• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/62

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

62 Cards in this Set

  • Front
  • Back

ALS


-umn and L____ signs in limbs and bulbar musculature


-D_____/Dysphagia (25% deficits associated with b____musculature)


-Yields S____ and/or flaccid dysarthria


((((initially pure, mixed, rarely equal presentat))

LMN


Dysarthria


bulbar


spastic

Loss of motor neurons in pre-p______ central cortex, corticos___ tracts, motor nuclei of c____ nervers, and a____ horns of spinal cord



ONSET- middle age to ______


--Death: r____ failure/infection (death 3-___ years onset) no cure

post


spinal


cranial


anterior


70


respiratory


5

Large # of ALS patients (__%) initially present with b____ symptoms

25


bulbar

CLINICAL FEATURES


*F____


*C___


*Fasciculations


*w_____


* muscle a______


*s______

fatigue


cramping


weakness


atrophy


spasticity

NONSPEECH


UMN (S___)


*jaw j__ and sucking reflexes


hyperactive g___ reflex


slow (s____) orofacial movements


P______ affect


D_____

SPASTIC


JERK


GAG


STIFF


PSEUDOBULBAR


DYSPHAGIA

LMN (F_____)


* Decrease g___ reflex


*weak c____


*decrease f______ tone


*lingual fasciculations and a______


D_______

Flaccid


gage


cough


facial


atrophy


Dysphagia

SPEECH:


*L___, slowly produced speech


*S___phrases and intervals between words and p___


*Grossly imprecise a______


*H_____


*Strain-S____ VOICE


*m______ AND MONOLOUDNESS

Labored


Short/ phrases


articulation


hypernasal


strangled


monopitch

MULTIPLE SCLEROSIS (White Matter)


- Most common of a____ d____ central nervous system diseases


-U___ cause (viral)


-Affects w____ matter scattered across the n______ system

acquired, demyelinating


Unknown


white


nervous

MS


*Lesions observed across time and s____


*Multiple episodes over time/episodes affect different s___/systems


*Destruction of m___ sheath that covers axon, and death of o_____ that produce myelin

space


structures


myelin


oligondendrocytes

*MS


Course of disease is u______


*Episode followed by complete r_____


*Episodes with constant p_____


*Exacerbating- r_____ progressing

unpredictable


remission


progression


remitting

Clinical Features:


*G____ decreased a______


*decreased S____ control


*C____ dysfunction


* D______


* N_______


* Intention T___

Gait


Ambulation


Cerebellar


Dysarthria


Nystagmus


Intention tremor

Nonspeech oral mechanism:


*Reduced v___ capacity and inadequate v_____


*Facial p____ (Bell's palsy)


*T____ neuralgia (sudden, brief periods of facial p___)


*facial m______ (persistent q______) muscles on cheeks

vital


ventilation


paralysis


Trigeminal


pain


myokymia


quivering

Multiple Sclerosis


*S____-a___ dysarthria most common (spastic with incoordination)


*Impaired L___ and pitch c___


* Suddenly a_____ breakdowns


* H_____


* S___ rate


* Reduced p___ variability

Spastic- axatic


loudness


control


articulatory


hypernasility


slow


pitch

MIXED DYSARTHRIA


- many other conditions yield mixed dys.


*Progressive s____ palsy


* Widespread a____


* W_____ disease


* Tumors, t_____

supranuclear


atrophy


wilson's


trauma

WILSON'S DISEASE


*Autosomal r___ metabolic disorder (25%)


*Unable to process c____ in food

recessive


copper

Wilson's Disease


*See n____ and hepatic (l____) dysfunction


may see hyperkinetic h___kin.. spastic.. ataxic


pg. 281 in DUFFY (Dont memorize)

neurologic


liver


hypokinetic


APRAXIA OF SPEECH


*Inability to acc produce v_____ speech movements.. independent of d_____ (muscle weakness, spasticity, rigidity, hypo/hyper) a_____ or other non-v____ apraxis (limb a____)

volitional


dysarthria


aphasia


verbal


apraxia


.."a phonetic-m_____ disorder of sp. production cause by inefficiencies in the t____ of a well-formed and filled phonologic frame to p____learned kinematic parameters assesmbled for carrying out the i_____ movement resulting in intra-and inter______ temporal and spatial segmental and p_____ distortions"

