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70 Cards in this Set

  • Front
  • Back
posterior iris epithelium
neural ectoderm
sphincter and dilator muscle of iris
neural ectoderm
posterior ciliary epithelium
neural ectoderm
neural ectoderm
neural ectoderm
Optic nerve fibers and glial
neural ectoderm
neural crest
parts of the orbit
neural crest
orbital septum and tarsal plate
neural crest
95% sclera
neural crest
secondary corneal stroma and endothelium
neural crest
stroma of the uveal tract
neural crest
ciliary muscle
neural crest
optic nerve meninges
neural crest
skin of the eye and derivatives
surface ectoderm
surface ectoderm
lacrimal gland
surface extoderm
primary corneal stroma
surface ectoderm
surface ectoderm
striated extraocular muscles
circulatory blood elements --> choriocapillaries
blood vessels
day rostral tube closes
day caudal closes
Cause of Retinochoroidal Coloboma
failure of the inferior fissure to fuse
Cause of ONH Coloboma
Fusion of the most proximal part of the fissure
Cause of Morning Glory/How is it different than Optic Nerve Head?
Failure to fuse.
- Morning glory tends to be unilateral, and has SEVERE VA effects 20/200. More often seen in women then men.
OHN has: an excavation below the optic nerve head and it does not lie within it, it has no glial tissue and normal vasculature
Morning glory: optic nerve WITHING excavation and is CENTRAL, it has a glial tissue, weird vasculature, and pigment is present surrounding it
Micropthalmia with cyst cause
defect in closure of the POSTERIOR fetal fissure, overgrowth of RPE and neural retina occur.
Congential Cystic eye cause
failure to invaginate the optic cup... fills with glial
Tilted Disc Syndrome cause
PARTIAL nonclosure of fissure. Superotemporal optic disc is elevated or swollen, appear longr horizontally then vertically. Accompanied by thinning tissue of fundus and weird blood vessel course
Cause of Iris coloboma
ANTERIOR fissure fails to close
CHPE Congential Hypertrophy of the RPE
black, flat, round, fundus lesions, may be surrounded by hypopigmente border. Due to INCREASE in RPE size and hyperpigmentation
primary eye malignancy in kids. develops from undifferentiated rpe cells. RBI1 gene
No melanin pigmentation
Nanopythalmia/pure micropthamus
marked hyperope
macular hypoplasia
late gluacoma. Disruption of optic cup not enlarging properly
Congential Cystic eye
non invagination of priamry optic vesicle
Reasons why infants eyesight is so bad:
-Incomplete migration of ganglion and bipolar cells
-TLC remains
-Immature cones (cones are short and thick instead of long and thin) create immature macula
-Incomplete myelination of axons
-Eye not full size
-Cells in the lateral geniculate are small
Which side is expected to have more damage in regard to vasculature?
Temporal! It is slower to develop so it will be most effected
Vascular component of Prelaminar
choriod vasculature and short posterior ciliaries
Vascular component of the Laminar
TRANSVERGE: circle of zihnn. Uses CRA and short posterior cilliares
Vascular component of the Retrolaminar
Uses Pial plexus, opthalmic artiers, musclar arteris, central retinal arteries, short posterior artery, and CRA
Tissue Dvelopment of Ganglion Cell Axons
Neural ectoderm
Tissue development of glial support
neural ectoderm
Tissue development of connective tissue support
neural crest
Tissue development of Blood supply
Tissue development of Meninges
Neural crest
Neural ectoderm
Lamina Cribrosa
Neural crest
Myelination begins at
the retrolaminar
Optic Nerve Hypoplasia arises from
failure of ganglion cell layer to develop, see small optic nerve head with a double ring of pigment.
Megalopapilla charactersticis
larger than 2.1 mm disc
healthy neural retinal rim
Congenital optic pits is due to
nonfunctional retina that has been pushed through a defect in lamina cribrosa
Lattice degeneration
Cigar shaped, RPE clumping
- At periphery retina.
PreRetinal Hemorrhage
Occurs at ILM and nerve fiber before retina, blood pools and makes a D --> see a big blotchy thing. IS red, then yellow
Dot Blot Hemorrhage
DEEP layers, occuring inner nuclear layer, outer plexiform layer. Deep retinal edema. Usually seen in diabetes. Breac can be in CRA. If blood pools between cells it looks like a blot. If it pools between fibers it looks like a dot.
Flame Shaped Hemorrhage
Occurs in Nerve fiber layer, sohrt lived. Area of LOCALIZED HYPOXIA. blood in contour of axons.
SubRetinal Hemorrhage occurs
At RPE! DESTRUCTIVE destroys photoreceptors.
Cotton Wool Spots
Focal infarcts in nerve fiber layer (inner retina layer --> CRA)
Gray White opacifications in retina due to:
macula edema, acute iscemia, CRA occlusion, Berlin's edema (can be due to a punch)
Optic nerve Hypoplasia is due to
GANGLION CELLS did not develop
See yellowish halo ring bordered by pigment
Congenital optic pits is due to
Nonfunctional retina that has pishe though a defect in the lamina cribrosa
Disc larger than 2.1 mm
BILATERAL, see cilioretinal artery present
healthy neural rim
The basement membrane of the ILM is reminscent of the
basement membrane on the BASAL side of the neural tube
The basement membrane of the RPE is reminscent of the
basement membrane of the APICAL side of the neural tube
Susensory retinal space's boundaries include
the APICAL surface of retinal pigmented epithelial cells
Area of brain responsible for visual perception
Lateral geniculate in thalamus
Area of brain responisble for control of eye movements
superior colliculus of midbrain
Area of brain rsponisble for control of papillary light reflex
pretectal area of midbrain
Area of brain responisble for control of diurnal rhythms and hormonal changes
Suprachiasmatic nucleus of hypoTHALAMUS