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310 Cards in this Set
- Front
- Back
Risk factors for eye disorders...
|
Aging
Trauma Heredity Diabetes |
|
Which of the following would not be considered an age-related disorder of the eye?
a) Lens opacities b) Increased orbital fat c) Loss of skin elasticity d) Loss of accomodation |
b) Increased orbital fat
|
|
What is Glaucoma?
|
Increased intraocular pressure
|
|
What does Glaucoma cause?
|
-Optic nerve damage
-progressive loss of peripheral and night vision |
|
What are the clinical manifestations of Glaucoma?
|
Halos
Headache |
|
Types or Glaucoma
|
Acute closed angle
Chronic closed angle Chronic open angle |
|
What type of Glaucoma causes a sudden increase of pressure in the eye?
|
Acute closed angle
|
|
Which type of Glaucoma is considered the "silent thief of sight'?
|
Chronic open angle
|
|
What does Glaucoma lead to?
|
damage to the optic disc
|
|
What is Cataracts?
|
Opacity of the lens
|
|
What are the clinical manifestations of Cataracts?
|
Blurred vision
Decreased visual acuity Decreased color perception |
|
What medications are used for cataracts?
|
Mydriatics
Cycloplegics |
|
What type of surgery is done for cataracts?
|
Removal of the lens
-extracapsular extraction -intracapsular extraction |
|
Nursing management of Glaucoma
|
Prevent further vision loss
Explain action and effects of medications Aid the patient in adjusting to vision loss or potential vision loss |
|
Nursing management of Cataracts
|
Preoperative care
Dilating eyedrops or other meds Call physician for vision changes, flashing lights, redness, swelling, significant pain |
|
Corneal Transplant Surgery
R.S.V.P. |
R-Redness
S-swelling V-vision decrease P-pain |
|
What is retinal detachment?
|
separation of the retina (light-sensitive membrane) of the eye
|
|
Is there pain with retinal detachment?
|
No Pain
|
|
What are the clinical manifestations of Retinal detachment?
|
Flashes of light
Floaters Increased blurred vision Loss of a portion of FOV |
|
Medical management of Retinal detachment
|
Surgery is the only treatment
|
|
Post-op Nuring considerations for Retinal detachment
|
Same as corneal transplant
Avoid reading for 3-5 weeks |
|
What is Macular degeneration?
|
Deterioration of the macula
|
|
What is the macula?
|
the area of the retina where light rays converge.
|
|
What causes Macular degeneration?
|
Aging - thinning of the macular tissues
|
|
What are the clinical manifestation sof Macular degeneration?
|
Decrease in central vision Decrease in near vision
Blurred vision |
|
How is Macular degeneration diagnosed?
|
Amsler grid
|
|
How is Macular degeneration treated?
|
Laser
|
|
Infectious and Inflammatory eye disorders?
|
Dry eye syndrome
Conjunctivitis Uveitis Orbital cellulitis |
|
What is Conjunctivitis?
|
"Pink Eye"
Can be bacterial, viral, fungal, parasitic, allergic, or toxic |
|
Is conjunctivitis contaigious?
|
Yes...All forms
|
|
What is Uveitis?
|
inflammation of the middle layer of the eye
-causes photophobia |
|
What are the signs of an eye infection?
|
Fever
Pain Red eyelids Swelling |
|
What is Diabetic Retinopathy?
|
vascular changes in retinal blood vessels
|
|
What is the most common incidence in diabetics?
|
diabetic retinopathy
|
|
What is the most common manifestation of diabetic retinopathy?
|
floaters
|
|
What is the best way to prevent diabetic retinopathy?
|
Teach good diabetes management
|
|
What is conductive hearing loss?
|
sound waves are blocked due to external or middle ear disorders
|
|
Is conductive hearing loss permanent?
|
No
|
|
How is conductive hearing loss corrected?
|
with minimal or no damage to hearing
|
|
What is sensorineural hearing loss?
|
Pathological process of inner ear
|
|
Is sensorineural hearing loss permanent?
