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170 Cards in this Set

  • Front
  • Back
four threats to the newborn
hypothermia

hypoxia

hypoglycemia

infection
methods of heat exchange
convection

conduction

evaporation

radiation
convection heat
heat gain/loss by air motion

loss: AC
gain: heated isolette (warm draft)
conduction heat
loss of heat to cool surface by Direct contact - cool hands, stethescompe, scales

gain: warm blankets, heat isolette pad (like a heating pad)
evaporation heat
loss: when water changes to vapor (wet from birth, baths, wet clothes, wet diapers)

gain: vapor to water (humidity provided in vent circuit)
radiation heat
transfer of heat to cooler surfaces through absorption and emission of infrared rays - not direct contact.

loss: laying next to a cold window.
Gain: next to a hot window
vernix caseosa
waxy cheeselike substance covering body
purpose to protect skin from 9 months of water exposure

starts developing at 28-29wks
infant thermogenesis
increased metabolic rate
muscular activity
chemical thermogenesis (nonshivering thermogenesis)
- this is unique to newborn. they use brown fat(colored bc it is enriched by blood supply, dense cellular content, abundant nerve endings)
- brown fat appears at 26-30wks and continues to increase until 2-5wks after birth
- brown fat deposited in midscapular area, around neck, in axillas
- deeper placement around tarachea, esoph, abd aorta, kidneys, adrenals
consequenses of low temp:
cold stress! Temp drops and they try to raise it. Have an increased 02 demand but the blood suply is low so you get hypoxic bc of peripheral and pulmonary vasoconstriction
infant stomach can hold
~ 30mL
if infant is hypoglycemic they might act
jittery or lethargic
vitamin K injection for newborns
give in L vastus lateralis muscle

purpose - clotting. since we need bacteria from the gut to make our own vitamin k, and infants don't make this until they are like 1wk old, we have to give it to them!
erythromycin med
ointment antibiotic for eyes

purpose - to prevent any opssible infection that baby could pick up on their way down the birth canal.
- gonorrhea and chlamydia can cause vision probs and blindness
physiologic wt loss for newbs

term
preterm
term - 5-10%
preterm - up to 15%
head circumference needs to be about ___ cm _____ than chest circumference
2cm larger
plethora color
red all over not only when crying
what does peeling feet mean for a newb?
baby is post term
apgar score performed when
at 1 and 5 mins
lanugo
soft downy fine hair covering baby beginning around 5 months gestation. usually shed and replaced by vellus at 33-36 wkns gestation. contribues to meconium.

presence is a sign of preemie. best place to see it is on back btwn shoulder blades
pathological jaundice
never normal
acrocyanosis
due to poor periph circulation which results in vasomotor instability and cap stasis when baby exposed to cold. common during initial 48h after birth.

if it perisists, think about periph vasoconstircion secondary to sepsis, shock, heart failure
central cyanosis
advanced hypoxia.
best assessed on tonuge and mucus membranes.
urgent investigation required if it persists beyond first few mins of life
circumoral cyanosis
bluish around mouth usually occurs in feedings, esp in preemies with poor oromotor coordination and true oxygen desat with feeds.
harlequin sign
periph cyanosis or pale on one side or one quadrant of body (red in dependent areas, cyanotic/pale in superior aspect). If baby rotates to the other side, skin color reverses. skin of affected area is warm.

seen only in immediate newborn period adn self resolves. may last 1-30 mins. ascribed to "vasomotor instability" which is a temp imbalance of the autonomic regulatory mechanism of the cutaneous vessels
mongolian spots
normal.

flat bluish/gray marks often mistaken for burises. more common in dark skin. mostly on back/butt
erythema toxicum
small yellow/white papules, surrounded by red skin. in 50% of all newborns
telanictatic nevi
flat deep pink localized areas of capilary dilation. Birth marks!
cafe au lait
cardinal dx feature of neurofibromitosis type 1 if there are 6 or more >5mm before puberty or >15mm after puberty
milia
baby acne. occurs when dead skin becomes trapped in small pockets at surface of skin. disappears within a few wks, don't squeeze them!
caput
long labor. disappears in one week
crosses suture line
cephalohematoma
trauma during birth. may perisst up to 3 months. does not cross suture line
anterior fontanel

