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164 Cards in this Set
- Front
- Back
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DESTRUCTION OF THE LMN
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ALS
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TRANSMISSION ACROSS SYNAPTIC CLEFT
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MYASTHENIA GRAVIS
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TEMPORARILY DAMAGED MYELIN SHEATHS
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GUILLIAN BARRE
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DESTRUCTION OF DOPAMINE PRODUCTION
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PARKINSONS
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DEMYELINZATION AND SCARRING
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MS
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DIFFICULTY SWALLOWING
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DYSPHAGIA
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NASAL SPEECH
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DYSPHONIA
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DOUBLE VISION
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DIPLOPIA
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TWITCHING OF THE MUSCLE
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FASCICULATION
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CLUMSY JERKING GAIT
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ATAXIA
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MUSCLE RELAXANT
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BACOLFEN, VALIUM
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DRUG CLASS USED FOR DROOLING
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ANTICHOLINERGICS
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ANTIDOTE FOR MESTINON
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ATROPINE
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SHUTS DOWN THE INFLAMMATION AND DECREASES RELAPSE
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BETA INTERFERON PRODUCTS
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NO EFFECT ON FREQUENCY OF ATTACK OR PROGRESSION IN MS
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STEROIDS
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WILL SHOW PLAQUES
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CT OR MRI
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LAB USED TO ASSESS RESPIRATORY FUNCTION
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ABG
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TEST INNERVATIONS OF MUSCLES BY NERVES
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EMG
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WILL SHOW INCREASED STRENGTH IF THIS SUBSTANCE IS GIVEN
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TENSILON TEST
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WILL SHOW INCREASED PROTEIN WBC AND T CELLS
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SPINAL FLUID
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EXACERBATIONS AND REMISSIONS
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MS
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SYMPTOMS WORSEN DURING THE DAY
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MG
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RELENTLESS AND PROGRESSIVE
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ALS
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DISABILITY IN 10-20 YEARS
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PARKINSONS
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FULL RECOVERY IN 1-2 YEARS
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GB
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DECREASED VITAL CAPACITY
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RESPIRATORY DISTRESS
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VOIDING SMALL AMOUNTS
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URINARY RETENTION
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FEVER PRODUCTIVE COUGH
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PNEUMONIA
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IRRITABILITY, BRADYCARDIA, INCREASED SALIVATION
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CHOLINERGIC CRISIS
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ARRHYTHMIAS, BRADYCARDIA, BLOOD PRESSURE INSTABILITY
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SYMPATHETIC AND PARA EFFECTS IN GB
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TAKE SMALL BITES EAT SLOW
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ASPIRATION PREVENTION
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USE CANE OR ASSISTIVE DEVICE
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FALL PREVENTION
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BLINKING, LETTER BOARD
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COMMUNICATION
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FLUIDS, FIBER, INCREASED ACTIVITY
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CONSTIPATION
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DAILY ROUTINE LISTS
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MEMORY WITH MS
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HELPS WITH MOBILITY AND DECREASES RIGIDITY
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LEVODOPA PRODUCTS
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PROLONGS ACH AT NEUROMUSCULAR JUNCTION
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MESTINON
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CAN CAUSE HEMORRHAGIC CYSTITIS
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IMMURAN
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LOOKS LIKE MYELIN
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COPAXANE
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CARDIO TOXIC CHEMO DRUG
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NIVANTRONE
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ROM AND POSITIONING
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CONTRACTURE PREVENTION
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TAKE MEDS 30-60 MIN BEFORE MEALS
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IMPROVE SWALLOWING
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DRUG NOT TO GIVE TO MG PATIENTS
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MUSCLE RELAXANTS
