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170 Cards in this Set
- Front
- Back
four threats to the newborn
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hypothermia
hypoxia hypoglycemia infection |
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methods of heat exchange
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convection
conduction evaporation radiation |
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convection heat
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heat gain/loss by air motion
loss: AC gain: heated isolette (warm draft) |
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conduction heat
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loss of heat to cool surface by Direct contact - cool hands, stethescompe, scales
gain: warm blankets, heat isolette pad (like a heating pad) |
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evaporation heat
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loss: when water changes to vapor (wet from birth, baths, wet clothes, wet diapers)
gain: vapor to water (humidity provided in vent circuit) |
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radiation heat
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transfer of heat to cooler surfaces through absorption and emission of infrared rays - not direct contact.
loss: laying next to a cold window. Gain: next to a hot window |
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vernix caseosa
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waxy cheeselike substance covering body
purpose to protect skin from 9 months of water exposure starts developing at 28-29wks |
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infant thermogenesis
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increased metabolic rate
muscular activity chemical thermogenesis (nonshivering thermogenesis) - this is unique to newborn. they use brown fat(colored bc it is enriched by blood supply, dense cellular content, abundant nerve endings) - brown fat appears at 26-30wks and continues to increase until 2-5wks after birth - brown fat deposited in midscapular area, around neck, in axillas - deeper placement around tarachea, esoph, abd aorta, kidneys, adrenals |
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consequenses of low temp:
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cold stress! Temp drops and they try to raise it. Have an increased 02 demand but the blood suply is low so you get hypoxic bc of peripheral and pulmonary vasoconstriction
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infant stomach can hold
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~ 30mL
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if infant is hypoglycemic they might act
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jittery or lethargic
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vitamin K injection for newborns
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give in L vastus lateralis muscle
purpose - clotting. since we need bacteria from the gut to make our own vitamin k, and infants don't make this until they are like 1wk old, we have to give it to them! |
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erythromycin med
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ointment antibiotic for eyes
purpose - to prevent any opssible infection that baby could pick up on their way down the birth canal. - gonorrhea and chlamydia can cause vision probs and blindness |
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physiologic wt loss for newbs
term preterm |
term - 5-10%
preterm - up to 15% |
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head circumference needs to be about ___ cm _____ than chest circumference
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2cm larger
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plethora color
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red all over not only when crying
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what does peeling feet mean for a newb?
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baby is post term
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apgar score performed when
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at 1 and 5 mins
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lanugo
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soft downy fine hair covering baby beginning around 5 months gestation. usually shed and replaced by vellus at 33-36 wkns gestation. contribues to meconium.
presence is a sign of preemie. best place to see it is on back btwn shoulder blades |
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pathological jaundice
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never normal
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acrocyanosis
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due to poor periph circulation which results in vasomotor instability and cap stasis when baby exposed to cold. common during initial 48h after birth.
if it perisists, think about periph vasoconstircion secondary to sepsis, shock, heart failure |
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central cyanosis
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advanced hypoxia.
best assessed on tonuge and mucus membranes. urgent investigation required if it persists beyond first few mins of life |
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circumoral cyanosis
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bluish around mouth usually occurs in feedings, esp in preemies with poor oromotor coordination and true oxygen desat with feeds.
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harlequin sign
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periph cyanosis or pale on one side or one quadrant of body (red in dependent areas, cyanotic/pale in superior aspect). If baby rotates to the other side, skin color reverses. skin of affected area is warm.
seen only in immediate newborn period adn self resolves. may last 1-30 mins. ascribed to "vasomotor instability" which is a temp imbalance of the autonomic regulatory mechanism of the cutaneous vessels |
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mongolian spots
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normal.
flat bluish/gray marks often mistaken for burises. more common in dark skin. mostly on back/butt |
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erythema toxicum
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small yellow/white papules, surrounded by red skin. in 50% of all newborns
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telanictatic nevi
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flat deep pink localized areas of capilary dilation. Birth marks!
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cafe au lait
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cardinal dx feature of neurofibromitosis type 1 if there are 6 or more >5mm before puberty or >15mm after puberty
|
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milia
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baby acne. occurs when dead skin becomes trapped in small pockets at surface of skin. disappears within a few wks, don't squeeze them!