|
Yes
|
|
Causes of sensorineural hearing loss
|
Damage to inner ear structures
Damage to cranial nerve VII Prolonged exposure to loud noise Medications Trauma Infections Meniere's Syndrome |
|
Medical Management of Hearing loss
|
Cochlear Implant
Hearing Aids |
|
Which type of hearing loss are hearing aids most effective in?
|
Conductive Hearing Loss
|
|
What is Presbycusis?
|
Degeneration of ganglion cells in the cochlea
Loss of elasticity of Basilar membranes |
|
Clinical Manifestations of Presbycusis
|
Gradual, Bilateral hearing loss
Pt thinks people are mumbling Difficulty understanding words |
|
What is the best tone to use to spek to a person with presbycusis?
|
Low, pitched voice
|
|
What is External Otitis?
|
Infective inflammatory or allergic response
"Swimmer's Ear" |
|
What are the clinical manifestations of External otitis?
|
Redness
Swelling Pain Conductive Hearing Loss |
|
What does External Otitis usually stem from?
|
Staph Infection
|
|
What is Otitis Media?
|
Acute or Chronic infective inflammatory or allergic response involvong structures of the middle ear.
|
|
If otitis media is left untreated, what is the result?
|
Mastoiditis
|
|
Clinical manifestations of otitis media
|
Pain from pressure
Hearing loss Tinnitis, dizziness, vertigo Fever, malaise N&V Bulging Tympanic membrane |
|
Which type of ear infection is most common in children
|
Otitis Media
|
|
What is Otosclerosis
|
Disease of the Labyrinthine capsule of the middle ear
|
|
What does otosclerosis result in?
|
bony overgrowths, causing fixation of bones
|
|
What does otosclerosis lead to?
|
Conductive hearing loss
|
|
What happens if otosclerosis spreads to the inner ear?
|
sensorineural hearing loss
|
|
Clinical manifestations of otosclerosis
|
Slow, progressive hearing loss
Tinnitus May involve both ears Pinkish coloring of Tympanic membrane |
|
What is Gout?
|
A type of arthritis where uric acid builds up in the joints
|
|
Acute gout
|
typically affects one joint
|
|
Chronic gout
|
repeated episodes of pain and inflammation
May involve more than one joint |
|
Symptoms of Gout?
|
Develop suddenly and usually only involve only one or a few joints
May be fever attack may go away in several days |
|
Which joints are most commonly affected in gout?
|
Big toe, knee, ankle
|
|
When does gout pain usually start?/
|
During the night
|
|
What does a gouty joint look like?
|
Warm, red and very tender
|
|
What kind of pain is associated with gout?
|
throbbing, crushing, excruciating
|
|
Tx for Gout
|
NSAIDs
Prescription strength pain meds for 4-10 days Corticosteroids |
|
Foods to avoid with Gout
*fatty, |
Alcohol
Anchovies, sardines, oils, herring Organ Meat (liver, kidney) Legumes Gravies Mushrooms Spinach, Asparagus, cauliflower |
|
What is Labyrinthitis?
|
INfection of the labyrinth of the inner ear
|
|
What is Labyrinthitis a complication of?
|
acute or chronic otitis media
|
|
What are the S/Sx of labyrinthitis?
|
Hearing loss on affected side
Tinnitus Vertigo N/V Nystagmus to affected side |
|
What is Meniere's Syndrome?
|
Build up of fluid in the inner ear
|
|
What is the pathophysiology of Meniere's Syndrome?
|
dilation of the endolymphatic system by overproduction or decreased reabsorption of endolymphatic fluid
|
|
What does repeated damage to the cochlea lead to?
|
Permanent hearing loss
|
|
What is the most important nursing interaction with Meniere's Syndrome?
|
SAFETY!!
|
|
What are the clinical manifestations of meniere's Syndrome?
|
Feeling of fullness in the ear
Tinnitus Hearing loss worsens during attacks Intense vertigo N/V Nystagmus Headaches |
|
What is Nystagmus?
|
twitching muscle spasm of the eye
|
|
What is acoustic neuroma?
