and after intercourse.
5. women avoid bubble baths, feminine sprays, and douches
6. wear cotton underwear
7. drink 2 glasses of cranberry juice/day
8. avoid excess milk and milk products, and sodium bicarb.
list s/s of chronic renal failure
s/s - headache, lethargy, metallic taste, anorexia, pruritis, anuria, muscle cramps, dusky color, <LOC, proteinuria, anemia, fluid overload, CHF
renal failure- 3 dietary modifications to teach pts prior to dialysis.
high carb
low K
low phosphorus

restrict fluids and NA
2 Major causes of chronic renal failure
Diabetes
Hypertention
kidneys effect
Fluid balance
electrolytes
BP
erythropoietin-RBC production
2 major signs of acute renal failure
-oliguria (urine output less than 400ml/day
-rising BUN or serum creatinine levels
Major categories of causes of acute renal failure (3)

give 2 examples of each
PRERENAL
-Impaired blood flow to kidney

hemorrhage, dehydration, HF, shock

INTRARENAL FAILURE
-acute tubular necrosis (ATN): -tubular cell damage due to prolonged ischemia and/or exposure to nephrotoxins; acute kidney disease.

ischemia, nephrtoxic drugs or substances, heatstroke

POSTRENAL FAILURE
obstruction of urine outflow

urethral obstruction by enlarged prostate or tumor, ureteral or kidney pelvis obstruction by calculi
3 classifications of nephrotoxic drugs that can cause acute renal failure
Aminoglycosides
NSAIDS
chemotherapy
cox 2 inhibitor
cocaine
venom
Compare Hemodialysis with Peritoneal Dialysis
HEMODIALYSIS
-3x per wk for 4-6hr
-pt blood in one compartment , dilysate in another
-clients blood into dialyzern(movement from blood to dialysate by diffusion, osmosis
ACCESS
subclavian and femoral catheter (short-term)
AV shunt- external arteriovenous shunt
AV fistula/AV graft- internal arteriovenous fistula

PERITONEAL DIALYSIS
-done at home; different schedules (usually at night), uses gravity.
-pertoneum is dialyzing membrane and substitues for kidney function.
ACCESS
surgical insertion of a catheter into the abd cavity 3-5cm below umbilicus.
BETTER TOLERATED
Steps of RAAS system
RENIN ANGIOTENSIN ALDOSTERONE SYSTEM

1.Decr. Blood flow to kidneys cause kidneys to secrete renin

2. Renin Converts angiotensinogen to angiotensin I

3. Angiotensin I goes to lungs where ACE converts it to angiotensin II

4. Angiotensin II causes
a) thirst mechanism stimulated
(incr blood volume and CO)
b)adrenal gland to release
aldosterone (NA retention,
H20 retention, and K
excretion)
c) vasoconstriction (incr BP)
Decr BP causes the pituitary to release
ADH
vital sign to monitor related to RAAS system and ADH
BP
What is BPH?
list 2 manifestations?
Benign Prostatic Hyperplasia
enlargement of the prostate gland

Nocturia
Diminished force of urinary stream
Hesitency in starting voiding
Dribbling after voiding
Incomplete bladder emptying
frequency, urgency
urge incontinence
dysuria, hematuria
what drug manages symptoms of BPH?
Finasteride (Proscar) causes gland to shrink

Alpha1 blockers terazosin (Hytrin), doxazoin (Cardura), and tamsulosin (Flomax) - relax smooth muscle reducing obstruction.
TURP (Transurethral Resection of the Prostate) NURSING CARE
BEFORE SURGERY
-provide routine pre-op care and teaching
-inform pt they will return from surgery w/ a urinary catheter.

AFTER SURGERY
-provide routine post-op care
-assess and manage pain (urethral discomfort, bladder spasms, and abdominal cramping due to gas)
-use aseptic technique when managing urinary drainage and irrigation.
-maintain accurate I&O
-encourage fluids when allowed
-frequently asses catheter patency, record color and characteristics of urine. monitor for hemorrhage for first 24-48 hours.
TURP - 2 things related to urine output post-op
1st 24hrs - light red urine w/ small clots

24-48hrs - light pink to yellow urine
ABCs of trauma care

A-E primary survey
(list 2 things to assess for for each)
A - AIRWAY W/ SPINAL STABILIZATION
-Jaw thrust
-Secretions, Vomitus, edema, bleeding, loose teeth, debris