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DECREASE PROTEIN AND B6
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TO IMPROVE EFFECTIVENESS OF LEVADOPA
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AVOID EXTREMES IN TEMPERATURE
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TO AVOID MS ATTACKS
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SLOWNESS OF MOVEMENT
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BRADYKINESIA
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REMOVES ANTIGEN ANTIBODY COMPLEXES
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PLASMAPHERESIS
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DYSKINESIAS AND HYPOTENSION
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SIDE EFFECTS OF LEVODOPA PRODUCTS
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REPOSITIONING, DISTRACTION AND HOT\COLD
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NON PHARM TX FOR PAIN IN GB
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COMMON CAUSE OF DEATH WITH PARKINSON'S
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PNEUMONIA
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DEFICIENCY OF DOPAMINE
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PARKINSONS
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LETTER, BOARDS FINGER MOVEMENT AND EYE BLINKS MAY BE USED TO FACCILITATE THIS
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COMMUNICATION
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SYMPTOMS WORSEN AS THE DAY GOES ON
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MYASTHENIA
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DRUG USED TO TEST FOR MYASTHENIA GRAVIS
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TENSILON
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MAY SHOW PLAQUES ASSOCIATED WITH MS
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MRI
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FILTERS BLOOD TO REMOVE LARGE COMPLEXES
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PLASMPHERESIS
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EXCESS OF THIS CAUSES SPASMS, INCREASED BRONCHIAL SECRETIONS
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CHOLINERGIC
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AFFECTS MOTOR NEURONS
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ALS
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MUST MONITOR FREQUENTLY IN GB
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RESPIRATORY
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DECREASES DAMAGE AND RELAPSE RATE IN MS
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BETASERON
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MAY HAVE HAD THIS IN THE WEEKS PRIOR TO GB
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VIRUS
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NEEDED TO ACTIVATE MUSCLE CONTRACTION
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ACETYLCHOLINE
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CAUSES DEMYELINATION AND SCARRING
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MS
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VOICE CHANGES
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DYSPHONIA
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MUST MONITOR FOR IF TAKING IMMURAN
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INFECTION
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CHIN TUCK, SMALL BITES AND SITTING UPRIGHT WILL HELP THIS
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SWALLOWING
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ASCENDING WEAKNESS AND PARALYSIS
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GB
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NEUROLOGIC ASSESSMENT TOOL
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GLASGOW COMA
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OSMOTIC DIURETIC USED TO TREAT BRAIN SWELLING
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MANNITOL
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SIDE OF BRAIN THAT CONTROLS RIGHT MOTOR FUNCTION
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LEFT SIDE
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WATER IN THE BRAIN
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CEREBRAL EDEMA
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MOST COMMON CAUSE STROKE
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HYPERTENSION
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TYPE OF STROKE WHERE YOU BLEED IN THE BRAIN
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HEMORRHAGIC
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80% OF THE VOLUME IS CONTAINED BY THE SKULL
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BRAIN TISSUE
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PART OF BRAIN THAT CONTROLS BREATHILNG, HEART RATE, CONSCIOUSNESS
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BRAINSTEM
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PRIMARY EXAM USED TO DX STROKE
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CAT SCAN
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STEROID MEDICATION USED TO TX BRAIN SWELLING
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DECADRON
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STIFF NECK, CHANGE IN LOC COULD BE A SIGN OF THIS
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MENINGITIS
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TYPE OF CELL THAT HIV ATTACTS
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T-4 LYMPHOCYTE
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SYSTEM AFFECTED BY LONG TERM USE OF MEDS FOR HIV
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CARDIOVASCULAR
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COMBINATION OF DRUGS TO TX HIV
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HARRT
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TYPE OF PNEUMONIA WITH AIDS
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PNEUMOCYSTIS
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WHAT THE EIA ENZYME IMMUNOASSAY DETECTS
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ANTIBODIES
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VAGINAL OR ORAL INFECTION
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CANDIDIASIS
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CATEGORY OF INFECTIONS PTS WITH HIV GET
