Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
54 Cards in this Set
- Front
- Back
What causes shock?
|
inadequate perfusion; or imbalance between oxygen supply and demand
|
|
What is shunting?
|
moving oxygenated blood into selected areas while bypassing others
|
|
When is the initial stage of shock present?
|
when client's MAP is decreased by less than 10mmHg
|
|
What are the characteristics of Stage 1 or compensatory shock?
|
MAP decrease 10-15mmHg, RAAS increases; patient looks right but vitals are unstable
|
|
What labs might indicate shock?
|
signs of metabolic acidosis, decreased bicarbonate, increased potassium and lactic acid
|
|
What occurs during Stage 2?
|
MAP is decreased 20mmHg, O2 not sufficient, hypoxia, hyperkalemia, lactic acid accumulation, hypotensive
|
|
How is hypotension corrected?
|
fluid resuscitation and or vasopressors
|
|
What are the characteristics of Stage 3?
|
mega cell death, shock is irreversible due to prolonged damage
|
|
What are the cardiac manifestations of shock?
|
myocardial cells become hypoxic, HR increases, nonpalpable pulses, dysrhymthmias due to potassium
|
|
What are the respiratory manifestations of shock?
|
increased RR, CO2 increases causing respiratory acidosis, ARDS can occur
|
|
What are the GI manifestations of shock?
|
organs become ischemic, stress uclers, paralytic ileus
|
|
What are the liver manifestations of shock?
|
gluconeogenesis leading to hypoglycemia, fat metabolism impaired
|
|
What are the mental manifestations of shock?
|
changes in mental status and orientation, lethargic
|
|
What are the renal manifestations of shock?
|
blood is shunted to brain and heart, oliguria, renal failure
|
|
What are the types of shock?
|
hypovolemic, cardiogenic, obstructive, distributive, septic, neurogenic, anaphlactic
|
|
When does hypovolemic shock occur?
|
when too little circulating blood volume causes a decreased MAP
|
|
What are the clinical manifestations of hypovolemic shock?
|
increased HR, absent PP, decreased O2, skin pallor, decreased systolic BP
|
|
What is the Rx for hypovolemic shock?
|
oxygen, fluids, crystalloid fluids, LR, isotonic
|
|
What does LR contain?
|
sodium, chloride, calcium, potassium, and lactate dissolved in H20
|
|
What is isotonic fluid?
|
helps expand volume and the lactate helps buffer any acidosis
|
|
What are vasoconstricting drugs used to treat hypovolemic shock?
|
dopamine, NE
|
|
Which drug enhances myocardial perfusion?
|
sodium nitroprusside
|
|
What is cardiogenic shock?
|
the heart cannot maintain cardiac output
|
|
What is the most common cause of cardiogenic shock?
|
acute MI
|
|
What are the manifestations of cardiogenic shock?
|
visible distress, dyspnea, tachycardia, hypotension, JVD, murmurs
|
|
What is the Rx for cardiogenic shock?
|
fluids, vasopressors, dopamine (increases BP), morphine for pain,
|
|
What is obstructive shock?
|
flow of blood is obstructed; impedes perfusion and can cause cardiac arrest
|
|
What is distributive shock caused by?
|
decreased systemic vascular tone characterized by a decreased systemic vascular resistance; hypotension
|
|
What is the most common type of vasodilatory shock?
|
septic shock
|
|
What is septic shock associated with?
|
severe infection and the release of inflammatory mediators
|
|
What is often released during septic shock?
|
endotoxins
|
|
Which type of bacteria most often causes septic shock?
|
gram-negative
|
|
What is the criteria for septic shock?
|
temp >38C, HR >90bpm, RR>20,
WBC >12,000 |
|
What are cytokines?
|
promote endothelial leukocyte adhesion, cell damage proteases, prostaglandins and activation of clotting cascade
|
|
What is the result of too many cytokines in septic shock?
|
major cap permeability and vasodilation....fluid shifts
|
|
Why does the skin become flushed and warm in the early stages of septic shock?
|
because of the vasodilation
|
|
What are some of the late signs of septic shock?
|
tachycardia, rapid respirations, cool, pale skin, lethargic to comatose, oliguria, decreased CVP
|
|
What lab MUST be checked for septic shock?
|
lactate levels (if >4mmol/dl) indicates hypo-perfusion
|
|
Which increased lab value is associated with low rates of survival?
|
WBC > 50,000
|
|
What is the Rx for septic shock?
|
control the causative agent (right ABX), PA cath, oxygen, fluid resuscitation, dopamine, NE
|
|
What is recombinant human activated protein C?
|
an endogenous protein that not only promotes fibrinolysis and thrombosis and inflammation but also shows signs of modulating the coagulating and inflammatory mediators in severe sepsis
|
|
Why is tight glycemic control important for the Rx of septic shock?
|
shown to decrease mortality rates; blood sugar should be maintained from 80-110mg/dL
|
|
What is used only in severe sepsis and is indicated for the reduction of mortality in adults with high risk?
|
Xigris
|
|
What is neurogenic shock?
|
occurs when a condition increases parasympathetic stimulation or inhibits sympathetic stimulation of smooth muscle
|
|
How is neurogenic shock characterized?
|
loss of vascular tone and reflexes
|
|
What is the most severe systemic allergic reaction?
|
anaphylactic shock
|
|
What is distributed into the blood stream in anaphylactic shock?
|
histamine and other vasoactive amines
|
|
What is the treatment for anaphylaxis?
|
Epi for severe, Benadryl, O2, steroids, bronchodilators
|
|
This is the formation of fibrin clots and thrombotic occlusion of small and mid-sized vessels caused by endotoxins and inflammation?
|
Disseminated Intravascular Coagulation
|
|
How is DIC characterized?
|
widespread clotting and bleeding
|
|
What are the manifestations of DIC?
|
hemorrhage, oozing of bleeding, purpura, petechiae, bruising, GI bleeding, blood sputum
|
|
This indicates the presence of abnormally high levels of fibrin degredation products in the body.
|
D-dimer/FDPs
|
|
This inhibits further thrombogenesis?
|
heparin
|
|
This inhibits fibrinolysis through the inhibition of plasminogen activator substances
|
Aminocapric acid (Amicar)
|