B - BREATING
-Rate, rhythm, spontaneous, vocalization, color of skin, breath sounds equal bilaterally, trachea, subq emphysema
-non-rebreather mask

C - CIRCULATION
-Pulses, pressure on external bleed, type and cross match, 2 #14IV, run one LR and NS w/blood tubing.
-beeding, capillary refill, pupils,

D - DISABILITY
-Pain, broken bones, LOC, PERRL, AVPU

E - EXPOSE/ENVIRONMENT
-remove clothing, keep warm
ABCs of trauma care

F-I secondary survey
F - FIVE INTERVENTIONS
-NG, cath, pulse-ox, EKG, full set of vitals
-lab tests
-family support

G - GIVE SUPPORT
Control pain/anxiety

H - HISTORY
-Head to toe exam
-Splint as necessary

I - INSPECT POSTERIOR SURFACE
types of shock (5)
Hypovolemic
Anaphylactic shock
Septic shock
Neurogenic shocl
Cardiogenic shock
Hypovolemic Shock
-not enough blood, low-volume shock, lack of circulating blood volume.
Anaphylactic Shock
BP tanks, immune reaction triggers abnormal dilation of blood vessels.
Septic Shock
Overwhelming infection, usually gram-negative; cause dilation of blood vessels
Neurogenic Shock
when nerves are cut which causes less venous constriction due to absent nerve stimulation; also spinal anesthesia and barbiturate overdose.
Cardiogenic Shock
Volume overload, not volume deficit; caused by faiure of hearts pumping action.
Shock

3 things every cell needs
body isn't able to provide the cell with a constant supply of O2 and removal of wastes.

3 things every cell needs
-adequate blood flow
-correctly functioning heart
-normal blood vessel diameter
3 stages of shock
Compensatory Stage
Progressive Stage
Irreversible Stage
Manifestations of Compensatory stage of Shock
(LOC, BP, P, R, Skin, Urine)
LOC -oriented but restless
BP -Normal to slightly decreased
P - >100bpm
R - >20/min
Skin - pale, cool, moist
Urine- Less than 30mL/hr
Manifestations of Progressive stage of Shock
(LOC, BP, P, R, Skin, Urine)
LOC - confused; decr response to pain
BP - <90mmHg
P - >150bpm
R - >30/min, shallow, possible crackles
Skin - Cold, clammy; possible cyanosis
Urine - Less than 20mL/hr
Manifestations of Irreversible stage of Shock
(LOC, BP, P, R, Skin, Urine)
LOC - Lethargy to coma, no relfex response
BP - Falling to unobtainable
P - Slow and Irregualr
R - Slow w/ Chayne-Stokes respirations
Skin - Cold, cyanotic, mottled
Urine - Anuria
alcoholism can cause both
low magnesium and low phosphorus
Fluid volume deficit
-manifestations
- lab values
MANIFESTATIONS
fatigue
postural hypotension
tachycardia
weak, thready peripheral pulse
weight loss
flat neck veins, decr central veous pressure
dry skin, poor turgor
decr urine output, concentrated urine

LAB VALUES
Incr osmolality
Incr Hematocrit
Incr Urine Specific Gravity
Fluid Volume Excess
-manifestations
-lab values
MANIFESTATIONS
Hypertension
Tachycardia
Full, bounding peripheral pulses
Incr respiratory rate
cough, dyspnea, orthopnea
moist crackles, wheezes
weight gain
distended neck veins, incr CVP
Dependent edema

