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53 Cards in this Set
- Front
- Back
list % of dehyd for mild, mod, severe for infant vs child?
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infant: 5, 10, 15%
child: 3,6,9 % |
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poor skin turgor, sunken fontanel, lack of tears, lethargy, taccy ? how much dehyd?
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mild; infant: 5%; child 3%
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orthostatic hyptn, sign tacy, oliguria, deepeneing lethargy? how much dehyd
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moderate
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shock w/ dehyd; how much dehyd?
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severe
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FeNA < 1 % or (2.5% in NB) is what?
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dehydration; renal Na conservation;
-kidney compensates by reabsorbing Na |
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% FeNa =
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urine Na/plasma Na // urine Cr / plasma Cr x 100
= urine na/plasma Na x plasma cr/ urine cr x 100 |
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? how much basal caloric expenditure for every degree > 37.8?
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add 12 % extra
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how calc fliuds in 24 hour?
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4/2/1:
10/10/next; then x 24 hours |
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BSA: ? ml/m2/24 hours;
Na? mEq / m2 / 24 hrs K ? meQ/ m2 / 24 hrs |
BSA: 1500 ml/m2/24 hours;
Na 30-50 mEq / m2 / 24 hrs K 20-40 meQ/ m2 / 24 hrs |
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? correction of deficits:
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bolus 20/kg
-mainteince + ongoing losses -volume repletion: give first 50% in first 8 hrs, remainder in next 16 hrs |
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ORS: for mild vs moderate? vs severe?
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5-10 cc q 5-10 min;
Mild: 50 cc/kg/ x 4 hrs; Mod: 100 cc/kg over 4 hrs; not effect in severe dehydration: shock, obtundation; > 10 cc/k/hr |
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calc serum osmolality?
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N x 2 + K x 2 + glucse/18 + BUN/ 3;
NL ~ 290-300; serum Na major player |
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head trauma, pituatary infarction/ tumors leads to ? that causes hypernatremia
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Central DI
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hypernetremia, boys; inheritance?
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nephrogenic DI; loss of V2 receptor activity
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low water intake, water los, inapp salt poison; leads to hi NA ~ also associated w/ other cause?
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inapp prepared formula
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hypernatremia in baby being Breast fed is due to what?
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CF in mom; mastitis
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Irritable, high pitched cry, doughy skin texture, convulsions; stupor paralysis, CNS damage;
-death |
signs of hypernatremia
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if Na < 170 , how fast correct vs > 170?
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< 170, correct over 48 hours;
> 170: correct over 3 days (reduce by 15 mEq/ day) |
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kids look more hydrated; taccy, low BP may present w/ o w/o dehdyration
-Neuro: Ha, obtundation, coma, status, cerebral edema, herniation; |
hyponatremia;
-rapid correction: central pontene myelinolysis |
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Na < 130; Posm < 280, urine that is not m aximally dilute U osm > P osm; but normal renal fxn
-? causes |
SIADH:
causes: cns diz, pulm diz, surgery, N/V/pain, stress |
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Normal Urine osm
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nl urine osm >> 290; if
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SIADH tx?
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3 % saline if severe (Na < 120;
-otherwise fliud restriction |
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? causes Hi ADH: drugs?
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cytoxan, vincristine
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6 yo w/ abd pain, vomiting, confused;
Lytes: 125/5/93/7 20/1/ gluc 900; serum osm (317) urine osm 600 |
DM w/ Pseudohyponatremia;
-hi glucose can contribute to hypnoatremia low na, |
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6 yo w/ abd pain, vomiting, confused
122/3.6/93/22/10/0.6 glc 100 serum osm: 260; urine osm 350 |
SIADH: vomiting or CNS process
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low aldo, hi K ; which type of RTA? ; -what meds also cause low aldo?
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type 4;
ACE I -NSAIDS; |
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normal or hi aldo w/ renal tubular unresponsive to mineralcorticoids
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obstructive uropathy, Sickle cell SLE
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? peaked T waves, long PR, wide QRS; ST depression due to what? treatment?