|
benign tumor of the vestibular or acoustic nerve
|
|
What happens in Acoustic neuroma?
|
May cause damage to hearing and to movement/sensation
Rarely recurs after removal |
|
What are the S/Sx of acoustic neuroma?
|
Gradual sensorineural hearing loss
Damage to adjacent cranial nerves |
|
Medical management of acoustic neuroma
|
Surgical removal via craniotomy
|
|
Does acoustic neuroma respond well to chemo and radiation?
|
No
|
|
What are post-op considerations for acustic neuroma?
|
Monitor for facial nerve paralysis
Drainage Cerebral Edema |
|
Important consideration for appendicitis?
|
DO NOT LIE FLAT!
|
|
What is the purpose of a cast?
|
Immobilizes body part
|
|
Types of cast composition? Benefits to each?
|
Plaster - Stronger
Fiberglass - waterproof |
|
Nursing care of client with a cast
|
Neurovascular checks
Assess for infection Teach proper crutch walking |
|
Complications R/T fractures
|
Compartment Syndrome
Fat embolism DVT Reflex sympathetic dystrophy |
|
What is compartment syndrome?
|
Increased pressure from hemorrhage or edema
Pressure from cast being too tight |
|
What does compartment syndrome result in?
|
Nerve, vessel, and muscle damage
|
|
What is priority nursing consideration for patient with compartment syndrome?
|
Maintain affected extremity at heart level
|
|
What is Fat embolism syndrome?
|
globules of fat, released from fractured bone, mix with platelets
|
|
S/Sx of Fat embolus
|
Dyspnea, hypoxia, agitation
Same as pulmonary embolus |
|
What is the difference between a fat embolus and a pulmonary embolus?
|
Petechiae
|
|
Tx of fat embolus
|
Maintain pulmonary function
Fluid balance Steroid treatment STABILIZE LONG BONE FX!! |
|
What is reflex sympathetic dystrophy?
|
excitation of nervous tissue
|
|
S/Sx of reflex sympathetic dystrophy
|
Persistent pain
Hyperasthesias Swelling Skin color changes Temperature changes |
|
Tx of reflex sympathetic dystrophy
|
Threat Symptoms
Alpha or Beta blockers |
|
Post op considerations for hip replacement
|
Hip precautions for 6 weeks
No leg crossing, internal rotation, or adduction |
|
Potential complications of hip replacement
|
Hemorrhage
Peripheral neurovascular dysfunction DVT Pulmonary complications Pressure ulcers |
|
Pain relief after hip fx/replacement
|
Administer analgesics
Buck's traction Handle extremity gently Support extremity with pillows Positioning Alternative pain relief measures |
|
Interventions for hip fx/replacement
|
Orient to environment
Safety Participation in self care Coughing, deep breathing Adequate hydration TED hose or SCDs Ankle exercises Teaching (pt & fam) |
|
What are the common causes for an amputation?
|
PVD
Trauma Infections Tumors |
|
Goals of care for amputation
|
Pain relief
Wound healing Acceptance of altered body image Resolution of grieving processes absence of complications |
|
Teaching for a client with an amputation
|
Wrap stump
Positioning stump Stump exercises Physical activities Household modifications Referrals for social services |
|
How to wrap a stump
|
figure 8 pattern
|
|
How to position stump for first 24 hours after amputation?
|
elevate
|
|
How to position a stump after the 1st 24 hours
|
Gentle range of motion to prevent contracture
|
|
Pain relief for amputation
|
Analgesics
Position changing TENS unit Light sandbag on residual limb |
|
Nursing care of the patient with low back pain
|
Pain relief
Improved physical mobility Proper body mechanics Improved self esteem Weight reduction |
|
What is Osteoporosis?
|
Loss of total bone mass, by increased rate of bone resorption, decreased rate of bone formation
|
|
What happens to the bones in Osteoporosis?
|
become porous, brittle, fragile, and break easily under stress
|
|
What does Osteoporosis result in?
|
Compression fractures of the spine
Femur/hip fractures Colles fx of the wrist |
|
Prevention of Osteoporosis
|
Balanced Diet
High calcium and Vitamin D intake Regular weight bearing exercises Calcitonin |
|
What does regular weight bearing exercise promote?
