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14 Cards in this Set
- Front
- Back
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How is Borrelia burgdorferi different
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1. Linear genome
2. Uses manganes or Mg instead of iron 3. doesn't make own protiens |
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Reservoir for Borrelia burgdorferi
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animals: rodents, ticks, deeer, zoonotic
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Virulent factors for Borrelia Burgdorferi
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OspA: help stick to midgut
OspC: interact w/ human tissue (temp controls expression) OspE: inhibits complement |
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What happens in Borrelia Burgdorferi when temp incr.
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OspA is no longer produced & OspC & OspE are produced preparing the spirochete to leave the tick and thrive in mammal
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Stages of B. burgdorferi
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1. Primary: skin infection, bulls-eye rash
2. Secondary: systemic infection 3. Tertiary: chronic Think: lyme juicy dripping into skin, nervous, heart, and joints |
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Lyme disease
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Borrelia Burgdorferi
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Difference btween T. Pallidum and B. Burgorferi
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T. Pallidum: sexually transmitted
B. Burgoderi: zoonotic |
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Bacteria that does not have a peptidoglycan layer
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Clamydia tachomatis
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Think of EB as an elementary weapon, like a cannon ball fired from host cell to host cell spreading infection
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Chlamydia Tachomatis
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How does chlamydia spread?
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1. EB enters by endocytosis
2. Develop into RBs 3. RBs change back into EBs before exiting cell RBs line up on outside of vacuole while EBs toward center |
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LGV: lymphogranuloma venereum, or granuloma of inguinal lymph nodes
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Chlamydia trachomatis
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Trachoma: conjunctivitis can cause blindness until eyelids curl so eyelashes scrape eye
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Chlamydia trachomatis
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Effects eyes and genitalia
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Picture a flower child w/ groovy clam eyeglasses and clam bikini
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In men causes urethritis
women: vaginitis |
Infect fallopian tubes can result from sinister clam causing PID
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