posterior fontanel
diamond. 2-3 x 3-4cm

triangle 1-2cm
whorls
the crown swirl

develps from same embryonic cells as nervous system. abnormal placement/absence can indicate abnorm brain development.
strabismus

nystagmus
cross eyed

jerky eye
occlude one nostril at a time to check for
choanal atresia
epstien pearls
in 80% newbs

protien filled cysts. harmless but they may worry parents

disappear in 1-2wks of borth. no tx
absence of pulses or b/p can raise questions about possible
coartation of aorta
baby pmi
lateral to midclavicular line at 3 or 4 intercostal space
neonatal period
28 days
initial breath
estabs pulm ventilation through lung expansion

increase in pulm circulation
initiation of breathing
chemical, mechanical, thermal, sensory factors
chemical stimuli for breathing
stimulated to breathe bc of transitory asphyxia (bc cord is cut)

initial breath is a gasp triggered by elevation in pco2 and decrease ph and p02

changes stimulate aortic and chemoreceptors that trigger medullas resp system

catecholamine production critical in changes involved in transition to extrauterine life - stimulate CO and contractility, surfactant release and promotion of pulm fluid clearance
mechanical events initial breathing
chest is recoiled after birth of trunk which makes a negative intrathoracic pressure (makes small passive insp of air replacing fluid in lungs)

high intrathoracic pressures help distibute air througout lungs and est functional residual capacity (amount of vol left in lungs after exhalation to prevent lung collapse)
thermal stimuli
dramatic decrease in temp stims breathing! get a teensy bit of cold stress -- > need to control this so it doesn't turn to hypoxia.

skin nerve endings stimulated by cold environment (increases metabolism)
sensory stimuli breathing
tactile, auditory, and visual stim help start breathing.
- gravity, light, sound, joint movement, touching/handling infant at birth

appropriate tactile stimuli include drying infant, encouracging skin to skin with mom, promoting thermoreg
surfactant
surface active phospholipids critical for alveolar stability. lower surface tension of alveoli during exhalation.

mature lungs have lecithin and sphingomylein (L/S) ratio of 2:1
as p02 rises in alveolii, stimulation of relaxation of _________ occurs so ________ decreases
relax of pulm arteries

pulm vasucalr resistance decreases
vascular flow in lungs achieves 100% by
24h of life
delivery of greater blood vol to lung contributes to
conversion from fetal circ to newb circ
percent hgb that is fetal
70-90
percent of hgb that is adult
10-30
fetal hgb has _______ affinity to
02
periodic breathing
pauses 5-15s. should not include skin color or hr changes
breathing stopped for longer than ____ = apnea
20s
resp distress

signs
<30 or >60 bpm at rest, dyspnea, cyanosis, nasal flaring, resp grunts, retractions

nasal flarse, intercostal or subcostal retractions, grunts, abd breathing, head bob
signs of risk for cv probs in newborn
tachycardia - +160. anemia, hypovolemia, hyperthermia, sepsis

bradycardia - less than 100. congenital heart block or hypoxemia

color - pallor. anema, vasoconstriction d/t asphyxia or sepis. prolonged cyanosis other than acrocyanosis can indicate rep and/or cardiac probs
lung expansion

increases______-
decreases ___________
pulm blood flow

pulm vascular resistance
foraman ovale

fetal circ
transition
allows oxygenated blood going to RA from inferior vena cava to pass into left atrium so it can bypass pulm circulation. this lets 02 go to other parts of the fetal body esp brain

after the first breath, air pushes into the lungs triggering an increase in pulm blood flow. as a result pressure in left atrium gets higher than right atrium. the increased pressure causes FO to close, allowing output from right vent to flow entirely to lungs
ductus aortis

fetal circ
transition
protects lungs against circ overload by shunting blood from right to left in descending aorta. patency during fetal life promoted by continual production of prostaglandin (so don't give mom antiprostaglandins in last bit of preg).
want this to stay open form reemies to keep lungs from being overloaded so we give them prostaglandins

closure depends on high o2 content of aortic blood resulting from aerating lungs. pulm vasc resistance decreases so pulm blood flow is increased and 02 exchange occurs.
occurs secondary to increase in p02 coinciding with first breath and umbilical cord occlusion
ductus venosis

fetal circ
transition
shunts blood from left umb vein to IVC. plays critical role in shunting oxygenated blood to fetal brain.