|
Stimulates bone mineral density
|
|
What do you want to assess for in a pt with osteoporosis?
|
Corticosteroid use
Alcohol, caffeine intake Smoking |
|
Interventions for osteoporosis
|
Pain relief
Improve bowel elimination Injury prevention |
|
What is Osteomalacia?
|
metabolic bone disease characterized by inadequate bone mineralization
|
|
What does osteomalacia do?
|
softens and weakens the long bones, causing pain, tenderness and deformities
|
|
What causes osteomalacia?
|
Activated vitamin D deficiency
-GI disorders -severe renal insufficiency -hyperparathyroidism -dietary deficiency |
|
Treatment of Osteomalacia
|
Correct underlying cause
Increased Vitamin D and calcium intake Handle pt carefully Address pain and discomfort |
|
Why must the patient with osteomalacia be handles carefully?
|
increased risk of fractures
|
|
What is Paget's Disease?
|
Disorder of localized bone turnover
(rapid bone turnover) |
|
What is Paget's Disease also known as?
|
Osteitis Deformans
|
|
What are pts with Paget's Disease at risk for?
|
fractures, arthritis and hearing loss
|
|
S/Sx of Paget's Disease
|
Skeletal deformities
Mild to mod aching pain Tenderness and warmth over the bones May be attributed to old age or arthritis |
|
What is Osteomyelitis?
|
Infection of the bone
|
|
Why does osteomyelitis occur?
|
Extension of sift tissue infection
Direct bone contamination Blood-borne spread from another infection site |
|
What causes Osteomyelitis?
|
Staphylococcus aureus
Pseudomonas E. coli |
|
What are risk factors for Osteomyelitis?
|
Elderly
Obesity Lupus Previous bone injury |
|
S/Sx of Osteomyelitis
|
localized pain
edema erythema fever drainage |
|
when does fever occur with chronic osteomyelitis?
|
low grade fever in the afternoon or evening
|
|
Interventions for osteomyelitis
|
Pain relief
Restricted activity Gentle ROM ADLs Vitamin C and protein Adequate hydration |
|
#1 remedy for sprains and strains
|
RICE!
Rest Ice Compression Elevation |
|
Contusion
|
soft tissue injury produced by blunt force
|
|
Strain
|
pulled muscle injury to the musculoteninous unit
|
|
Sprain
|
Injury to ligaments and supporting muscle fiber around a joint
|
|
Dislocation
|
articular surfaces of the joint are not in contact
|
|
S/Sx of contusion
|
pain, swelling, and discoloration
|
|
S/Sx of Strain
|
Pain, edema, muscle spasms, ecchymosis, loss of function
|
|
S/Sx of Sprain
|
Joint is tender, movement is painful, edema, disability
Pain increased during first 2-3 hours |
|
S/Sx of Dislocation
|
change in contour, axis and length of limb, loss of mobility
|
|
What is tendonitits?
|
inflammation of tendon from overuse
|
|
What is GERD?
|
Gastroesophageal reflux disease
acidic secretions reflux into the esophagus |
|
S/Sx of GERD
|
Heartburn
Regurgitation Dysphagia Bleeding |
|
Complications of GERD
|
Aspiration
Scar tissue |
|
How is GERD diagnosed?
|
Barium Swallow
Esophagoscopy |
|
GERD Interventions
Lifestyle changes |
Dietary (avoid alcohol, sweets, caffeine, spicy, acidic)
|
|
medical interventions for GERD
|
Antacids
H2 receptor antagonists (prevacid) Proton pump inhibitors (nexium) |
|
Why to avoid dairy products with GERD
|
Stimulates acid secretion
|
|
What is gastritis?
|
inflammation of the stomach lining
|
|
What are the S/Sx of gastritis?
|
Abdominal pain, nausea, anorexia
|
|
Tx of Gastritis
|
Remove irritating substance
-Meds, aspirin, NSAIDS, steroids Bland diet Antacids Small, frequent meals |
|
Common sign of gastritis
|
Bloating, belching
|
|
When do antacids work the best?