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caput
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long labor. disappears in one week
crosses suture line |
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cephalohematoma
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trauma during birth. may perisst up to 3 months. does not cross suture line
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anterior fontanel
posterior fontanel |
diamond. 2-3 x 3-4cm
triangle 1-2cm |
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whorls
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the crown swirl
develps from same embryonic cells as nervous system. abnormal placement/absence can indicate abnorm brain development. |
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strabismus
nystagmus |
cross eyed
jerky eye |
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occlude one nostril at a time to check for
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choanal atresia
|
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epstien pearls
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in 80% newbs
protien filled cysts. harmless but they may worry parents disappear in 1-2wks of borth. no tx |
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absence of pulses or b/p can raise questions about possible
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coartation of aorta
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baby pmi
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lateral to midclavicular line at 3 or 4 intercostal space
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neonatal period
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28 days
|
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initial breath
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estabs pulm ventilation through lung expansion
increase in pulm circulation |
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initiation of breathing
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chemical, mechanical, thermal, sensory factors
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chemical stimuli for breathing
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stimulated to breathe bc of transitory asphyxia (bc cord is cut)
initial breath is a gasp triggered by elevation in pco2 and decrease ph and p02 changes stimulate aortic and chemoreceptors that trigger medullas resp system catecholamine production critical in changes involved in transition to extrauterine life - stimulate CO and contractility, surfactant release and promotion of pulm fluid clearance |
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mechanical events initial breathing
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chest is recoiled after birth of trunk which makes a negative intrathoracic pressure (makes small passive insp of air replacing fluid in lungs)
high intrathoracic pressures help distibute air througout lungs and est functional residual capacity (amount of vol left in lungs after exhalation to prevent lung collapse) |
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thermal stimuli
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dramatic decrease in temp stims breathing! get a teensy bit of cold stress -- > need to control this so it doesn't turn to hypoxia.
skin nerve endings stimulated by cold environment (increases metabolism) |
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sensory stimuli breathing
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tactile, auditory, and visual stim help start breathing.
- gravity, light, sound, joint movement, touching/handling infant at birth appropriate tactile stimuli include drying infant, encouracging skin to skin with mom, promoting thermoreg |
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surfactant
|
surface active phospholipids critical for alveolar stability. lower surface tension of alveoli during exhalation.
mature lungs have lecithin and sphingomylein (L/S) ratio of 2:1 |
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as p02 rises in alveolii, stimulation of relaxation of _________ occurs so ________ decreases
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relax of pulm arteries
pulm vasucalr resistance decreases |
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vascular flow in lungs achieves 100% by
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24h of life
|
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delivery of greater blood vol to lung contributes to
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conversion from fetal circ to newb circ
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percent hgb that is fetal
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70-90
|
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percent of hgb that is adult
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10-30
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fetal hgb has _______ affinity to
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02
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periodic breathing
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pauses 5-15s. should not include skin color or hr changes
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breathing stopped for longer than ____ = apnea
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20s
|
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resp distress
signs |
<30 or >60 bpm at rest, dyspnea, cyanosis, nasal flaring, resp grunts, retractions
nasal flarse, intercostal or subcostal retractions, grunts, abd breathing, head bob |
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signs of risk for cv probs in newborn
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tachycardia - +160. anemia, hypovolemia, hyperthermia, sepsis
bradycardia - less than 100. congenital heart block or hypoxemia color - pallor. anema, vasoconstriction d/t asphyxia or sepis. prolonged cyanosis other than acrocyanosis can indicate rep and/or cardiac probs |
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lung expansion
increases______- decreases ___________ |
pulm blood flow
pulm vascular resistance |
|
foraman ovale
fetal circ transition |
allows oxygenated blood going to RA from inferior vena cava to pass into left atrium so it can bypass pulm circulation. this lets 02 go to other parts of the fetal body esp brain
after the first breath, air pushes into the lungs triggering an increase in pulm blood flow. as a result pressure in left atrium gets higher than right atrium. the increased pressure causes FO to close, allowing output from right vent to flow entirely to lungs |
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ductus aortis
fetal circ transition |
protects lungs against circ overload by shunting blood from right to left in descending aorta. patency during fetal life promoted by continual production of prostaglandin (so don't give mom antiprostaglandins in last bit of preg).
want this to stay open form reemies to keep lungs from being overloaded so we give them prostaglandins closure depends on high o2 content of aortic blood resulting from aerating lungs. pulm vasc resistance decreases so pulm blood flow is increased and 02 exchange occurs. occurs secondary to increase in p02 coinciding with first breath and umbilical cord occlusion |
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ductus venosis
fetal circ transition |
shunts blood from left umb vein to IVC. plays critical role in shunting oxygenated blood to fetal brain.