motoric


translation


phonologic


intended


articulator


prosodic

AOS


* Distortions of s___ and intersegment transitionalization resulting in extended duration of c____, vowels, and t__ between sounds, syllables, and w)___... These distortions often perceived as sound s____ and as the misassignment of s___ and other phrasal /sentence-level p____ abnormalities

segment


consonants


time


words


substitutions


stress


prosodic

AOS


*Errors are relatively c____ in location within the utterance and in_____ in type. It is not attributable to deficits of m___ tone or reflexes, not deficits in processing of a___,tactile, kinesthetic, propropceptive, or l_____ info... In its extremely infreq. occurring "pure" form, not accompanied by m___physiology, perecept/lang

consistent


variable


muscle


auditory


language


AOS


*Disturbance in the p_____ or speech movements


*Muscles capable of n______ function

programming


normal

PLANNING


- planning for speech takes place in m_____ association areas


*P____/Broca's Area/ S_____ Motor area/ Prefrontal/p___association areas/ c_____circuit of the Basal Ganglia/ W_____ Area

motor


premotor


supplementary


parietal


caudate


wernickes

PROGRAMMING


*Speech "programming" is.. a set of m___commands that are structured before a movement s____ begins which can be delivered without reference to e___ feedback


*Programs specify muscle: tone, m____ direction, force, r___, rate and mechanical s____ or joints

muscle


sequence


external


movement


range


stiffness

PROGRAMMING BREAKDOWN


*Programming for speech take place in:


-B____ Ganglia (caudate/putamen)


- Lateral C____


*S____ motor areas


*premotor c___
*P____ motor cortex


*Frontolimbic system

Basil


cerebellum


supplementary


cortex


primary


AOS


*Often coexists with d____ and/or a_____


*Nearly always result of pathology in l_____ cerebral hemisphere

dysarthria


aphasia


left

AOS


-Anatomy and Physiology


*Motor and speech programming involves many areas


*L_____ cerebral hemi involved in m___speech p____

Left


motor


programming

Left cerebral hemi


-L____ Input to MSP from left p____ area (temproprarietal cortex and posterior portions of frontal lobe) and b___ ganglia and t____

linguistic


perisylvian


basal


thalamus

AOS


-Overlap between l___areas and MSP; so da,age to the p___ zone results in co-o____ of AOS and a____



-MSP transforms a___ phonemes to a neural code, which is to be executed by the m___ system

linguistic


perisylvian


occurrence


aphasia


abstract


motor

AOS


cortical areas involved


*p___ area and b____ area


*s____ motor area (medial aspect of frontal love)


*P___ lobe somatosensory cortex


*i______


*b____ gang- caudate/putamen

premotor, Broca's


supplementary


parietal


insula


basal

lesions that result in AOS; L___ posterior f___ or p___ lobes/ in the i_____ or in the b___ ganglia

left


frontal


parietal


insula


basal

AOS


Etiologies


*V____ lesions (stroke_ most common; l___hemi middle c____ artery


*Diffuse diseases rarely produce i_____ AOS

Vascular


left


cerebral


isolated

Nonverbal oral mechanism


*May be no evidence of w_____; g__ reflex and chewing/s___ may be normal


*Lesions are usually l__ enough, may be concomitant unilateral c_____ damage.....d____



*Possible s_____ deficits

weakness


gag


swallowing


large


corticobulbar


dysarthria


sensory

AOS


*Nonverbal oral apraxia


-inability to i___ or follow commands/perform v____ movements of speech structures


* Can occur independently from AOS


* (((box 3-1 pg 88))) sequencing is important (BOX 3-3)

Imitate


volitional

AOS


SPEECH


*p___ level errors


*I___ ERRORS (Errors vary as complex. artic. output varies)


*I___ performance is poor


*Patient is a___ of errors/ cant correct them

phoneme


inconsistent


imitative


aware

*When patient tries to monitor speech to anticipate errors P____ deficits result


*Oral n___ apraxia is often, but not always in conjunction with A_____

pROSODIC


AOS

OTHER


*automatic-reactive speech better than v___-purposive speech


*Artic may be better both a__and v__ stim than either one alone


*___ work no effect/single word


*I___ better than spontaneous speech


*A___ may increase on c______ attempts

volitional


auditory/ visual


Mirror


Imitation


accuracy /consecutive

M___/inspirational instruct. effect accuracy


Response d____ intervals/dont influence accuracy


B___masking has no postive efffect


d___auditory feedback may have n____ affect on accuracy


a____ discrim bbetter than v____ production


o____ perception and s___ may be impaired

Motivation


delay


Binaural


delayed... negative


auditory / verbal


oral/ sensation

AOS


*Speech assessment/Diagnosis


*Use speech s____ motion rates (SMRs) and i___ of complex m_____ words and sentences


