Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
30 Cards in this Set
- Front
- Back
autonomic ganglia are what type of cells?
|
multipolar
|
|
what cells are pseudounipolar cells?
|
spinal and cranial nerve ganglia
|
|
______are responsible for myelination and begins at age_____ and is complete at age____
|
oligodendrocytes, begins at 4mo after conception, complete at age 2
|
|
Clinical features:
cardiac septal defects, facial malformations including widely spaced eyes and long philtrum, growth retardation. |
fetal alcohol syndrome (most common cause of mental retardation).
|
|
clinical features:
from improper closure of neuropore has several forms with presentations varying from tufts of hair around back(spinal cord area), meninges that extend out of defective spinal canal, and neural tissue that is visible externally |
spina bifida
|
|
what is it called when the neural tissue is visible externally?
|
rachischisis
|
|
what is it called when the meninges AND spinal cord extend out of the spinal canal
|
meningomyelocele (without spinal cord is called spinal meningocele or spina bifida cystica)
|
|
hydrocephaly- what is it?
|
accumulation of CSF in ventricles and subarachnoid space
|
|
how is hydrocephaly caused?
|
by congenital blockage or cerebral aquaducts, CMV, or toxoplasmosis infxn, leading to increased head circumference.
also a non obstructive type which is the failure of csf absorption. |
|
dandy walker malformation
|
dilation of the 4ht ventricle, leading to hypoplasia of the cerebellum, failure of foramina of luschka and magendie to open, from riboflavin inhibition, posterior fossa trauma, or viral infxn.
|
|
failure of the brain to develop, from lack of closure of anterior neuropore, associated with increased maternal a-fetoprotien (AFP), decreased head circumference in neonates.
|
anencephaly
|
|
herniation of the cerebellar vermis through foramen magnum, hydrocephaly, myelomeningocele
|
arnold-chiari malformation
|
|
receptors of the symp and parasymp NS:
site of action=smooth muscle, skin and viscera |
symp= contracts via a1 receptor
para= relax via muscarinic receptor |
|
receptors of the symp and parasymp NS:
smooth muscle and skeletal muscle |
a1, b1 = symp=a1=contract, b1=relax,
muscarinic= parasymp= relax |
|
receptors of the symp and parasymp NS:
smooth muscle of the lung |
b2= symp = relax
muscarinic= parasymp= contract |
|
receptors of the symp and parasymp NS:
sm. muscle of gi tract |
a1 = contract sphincters = symp
b2= relax intest wall muscarinic= parasymp= contract gi wall and relax sphincters |
|
receptors of the symp and parasymp NS:
heart, sa node |
b1= increase heart rate
musc = para= decrease rate |
|
receptors of the symp and parasymp NS:
heart ventricles |
b1= symp= inc. contractility and contraction velocity
muscarinic= parasymp=small decrease in contractility |
|
receptors of the symp and parasymp NS:
eye- iris- radial muscle |
a1-symp- mydriasis (pupil dilation)
parasymp= n/a= no receptors |
|
receptors of the symp and parasymp NS:
eye- ciliary muscle |
b2- symp- relax
muscarinic- contract= near vision |
|
receptors of the symp and parasymp NS:
eye- sphincter muscle |
no sympathetic inn.
parasymp= muscarinic, miosis (pupil constriction) |
|
receptors of the symp and parasymp NS:
bladder |
symp= b2 relax wall, contract sphincter
para= musc= contract wall,relax sphincter |
|
receptors of the symp and parasymp NS:
uterus |
symp= a1 contract, b2 relax
para= musc= contract |
|
receptors of the symp and parasymp NS:
penis |
a2= ejaculate
muscarinic- para- erection |
|
receptors of the symp and parasymp NS:
sweat glands |
all sympathetic via MUSCARINIC receptors cause secretion.
|
|
most common cause of cns pathology
|
cerebrovascular disease. = 3rd major cause of death in U.S.
|
|
botulinum mech of action
|
stops ach release?
|
|
cocaine and tca MOA
|
stops NE reuptake
|
|
guanethidine MOA
|
stops NE release
|
|
reserpine moa
|
stops dopamine conversion to NE
|