closes within a few days after birth bc shunting no longer needed bc liver is activated. the liver takes over functions of placenta |
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hematopoietic adaptations of newb
rbc bv leukocytes platelets |
rbc prod diminishes after birth bc of increase in 02 production causing initial decline in hgb production = phyisologic anemia of infancy
bv 80-85ml/kg of body wt. effected by - delayed cord clamping (get blood from placenta so it's almost like autotransfusion), gestational age (preemies have less bv), prenatal hemorrhage, site of blood sample leukocytes - wbc of about 18000 at birth and increases to 23000 at day 1. diminishes quickly and returns to birth wbc count. increase may indicate infection but some experience sepsis without a wbc increase platelets 100,000-300,000 same as adults |
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renal adaptations of newb
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daily fluid requirement 60-80ml/kg
babies should void within first 24h of life. if they don't in 48h it may indicate renal impairment may pass uric crystals - brick dust spots. beware dehydration! |
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GI adapts of newb
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digestion regulated by enzymes present at birth (except amylase and lipase)
newb requires 110 cal/kg/day to gain weight |
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newb iron storage
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have enough to last 4-6 mo
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conjuagtion of bili
physiologic jaundice patho jaund |
can cross bbb
normal. babes with larger bv are more prone occurs before 24h or first day. means they have an rh prob - kernicterus - can cause perm brain damage |
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developmental dysplasia of the hip (congenital hip dislocation)
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ortolani's manuver - limited hip abduction as seen in flexion
barlow's manuver - middle finger over greater trochanter and thumb on midthigh. hip flexed to 90 degrees adn adducted followed by gentle dowward pushing of femoral head. if hip can be dislocated, femoral head moves out adn you feel a clunk |
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neurmusc adapts of newb
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almost fully developed at birth. can have transient tremors. muscle tone adn strength directly related.
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self quieting
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sensory adaptation. using own resources to quiet and comfort. suckiing on fist or tongue
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habituation
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sensory adaptation. ability to process complex sitches. slight startle with stimulius but when repeated, may be able to diminish rxn to stim
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orientation
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sensory adaptation. fixate on stimuli like face, black and white images or shapes. when they hear a sound, cardiac rate rises and startle may be seen.
baby able to distinguish mom smell from other mothers. |
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first period of reactivity
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up to 30 mins after birth. bonding so important!
hr increases to 160-180 but grad decreases. after this period, they either sleep or have marked decrease in motor activity. this lasts for 60-100 mins |
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second period of reactivity
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2-8h after birth lasting 10 mins to several hours.
tachycard, tachypnea, increased muscle tone. improved skin color, mucus production, meconium passed |
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gest age assessment completed when?
more comprehensive assessment when? |
within first hours in a stable baby
within 24h |
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apgar score
0-3 4-6 7-10 |
severe distress
mod difficulty minimal or no difficulty |
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normal assessment findings on newb
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temp - 97.7-98.9
hr - 120-160 b not assessed right at birth bc it falls to a min by 3h after nirth then steadily climbs and plateaus at 4-6d after birth |
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BL measurements
wt head circ body length |
5lb 5oz - 8lb 8oz
measure at occiptiofrontal diameter. 32-36.8cm 45-55cm |
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reflexes needed to have proper growth
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sucking and rooting
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preterm/premature
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born before 37w
|
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late preterm
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born btwn 34-36.7wks
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postterm
postdate |
after 42 wks
after due date |
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umbilical cord falls off when?
|
10-14days bc of dry gangrene.
