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53 Cards in this Set

  • Front
  • Back
CV system
heart, blood, blood vessels

transport o2, remove wasteq
efficiency of CV system depends on
ability of heart to pump
patency of blood vessels
quality of blood
quantity of blood
Right heart pumps to
lungs
left heart pumps to
body
layers of heart
endocardium (lines chamber)
myocardium (muscle)
coronary arteries
epicardium - only a few cells thick
left main coronary artery
widow maker
blocks circulation to all rest of LV
coronary artery fills during
diastole
empties to RA
workhorse of heart
Left Ventricle
SA node
pacemaker
intrinsic rate 60-100
AV node
intrinsic rate 40-60
bundle branch rate
20-30
which cell is pacemaker?
cell that beats fastest
order of conduction system
SA, AV, bundle of His, left/right bundle branch
Electrolytes in heart at rest for polarization
Na out of cell
K in cell
Electrolytes in heart during contraction
na moves into cell
K moves out of cell
electrolytes in heart when recovering
na moves back outside cell
k moves back into cell
tall T, arrythmias
Hyperkalemia
lo T, u wave
hypokalemia
calcium realated to contractility
aids in contractility

if high (hypercalcemia) irritability happens - cause arrythmias
heart and PSNS
resting time
negative chronotropic - slows HR
negative inotropic - less work for heart
heart and SNS
postive chronotropic - beat fast
positive inotropic - heart beats hard
catecholamines
epinephrine
norepi
dopamine

produce SNS effect
thyroid hormone
can increase HR higher than normal

(BMR)
capillaries
all exchange occurs - o2, nutrients, waste
lymphatic
feeds into veins
what % blood plasma
55
(90% water, albumin, gamma globulin, fibrinogen, etc)
what % blood is solid particles
45, includes
WBC
RBC
WBC
leukocytes
5,000-10,000
short lifespan
hematocrit
% RBC

men: 42-50%
women: 40-48%
hemoglobin
male: 13-18
female: 12-16
Hg:Hct ratio
1:3
thrombocytes
platelets
100,000-400,000
digoxin
listen at PMI for 60s
digoxin slows a too fast HR
if HR less than 60, hold digoxin
Cardiac output
CO = SV x HR
stroke volume
amount of blood ejected from the heart with each contraction

usually 70-80
preload
amount of volume returning to Right heart
dependent on venous return
preload in FVD
decreased preload
preload in FVE
hi preload
afterload
arterial resistance

LV has to overcome aortic pressure
indirect measures of CO
2+ pulses
skin warm, dry
good capillary refill
BP WNL
good Urine outpt
AOx3
HR WNL
RR WNL
clear breath sounds
direct measures of CO
swan ganz
cardiac cath
baroreceptors
respond to change in BP

lo BP - inc SNS
chemoreceptors
respond to cahnge in acidosis, hypoxia
EKG, holter monitor
looks at electrical activiyt of the heart
12 lead ECG
diagnose exactly what's going on
more complicated diagnosis
see 12 angles of heart
echo
look at sound waves
same principle as sonogram
external echo
lay on L to get heart closer to chest
internal echo
if obese, inc chest muscle

trans esophogeal EKG - small transducer in esophogas
stress test
non invasive
stress pt under controlled condition
exercise or pharmacological
contraindications for stress test
severe aortic stenosis
MI
severe hypertension - could cause stroke
RN interventions for stress test
usually npo 4-6 h
no caffiene, tobacco before
stop beta blockers 24 h before
coronary artery can dilate
up to 4x normal to deliver o2 under stress
radionucleotide imagery
thallium 201
technetium