• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/34

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

34 Cards in this Set

  • Front
  • Back
EFE
endocardial fibroelastosis:
-hypertrophy of the wall of the LV and conversion of the endocardium into a thick fibroelastic coat, resiulting is a stiff heart(restrictive)
what are the signs and symptoms of endocardiaofibroelastosis?
-loss of consciousness
-apnea
-seizures
-heart murmur-MV
what is the echo presentation of endocardiofibroelastosis?
-primary congestive cardiomyopathy or restrictive form of cardiomyopathy
-layer of fibrosis surrounds the endocardium(bright walls)
-may or may not occur w/ eoisniohlla
-obliteration of the apex
-sucesceptible to cardiac thrombi and peripheral embolization
-numerous echogenic areas evident along the endocardium ad MV
HCM
-Hypertrophic cardiomyopathy
-hypertrophy of the myocardium w/out dialation of the ventricular cavity
-certain areas are more hypertrophied than others depending on the type
what are the signs and symptoms of hypertrophic cardiomyopathy?
-asymptomatic(without obstruction)
-dyspnea upon exertion
-chest pain
-palpitations and fatigue
-pulmonary hypertension
Name some facts and the echo presentation of HCM?
-most common site of hypertrophy is the septum
-20-25% have LVOTO
-severe HCM seen in infancy leads quickly to CHF and carries a poor progosis
how is HCM treated?
beta blockers
surgery to remove some of the muscle
GSD
glycogen storage disease:
-inheritable disorder resulting from specific enzymatic defects
-aka pompe's disease
-classic form of infantile GSD and is autosomal recessive
-progressive deposition of glycogen in all tissues, esp. the myocardium
What is the echo presentation of GSD?
Glycogen storage disesase:
-severe thickening of the IVC, free wall, and post LV wall
-tumor like appearance of the pap muscles
-small LV cavity
-poor global LV systolic function
what are some systemic diseases w/ cardiovscular manifestations?
-hypertension
-diabetes mellitus
-pregnancy
-chronic renal insufficiency
What is the echo presentation in a patient w/ hypertension?
-LVH
-diastolic dysfunction
-later systolic dysfunction
-MAC
-Mild AO thickening
-Dialation of the Asc. AO-AI
What is the echo presentation of Diabetes mellitus?
-diastolic dysfunction
-premature coronary artery disease
-lipid disorders=hypertension
What will change in the heart w/ pregnancy?
-since in the 3rd trimester, there is an increase in the volume of blood by 50%, there will be a mild increased in chamber dimensions due to an increase in SV
-Altered MV coaptation, TR
-Small pericardial effusions
-Pseudo wall motino abnormalities due to the enlarged UT pressing on the heart
-benign arrythmias
Explain chronic renal insufficiency, and what it will present as on echo?
-elderly patients
-hypertension or diabetes=coronary artery disease
-calcification of mitral annulus
-LVH
-small pericardial effusion
-
Name 3 connective tissue diseases?
-systemic lupus erythematosus
-scleroderma
-marfans
Explan the echo presentation w/ Sytemic lupus Erythmatosus(SLE)?
-non infectous endocarditus(libman-sacks vegetation)-more common on MV, not mobile
-inflammatory component results in leaflet deformity and regurge
-coronary vasculitis=global dysfunction which can mimic cardiomyopathy
-acute pericarditis(PE)
Marfan's syndrome
-heritable disorder of connective tissue
-cystic medial necrosis=degeneration of the mdial layer of the AO
-Aortic dimention of >55mm-indication for surgery
-MVP
-Risk of AO dissection
What will be seen on echo w/ chronic liver disease/ cirrhosis?
-poor cardiac output
-RV dysfunction
what are the causes of cirrhosis?
-drugs
-viruses
-unknown
-alcohol abuse
-heredity
sarcoidosis
-inflammatory multisystem disease of uncertain etiology(lungs and lymphatic system)
-40% have cardiac involvement
-global LV dysfunction-MR
-granulomas seen in pericardium, conducting system or myocardium
hemochromatosis
-infiltrative pattern similar to amyloidosis
-dialated cardiomyopathy
Explain how muscular dystrophy relates to echo?
-hypertrophic or dialated cardiomyopathy
-may be greater regional variation in LV dysfunction
Freidreich ataxia-what is is?
-slowly pregressive disorder of the nervous sytem and muscles
-inability to coordinate voluntary muscle movement
-premature death of nerve cells
-presents usually btw 5-20
what is the echo presentation w/ fredrich ataxia?
-dialated cardiomyopathy
-posterior wall abnormalities
-arrythmias
hyperosinophilic
-due to eosinophilic leukenia
-obliteration of the lt or rt ventricular apex by laminar thrombus
-see thrombus in the apex when the walls are moving good.
what is the most common cause of AO dissection?
hypertension
what does effacement of the sinus of valsava mean?
the walls around it are enlarging to meet its size, so the sinus isn't distinct anymore.
sickel vell anemia
-high output state=ventricular dialation=dialated cardiomyopathy
-microinfarction and ventricular dysfunction
-may also develop arterial hypertension
what can occur in the heart w/ HIV?
-endocardidits
-pericardidits
-pulmonary hypertension
-dialated cardiomyopathy
-
what can diet drugs in the heart cause?
-valvular heart disease
Explain some neurogenic events and cardiac sources of embolus?
-LA appendage thrombus
-AO atheroma
-PFO
What will happen in the heart w/ hyperthyroidism?
-there is an increse in total blood
-increase in LV contractility
-decrease in systemic vascular resistance
-high output state
-increased SV
-sinus tachycardia and a-fib
What can occur in the heart w/ hashimotos disease?
-MVP
-MR
What can advanced age do to the heart?
-cause a sigmoid shape to the proximal ventricular septum
-don't mistake for LVOTO