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33 Cards in this Set

  • Front
  • Back
What is non-fluent aphasia?
Non-fluent; perservative speech
What types of words dominate non-fluent aphasia?
Word selection dominated by content words,
How is paraphrases used in non-fluent aphasia?
rarely few paraphrases
Do non-fluent aphasic monitor their speech?
preserved self-monitoring
How long are non-fluent aphasic phrases?
Phases < 4 words
What is fluent aphasia?
Fluent speech w/ few perservations
What types of words dominate fluent aphasia?
Uses both content & function words
How is paraphrases used in fluent aphasia?
Prominent paraphrasis
How long are fluent aphasic phrases?
Phrases > 5-6 words
Do fluent aphasic monitor their speech?
Self-monitoring is poor
What is the distribution of fluent & non-fluent aphasics in acute & chronic stages?
About 1/2 & 1/2. Over time non-fluents become fluent (79%); b/c of practice
What is the range of the aphasic severity scale?
0= No usable speech 5= Minimal handicap
What are the types of nonfluent aphasia syndromes?
Global, Broca, & Transcortical motor
What are the types of fluent aphasia syndromes?
Wernicke's, Anomic, Conduction, & Transcortical
What are the basic assessment tasks to diagnose aphasic syndrome?
Spontaneous speech production, Repetition, Comprehension, Naming, Reading, & Writing
Describe global aphasics.
Not able to say much (single words or over learned phrases), severe deficits in all language modalities, spares non-verbal intelligence
How do global aphasic appear when they communicate?
Appear to communicate more purposefully by the use of gestures & facial expressions
Describe Broca's aphasia.
Uses real words, speak haltingly, trouble producing speech, naming, & repeating. Comprehension in tact
How do Broca's aphasic patients sound when they speak? Look?
Telegraphic; articulation maybe impaired. Uses gestures to compensate
Are Broca's patients aware of deficits?
Fair-Good awareness
What is the most common type of non-fluent aphasia?
Broca's
Describe the verbal output of Wernicke's patients?
Uses jargon (unaware of deficits)
What is impaired in Wernicke's patients?
Repetition, confrontation, naming, & auditory comprehension
Describe anomic aphasic patients?
"tip of tongue" (aware of deficit)
Describe the verbal output of anomic patients?
Complete utterances marked by inability to retreive words & circumlocutions
What lesion causes anomic aphasia?
Angular gyrus
Describe the verbal output of conduction aphasia patients.
Conversational speech appears normal but repetition & confrontation naming is poor (good auditory conprehension). Some paraphrasis & circumlocutions (aware of deficit)
What lesion causes conduction aphasia?
Damage to arcuate fasiculus (connects Broca & Wernicke)
Which aphasias are the mose severe?
Non-fluent tends to be most severe
Of the fluent aphasia, which is the most severe?
Wernicke's
How do aphasias evolve?
Non-fluent evolves into fluent, butnever the reverse
How do Broca's patients evolve?
Into anomic
How do Wernicke's patients evolve?
Into conduction or anomic