|
When taken after a meal
|
|
What bacteria causes gastritis?
|
helicobacter pylori
|
|
what does chronic gastritis lead to?
|
Pernicious Anemia
|
|
What is the biggest complication of Gastritis?
|
Malnutrition
|
|
The biggest complicatiopn of gastritis?
|
Malnutrition
|
|
What is dumping syndrome?
|
Rapid gastric emptying
|
|
Complications of gastric surgery
|
Hemorrhage
Gastric distention Nutritional problems |
|
What is necessary after gastric surgery?
|
Lifelong B12 injections
|
|
Complications of gastric surgery
|
Steatorrhea
Pyloric obstruction Dumping syndrome |
|
What is the first sign of appendicitis?
|
Pain around belly button (periumbilical)
|
|
Where does appendicitis pain radiate?
|
RLQ - McBurney's Point
|
|
What does sudden pain relief in appendicitis indicate?
|
ruptured appendix
|
|
What is peritonitis?
|
Abdominal infection caused by ruptured appendix
|
|
what can peritonitis lead to?
|
septic shock
|
|
What are risk factors for diverticulitis/osis?
|
Low fiber diet
Increased age Chronic constipation |
|
Therapeutic interventions for diverticulitis/osis?
|
Prevent constipation
IV antibiotics Pain control Surgery |
|
What are fistulas?
|
Abnormal tunneling connecting two body cavities
|
|
Tx of fistulas
|
surgery
|
|
What is ulcerative colitis?
|
inflammatory bowel disease of the large colon and rectum
|
|
S/Sx of ulcerative colitis
|
Abdominal pain
5-20 stools per day Rectal bleeding Fecal urgency Weight loss Vomiting |
|
Ulcerative causes increased risk of...?
|
colon cancer
|
|
Therapeutic interventions for ulcerative colitis
|
Avoid offending foods
-spicy, fatty, gaseous, dairy, acidic |
|
Medications used for ulcerative colitis
|
Antiinflammatories
Antidiarrheals Immunosuppressants Corticosteroids |
|
What is irritable bowel syndrome?
|
Altered intestinal motility
Increased sensitivity to visceral sensations |
|
What are risk factors for colon cancer?
|
Polyps
Increased red meat intake Age over 60 |
|
Treatment of colon cancer
|
depends on the stage of the cancer
|
|
Treatment of Irritable Bowel Syndrome
|
Increase fiber
Avoid trigger foods Smaller, more frequent meals Stress management |
|
What is Hepatitis?
|
Inflammation of the cells of the liver
Usually caused by a virus |
|
How is Hepatitis A transmitted?
|
Fecal/Oral
|
|
How are Hepatitis B and C transmitted?
|
Blood/Body fluid
|
|
Which Hepatitis viruses cause cirrhosis of the liver?
|
B and C
|
|
Which Hepatitis virus causes no permanent liver damage?
|
A
|
|
How to prevent Hepatitis
|
Cleanliness
Vaccines (A&B only) Standard precautions |
|
Therapeutic interventions for Hepatitis
|
Rest
Nutrition INTERFERON Antivirals |
|
What causes cirrhosis of the liver?
|
Chronic alcohol ingestion
Hepatotoxins Hepatitis Gallbladder obstruction Heart failure |
|
Is cirrhosis reversible?
|
NO
|
|
S/Sx of Cirrhosis
|
Hepatic encephalopathy
Jaundice Oliguria Bruising Abnormal Bleeding |
|
What is esophageal varices?
|
dilated blood vessels in the esophagus
|
|
Sign of esophageal varices?