-zip/zipper/zippering

sequential


imitation


multisullabic

AOS


*look for t__ at which patient s__ or fails on tasks reflecting contimuum of speech programming demands (vowels, CV,CVC, AMRs, s___/speaking words)


- pg 86-87

threshold


succeeds


singing


Patient Complaints:


*s__ wont come out right


a___ problems *not respiration/phonation/reson


*speak s___ and carefully


*l____ words difficult to pronounce

Speech


artic.


slowly


long

AOS


*TX- consider m___ Learning


*R___ MOTOR DRILL: lots of trials


*M___DRILL- quick dev.(poor generalization)


*D__ DRILL


slower development and good g_____

motor


repetitive


mass


distributed- generalization

Knowledge of Results


*f___
* how was it right/w___


*not too much detail


INFLUENCE OF R___


*speed/accuracy trade-off


* vary, working toward natural p____

frequent


wrong


rate


prosody

*phonetic p____/manipulate a____


* Phonetic derivation: use intact n__-speech gestures to elicit target s____ sounds


*I____/integral stim: watch, listen, s____ produce, delay to produce

placement/ articulators


non


speech


Imitation


simultaneously

AOS


*C_____ stress drill/change stress/rate/r___/intonation


*M___ INTONATION THERAPY


*Increase s___; contextual complexity

contrastive


rate


Melodic


speed

family c____/education


total c___


a___ communication

counseling


communication


augmentative

AOS by definition a m___speech disorder


*Essential nature and characteristics of AOS must be consistent with the models of s____ production and with known p____ at the motor p___/programming levels

motor


speech


pathology


planning

AOS


P____, lexical /syntactic errors should not be lumped under AOS


*Freq. concomitant errors at p__, syntactic, phonologic, lexical levles


* happens because the responsible a______ areas very close in proximity and b/c many pathologies not strictly localizable (NOT AOS)

Phonological


phonetic


anatomical

DIFFERENTIAL DIAGNOSIS


*Dysarthria (neuromuscular)


- alterations in s___,tone, r____



AOS (neuromuscular)


- no c____

strength


range


changes

DYSARTHRIA


- All components of s__ maybe affected



AOS


- A____

SPEECH


ARTICULATION

DYSARHTIRA


Aphasia (freq N_____ ASSOCIATED)



AOS


Aphasia freq A____ associated with aphasia

not


always?

DYSARTHRIA


Variability - speech generally c___ (not influenced by a___/stimulus modality/linguistic variables )



AOS


Variability (speech is v____) automatic=OK


propositional (repeat what i say)/repetition influenced by multiple factors

consistent


automaticity



variable

DYSARTHRIA


*Error Types (d___/simplifications)



AOS


*Perceived s___, additions, r___, prolongations, complications of t____ sounds

distortions



substitutions


repetitions


targeted

Dysarthria


g______ rarely observed



AOS


G____ highly prevalent

groping


groping

ATAXIA


- SPEECH AMRs I______



AOS


-SPEECH AMRs R____

Irregular



Regular

ATAXIA


- NORMAL S____ (Rhythm is off)



AOS


-Sequencing of SMR'S


- A___

sequencing



abnormal

ATAXIA


P____ HIGHLY VARIABLE



AOS


Prosody less v____

prosody


variable

ATAXIA


islands error- free speech (u___)


E ___ (Automatic/propositional speech)



AOS (Typical lslands of e___-free speech)


a___ speech better than propositional speech



frequent s_______


unusual


Equal


error


automatic


substitutions

Treatment


- know when it will end!


-remember e____ options/P____AAC/PACERS (Dont go there first)



WHO- I___/disability/ handicap

external


prostheses



Impairment

*Speaker Oriented vs. Communication Oriented approaches


*Speaker oriented


Restore/C_____ (Reduce I_____)


Then work on Efficiency, N____&Quality of communication


Compensate


impairment


naturalness

Communication oriented


modify the communicative i_____

interaction

Treatment ... counseling.. e___ intervention; know the m___ plus the speech diagnosis



*Physiological support f___ then other interventions


*learn about m___ learning


* F_____ is often key!!

early


medical


first


motor


flexibility


MIXED

MIXED