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OPPORTUNISTIC
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IMPORTANT TO TAKE AS DIRECTED WITH HIV
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MEDS
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WILL REDUCE RISK OF HIV
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CONDOMS
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TYPE OF LYMPHOMA WITH AIDS
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SARCOMA
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NEGATIVE SKIN TEST CAN NOT RULE THIS OUT IN HIV
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TUBERCULOSIS
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CAN CAUSE SKIN IRRITAION AND MALNUTRITION
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DIARRHEA
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TYPE OF INFLAMMATORY ARTHRITIS
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RHEUMATOID
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PROTEIN, RED CELLS AND CASTS INDICATE A PROBLEM WITH
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RENAL
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HARSH HAIR PRODUCTS CAN LEAD TO THIS
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ALOPECIA
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TISSUE AFFECTED BY RA
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SYNOVIAL
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MUST WATCH FOR VISUAL PROBLEMS WITH THIS DRUG
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PLAQUENIL
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WHY PTS WITH LUPUS MUST AVOID SUNLIGHT
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PHOTOSENSITIVITY
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RESULT OF SYSTEMIC EFFECTS OF RA
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ANEMIA
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LAB TEST THAT INCREASES WITH A FLARE
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ESR
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TYPE OF RASH WITH LUPUS
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BUTTERFLY
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TINNITUS
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SIDE EFFECT OF ASA THERAPY
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STEROIDS REDUCE THIS, BUT MAY CAUSE THIS
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INFLAMATION, HYPERGLYCEMIA
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MUST MONITOR FOR THIS IF ON IMMUNE SUPPRESSANTS
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INFECTION
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GIVEN SQ, INHIBITS TUMOR NECROSIS FACTOR
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ETANERCEPT
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CONTROLS MOTOR FUNCTION OF THE LEFT SIDE OF THE BODY
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RIGHT BRAIN
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CONTROLS MOTOR FUNCTION ON THE RIGHT SIDE OF THE BODY
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LEFT BRAIN
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CONTROLS EMOTIONS, THINKING SKILLS, NONVERBAL COMMUNICATION AND SPATIAL ORIENTATION OR SENSE OF BODY POSITION
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RIGHT BRAIN
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MEMORY AND JUDGMENT FOR SAFETY COULD BE AFFECTED IN ?
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RIGHT BRAIN INJURY
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LANGUAGE IS CONTROLLED BY THE ____ SIDE OF THE BRAIN.
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LEFT
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MOOD, BEHAVIOR AND LANGUAGE SKILLS WILL BE AFFECTED IN?
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LEFT BRAIN INJURY
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INITIATION OF ACTIVITIES, PROBLEM SOLVING, JUDGMENT, PERSONALITY, EMOTIONS, AWARENESS OF ABILITIES, LIMITATIONS, ORGANIZATION, CONCENTRATION, EXPRESSIVE LANGUAGE
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FRONTAL LOBE
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MEMORY, HEARING, UNDERSTANDING LANGUAGE, ORGANIZATION AND SEQUENCING
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TEMPORAL LOBE
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VISION
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OCCIPITAL LOBE
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BALANCE, COORDINATION, SKILLED MOVEMENT ACTIVITIES
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CEREBELLUM
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BREATHING, HEART RATE, CONSCIOUSNESS, AROUSAL, SLEEP, WAKE FUNCTIONS, ATTENTION, CONCENTRATION
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BRAIN STEM
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MOST COMMON MOTOR NEURON DISEASE
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ALS OR LOU GEHRIG'S DISEASE
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MORE IN MEN THEN WOMEN, AGES 40-70, THEORIES INCLUDE HEAVY METALS, VIRUS, LYMPHOMA, HIV, GENETICS, AUTOIMMUNE,
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ALS
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LIFE EXPECTANCY ONLY 2-5 YEARS
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ALS
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DEMYELINIZATIN OCCURS IN UMN AND LMN CAUSING DESTRUCTION, AFFECTS MUSCLES OF RESPIRATION
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ALS
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DESTRUCTION OF THE ACETYLCHOLINE RECEPTORS ON THE MUSCLES SO THE ACETYLCHOLINE CAN'T ACTIVATE MUSCLE CONCRTACTION. ACETYLCHOLINE COMES FROM THE NERVS
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MYASTHENIA
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DEGENERATIVE CHANGES IN THE CUBSTANTIA NIGRA AND OTHER PORTIONS OF THE BRAIN LEAD TO DECREASED PRODUCTION OF DOPAMINE. THE USUAL BALANCE OF ACTIVITY BETWEEN DOPAMINE AND ACETYLCHOLINE IN THE BRAIN IS DISRUPTED; ACETYLCHOLINE IS NO LONGER INHIBITED BY DOPAMINE. THIS IMBALANCE LEADS TO MANIFESTATIONS OF THE DISORDER
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PARKINSONS
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AN ACUTE POLYNEUROPATHY CHARACTERIZED BY INFLAMMATIN AND DEMYELINIZATION. IT RESULTS IN AREFLEXIC MOTOR PARALYSIS WITH POSSIBLE SENSORY DEFICITS.