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hi K;
1) IV calcium gluconate (protect heart) 2); sodium bicarb: correct acidosis; 3)insulin, dextrose; albuterol Lasix: renal K wasting |
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T wave flattening, constipation; weakness, cramps, rhabdo:
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hypokalemia;
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alkalosis and hypokalemia ~ ?
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vomiting
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hypokalemia and acidosis,
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diarrhea
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12 wt loss P 80/30;
bmp: 140/6/120/10/10/0.6: next step? |
Adrenal insufficiency (addison's): no aldo--> hi renin: low BP, hi K
-next: serum renin (should be high and aldo should be low -tan ( no melanin) |
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12 yo wt loss, short, anemia, bp: 80/30
bmp: 140/6/120/10/75/5.2 cause? |
Chronic kidney disease: afects bone/blood
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1) low bicarb, low PcO2:
2) Hi bicarb, hi PCO2 3) hi pCO2, HI bicarb: causes? 4) low pCO2, low bicarb? |
1) met acidosis
2) met alkalosis: ~ hypokalemia; w/u: check Urin CL 3) resp acidosis 4) resp alkalosis |
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urin Cl and BP: ? for metab alkalosis?
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Nl urine cl: (> 15);
1) - if urine cl < 15 w/ nL BP: Pyloric stenosis, CF; 2) urine cl > 15: renal cause or lasix; |
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? hypercalciuria/nephorcalcinosis; hearing loss;
what's causes and acid/base disturbance |
loop duirteics; metab alkalosis
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polyhydramnios in utero; hypercalcuria; +/- hearing loss; hi urine cl
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barter's; looks like person on loop diuretics
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alkalotic, hypertensive, hi urine cL, ;
-low renin/low aldo: |
Liddle
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alkalotic, hypertensive, hi urine cL,
-ambig genitalia; ? most common kind and inheritence? |
CAH (11) ~ HTN; most common is 21
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hi renin/high aldo; alkalotic, hypertensive, hi urine cL,
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RAS
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cal AG?
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Na - (cl + HCO3)
NL 9-12 |
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name + AG met acidosis:
-what are most common? |
Methanol
Uremia DKA*** Paraldehyde Isoniazide/iron lactic acidosis*** dehydration/sepsis an ethylene glycol~ anti freeze salicylates~ metabolic acidosis/resp alkalosis |
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Most common cause of non-gap acidosis? *** boards
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GI losses - diarrhea, fistulas,
-RTA |
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HTN, hi urine cl, low renin/high aldo?
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aldosterone escess
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5 wk, bp 70/30;
bmp: 128/2.8/82/35 10/0.6 glc 100; BG: 7.5/45 urine Cl 75 Urine ca/cr: 1 |
Barter's syndrome ( not CF - kidney would hold on Cl)
alkalotic; pcO2; hi bicarb: metabl alkalosis hypokalemia; hi ur ca/cr ratio (nl < 0.2); hi urine cl (nl 15) |
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hyperchloremia
hypokalemia Non gap acidosis short, nephrocalcinois + rickets |
RTA:
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? urine anion gap:?
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urine (na + K) - Cl;
type I: gap + type II: gap neg |
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proximal form, wasting bicarb in prox tubule; decreased threshold; distal tube can secrete H;
-aciditic urine; ; urine pH < 5.5; neg urine AG; low K - |
type II
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low aldo: hihg K; urine Ph > 5.5
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type IV
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low K, urine pH > 5.5; urine AG: 0 to +
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distal I RTA
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sensorineural deafness, rickets, nephrocalcinosis/hi ur ca/cr ratio? RTA?
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type I distal hypkalemic
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which RTA ~ fanconi's syndrome, renal wasting phosphate, aa, bicarb; urate, glucose (cystinosis); rickets?
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type II (proximal)
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which RTA ~ hypoaldosteronism, addison's renal resistance to aldo; obstructive uropathy?
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type IV (distal hyperkalemic)
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