assess with each diaper change for edema, redness, purulent drainage odor alone is not an indicator of ompalitis bc it deteriorates through dry gangrene some blood can be seen in umbilicus at separation |
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maternal levels of ___ and ____ increase with early ___
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prolactin and oxytocin
breastfeeding |
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active immunity for newborn begins
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as infant digests antibodies from mom's colustrum
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physiologic jaundice
assessment tcB monitors high risk if |
pressure points (forehead and nose)
conjuctiva and buccal mucosa in natural light visual assessment not acurate tcb monitor decresases need for serum bili levels. provides acurate measurements within 2-3 mg if serum level below 15mg/dl. not effective after light therapy started. gest age <38w, breastfeeding, previous sib with sig jaundice |
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hypoglcemia
s/s if at risk, you should be assessed withing ______ |
less than adequate to support neuro, organ, tissue function
tx warranted usually <40mg/dl but some recommend <50 concern about neuro injury as result of sever or prolonged hypoglycemia esp in combo with ischemia. jittery, lethargy, poor feeds, hypotonia, temp instability (hypothermia), resp distress, apnea, siezures first hour of life |
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maternal risks for hypoglycemia
neonatal risks |
maternal dm, gest htn, tocolytic therapy
prematurity, lga, sga, perinatal hypoxia, infection (metab increased), hypothermia, congenital malformations |
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hypocalcemia
occurs in s/s |
levels <7.8 - 8mg/dl in term infants and 7mg in preemies
infants of moms with dm, perinatal asphyxia or trauma, lbw and preemies jittery, high pitched cry, irritable, apnea, intermittent cyanosis, abd distention, laryngospasm, asymptomatic |
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early onset hypocalc usually resolves in
hypocalc tx |
1-3d
human milk or preterm infant formula |
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all states screen for __ and ___
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PKU and hypothyroidism
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newborn hearing screening
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1 in 1000 births. when id'd can help prevent early developmental delays. inital screening done with evoked otoacoustic emissions . auditory brainstem response used as follow up if screening is abnormal
|
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heel stick
comp repeated sticks can cause |
minimizes pain and maxes accuracy of the test.
necrotizing osteochondritis resulting from lancet penetration to bone fibrosis and scarring leading to probs with walking later in life they need to be cuddled and comforted! |
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veinipuncture
can be drawn from _____ these things can affect blood gas values apply pressure for ______ with ______ observe frequently within ______ |
antecub, saph, superficial wrist, scalp (rare)
crying, fear, agitation 3-5 mins with dry gauze to prevent bleeding for 1h after assessing for bleeding or hematoma |
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obtaining urine specimen on newborn
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should be fresh and analyzed within 1h of collection
wash genetalia thoroughly and dry. 24h urine collection - drain into receptacle at intervals and observe skin closely for irritation |
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IM injections on newbs
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25 gauge, 5/8" needle
they don't tolerate more than 0.5ml injection use vastus lateralis insert at 90 degrees. |
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hyperbilirubinemia therapy
best is _____ |
prevention!
|
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phototherapy (hyperbili)
|
uses energy light to change shape and structure of unconj bili and convert it to molecules that can be excreted.
closely monitor baby's temp lights increase rate of insensible water loss -- mon dehydration monitor urine output - can be decreased or be dark brown or gold record number and consistency of stools - excretion can increase motility and loose stools which can break down their skin |
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circumcision
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removes part of prepuce
3rd most common surg in US there are potential benefits, but not enough to recommend it. routine analgesia used. proponents - decreasee UTIs in babies up to 1y, decrease risk penile CA, phimosis, paraphimosis, balantis, decrease stds (hpv and hiv), low rate of comps and no substantial neg effect on sexual funct opponents - unnatural and unnecessary risks - hemorrhage, infection, penile injury, long term probs like advers sexual funct/pleasure, acute pain, psych effects MATTER OF PERSONAL CHOICE |
|
Anesthesia used during circumcision
nonpharm methods |
ring block
dorsal penile block topical anesthesia concentrated oral glucose non nutritive sucking (shular), containment, swaddling |
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postop care for circumsicion
|
check q15-30 mins first h then hourly next 4-6h for bleeding and pain.
if bleeding occurs, gentle pressure with folded sterile gauze. if not able to control, one nurse applies pressure while another calls dr who may have to ligate a vessel DO NOT USE COMMERCIAL WIPES they have alcohol and that delays healing and causes discomfort! |
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neonatal pain
pysch physiologic |
psych - they have the sturctures in place that can transmit pain around 24weeks gest
phys - the pain response can cause low tidal vol, increase demands on cv system, increase metab, neuroendocrine imbalance |
|
neonatal infant pain scale: CRIES
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Crying
Requiring more 02 Increased VS Expression Sleeplessness each scored from 0-2. worst pain possible = 10. pain scale >4 = significant |
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local anesthesia use in babies
topical anesthesia use |
chest tube insertion, circumcision
circumsicion, LP, veinipuncture, heelstick |
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highest maternal mortality
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ruptured ectopic and abruptions
|
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threatened miscariage
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unexplained bleeding, cramps, cx closed, no dilation
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inevitable miscarage
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mod-heavy bleed, cramps, os dilates
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incomplete miscariage
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mod-heavy bleeding, os dilated, poc retained/placenta
|
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complete miscariage
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all poc expelled, uterus contracted, os may close
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missed miscariage
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fetus dies, not delivered, growth ceaseed, breast regress, brownish disccharge, cx closed before 20w
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reucrrent
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3 losses before 20w
|
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septic miscariage
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presence of infection. rupture and dont know ro rupture and didnt go to dr. (diy abortion years ago)
|
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incompetenet cx
|
passive and painless dilation of cx during 2nd trimester
tx - BR, antiboitics, nsaids (indocen, motrin - stop at 32w), progesterone (maintain preg) |
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cerclage
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11-15w prophylacticlally or rescue
cx <2-2.5 before 23-24w tx - BR, no sex for few days, tocolytic meds, s/s for PTL, infection, ROM |
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cerclage immediate return to hosp -
|
ctx strong q5m rom, severe peritoneal pressure, urge to push --- rips cx to pieces
|
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leading cause of infertility
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ectopic preg
|
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tubal preg
|
abd pain starts as a dull throb, as tube strethces it becomes shar, stabbing pain due to growth.