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GUILLAIN BARRE
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CHARACTERIZED BY INFLAMMATORY DEMYELINATION AND GLIOSIS (SCARRING) OF THE CENTRAL NERVOUS SYSTEM
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MS
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WEAKNESS, WASTING AND ATROPHY OF THE AFFECTED MUSCLES. SENSATION AND MENTAL STATUS IS UNCHANGED, UPPER BODY THEN LOWER BODY. PATIENTS NOTICE CLUMSINESS, TRIPPING AND FATIGUE
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ALS
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DIPLOPIA AND PTOSIS ARE EARLY SX OF?
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MYASTHENIA
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TIREDNESS AND MOVING MORE SLOWLY, PILL ROLLING, TREMOR AT REST THAT WORSENS WITH STRESS. DECREASED FINE MOTOR DEXTERITY, WRITING, EATING AND DRESSING
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PARKINSONS
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MUSCLE WEAKNESS AND PAIN, PAIN, PAIN PROGRESSING FLACCID PARALYSIS THAT BEGINS DISTALLY AND PROGRESSES IN A PROXIMAL PATTERN
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GUILLAIN BARRE
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FATIGUE, WEAKNESS, STIFF UNSTEADY GAIT, CLUMSY, ATAXIA, DYSARTHRIA, FLEXOR SPASMS AT NIGHT, INTENTION TREMOR, INCREASED REFLEXES, POSITIVE BABINSKI, BLURRED VISION, DIPLOPIA, DECREASED VISUAL ACUITY
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MS
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R/O OTHER DX, EMG SHOWS MUSCLE DENERVATION WITH FIBRILLATION AND FASCICULATIONS, SERUM, CPK MAY BE ELEVATED, MUSCLE BIOPSY
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ALS
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IV ENDROPHONIUM (TENSILON) CONFIRMS DX BY IMMEDIATELY IMPROVING MUSCLE WEAKNESS, SX LASTS FIVE MINUTES. ELEVATED ACH RECEPTOR ANTIBODY TITER
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MYASTHENIA
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THERE IS NO TEST THAT CLEARLY DIFFERENTIATES FROM OTHER NEUROLOGIC DISORDERS. POSITRON EMISSION TOMOGRAPHY SCAN WILL SHOW DECREASED UPTAKE OF FLUORO DOPA
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PARKINSONS
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CEREBROSPINAL FLUID ANALYSIS, ELECTROMYOGRAPHY WILL DX
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GUILLAIN BARRE
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MRI, CEREBRAL FLUID ANALYSIS, CT, PET, AND EVOKED RESPONSE TESTING WILL DX
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MS
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RILUZONE (RILUTEK) FOR EARLY SX, ANTI-GLUTAMATE. ANTIVIRALS, PLASMAPHORESIS, ANTICHOLINERGICS FOR DROOLING, QUININE FOR MUSCLE CRAMPS
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ALS
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MESTINON, MYTELEASE, PROSTIGMIN, ANTICHOLINESTERASE AGENTS, PREDNISONE, IMURAN, PLASMAPHERESIS, THYMECTOMIES
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MYASTHENIA
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LEVODOPA, CARBIDOPA, CATECHOL OMETHYLTRANSFERASE INHIBITORS, MONOAMINE OXIDASE INHIBITORS, DOPAMINE AGONISTS, ANTICHOLINERGICS
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PARKINSONS
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PLASMAPHERESIS OR IV IMMUNOGLOBULIN PLASMA EXHANGE
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GUILLAIN BARRE
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INTERFERON, BETASERON, AVONEX, REBIF, COPAXONE AMINO ACIDS, TYSABRI MONOCLONAL ANTIBODY, IMURAN, CYTOXAN, NIVANTRONE, MUSCLE RELAXANTS, BACLOFEN, VALIUM
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MS
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SENSORY, COGNITIVE AND SPHINCTER FUNCTION REMAIN INTACT THROUGHOUT THE DISEASE. DEATH USUALLY RESULTS IN 2-5 YEARS AFTER ONSET, USUALLY FROM RESPIRATORY FAILURE.