closes within a few days after birth bc shunting no longer needed bc liver is activated. the liver takes over functions of placenta
hematopoietic adaptations of newb

rbc
bv
leukocytes
platelets
rbc prod diminishes after birth bc of increase in 02 production causing initial decline in hgb production = phyisologic anemia of infancy

bv 80-85ml/kg of body wt. effected by - delayed cord clamping (get blood from placenta so it's almost like autotransfusion), gestational age (preemies have less bv), prenatal hemorrhage, site of blood sample

leukocytes - wbc of about 18000 at birth and increases to 23000 at day 1. diminishes quickly and returns to birth wbc count. increase may indicate infection but some experience sepsis without a wbc increase

platelets 100,000-300,000 same as adults
renal adaptations of newb
daily fluid requirement 60-80ml/kg

babies should void within first 24h of life. if they don't in 48h it may indicate renal impairment

may pass uric crystals - brick dust spots. beware dehydration!
GI adapts of newb
digestion regulated by enzymes present at birth (except amylase and lipase)

newb requires 110 cal/kg/day to gain weight
newb iron storage
have enough to last 4-6 mo
conjuagtion of bili

physiologic jaundice
patho jaund
can cross bbb

normal. babes with larger bv are more prone

occurs before 24h or first day. means they have an rh prob
- kernicterus - can cause perm brain damage
developmental dysplasia of the hip (congenital hip dislocation)
ortolani's manuver - limited hip abduction as seen in flexion

barlow's manuver - middle finger over greater trochanter and thumb on midthigh. hip flexed to 90 degrees adn adducted followed by gentle dowward pushing of femoral head. if hip can be dislocated, femoral head moves out adn you feel a clunk
neurmusc adapts of newb
almost fully developed at birth. can have transient tremors. muscle tone adn strength directly related.
self quieting
sensory adaptation. using own resources to quiet and comfort. suckiing on fist or tongue
habituation
sensory adaptation. ability to process complex sitches. slight startle with stimulius but when repeated, may be able to diminish rxn to stim
orientation
sensory adaptation. fixate on stimuli like face, black and white images or shapes. when they hear a sound, cardiac rate rises and startle may be seen.
baby able to distinguish mom smell from other mothers.
first period of reactivity
up to 30 mins after birth. bonding so important!
hr increases to 160-180 but grad decreases.

after this period, they either sleep or have marked decrease in motor activity. this lasts for 60-100 mins
second period of reactivity
2-8h after birth lasting 10 mins to several hours.
tachycard, tachypnea, increased muscle tone. improved skin color, mucus production, meconium passed
gest age assessment completed when?

more comprehensive assessment when?
within first hours in a stable baby

within 24h
apgar score

0-3
4-6
7-10
severe distress

mod difficulty

minimal or no difficulty
normal assessment findings on newb
temp - 97.7-98.9

hr - 120-160

b not assessed right at birth bc it falls to a min by 3h after nirth then steadily climbs and plateaus at 4-6d after birth
BL measurements

wt
head circ
body length
5lb 5oz - 8lb 8oz

measure at occiptiofrontal diameter. 32-36.8cm

45-55cm
reflexes needed to have proper growth
sucking and rooting
preterm/premature
born before 37w
late preterm
born btwn 34-36.7wks
postterm

postdate
after 42 wks

after due date
umbilical cord falls off when?
10-14days bc of dry gangrene.

assess with each diaper change for edema, redness, purulent drainage

odor alone is not an indicator of ompalitis bc it deteriorates through dry gangrene

some blood can be seen in umbilicus at separation
maternal levels of ___ and ____ increase with early ___
prolactin and oxytocin

breastfeeding
active immunity for newborn begins
as infant digests antibodies from mom's colustrum
physiologic jaundice

assessment

tcB monitors

high risk if
pressure points (forehead and nose)
conjuctiva and buccal mucosa
in natural light
visual assessment not acurate

tcb monitor decresases need for serum bili levels. provides acurate measurements within 2-3 mg if serum level below 15mg/dl. not effective after light therapy started.

gest age <38w, breastfeeding, previous sib with sig jaundice
hypoglcemia

s/s

if at risk, you should be assessed withing ______
less than adequate to support neuro, organ, tissue function

tx warranted usually <40mg/dl but some recommend <50

concern about neuro injury as result of sever or prolonged hypoglycemia esp in combo with ischemia.