|
Increased amount of blood in emesis
|
|
Tx of Cirrhosis
|
Decrease alcohol intake
Low protein diet Lactulose Better nutrition |
|
Signs of liver transplant rejection
|
Pulse >100bpm
Temp >101 RUQ pain Increased jaundice Elevated liver enzymes Decreased bile from t-tube |
|
Cancer of the liver
|
Usually mets from another site
|
|
Risk factors for liver cancer
|
Chronic Hep B
Nutritional deficiencies Exposure to hepatotoxins |
|
S/Sx of liver cancer
|
Encephalopathy
Bleeding Jaundice Ascites |
|
Cholecystitis
|
Inflammation of the gall bladder
|
|
cholelithiasis
|
Gall stones
|
|
Choledocholithiasis
|
Stones in common bile ducts
|
|
Pain in appendicitis
|
Starts dull and steady in periumbilical area, progresses to RLQ over 4-6 hours
|
|
#1 sign of peritonitis
|
Board-like abdomen
|
|
Biggest nursing consideration for bowel obstruction
|
Identify!!
|
|
the higher the bowel obstruction...
|
...the quicker the symptoms
|
|
Vomiting with bowel obstruction...
|
...may turn malodorous with fecal smell
|
|
S/Sx of bowel obstruction
|
Extreme constipation
Failure to pass flatus |
|
When does Crohn's Disease peak?
|
ages 15-40
|
|
Later S/Sx of Crohn's Disease
|
Weight loss
Dehydration Electrolyte imbalance Anemia |
|
Which GI disease affects both the large and small intestines?
|
Crohn's Disease
|
|
Important nutritional considerations with Dumping syndrome
|
No fluids with meals
No high carbs Eat small meals Watch carbs and starches |
|
Osteoporosis Risk factors
*ACCESS* |
Alcohol use
Corticosteroid use Calcium low Estrogen low Smoking Sedentary lifestyle |
|
Access leads to...
|
Osteoporosis
|
|
Hepatitis with a vowel comes from the...
|
Bowel
|
|
Hepatitis D
|
Super infection with Hep B
|
|
Increased intracranial pressure S/Sx
(opposite if shock) |
Increased BP
Decreased pulse Decreased respirations |
|
What is Parkinson's Disease?
|
A progressive degenerative disease of the extrapyramidal system
|
|
What is Parkinson's associated with?
|
Dopamine deficiency
|
|
Causes of Parkinson's
|
Imbalance of dopamine and acetylcholine in basal ganglia
Cerebrovascular disease Drugs Head trauma |
|
Key S/Sx of Parkinson's
|
"Pill rolling" tremors
Shuffling gait Stiff joints Masklike facial expression Dyskinesia Stooped posture "Cogwheel" rigidity |
|
Treatment of Parkinsons
|
High-residue, High-calorie, high-protein diet
Soft foods Anticholinergics Antidepreesants Dopamine receptor agents |
|
Key Nursing interventions for Parkinsons
|
Assess neurovascular and respiratory status
Prevent falls Reinforce gait training |
|
Key complications of Parkinsons
|
Depression
Injury Aspiration Corneal ulceration |
|
What is Multiple Sclerosis (MS)?
|
Progressive immune-mediated demyelinating disease of motor and sensory neurons
|
|
Causes of MS
|
Autoimmune disease
Virus Genetic Disposition |
|
Key S/Sx of MS
|
Weakness
Nystagmus Diplopia Blurred vision Paresthesia Impaired sensation Optic neuritis |
|
Treatment of MS
|
Plasmapheresis
Muscle relaxant Glucocorticoids Immunosuppressant Skeletal muscle relaxant |
|
Key nursing interventions for MS
|
Assess neurologic status
Maintain active and passive ROM Establish bowel and bladder elimination |
|
Key complications of MS
|
UTI
Rep tract infection Depression |
|
What is Myasthenia Gravis?
|
Neuromuscular disorder that results in weakness of voluntary muscles
|
|
Causes of Myasthenia Gravis
|
Insufficient acetylcholine
Autoimmune disease |
|
Key S/Sx of Myasthenia Gravis
|
Muscle weakness (increases with activity)
Dysphagia Dysarthria Dysphonia |
|
Treatment of Myasthenia Gravis
|
High-calorie diet
Activity as tolerated Plasmaphoresis anticholinasterases |
|
Key nursing interventions for Myasthenia Gravis
|
Assess neurologic and Respiratory status
Assess swallow and gag reflexes Administer meds before meals to maximize muscles for swallowing. |
|
What does interferon do?