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ALS
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APPROXIMATELY 75% OF CLIENTS HAVE DYSPLASIA OF THE THYMUS GLAND, THYMECTOMY IS OFTEN PERFORMED WITHIN THE FIRST 2 YEARS OF DX. MORE THAN HALF OF PTS IMPROVE AFTER A THYMECTOMY
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MYASTHENIA
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TOTAL DISABILITY IN 10-20 YEARS. DEATH FROM PNEUMONIA
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PARKINSONS
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ABOUT 30% REQUIRE VENTILATORY SUPPORT DUE TO RESPIRATORY MUSCLE PARALYSIS. RECOVERY IS SPONTANEOUS AND COMPLETE IN 80% -90% OF AFFECTED PERSONS. NEUROLOGIC DEFICITS SUCH AS WEAKNESS MAY PERSIST, ESPECIALLY N THE LOWER EXTREMITIES
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GUILLAIN BARRE
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FOLLOWS A PROGRESSIVE, RELAPSING, REMITTING COURSE. DURING REMISSION, REMYELINATION CAN OCCCUR AND SX IMPROVE. THE DX USUALLY PROGRESSES SLOWLY WITH LONG PERIODS OF REMISSION, BUT MAY PROGRESS RAPIDLY IN SOME PEOPLE.
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MS
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5-10% OF CLIENTS INHERIT IT AS AN AUTOSOMAL DOMINANT TRAIT, THE MAJORITY OF CASES OCCUR RANDOMLY. IN MOST THE CAUSE IS UNKNOWN, ABNORMAL GLUTAMATE METABOLISM AND HYDROGEN PEROXIDE PRODUCTION ARE BEING STUDIED. VIRAL AND ENVIRONMENTAL FACTORS ARE BEING RESEARCHED
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ALS
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DESTRUCTION OF ACH RECEPTORS ON SKELETAL MUSCLE CELL MEMBRANE OF MYONEURAL JUNCTION, MOST COMMON FORM. RARE GENETIC TYPES, OTHER IMMUNE DISORDERS, THYMOMAS, AND SMALL CELL CARCINOMA OF LUNG MAY COEXIST
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MYASTHENIA
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THE CAUSE IS UNKNOWN, GENETIC FACTORS MAY PLAY A ROLE, SECONDARY CAN RESULT FROM TRAUMA OR INSULT
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PARKINSONS
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USUALLY FOLLOWS A VIRAL INFECTION BY ABOUT 1-3 WEEKS, MOST OFTEN WITH EPSTEIN BARR VIRUS OR CYTOMEGALOVIRUS
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GUILLAIN BARRE
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BELIEVED TO RESULT FROM AN AUTOIMMUNE RESPONSE TO A PRIOR VIRAL INFECTION IN A GENETICALLY SUSCEPTIBLE PERSON, THE INFECTION, WHICH IS THOUGHT TO OCCUR EARLY IN LIFE, ACTIVATES T-CELLS
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MS
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MUSCLE ATROPHY
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FLACCIDITY
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TWITCHING
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FASCICULATION
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FLACCIDITY, FASCICULATION (early), DECREASED DTR, MUSCLE CRAMPING, GENERALIZED FATIGUE, PROGRESSIVE AND RELENTLESS COURSE
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ALS
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UMN LESIONS CAUSE HYPERACTIVE DTR +BABINSKI, JAW CLONUS, TONGUE FASCICULATION, SLURRED SPEECH, DYSPHAGIA, DYSARTHRIA, DIMINISHED GAG AND COUGH REFLEX