methotrexate - antimetabolite (interferes with growth) and folic acid antagonit (destroys rapid dividing cells) NO ALCOHOL, NO FOLIC ACID |
|
cullen sign
|
echymotic blueness at umbilicus, hematoperitoneum
|
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gest trophoblastic
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abnorm cells that would become the placenta
hydatiform mole - benign growth of placental trophoblast. grapelike cluster |
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partial molar preg
|
2 sperm and one egg so you get whole extra set of chromosomes
fetal parts and amniotic sac but never baby. often mistaken for incomplete or missed abortion. smaller vessicles. dx transvaginal us and serum hCG |
|
complete molar pret
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1 or two sperm with empty egg - no dna material.
no fetus, placenta, amniotic membranes or fluid early on, uterus is sig larger than expected. hypdropic vessicles grow quickly, anemic, excessive n/v, abd cramps from distention later, prune juice color bleeding. continues for days or intermittently for weeks starting at 4wks and could last till 2nd trimester. dx at <24wks bc many get preeclamptic many get hyperthyroidism when having molar preg but tx restores its function |
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placenta previa
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painless vag bleeding. placenta located in lower uterine segment or over cervical os instead of fundus. can lose upt to 40% blood without displaying signs of shock.
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placenta acreta
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chronic vili Attach directly to myometrium
|
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placenta increta
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myomet Invaded
|
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placenta percreta
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myomet Penetrated
|
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placental abrubtion-
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extreme pain, rock hard rigid abdomen. ctx qivering, no resting tone. vag bleeding. 40% get DIC.
|
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marginal abruption
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vag bleeding - passes btwn fetal membranes and uterine wall. separation at periph of placenta
|
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central abruption
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placenta separates centrally. blood trapped btwn placenta and uterine wall. edges intact, bleed behind (concealed)
|
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complete abruption
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massive bleed, presence of almost total separation. baby dead, placenta and him are free floating in uterus
|
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velamentous insertion of cord
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rare. assoc w previa and multiple gestation.
cord vessels branch at membranes then onto placenta. |
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vasa previa
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presentation of umb blood vessels in advance of baby head
|
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battledore placenta
placenta succenturiate |
connects to one side. thick edge
main placenta and little lobe - still functions bc it has some blood vessels. |
|
DIC is always a
|
secondary diagnosis
results from triggering clotting cascade. disruption in hemostasis in response to underlying disease or trauma. most common cause is abruption. also undelivered fetal demise and anaphylactoid syndrome of pregnancy tx - volume replacement rapidly! |
|
preterm labor and birth
|
labor - 20-37wks gestatioon. 80% effaced, greater than or equal to 2cm dilated
birth - before 37w gest |
|
preterm birth
|
length of gestation regardless birth wt
<37w more dangerous! causes - spontaneous, indicated (births to resolve maternal or fetal risk, preeclamp, fetal distress, abrubtion, DM, rh sensie) |
|
LBW
|
only wt at birth.
2500g or less |
|
predicting spontaenous preterm labor and birth
|
biochem marker - fetal fibronectin (glue like protein that you don't have till you'r ein labor. before doing a vag exam, they ask if you have bled in past 24h and had sex in last 24h then they swab till you hit fx. want it to be neg meaning you prob wont deliver in next 2w). cervical length
|
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only known cause of preterm labor
|
infection. bacterial cx or uti. periodiontal infection - prostaglandin relase (ripens cx), placental site bleeding - ischemia at decidual layer of placenta may somehow activate PTL
|
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PTL symps
|
empty bladder, lie down, tilt to side, drink 3-4c fluid, palpate ctx, warm tub soaks with uterus completely submerged
|
|
suppresion of uterine activty - tocolytics
yutopar mag terbutaline nifedipine indomethicin |
only FDA approved. not used.
