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53 Cards in this Set

  • Front
  • Back
list % of dehyd for mild, mod, severe for infant vs child?
infant: 5, 10, 15%
child: 3,6,9 %
poor skin turgor, sunken fontanel, lack of tears, lethargy, taccy ? how much dehyd?
mild; infant: 5%; child 3%
orthostatic hyptn, sign tacy, oliguria, deepeneing lethargy? how much dehyd
moderate
shock w/ dehyd; how much dehyd?
severe
FeNA < 1 % or (2.5% in NB) is what?
dehydration; renal Na conservation;
-kidney compensates by reabsorbing Na
% FeNa =
urine Na/plasma Na // urine Cr / plasma Cr x 100
= urine na/plasma Na x plasma cr/ urine cr x 100
? how much basal caloric expenditure for every degree > 37.8?
add 12 % extra
how calc fliuds in 24 hour?
4/2/1:
10/10/next; then x 24 hours
BSA: ? ml/m2/24 hours;
Na? mEq / m2 / 24 hrs
K ? meQ/ m2 / 24 hrs
BSA: 1500 ml/m2/24 hours;
Na 30-50 mEq / m2 / 24 hrs
K 20-40 meQ/ m2 / 24 hrs
? correction of deficits:
bolus 20/kg
-mainteince + ongoing losses
-volume repletion: give first 50% in first 8 hrs, remainder in next 16 hrs
ORS: for mild vs moderate? vs severe?
5-10 cc q 5-10 min;
Mild: 50 cc/kg/ x 4 hrs;
Mod: 100 cc/kg over 4 hrs;
not effect in severe dehydration: shock, obtundation; > 10 cc/k/hr
calc serum osmolality?
N x 2 + K x 2 + glucse/18 + BUN/ 3;
NL ~ 290-300; serum Na major player
head trauma, pituatary infarction/ tumors leads to ? that causes hypernatremia
Central DI
hypernetremia, boys; inheritance?
nephrogenic DI; loss of V2 receptor activity
low water intake, water los, inapp salt poison; leads to hi NA ~ also associated w/ other cause?
inapp prepared formula
hypernatremia in baby being Breast fed is due to what?
CF in mom; mastitis
Irritable, high pitched cry, doughy skin texture, convulsions; stupor paralysis, CNS damage;
-death
signs of hypernatremia
if Na < 170 , how fast correct vs > 170?
< 170, correct over 48 hours;
> 170: correct over 3 days (reduce by 15 mEq/ day)
kids look more hydrated; taccy, low BP may present w/ o w/o dehdyration
-Neuro: Ha, obtundation, coma, status, cerebral edema, herniation;
hyponatremia;
-rapid correction: central pontene myelinolysis
Na < 130; Posm < 280, urine that is not m aximally dilute U osm > P osm; but normal renal fxn
-? causes
SIADH:
causes: cns diz, pulm diz, surgery, N/V/pain, stress
Normal Urine osm
nl urine osm >> 290; if
SIADH tx?
3 % saline if severe (Na < 120;
-otherwise fliud restriction
? causes Hi ADH: drugs?
cytoxan, vincristine
6 yo w/ abd pain, vomiting, confused;
Lytes: 125/5/93/7 20/1/ gluc 900;
serum osm (317)
urine osm 600
DM w/ Pseudohyponatremia;
-hi glucose can contribute to hypnoatremia

low na,
6 yo w/ abd pain, vomiting, confused

122/3.6/93/22/10/0.6 glc 100
serum osm: 260; urine osm 350
SIADH: vomiting or CNS process
low aldo, hi K ; which type of RTA? ; -what meds also cause low aldo?
type 4;
ACE I
-NSAIDS;
normal or hi aldo w/ renal tubular unresponsive to mineralcorticoids
obstructive uropathy, Sickle cell SLE
? peaked T waves, long PR, wide QRS; ST depression due to what? treatment?
hi K;
1) IV calcium gluconate (protect heart)
2); sodium bicarb: correct acidosis;
3)insulin, dextrose;
albuterol
Lasix: renal K wasting
T wave flattening, constipation; weakness, cramps, rhabdo:
hypokalemia;
alkalosis and hypokalemia ~ ?
vomiting
hypokalemia and acidosis,
diarrhea
12 wt loss P 80/30;
bmp: 140/6/120/10/10/0.6:
next step?
Adrenal insufficiency (addison's): no aldo--> hi renin: low BP, hi K
-next: serum renin (should be high and aldo should be low
-tan ( no melanin)
12 yo wt loss, short, anemia, bp: 80/30
bmp: 140/6/120/10/75/5.2
cause?
Chronic kidney disease: afects bone/blood
1) low bicarb, low PcO2:
2) Hi bicarb, hi PCO2
3) hi pCO2, HI bicarb: causes?
4) low pCO2, low bicarb?
1) met acidosis
2) met alkalosis: ~ hypokalemia; w/u: check Urin CL
3) resp acidosis
4) resp alkalosis
urin Cl and BP: ? for metab alkalosis?
Nl urine cl: (> 15);
1) - if urine cl < 15 w/ nL BP: Pyloric stenosis, CF;
2) urine cl > 15: renal cause or lasix;
? hypercalciuria/nephorcalcinosis; hearing loss;
what's causes and acid/base disturbance
loop duirteics; metab alkalosis
polyhydramnios in utero; hypercalcuria; +/- hearing loss; hi urine cl
barter's; looks like person on loop diuretics
alkalotic, hypertensive, hi urine cL, ;
-low renin/low aldo:
Liddle
alkalotic, hypertensive, hi urine cL,
-ambig genitalia; ? most common kind and inheritence?
CAH (11) ~ HTN; most common is 21
hi renin/high aldo; alkalotic, hypertensive, hi urine cL,
RAS
cal AG?
Na - (cl + HCO3)
NL 9-12
name + AG met acidosis:
-what are most common?
Methanol
Uremia
DKA***
Paraldehyde
Isoniazide/iron
lactic acidosis*** dehydration/sepsis an
ethylene glycol~ anti freeze
salicylates~ metabolic acidosis/resp alkalosis
Most common cause of non-gap acidosis? *** boards
GI losses - diarrhea, fistulas,
-RTA
HTN, hi urine cl, low renin/high aldo?
aldosterone escess
5 wk, bp 70/30;
bmp: 128/2.8/82/35 10/0.6 glc 100;
BG: 7.5/45
urine Cl 75
Urine ca/cr: 1
Barter's syndrome ( not CF - kidney would hold on Cl)
alkalotic; pcO2; hi bicarb: metabl alkalosis
hypokalemia; hi ur ca/cr ratio (nl < 0.2);
hi urine cl (nl 15)
hyperchloremia
hypokalemia
Non gap acidosis
short,
nephrocalcinois + rickets
RTA:
? urine anion gap:?
urine (na + K) - Cl;
type I: gap +
type II: gap neg
proximal form, wasting bicarb in prox tubule; decreased threshold; distal tube can secrete H;
-aciditic urine; ; urine pH < 5.5; neg urine AG; low K
-
type II
low aldo: hihg K; urine Ph > 5.5
type IV
low K, urine pH > 5.5; urine AG: 0 to +
distal I RTA
sensorineural deafness, rickets, nephrocalcinosis/hi ur ca/cr ratio? RTA?
type I distal hypkalemic
which RTA ~ fanconi's syndrome, renal wasting phosphate, aa, bicarb; urate, glucose (cystinosis); rickets?
type II (proximal)
which RTA ~ hypoaldosteronism, addison's renal resistance to aldo; obstructive uropathy?
type IV (distal hyperkalemic)