LAB VALUES
Decr serum osmolality
Decr Hematocrit
Decr Urine specific gravity
prehospital emergency care of the client experiencing hemorrhage
1. scan environment for hazards
2. call for help
3. ensure adequate airway
4. assess for cause of hemorrhage
5. control external bleeding with direct pressure and pressure points.
6. turniquet as LAST RESORT
7. assess for shock
8. if no head injury keep head and back flat and elevate legs.
9. cover client for warmth
10. NPO
11. use touch and verbal communication to reduce anxiety.
Nursing responsibilities for a blood transfusion
ID client and blood.
monitor for febrow reaction (occurs w/in the 1st 15 minutes)
waste products removed by kidneys
creatinine and BUN
positioning for dyspnea
high fowlers
renal calculi prevention
drink 8-10 gasses of water a day
manifestations of fluid volume deficit
(HR, BP, Skin)
incr HR, decr BP, dry skin
lab results post hemodialysis
decr serum creatinine
normal body temp in deg celcius
37.0 deg celcius
indication of peritonitis during peritoneal dialysis
cloudy dialysate
secreted by kidney, resonsible for RBC production
erythropoietin
edema assessment of a pt on bed rest
sacral area
manifestations of peritonitis
absent bowel sounds, rigid board like abdomen, cloudy dialysate
periorbital edema
nephrotoxic syndrom
healthy adults feel the urge to void when how much urine has collected in the bladder?
300-500mL
post IVP nursing actions
1. monitor injection site
2. report signs of reaction to dye
3. monitor vitals and urine output
manifestations of hypocalcemia
tetany, muscle cramps, tingling
electrolyte imbalance, tall peaked T waves on ECG
hyperkalemia
manifestations of hyponatremia
headache, anorexia, N/V
3rd spacing
fluid moves out of intervascular space into surrounding area
low risk for third spacing
diabetes mellitus - fluid moves into the blood
high risk for fluid volume deficit
ileostomy
medication that lowers K levels in body
kayexalate
electrolyte that incr metabolic acidosis and decr metabolic alkosis
potassium
electrolyte effected by by thyroid and PTH gland
calcium
2 things that must be present before you can give high dose potassium
adequate urinary output and a cardiac monitor.
monitor for what after giving opiods
respiratory depression
medication frequently given pre-op to reduce oral secretions and prevent aspiration
atropine sulfate
L sided HF manifestations
dyspnea, orthopnea, cough
lab values for a client with hematuria, melena (back tarry stools), and purpura (subq bleeding) are likely to show
thrombocytopenia
sickle cell crisis triggered by
dehydration, hypoxia, infection, excessive exercise
in pernicous anemia deficiency of this results in malabsorption of Vit B12
intrinsic factor
if this was found in a chest tube drainage further action would be needed
vigorous bubbling in H2O seal chamber
breathing technique that prolongs exhalation, helping remove C02 from lungs
pursed lip breathing
impaired liver function will not effect
production of amylase
medication that decr serum ammonia by inhibiting ammonia absorption in the gut and promoting ammonia excretion in the stool
lactulose (cephalac)
new diagnosis of A fib monitor for
irregular pulse
pink frothy sputum is a sign of
pulmonary edema
nursing responsibility for plasmapheresis
check for signed consent
manifestations of pulmonary embolism
chest pain, cough, anxiety, dyspnea
pulmonary hypertension can cause this
cor pulmonale

(R sided HF w/ or w/o HF)
flow of blood though the heart
right atrium
tricuspid valve
right ventricle
pulmonic valve
pulmonary trunk
pulmonary arteries
lungs
pulmonary veins
left atria
bicuspid valve
left ventricle
aortic valve
aorta
arteries
arterioles
capilaries
venules
veins
inferior and superior vena cava
hyponatremia
LOW SODIUM
-affects function of voluntary and involuntary muscles.
-brain cells swell leading to NEURO s/s

s/s
anorexia, N/V, abd cramping, diarrhea, mental status change, hyperreflexia, twitching, tremors, convulsions, coma

LABS
serum sodium<135
serum osmolality<280
hypernatremia
EXCESS SODIUM
-changes in LOC
-brain cells shrink
-dry, sticky, membranes

s/s
thirst, restlessness, weak, altered mental state, LOC changes, muscle irritability, dry sticky mucous membranes, postural hypotension, hot, dry skin, fever, decr sweating

LABS
serum sodium >145
serum osmolality >295
hyper/hyponatremia effects:
fluid shifting causes neuro changes
Potassium Imbalance efects:
the heart
hypokalemia
LOW POTASSIUM
s/s dysrythmia and EKG changes. FLATTENED T-WAVE; U WAVE; n/v, anorexia. <BS, muscle weakness, leg cramps.

serum potassium <3.5
hyperkalemia
HIGH POTASSIUM

MOST DONGEROUS OF ALL ELECTROLYTE IMBALANCES!!

s/s
TALL< PEAKED T-WAVES, dysrythimia, heart block, cardiac arrest, nausea, abd cramping, muscle weakness, paresthesias, flacid paralysis.

lab K>5.3
hypo/hypercalcemia affect:
muscle irritability
hypocalcemia
s/s

neuromuscular excitability increases.
tetany, numbness, tingling.
positive Chvostek's sign, positive Trousseau's sign.
abd cramping, diarrhea
<BP, <CO, dysrhythmia
hypercalcemia
s/s
muscle weakness
fatigue
<tendon reflexes
constipation
anorexia, n/v
>BP, dysrhythmias
>urine output