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ALS
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TX- FAMILY AND PT SUPPORT, ENCOURAGE END OF LIFE DECISIONS, VENTILATOR, ENTERAL FEEDINGS ARE HEARTBREAKING TO FACE, NOT MUCH PHARM SUPPORT
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ALS
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RILUZONE (RILUTEK) FOR EARLY SX ANTIGLUTAMATE, ANTIVIRALS, PLASMAPHORESIS, ANTICHOLINERGICS FOR DROOLING (ARTANE, COGENTIN, BENADRYL) QUININE FOR MUSCLE CRAMPS, PROMOTE GENERAL HEALTH
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ALS
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RISK FOR ASPIRATION, INEFFECTIVE AIRWAY CLEARANCE, ALTERED NUTRITION, INEFFECTIVE COPING
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NURSING DX ALS
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ENCOURAGE TUCKED CHIN POSITION WHILE EATING, KEEP PT UPRIGHT WHILE EATING, OBSERVE FOR BREATHING PATTERN CHANGES, ASSESS LUNGS, PAPASE ENZYME PRIOR TO EATING REDUCES STICKY SALIVA
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ALS
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SMALL MEALS, SOFT CONSISTENCY, THICKENERS, ALLOW PLENTY OF TIME TO EAT, AVOID EXTREME FOOD TEMPERATURES
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ALS
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REFERRAL TO SUPPORT GROUP THROUGH MD ASSOCIATION
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ALS SUPPORT GROUP
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AUTOIMMUNE DISEASE THAT AFFECTS THE NEUROMUSCULAR TRANSMISSION OF IMPULSES ACROSS THE SYNAPTIC CLEFT
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MYASTHENIA
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PRESENTS AS MUSCULAR WEAKNESS AND FATIGUE WHICH WORSENS AS THE DAY PROGRESSES AND IMPROVES WITH REST
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MYASTHENIA
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DESTRUCTION OF THE ACH RECEPTORS ON THE MUSCLES SO THE ACH CAN'T ACTIVATE MUSCLE CONTRACTION, ACETYLCHOLINE COMES FROM THE NERVES
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MYASTHENIA
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ANTIBODIES ARE FORMED AGAINST ACH RECEPTORS, 25% HAVE THYOMAS, 80% SHOW THYMUS CHANGES, INFANTS OF AFFECTED MOTHERS SHOW SX AT BIRTH BUT DISSIPATE AFTER SEVERAL WEEKS
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MYASTHENIA
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MOTOR D\O, EXTREME MUSCLE WEAKNESS AND EASILY FATIGUED, DIPLOPIA AND PTOSIS ARE EARLY SX, DYSPHONIA, DYSPHAGIA WILL INCREASE ASPIRATION RISK, UPPER EXTREMITIES USUALLY WEAKER THAN LOWER, FINE MOTOR PROBLEMS (DEXTARITY)
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MYASTHENIA
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PROGRESSIVE WEAKNESS OF DIAPHRAGM AND INTERCOSTAL MUSCLES MAY PRODUCE RESPIRATORY FAILURE, NASAL QUALITY TO SPEECH, SLEEPY, MASK LIKE FACE
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MYASTHENIA
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DX BASED ON H&P, IV EDROPHONIUM (TENSILON) CONFIRMS BY IMPROVING MUSCLE WEAKNESS, LASTS FIVE MIN., ELEVATED ACH RECEPTOR ANTIBODY TITIER
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MYASTHENIA
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