• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/51

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

51 Cards in this Set

  • Front
  • Back

causes of short unconsciousness

- vasovagal syncope
- postural hypotension
-cardiac arrhytmias
- hypervent


- hypoxia
- hypoglycemia
- vertebro-basilar transient ischemic attacks
- epileptic seizures

vasovagal syncope

increased vagal pressure leading to decreased sympathetic activity -> slows heart and blood pools peripherally, CO decreases --> inadequate brain perfusion-> LOC

postural hypotension

abcsence of cardoio- acceleration and perioheral vasoconstriction when changing to erect position .

main cause of postural hypotension

drugs that decreases the sympathetic tone

hyperventilation mechanism forLOC

hypervent -> decreased CO2 -> cerebral vasoconstriction -> LOC

vertebro-basilar transient ischemic attacks mechanism

thromboembolic material from heart or carotid lodges in small A supplying the brain stem -> ischemia ->

vertebro-basilar transient ischemic attacks and LOC

rarely without other symptoms of breinstem dysfunc

what are the main parts of consciousness

- arousal
- awareness

arousal

wakefullness due to ascending reticular activating system

awarenes

due to cortical functions

vigilence

the quality or state of staying alert especially to possible danger

disorders of vigilance

torpidi/daze
- somnolence
- sopor
- coma
- brein death

torpidity/daze

mildes disorder of vigilence
- completly alert
- slowed down
- problems focusing

somnolence3

- superficial sleeping
- awakened easily by verbal or other sensory stimuly

sopor

- only awakened by stronger stimuli
- falls back asleep during conversation

coma

pt can NOT be awakened by any stimuli

superficial coma

superficial reflexes (ex cornral reflex) can be elicted

deep coma

- superficial reflexes can not be elicited
- inadequate breathing

awareness disorders

- confusion
- delirium
- tenebrosity
- peraistant vegitative state
- minimal conscious state

confusion

impaired orientation in respect to place, time and person

delirium

an acutely disturbed state of mind characterized by restlessness, illusions, and incoherence and hallucinations

T/F


delirium ids the most serious form for confusion

true

causes of delirium

alcohol
- tranqulizers
- intox
- withdrawal
- infections

tenebrosity

pt reacts slowly and do not behave adequatly
- aphasia may be present

tenebrosity may occur

following an epileptic seizure
- in transient global amnesia

persitant vegitative state

the pts sleep wake cycle is intact, reflexes are normal but the pt is unable to preform conscious activities. no for for communication is possible.

minimally conscious state

similar to tenebrosity but the pt is able to communicate a little for short periods of time

most common cases of impaired consciousness and coma

- brain abscess
- brain tumor
- head trauma
- subarachnoid hemorrhage
- seizured
- resp failure
- heart failure
- DKA
- hypoglycemia
- hypothyroidism
- wernickes encephalopathy
- encephalitis
- meningitis
- sepsis
- hyer/hypothermia
- alcohol
- sedatives
- CO posoning

GCS definition of coma

- failure to open eyes in response to verbal stimuli (E2)
- only weak flexion as motor response (M4)
- incomprehensive sounds in response to pain (v2)

coma results from

damage to RAS in the brainste, or extensive bilat cortical damage

classification of coma

coma without focal signs or meningism
- coma with meningism
- coma with focal signs

causes of coma without focal signs or meningism

hypoxic - ischemic injury
- metabolic
- toxic
- post ictal

causes of coma with meningism

- meningoencephalitis
- SAH

causes of coma with focal signs

hemorrhage
- infarction
- abscess
- tumor
- hypoglycemia

examination of comatose pt

- quick amnamnestic data and inspection
- general examination
- neurologic exam

general exam to be done in comatose pt

- pulse - circ and frequency maintained?
- breathing
- temperature
- skin colour
- breath smell
- abdominal defence?

what can periodic breathig in a comatose pt indicate

hcaynes stokes
- brainstem lesio
- heart failure

what can slow superficial breathing indicate in a comatose pt

drugs/narcotics

what can quick superficial breathing indicate in a comatose pt

brain stem problem

parts of neurologic examination in comatose pt

menigeal signs ( only if cervical trauma can be excluded)
- fudus of eye examination
- pupil size and pupillary reflex
- position of eyes

- spontaneous eye movement
- cornea reflex
- pharyngeal and soft palate reflex
- Muscle tone
- muscle movement
- Deep reflexes
- pyramidal signs

unilateral maximal dilated pupil that do not react to light can indicate

tentorial herniation (oculomotor N)

bilateral maximal dilated pupils that do not react to light can indicate

- brainstem lesion
- herniation
- atropine-like subs

tiny-pinpoint pupils may indicate

- damage of pons
- opiods

small but reactive pupils may indicate

thalamic lesion

skew deviation of eyes may indicate

brainstem lesion

conjugate lesion of eyes may indicate

ipsilat frontal gaze center
- brainstem

decorticate M tone

hemisferic lesion

ecerebrate M tone

brainstem lesion

investigations to be done in comatose pt

- metabolic screen
- CT/MRI
- EEG
- consider lumbar puncture

T/F
a normal CT can exclude elevated ICP

FALSE

T/F
the chance of making a good recovery from coma decreases with time in coma

true