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9 Cards in this Set

  • Front
  • Back
  • 3rd side (hint)
polio, Werdnig Hoffman disease
-what type of lesions?
-location?
-dysfxn?
-LMN lesions only
-anterior horns
-flaccid paralysis
multiple sclerosis
-what type of matter?
-distribution?
-mechanism of destruction
-sxs?
-white matter
-asymmetric and random
-demyelination
-SIN
-scanning speech, intention tremor, nystagumus
ALS
-what type of deficits
-not what type of deficits?
-location of lesions?
-UMN and LMN
-no sensory deficits
-anterior horn, lateral corticospinal tracts
occlusion of ventral artery
-what structures are spared?
-dorsal columns and tract of Lissauer
Tabes dorsalis
-seen in which disease?
-degen of which structures?
-what is impaired
-tertiary syphilis
-dorsal roots and columns
-proprioception, locomotor ataxia
syringomyelia
-which fibers are damaged?
-loss of what sensory?
-crossing fibers of spinothalamic tract
-bilateral of loss of pain and temperature
vitamin B12 neuropathy, Friedrich's ataxia
-what type of damage?
-where are the lesions?
-what sensory deficits?
-demyelination
-dorsal columns, lateral corticospinal tracts, spinocerebellar tracts
-ataxic gait, hyperreflexia, impaired vibration/position sense
Brown Sequard syndrome
-what damage?
-what are the ipsilateral findings? 3
-what are the contralateral findings?
-what type of damage at the level of the lesion?
-what happens if T1 is involved?
-hemisection of spinal cord
-ipsilateral: corticospinal tract (UMN), dorsal columns, loss of sensation at the level of damage
-contra: pain and temp (spinothalamic)
-LMN signs and loss of sensation
-Horner's syndrome
Horner's syndrome
-what are the symptoms?
-lesions above which level?
-what type of lesions may lead to Horner's?
-Ptosis
-anhydrosis
-miosis (pupil constriction)
-above T1
-Pancoast tumor, hemisection, syringomyelia (late stage)
-PAM