|
Helps regulate immune system
|
|
When does MS strike?
|
20-40s
|
|
What is Guillain-Barre syndrome?
|
Inflammation of spinal and cranial nerves
|
|
What is characteristic of Guillain-Barre?
|
Ascending paralysis
|
|
S/Sx of stage 1 Guillain-Barre
|
Abrupt onset weakness and paralysis
May affect respiration ANS effects |
|
S/Sx of stage 2 Guillain-Barre
|
Plateau
Progression stopped |
|
S/Sx of stage 3 Guillain-Barre
|
Recovery
|
|
Complications of Guillain-Barre
|
Repiratory failure
Infection Depression |
|
Treatment of Guillain-Barre
|
Steroids
Plasmapheresis Life support Rehab |
|
Simple partial seizure
|
symptoms are confined to one hemisphere
|
|
Complex partial seizure
|
Symptoms begin in one focal area, and spread to both hemispheres
|
|
Types of Generalized seizures
|
Absence (petit mal)
Myoclonic Clonic Tonic Tonic-Clonic (grand mal) Atonic Akinetic |
|
What is an absence (petit mal) seizure?
|
Loss of responsiveness
Continued ability to maintain posture control and not fall |
|
What is a tonic-clonic (grand mal) seizure?
|
Violent total body seizure
|
|
S/Sx of absence seizure
|
Can have 100 daily
Twitching eyelids Lip smacking Staring No postictal symptoms |
|
S/Sx of tonic-clonic seizure
|
Aura
Tonic first (20-40 seconds) Clonic next Postictal symptoms Incontinence |
|
Causes of seizures
|
Idiopathic
Head injury Brain tumor |
|
Treating seizure disorders
|
Seizure precautions
Anticonvulsants Diazepam (status epilipeticus) |
|
Care during seizure
|
Pad side rails
Prevent injury Monitor airway Observe and document |
|
Complications of seizure disorder
|
Musculoskeletal injury
Hypoxia |
|
Status Epilepticus
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Life threatening
30 minutes of seizure activity |
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What is a TIA?
|
"mini-stroke"
Temporary impairment of cerebral circulation Resolves within 24 hours |
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S/Sx of TIA
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Depends on part of the brain affected
-Visual disturbances -Speech difficulty -Weakness -Paralysis -Confusion |
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What causes TIA?
|
Atherosclerosis
Emboli |
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Treatment of TIA
|
Coumadin
Antihypertensives Antiplatelet agents |
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What is CVA?
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Stroke, Brain accident
Infarction and death of brain tissue Permanent Damage |
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What causes CVA?
|
Deficient blood supply (thrombotic, embolic)
Bleeding into the brain |
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Non-modifiable risk factors for CVA
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Age
male gender African american |
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Modifiable risk factors for CVA
|
Hypertension
CV disease Elevated cholesterol Obesity Diabetes Smoking Alcohol use |
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What does Right CVA affect?
|
Motor function
|
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What does Left CVA affect
|
Cognitive
|
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Effects of Right Sided CVA
|
Left side paralyzed
Spacial-perceptual deficits Short attention span Impaired judgement Impulsive |
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Effects of Left Sided CVA
|
Right side paralyzed
Impaired speech and language Slow performance Aware of deficits Impaired comprehension |
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Treatment of CVA
|
TIME IS BRAIN!!
Thrombolytic therapy must be given within 3 hours to be effective Anticoagulants Airway management Surgery |
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Long term effects of CVA
|
Motor function
Aphasia Emotional lability Impaired judgement Unilateral neglect |
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What is an aneurysm?
|
Weakness in artery wall
|
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What is a subarachnoid hemorrhage?
|
Bleed from aneurysm, AV malformation, or Head trauma
|
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S/Sx of hemorrhage
|
Severe headache
IICP Photophobia Vomiting Seizures Stiff neck |
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Treatment of hemorrhage
|
Craniotomy
Blood pressure control Invasive monitoring |
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Complications of hemorrhage
|
Rebleed
Hydrocephalus Vasospasm |
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What does Head trauma result in?
