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9 Cards in this Set
- Front
- Back
- 3rd side (hint)
polio, Werdnig Hoffman disease
-what type of lesions? -location? -dysfxn? |
-LMN lesions only
-anterior horns -flaccid paralysis |
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multiple sclerosis
-what type of matter? -distribution? -mechanism of destruction -sxs? |
-white matter
-asymmetric and random -demyelination -SIN |
-scanning speech, intention tremor, nystagumus
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ALS
-what type of deficits -not what type of deficits? -location of lesions? |
-UMN and LMN
-no sensory deficits -anterior horn, lateral corticospinal tracts |
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occlusion of ventral artery
-what structures are spared? |
-dorsal columns and tract of Lissauer
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Tabes dorsalis
-seen in which disease? -degen of which structures? -what is impaired |
-tertiary syphilis
-dorsal roots and columns -proprioception, locomotor ataxia |
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syringomyelia
-which fibers are damaged? -loss of what sensory? |
-crossing fibers of spinothalamic tract
-bilateral of loss of pain and temperature |
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vitamin B12 neuropathy, Friedrich's ataxia
-what type of damage? -where are the lesions? -what sensory deficits? |
-demyelination
-dorsal columns, lateral corticospinal tracts, spinocerebellar tracts -ataxic gait, hyperreflexia, impaired vibration/position sense |
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Brown Sequard syndrome
-what damage? -what are the ipsilateral findings? 3 -what are the contralateral findings? -what type of damage at the level of the lesion? -what happens if T1 is involved? |
-hemisection of spinal cord
-ipsilateral: corticospinal tract (UMN), dorsal columns, loss of sensation at the level of damage -contra: pain and temp (spinothalamic) -LMN signs and loss of sensation -Horner's syndrome |
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Horner's syndrome
-what are the symptoms? -lesions above which level? -what type of lesions may lead to Horner's? |
-Ptosis
-anhydrosis -miosis (pupil constriction) -above T1 -Pancoast tumor, hemisection, syringomyelia (late stage) |
-PAM
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