jittery, lethargy, poor feeds, hypotonia, temp instability (hypothermia), resp distress, apnea, siezures

first hour of life
maternal risks for hypoglycemia

neonatal risks
maternal dm, gest htn, tocolytic therapy

prematurity, lga, sga, perinatal hypoxia, infection (metab increased), hypothermia, congenital malformations
hypocalcemia

occurs in

s/s
levels <7.8 - 8mg/dl in term infants and 7mg in preemies

infants of moms with dm, perinatal asphyxia or trauma, lbw and preemies

jittery, high pitched cry, irritable, apnea, intermittent cyanosis, abd distention, laryngospasm, asymptomatic
early onset hypocalc usually resolves in

hypocalc tx
1-3d

human milk or preterm infant formula
all states screen for __ and ___
PKU and hypothyroidism
newborn hearing screening
1 in 1000 births. when id'd can help prevent early developmental delays. inital screening done with evoked otoacoustic emissions . auditory brainstem response used as follow up if screening is abnormal
heel stick

comp
repeated sticks can cause
minimizes pain and maxes accuracy of the test.

necrotizing osteochondritis resulting from lancet penetration to bone

fibrosis and scarring leading to probs with walking later in life

they need to be cuddled and comforted!
veinipuncture

can be drawn from _____
these things can affect blood gas values
apply pressure for ______ with ______
observe frequently within ______
antecub, saph, superficial wrist, scalp (rare)

crying, fear, agitation

3-5 mins with dry gauze to prevent bleeding

for 1h after assessing for bleeding or hematoma
obtaining urine specimen on newborn
should be fresh and analyzed within 1h of collection

wash genetalia thoroughly and dry.

24h urine collection - drain into receptacle at intervals and observe skin closely for irritation
IM injections on newbs
25 gauge, 5/8" needle

they don't tolerate more than 0.5ml injection

use vastus lateralis

insert at 90 degrees.
hyperbilirubinemia therapy

best is _____
prevention!
phototherapy (hyperbili)
uses energy light to change shape and structure of unconj bili and convert it to molecules that can be excreted.

closely monitor baby's temp

lights increase rate of insensible water loss -- mon dehydration

monitor urine output - can be decreased or be dark brown or gold

record number and consistency of stools - excretion can increase motility and loose stools which can break down their skin
circumcision
removes part of prepuce

3rd most common surg in US
there are potential benefits, but not enough to recommend it.
routine analgesia used.

proponents - decreasee UTIs in babies up to 1y, decrease risk penile CA, phimosis, paraphimosis, balantis, decrease stds (hpv and hiv), low rate of comps and no substantial neg effect on sexual funct

opponents - unnatural and unnecessary

risks - hemorrhage, infection, penile injury, long term probs like advers sexual funct/pleasure, acute pain, psych effects

MATTER OF PERSONAL CHOICE
Anesthesia used during circumcision

nonpharm methods
ring block
dorsal penile block
topical anesthesia
concentrated oral glucose

non nutritive sucking (shular), containment, swaddling
postop care for circumsicion
check q15-30 mins first h then hourly next 4-6h for bleeding and pain.

if bleeding occurs, gentle pressure with folded sterile gauze. if not able to control, one nurse applies pressure while another calls dr who may have to ligate a vessel

DO NOT USE COMMERCIAL WIPES they have alcohol and that delays healing and causes discomfort!
neonatal pain

pysch
physiologic
psych - they have the sturctures in place that can transmit pain around 24weeks gest

phys - the pain response can cause low tidal vol, increase demands on cv system, increase metab, neuroendocrine imbalance
neonatal infant pain scale: CRIES
Crying
Requiring more 02
Increased VS
Expression
Sleeplessness

each scored from 0-2. worst pain possible = 10. pain scale >4 = significant
local anesthesia use in babies