|
Hemorrhage
Contusion Laceration |
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Warning signs the first 24 hours after head injury
|
Changes in LOC
Seizures Drainage from the nose (blood, H2O) Pupils slow to react or unequal Vision problems Loss of sensation Slurred speech Projectile vomiting |
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Treatment for head injury
|
Surgical removal of hematoma
Control IICP Therapeutic coma |
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Complications of head injury
|
Diabetes Insipidus
acute hydrocephalus Labile vital signs Posttraumatic syndrome Cog and personality changes |
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Spinal cord injury does not mean...
|
...that the spinal cord is broken!
|
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What is a spinal cord injury?
|
damage to nerve fibers
*interference with blood flow and brain/body communication |
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Causes of spinal cord injury
|
MVA
Falls Sports injuries Assault |
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S/Sx of Cervical injury
|
Paralysis
Paresthesias Impaired respiration Loss of bladder/bowel control Quadriplegia/paresis |
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Spinal injury at C2 or above...
|
...fatal
|
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S/Sx Thoracic/Lumbar injury
|
Paraplegia/paresis
Bowel and bladder control affected |
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What is meningitis?
|
infection and inflammation of pia mater, subarachnoid and arachnoid space
Possible cranial nerve involvement |
|
S/Sx of meningitis
|
Similar to the Flu!!
-Severe headache -Fever -Photophobia -Petechial rash |
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Late S/Sx of meningitis
|
Nuchal rigidity
N/V Encephalopathy Changes in LOC |
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Complications of meningitis
|
Hydrocephalus
Seizures Resp impairment |
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Ensephalitis is passed by...
|
...mosquitos
|
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What is encephalitis?
|
Inflammation of brain tissue
|
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What does encephalitis result in?
|
nerve damage
IICP Neuro effects depend on area of brain affected |
|
S/Sx of encephalitis
|
Headache
Fever N/V Nuchal rigidity confusion seizures photophobia |
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Complications of encephalitis
|
cognitive disabilities
personality changes ongoing seizures motor deficits blindness |
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Treatment of encephalitis
|
treat the symptoms
|
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What is trigeminal neuralgia?
|
Neuropathy of the trigeminal nerve
Affects sensory portion of nerve |
|
S/Sx of trigeminal neuralgia
|
Pain on one side of face
|
|
TX of trigeminal neuralgia
|
Anticonvulsants (muscle relaxants)
Nerve blocks Surgery |
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What is Bell's Palsy?
|
Inflammation and edema of facial nerve
|
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What is Huntington's disease?
|
Degeneration of parts of the brain
|
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S/Sx of Huntington's
|
Dementia
Choreiform movements Dysphagia Depression Death |
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Tx of Huntington's
|
Antipsychotics
Antidepressants Antichorea agents Fetal tissue transplants |
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S/Sx of Stage 1 Alzheimer's
|
Increasing forgetfulness
|
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S/Sx of Stage 2 Alzheimer's
|
Progressive memory loss
Irritability Depression Aphasia Sleep disruption Hallucinations Seizures |
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S/Sx of Stage 3 Alzheimer's
|
Complete dependency
Loss of bowel and bladder control Loss of emotional control Inability to recognize significant others |
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Medical Treatment of Alzheimer's
|
Cholinesterase inhibitors
Antidepressants Antipsychotics Antianxiety agents |
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Best method of mental stimulation for Alzheimer's
|
Crossword puzzles
Memory games |
|
What is Amyotrophic Lateral Sclerosis
(ALS, Lou Gherig's Disease) |
Degeneration of peripheral motor neurons
Blocked nerve impulse transmission Muscle weakness and atrophy |
|
S/Sx of ALS
|
Progressive muscle weakness
Decreased coordination Muscle spasms Speech difficulty Pulmonary comprimise Death in 3-5 years |
|
Treatment of ALS
|
*Focus on symptomatic relief
Muscle relaxants PT/OT/ST Pain control Tube feedings Prevent infection |