• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/24

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

24 Cards in this Set

  • Front
  • Back
INFO Reading:
Bickley,LS, Bates’ Guide to Physical Examination and History Taking. 10th ed. Philadelphia, PA: Lippincott Williams & Wilkins; 2009: 323-371.
.
a. PMI
apical impulse/point of maximal impulse brief early pulsation of left ventricle-moving anteriorly during contraction, touching chest wall. Page 357, 20%.
(Red side note) palpable in 25-40% of healthy adults and 50% and left decubitus position.
S1

b. Page 355/356 Bates
first sound/start of systole/contraction of atrium's,
S2
second sound/start of diastole/contraction of ventricles,
S3
(Wikipedia) caused by the oscillation of blood back and forth between the walls of the ventricles initiated by inrushing blood from the atria., protodiastolic gallop, ventricular gallop, or informally the "Kentucky" gallop
S4
(Wikipedia) sound occurs just after atrial contraction ("atrial kick") at the end of diastole and immediately before S1, presystolic gallop or atrial gallop (Tennessee.) blood being forced into a stiff/hypertrophic ventricle.
c. Split S2 :


Bates page 365, 10%/Bates page 380 0%
Splitting

Why: either aortic valve shut early or pulmonary shut late.
Increased normal splitting [wider split when inspire] DDx:
• Delayed RV emptying (pulmonary stenosis, RBBB).  
d. Systole (systolic)


Bates 327 45 %/page 326 entirety
.
e. Diastole (diastolic)


Bates, 327, 20%/page 326 entirety
.
f. Pansystolic (holosystolic)


page 366, right, 50%
.
g. Continuous (as applied to heart sounds)
plateau murmur equals same intensity throughout. Page 367 left 60%
h. Murmur =
turbulent blood flow. page 329, 90%
i. Thrill (as applied to heart sounds)
vibration palpable from a murmur
j. Stenosis (as applied to each heart valve)
stenotic valve which has an abnormally narrowed valvular orifice that obstructs blood flow.

Page 329 Bates 95%.
k. Regurgitation (as applied to each heart valve)
back flow of blood through a valve. Page 329 98%
l. Insufficiency
the inability to carry sufficient amounts of oxygenated blood for the needed demand to the peripheral system. (Handout)
m. Crescendo
one marked by progressively increasing loudness.
n. Decrescendo
one with increasing intensity until mid- to late systole, then a decreasing intensity, giving a diamond-shaped tracing on phonocardiography. Characteristic of pulmonary stenosis.
Murmurs: grading
Graded on scale of 1 to 6.
1.Only cardiologist can hear.
2.Trained doctor can hear.
3.Student can hear. No thrill.
4.Thrill barely palpable.
5.Thrill easily palpable.
6.Can hear murmur by being in the room without a stethoscope.
Murmurs: general considerations
Can be either systolic or diastolic.
-Students will only hear systolic.
Mnemonic for whether murmur is systolic diastolic:
• PASS and PAID:

• Pulmonary, Aortic Stenosis = Systolic.

• Pulmonary, Aortic Insufficiency = Diastolic.

• Then mitral and tricuspid must be opposite to these.
Systolic click
a-Sound:
b-Where:
c-Why:
d--DDx:
a-Sound: high-pitched click, soon after S1. Click followed by AS or PS murmur.

b- Where:aortic, pulmonary ausc sites.

c- Why: stiff aortic valve suddenly opened.

d- DDx: AS(aortic stenosis).
Opening snap?

Sound:
Where:
Why:
DDx:
Sound: high-pitched click after S2.

Where: lower L sternal edge.

Why: stiff mitral valve suddenly opened.

DDx: • Mitral stenosis.
4th heart sound?
Sound:
Where:
Why:
DDx:
Sound: higher pitch, late diastole, "gallop sounding"

Where:

Why: ventricular filling resistance, always pathologic.

DDx: •HTN • MI • AS • Heart block
3rd heart sound
Sound:
Where:
Why:
DDx:
Sound: in early-mid diastole, low-pitched, "gallop sounding".

Where: apex, louder on expiration.

Why: ventricular distension, may be normal.

DDx: • Normal in children
• Constrictive pericarditis.
• Mitral regurgitation
• Tricuspid regurgitation
• LVF, RVF