• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/20

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

20 Cards in this Set

  • Front
  • Back
trophozoites cause ulcerative FLASK shaped lesions in lg intestinge. can cause granulomas in brain, spleen, liver, lungs and skin

sx. BLOODY diarrhea, leukocytosis, anemia, nausea, vomiting, abdominal pain
amebiasis

tx with metronidazole
what can occur with amebiasis in

Resp?
Hep?
Brain?
EYE?
Resp=infections w/ pain and cough

Hep=chills, fever,

Brain abscess- can kill


Donjunctivities w/ FETID ODOR
commensal bacterial co-infection on the eyelids and ocnjuncitva play a role. collaenase and proteolytic enzymes released by trphozoites

*PARTIAL or COMPLETE RING develops in eye

*antibody dependent cellular destruction is promoted

SX:
*uveitis, optic neuritis, corneal keratitis
Acanthoamoebic keratitis
what bacteria does acanth keratitis love?


what are cephalic problems?
what are skin problems?
loves pseudomonas

forms granulomatous amebic encephalitis in immunocompromised

skin lesions occure months before invasion of brain and are ulcers, nodules, abscesses and amebic granulomas
intestinal villi are infected and atrophy and fuse w/ infiltate

sx:
watery diarrhea, weight loss, abdominal pain, nausea,

keratitis(from microsporidia0
or pneumonitis if gets into lung
microsporidiosis
cercaria migrate under the skin of humans producing mechanical toxic damge but DO NOT ENTER blood vessels to become adults

sx:
-serpinginous tunnel lesions, vesicles, papules, rash with itching and secondary bacterial infection

shoudl go away in 4-5 days but can persist if hypersensitive
Schistosome dermatitis,

swimmers itch
adult worms lie attached by SPiens on rostellum of small intesting where metabolic by products interact with mucosal surface

sx:
-inflamation, rash, vomiting, diarrhea, nervouse syspmtos and allergic response
Dipylidiasis
intestinal obstruction causes inflammation and irritation at site of attachment of scolex. can also exist in larvae stage and displace tissue and provoke cellular inflammatory rxn

sx:
-abdominal pain and weight loss w/ weakness, dizziness, ha, vomit, diarhea and allergic response
adult worm taenisis
how does larvae of teaniasis present (cysticercus cellulosae)
HA, nervous, manifestations, subrentinal masses, keratitis, insomnia, seizure
larvae cause skin lesions and allergic rxns develop. can cause inflammation of alveoli w/ hemorrhaging if trapped in lungs. adult worms suck blood from small intesting producing inflammation, eosinophilia, blood, loss and Fe defic anemia

sx:
-infection produces skin rashes, Fe defic anemia, anorexia, weight loss, blurred vision, abdominal pain, nausea, vomit, diarrhea, malnutrition, wheeze and pneumonitis
Ancylostomiasis
which is an adult worm that results in intestinal blockage, malnutrition, eosionphilia and when distrubed may lodge into bile duct or appendix


sx. abdominal cramps, diarrhea, loss of appetitiis, malnutriton appendicits?
ascariasis
what happens if ascariasis gets into lungs?

what causes neurologic sx?
Loffler's syndrome in lungs (eosinophilia in lung tisse)

toxic an allergenic metabolites cause neurologic symtpoms(retinal granulomas)
adult female worms irritate anal area when laying eggs. itnese scratchin and sometimes bacterial infection

Sx:
-appendicitis and pelvic periotenal graulomas
-anal itching, scratching, insomnia
enterobiasis (human pin worm)
larvae penetrate skin adn wander around under dermis but never enter blood. local eosinophilia around tunnesl and persists for months if untreated

sx. serpinfinous tunnel with icthin, red papulea and edema
cutaneous larva migrans (creeping eruption)
migrating larvae produce inflammation, heper eosinophilia, skin rash , fever,

damage depends on #of eggs ingested, migration route of larvae adn stregnth of immune response

sx: fever, LOSS OF VISION, cough, pneumonitis, edema,
visceral larva migrans
early has malasie abdominalpain, nausea, vomit, diarrhewa, fever, myalgia for 5 days

progress to cough, INTENSE MUSCLE PAIN, periorbital edeam and myocarditis to CHF 7-14 dyas

after 2 weeks, cachexia of infection , myositis with pain and SWELLING OF ARM AND LEG MUSCLES, rash , eye problems, pneumonitis
Trichinosis
what causes inflammation, toxic effects and hypersensitivity of trich?


what causes inflammatory response w/ eosinophilia?
larvae and adult worms


larvae also produce inflam respons w/ eosin
Lice suck blood injecting saliva causing pustular lesions at site of bite

prim hypersenstivity attained 3-8 months after original infestation

sx:
-papules, ulceations, intense itching and discoloration
may have toxic frollicular conjucntivitys from feces
Lice infestaiton
larvae bit through skin of humans injecting saliva that is antigenic and contains lytic compounds to dissolve tissue.

DO NOT SUCK BLOOD OR BBURROW INTO SKIN

allergic rxn develops following first expose
re

SX:
-deep red lesions with attendant loss of sleep for days/weeks
-small itching red s pots develop into dermatitis where clothing is tight

can get secondary bact. infecton from intense scratching
Mites (trombidiosis)
mites live in serpinginous cutaneous burrow in humand and progress w/o symptoms while crawling and laying eggs. pap, vesic or pusutalr lesions produced by allergic rxn to eggs, larvae, mites or feces

sx:
-reddish elevated tracts in skin b/w fingers, wrists, forearms and bend of knee and elbow

also inguinal area,penis and shoulder blades that develop into SINGLE LESIONS OR RASH LIKE LEIOSN OCCURIN IN TRACKS

INENSE ITCHING leads to scratch, which spreads.
SEVEN YEAR ITCH, scabies