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71 Cards in this Set
- Front
- Back
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which herpes virus would you find in the mouth
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HHV-1
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how is HHV-1 spread
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saliva or perioral lesions
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primary infection with herpes
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initial exposure to somebody with out the antibodies
80% asymptomatic |
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HSV tolerated the external enviornment well
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FALSE - almost all primary infections occur as a result of contact with someone who is releasing the virus
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young patient (6months - 5 years)
anterior cervical lymphadenopathy fever generalized mouth lesions |
acute herpetic gingivostomatitis
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what throat infection may accompany primary herpes
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pharyngotosillitis
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fever blister
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noted with recurrent HSV-1, most commonly on the vermillion border
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where do you find intraoral recurrent HSV-1
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keratinized mucosa
attached gingiva and hard palate |
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herpetic whitlow
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recurrences on digits
result in parasthesia and permanant scarring |
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Tzanck cells
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acantholytic epithelial cells
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primary hhv-3
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chickenpox
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recurrent hhv-3
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shingles
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"dewdrop on a rose petal"
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common presentation of early chickenpox vesicle
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most frequently ivolved oral location for chicken pox
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palate and buccal mucosa
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difference between gingival lesions of chicken pox and HSV
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chicken pox in the gingiva are non painful
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treatment of chicken pox
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antipyretics other than aspirin
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where does the hhv-3 go latent
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dorsal spinal ganglia
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rash with respect to dermatome
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hhv -3 recurrant
shingles |
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ramsay hunt syndrome
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cutaneous lesions of auditory canal
damage to cn 7 and 8 |
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can lead to ramsay hunt syndrome
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shingles
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three stages of shingles
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prodrom, acute, and chronic
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when should you refer shingles to ophthalmologist
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if rash involves tip of the nose.... this shows nasociliary involvement
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coxsackivirus A
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herpangina
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most common location for herpangin
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tonsillar pilars and soft palate
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coxsackievirus A16
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hand foot and mouth disease
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borders of palms and soles
ventral surfaces of fingers and toes |
hand foot and mouth
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intraepithelial vesicle
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potential to form with herpangina and hand foot, mouth
caused by combination of intra and intercellular edema |
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yellow to pink nodules in soft palate or tonsillar pillars
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acute lymphonodular pharyngitis
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paramyxovirus
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Measles (rubeola)
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Koplik spots
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most telling initial stage oral manifestation of measles
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bluish - white macules on labial mucosa
"grains of salt on white background" |
koplik spots
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warthin finkeldy
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giant cells associated with rubeola
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togavirus
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rubella (german measles)
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congenital triad from rubella
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deafness, heart disease, cataracts
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EBV
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Infectious mononuclosis
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petechiae on the hard or soft palate
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mono
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orophryngeal tonsillar enlargement
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shown in 80% of mono patients
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immunocomprimised patients in mono can develop
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life threatening polyclonal B lymphocyte proliferation
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T pallidium
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syphillus
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characterizes primary syphillis
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chancer
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explosive and widespread strain of syphillis
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lues maligna
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mucous patches and condylomata lata
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syphilllis sypmtoms
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tertiary syphillis problems
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gumma (extensive tissue distruction)
ex/ perforation in hard palate |
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hutchinsons triad
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hutchinsons teeth, interstitial keratitis, eighth nerve deafness
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where can T. Palladium "escape" to
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lymph nodes or CNS
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treatment for syphillis
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penicillin
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miliary tuberculosis
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diffuse dissemination through vascular system
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lupus vulgaris
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tb involvement of the skin
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enlarged orophyrangeal lymphoid tissue
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scrofula
often cervical lymph nodes associated with TB |
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most common actinomyces in clinic
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A. israelii
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discharge of "sulfur granules"
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actinomyces
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actinomyces DOC
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penicillin
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note a fistula in submandibular or parotid region.... patient says it formerly felt "woody"
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think actinomyces
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see white "cottage cheese"
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candidiasis
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burning sensation or altered taste of tongue
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candidiasis
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well demarcated erythematous zone at the midline aspect dorsal tongue
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erythematous candidiasis
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corner of mouth
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angular chelitis
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what must you see to diagnose candidiasis
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hyphea or pseudohyphea
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what form of histoplasmosis might you see in the mouth
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disseminated
others are acute and chronic - these stay in the lung region |
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what other disease does histoplasmosis resemble
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SCC (ulcerated lesions with firm borders)
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treat disseminated histoplasmosis
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iv amphotericin B
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southewestern US mold
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coccidiodomycosis
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coccidiodomycosis becomes disseminated by
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spreading through the blood
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treatment of blastomycosis
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most patients require no treatment
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most common life threatening infection in AIDS patients
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cryptococcosis
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diffuse infiltrates and hilar masses in a chest film WITHOUT calcification
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blastomycosis
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valley fever
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hypersensitivity reaction resulting in painful erythematous inflammatory nodules in the subcutaneous tissue
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head and neck skin have erythromatous papules or pustules
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cryptococcosis
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zygomycosis treatment
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radical surgery
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bloody nasal discharge
facial paralysis |
think zygomycosis
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zygomycosis is particularly noted in
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patients with poorly regulated insulin dependant diabetes
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