competes w calcium bc ca cant get into cells so they cant contract beta adrenergic agonist - for asthma. use in ob bc it stims SNS ca blocker, check for low bp <32w. constircute of ductus arterious, oligo, neonatal pulm htn |
|
mag sulfate
levles etc |
1.6-2.4 is norm but for this case, 5-7 is norm. 4 or 6g loading dose over 20-30 min period. always give piggyback.
causes sedation, decreased reflexes, hypotension! maintence 2g/h excreted through kidneys. hyperfunct kidneys = mag just gets excreted. mag 10 = DTR disappear mag 12 = resp disappear mag 25 = cardiac arrest |
|
promotion of fetal lung maturity med for PTL
|
adraenal ccs - stresses moms body so it makes baby pruduce surfactant. want mom to stay preg at least 24h after does to take complete effect.
betamethasone or dexamethasone |
|
choriomaniionitis
|
horrid smell, green pus. comp of PROM
|
|
most common indication for c/s
|
dystocia
|
|
version
A B |
A - breech pushed up out of pelvic inlet while head pulled toward inlet
B - head pushed toward inlet while breech pulled upward do after 37w |
|
induction of labor
bishop score cervical ripening methods |
higher number = more ripened cx
chemical agents - cytotec: 25-50mcg into cervix. prostaglandin that ripens cx and causes ctx and tachysystole mechanical/physical - balloon cath - foley into cervical os, fill with 30-40 ml fluid it mechanically dilates cx other - amniotomy, oxytocin, augment labor |
|
forceps assisted birth
outlet low outlet midlpelvis |
scalp visible
head at least +2 station head engaged, station 0-2 |
|
meconioum stained amniotic fluid
|
emergency!
dark green - may be caused by norm phys function of maturity, breech. hypoxia, umb cord compression |
|
shoulder dystocia
|
apply suprapbic pressure then macroberts manuver
|
|
insulin ________ cross placenta
|
does not
|
|
screen for GDM at
|
24-28w if no risk
BS 130+ needs more testing |
|
norm preg woman is in resp _______
|
alkaloiss
02 higher, co2 lower |
|
gest htn
|
increased bp with no proteinuria. in latter part of preg, resolves by 6w pp
|
|
preeclampsia
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new onset htn and proteinuria >20w gestation
|
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chronic htn with superimposed preeclamp
|
new onset proteinuria >20w gest. Chtn
|
|
preeclamp
|
vascular damage.
headaches unrelieved by tylenol, visual probs (spots, floaters, stars), rapid wt gain (5-10lb over night), facial/head edema |
|
preeclamp pathophys
|
placental development altered.
endothel cell dysfunction - prostacyclin (vasodilator made by endothel cells, lowers bp). - thromboxane dominance - PTL causes constrict of vessels and plt to clump together uterine spiral arteries - vascular remod: widen, thick walled muscular vessels, thinner saclike vessels much larger diameter. |
|
leaky vessels fo preeclamp
|
fluid leaks to tissues. hematocrit rises - intravasc dry bc of leakage into 3rd space.
|
|
preeclamp is a disaseas process affecting blood vessels, ultimately causing
|
leaky vessles, vasospasms, microclotting
|
|
preeclamp bp levels
proteinuria levels |
systolic +140. diastolic +90
protien +300mg in 24h urine specimine or 1+ on dipstick |
|
preeclamp may damage
kidney liver brain heart eyes |
low gfr, oliguria, proteinuria, incerased serum uric acid
increaed liver enzymes clunus, seizures, edema, hemorrhage, increaseed CNS irritability |
|
HELLP
|
hemolysis
elevated liver enzymes low platelets |
|
ASD
|
left to right. most common
|
|
VSD
|
left to right
|
|
PDA
|
left to right
|
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acyanotic
|
coarction of aorta
|
|
cyanotic lesion
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tet of fallot
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mitral valve stenosis
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restrict sodium, shortening second stage o flabor
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mitral valve prolapse
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midsystolic click and late systolic murmur
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marfans
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autosomal dom genetic disorder
gen weakness of CT - joint deformities, ocular lens dislocation, weakness of aorta wall and root |
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peripartum cardiomyopathy
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CHF with cardiomyopathy
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take Fe pills when
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at opposite times of prenatal vitamins bc cells dont allow anymore fe. take with vitamin C to increase absorption
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