topical anesthesia use
chest tube insertion, circumcision

circumsicion, LP, veinipuncture, heelstick
highest maternal mortality
ruptured ectopic and abruptions
threatened miscariage
unexplained bleeding, cramps, cx closed, no dilation
inevitable miscarage
mod-heavy bleed, cramps, os dilates
incomplete miscariage
mod-heavy bleeding, os dilated, poc retained/placenta
complete miscariage
all poc expelled, uterus contracted, os may close
missed miscariage
fetus dies, not delivered, growth ceaseed, breast regress, brownish disccharge, cx closed before 20w
reucrrent
3 losses before 20w
septic miscariage
presence of infection. rupture and dont know ro rupture and didnt go to dr. (diy abortion years ago)
incompetenet cx
passive and painless dilation of cx during 2nd trimester

tx - BR, antiboitics, nsaids (indocen, motrin - stop at 32w), progesterone (maintain preg)
cerclage
11-15w prophylacticlally or rescue
cx <2-2.5 before 23-24w

tx - BR, no sex for few days, tocolytic meds, s/s for PTL, infection, ROM
cerclage immediate return to hosp -
ctx strong q5m rom, severe peritoneal pressure, urge to push --- rips cx to pieces
leading cause of infertility
ectopic preg
tubal preg
abd pain starts as a dull throb, as tube strethces it becomes shar, stabbing pain due to growth.

methotrexate - antimetabolite (interferes with growth) and folic acid antagonit (destroys rapid dividing cells)

NO ALCOHOL, NO FOLIC ACID
cullen sign
echymotic blueness at umbilicus, hematoperitoneum
gest trophoblastic
abnorm cells that would become the placenta

hydatiform mole - benign growth of placental trophoblast. grapelike cluster
partial molar preg
2 sperm and one egg so you get whole extra set of chromosomes

fetal parts and amniotic sac but never baby. often mistaken for incomplete or missed abortion. smaller vessicles.

dx transvaginal us and serum hCG
complete molar pret
1 or two sperm with empty egg - no dna material.

no fetus, placenta, amniotic membranes or fluid

early on, uterus is sig larger than expected. hypdropic vessicles grow quickly, anemic, excessive n/v, abd cramps from distention

later, prune juice color bleeding. continues for days or intermittently for weeks starting at 4wks and could last till 2nd trimester.

dx at <24wks bc many get preeclamptic

many get hyperthyroidism when having molar preg but tx restores its function
placenta previa
painless vag bleeding. placenta located in lower uterine segment or over cervical os instead of fundus. can lose upt to 40% blood without displaying signs of shock.
placenta acreta
chronic vili Attach directly to myometrium
placenta increta
myomet Invaded
placenta percreta
myomet Penetrated
placental abrubtion-
extreme pain, rock hard rigid abdomen. ctx qivering, no resting tone. vag bleeding. 40% get DIC.
marginal abruption
vag bleeding - passes btwn fetal membranes and uterine wall. separation at periph of placenta
central abruption
placenta separates centrally. blood trapped btwn placenta and uterine wall. edges intact, bleed behind (concealed)
complete abruption
massive bleed, presence of almost total separation. baby dead, placenta and him are free floating in uterus
velamentous insertion of cord
rare. assoc w previa and multiple gestation.
cord vessels branch at membranes then onto placenta.
vasa previa
presentation of umb blood vessels in advance of baby head
battledore placenta

placenta succenturiate
connects to one side. thick edge

main placenta and little lobe - still functions bc it has some blood vessels.
DIC is always a
secondary diagnosis

results from triggering clotting cascade. disruption in hemostasis in response to underlying disease or trauma.

most common cause is abruption. also undelivered fetal demise and anaphylactoid syndrome of pregnancy

tx - volume replacement rapidly!
preterm labor and birth
labor - 20-37wks gestatioon. 80% effaced, greater than or equal to 2cm dilated

birth - before 37w gest
preterm birth
length of gestation regardless birth wt

<37w

more dangerous!

causes - spontaneous, indicated (births to resolve maternal or fetal risk, preeclamp, fetal distress, abrubtion, DM, rh sensie)
LBW
only wt at birth.
2500g or less
predicting spontaenous preterm labor and birth
biochem marker - fetal fibronectin (glue like protein that you don't have till you'r ein labor. before doing a vag exam, they ask if you have bled in past 24h and had sex in last 24h then they swab till you hit fx. want it to be neg meaning you prob wont deliver in next 2w). cervical length
only known cause of preterm labor
infection. bacterial cx or uti. periodiontal infection - prostaglandin relase (ripens cx), placental site bleeding - ischemia at decidual layer of placenta may somehow activate PTL
PTL symps
empty bladder, lie down, tilt to side, drink 3-4c fluid, palpate ctx, warm tub soaks with uterus completely submerged
suppresion of uterine activty - tocolytics

yutopar

mag

terbutaline

nifedipine

indomethicin
only FDA approved. not used.

