Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
33 Cards in this Set
- Front
- Back
what are the lobes of the cerebrum and their functions?
|
frontal-
Temporal- parietal- |
|
What function do the frontal lobes serve? ho w are they divided
|
Regulate self awareness, responsibility, purposefulness, meaning , thinking , planning working memory, inhibitory control.
bilateral lesions here will cause- personality changes, lose determinitation, abulia, disinhibition, indifference, pseudobulbar palsy (dont use the lower cranial nerves well), apraxia (can move but poor dude to connections |
|
What is the function of the temporal lobes?
|
create personalized subjective emotional experience.
3 gyri - superior middle and inferior. Contains heschl's gyrus in the medial part its primary auditory cortex -contains the limbic system, amygdala(emotion), hippocampus (memory) lesions can give hallucinations, partial complex seizures, emotional and behavioral changes ( dont distinguish left or right ) bilateral lesions- amnesia w/ both hippocampi, Dominant side lesion- wernicke's aphasia ND- difficulty with spatial relations, agnosia for sounds and quality of music |
|
What are the different types of memory? describe
|
working- immediate recall.
short term- current events and object memory @ 3- 5 min long term- old memories from childhood and youth. Semantic- facts of common knowledge. encoded in temporal lobe retrieved by frontal Episodic- personal filmic memories. encoded by hippocampus stored in cortex Procedural- remembering how to do a task. stored in cerebellum and putamen, ingrained habits stored in caudate nuclei ( phobia compulsion) Fear- stored in amygdala. |
|
What is the gateway to the hippocampus?
|
Entorhinal cortex- all cortical input to the hippocampus goes through it.
In alzheimhers there is a large amount of tangles here. |
|
What are the characteristics of Broca's aphasia?
|
They can understand you but cannot speak. Language is formulated into words
- broken speech, non-fluent. lesion located posterior inferior frontal lobe |
|
What are the characteristics of conduction, transcortical motor/sensory aphasia?
|
conduction- repetition affected primarily, intact comprehension and fluent speech production, but hard time repeating. Damage to arcuate fasciculus
Transcortical- lesion to anterior superior frontal lobe. Good comprehension, good repetition, difficulty with complext converstation( long sentences polysyllabic words) Trancortical sensory- only one with normal rep but no comprehension or meaningful speech production. can finish a prayer, or song |
|
What structures are involved in memory storage and retrieval?
|
hippocampus, fronix, cingulate gyrus, mamillary bodies, several nuclei in thalami
|
|
what side for language, what side for spatial orientation?
|
language left (dominant), spatial right
|
|
What are the characteristics of Wernicke's aphasia?
|
It's a fluent aphasia
- comprehension of language is affected. they cannot understand what you say and they speak to you in jibberish.repetition affected. location- left temporal superior posterior lobe |
|
What maybe considered the most primitive part of the limbic system?
|
hypothalamus fight or flight most primitive emotions. no thinking .
|
|
what does the hippocampus do?
|
It tells you what memories you should store. "its the decider".
throws away irrelevant memories from becoming long term. stores and consolidates new memories |
|
what type of lesion is amnesia usually seen ?
|
bilateral hippocampal lesions.
Unilateral lesions generally do not cause a permanent memory deficit. |
|
Why is the WADA test used for ?
|
to find out the impact of each hemisphere on language and memory of their temporal lobes. Used when considering a temporal lobe resection for seizure treatment.
|
|
Where do memories destined to be long term go after the hippocampus?
|
Hippocampus relays they experience to cortex. ( auditory visual etc) eventually hippocampus no longer necessary.
|
|
Function : Amygdala
|
Generation of the most basic/primitive emotions. concious emotions linked to frontal lobes.
Fear, Love |
|
Fxn: Septal Nuclei
|
Produce extremes of emotions ( explosive violence, outbursts)
- lesions here can lead to "septal rage" |
|
Fxn: Cingulate gyrus
|
Links memory and emotion, generates emotion of speech, musicality.
|
|
What is the Papez circuit?
|
explains the idea of emotional awareness and response to cortical influences. Not just one area for emotion but a series of linked structures. ( limbic with memory systems) get pic**
|
|
what are the functions of the parietal lobe what structures do they contain?
|
Right side- attention to surrounding world, direction, position. Lesion disrupts ability to attend to a stimulus. contralateral. visual-spatial disoder= contruct. apraxia cant make puzzles or map
Lack of self awareness= Anosognosia dont recognize deficit. Visual field defect= pie on floor OKN. Contains sensory areas for contralateral sensory info. (touch) lesions here can result in asterognosis, agraphesthesia ( comprehend tactile stim), sensory extinction. |
|
Right parietal lobe lesion : Neglect
|
Pt can ignore side of body, deny existence of own body parts, Visual neglect recovers in 9wks while motor make take 1-2 years.
|
|
What does the left parietal lobe contain what does it ?
|
angular gyrus- involved in symbolic representation of language (read write). Comprehension of writing
|
|
What is alexia without agraphia? how do you get it?
|
Reading comprehension is gone. Letters can be seen but dont mean anything. Caused by lesion of intact angular gyru disconnected from visual input.
Writing still normal b/c angular gyrus connections to motor centers intact. aka - pure word blindness |
|
What is Gertsmann syndrome?
|
lesion to left parietal lobe that includes the angular gyrus. Includes: dysgraphia, dyscalculia, R/L confusion, finger agnosia.
|
|
What is Balint Syndrome?
|
Lesions to bilateral dorsolateral parieto-occipiutal association cortex. Contains triad of simultanagnosia, optic ataxia, ocular apraxia.
|
|
simultanagnosia?
|
impaired ability to percieve parts of a visual scenes as a whole. can describe parts but dont see big picture.
|
|
optic ataxia?
|
impaierd ability to reach for or point to objects under visual guidance despite intact motor function.
|
|
Ocular apraxia?
|
inability to direct gaze towards objexts in the peripheral vision through saccades. cannot will but can react to a clap or loud sound.
|
|
What is the function of the occipital cortex?
|
-process the visual info detected by the primary striate cortex.
- occi-temp Projections help say what is that, what color, what it means lesions here can cause achromatopsia, visual agnosia, alexia, prosopagnosia, topographagnosia. |
|
Bilateral occipital lesions can cause what ?
|
cortical blindness-
Visual halluciniation -macro/microopsia Anton's syndrome- denial of blindness ( dunno what missing) Achromatopsia/visual agnosia Inability to perceive motion |
|
What are some interhemispheric connections?
|
Corpus callosum, anterior posterior commisure and optic chiasm.
|
|
What is the corpus callosum?
|
thick band of 80 million mylenated axons. lesions not common and known as disconnection syndromes.
|
|
Lesion in the left occipital lobe and splenium of the corpus callosum causes what?
|
Right angular gyrus cannot communicate with left angular gyrus. This is alexia without agraphia.
|