competes w calcium bc ca cant get into cells so they cant contract

beta adrenergic agonist - for asthma. use in ob bc it stims SNS

ca blocker, check for low bp

<32w. constircute of ductus arterious, oligo, neonatal pulm htn
mag sulfate

levles etc
1.6-2.4 is norm but for this case, 5-7 is norm. 4 or 6g loading dose over 20-30 min period. always give piggyback.

causes sedation, decreased reflexes, hypotension!

maintence 2g/h

excreted through kidneys. hyperfunct kidneys = mag just gets excreted.

mag 10 = DTR disappear
mag 12 = resp disappear
mag 25 = cardiac arrest
promotion of fetal lung maturity med for PTL
adraenal ccs - stresses moms body so it makes baby pruduce surfactant. want mom to stay preg at least 24h after does to take complete effect.

betamethasone or dexamethasone
choriomaniionitis
horrid smell, green pus. comp of PROM
most common indication for c/s
dystocia
version

A

B
A - breech pushed up out of pelvic inlet while head pulled toward inlet

B - head pushed toward inlet while breech pulled upward

do after 37w
induction of labor

bishop score
cervical ripening methods
higher number = more ripened cx

chemical agents - cytotec: 25-50mcg into cervix. prostaglandin that ripens cx and causes ctx and tachysystole
mechanical/physical - balloon cath - foley into cervical os, fill with 30-40 ml fluid it mechanically dilates cx
other - amniotomy, oxytocin, augment labor
forceps assisted birth

outlet
low outlet
midlpelvis
scalp visible

head at least +2 station

head engaged, station 0-2
meconioum stained amniotic fluid
emergency!
dark green - may be caused by norm phys function of maturity, breech. hypoxia, umb cord compression
shoulder dystocia
apply suprapbic pressure then macroberts manuver
insulin ________ cross placenta
does not
screen for GDM at
24-28w if no risk

BS 130+ needs more testing
norm preg woman is in resp _______
alkaloiss

02 higher, co2 lower
gest htn
increased bp with no proteinuria. in latter part of preg, resolves by 6w pp
preeclampsia
new onset htn and proteinuria >20w gestation
chronic htn with superimposed preeclamp
new onset proteinuria >20w gest. Chtn
preeclamp
vascular damage.

headaches unrelieved by tylenol, visual probs (spots, floaters, stars), rapid wt gain (5-10lb over night), facial/head edema
preeclamp pathophys
placental development altered.

endothel cell dysfunction - prostacyclin (vasodilator made by endothel cells, lowers bp).
- thromboxane dominance - PTL causes constrict of vessels and plt to clump together

uterine spiral arteries - vascular remod: widen, thick walled muscular vessels, thinner saclike vessels much larger diameter.
leaky vessels fo preeclamp
fluid leaks to tissues. hematocrit rises - intravasc dry bc of leakage into 3rd space.
preeclamp is a disaseas process affecting blood vessels, ultimately causing
leaky vessles, vasospasms, microclotting
preeclamp bp levels
proteinuria levels
systolic +140. diastolic +90

protien +300mg in 24h urine specimine or 1+ on dipstick
preeclamp may damage

kidney
liver
brain
heart
eyes
low gfr, oliguria, proteinuria, incerased serum uric acid

increaed liver enzymes

clunus, seizures, edema, hemorrhage, increaseed CNS irritability
HELLP
hemolysis
elevated liver enzymes
low platelets
ASD
left to right. most common
VSD
left to right
PDA
left to right
acyanotic
coarction of aorta
cyanotic lesion
tet of fallot
mitral valve stenosis
restrict sodium, shortening second stage o flabor
mitral valve prolapse
midsystolic click and late systolic murmur
marfans
autosomal dom genetic disorder
gen weakness of CT - joint deformities, ocular lens dislocation, weakness of aorta wall and root
peripartum cardiomyopathy
CHF with cardiomyopathy
take Fe pills when
at opposite times of prenatal vitamins bc cells dont allow anymore fe. take with vitamin C to increase absorption