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18 Cards in this Set

  • Front
  • Back
hemolytic-uremic syndrom
Common in children
Causes acute renal failure
Commonly caused by E.coli (O157:H7)
Toxins damage endothelium, reduce NO, promote vassoconstriction and necrosis, promote thrombus
- Microangiopathic hemolytic anemia
Wegener's granulomatosis
Vasculitis in adults
IgA nephropathy
Most often in young adults.
Expanded Mesangial matrix.
Electron dense humps
In post infectious GN
IgG and C3
Variable thickening of BM
Mesangial Matrix expanded
DM, IgA nephropathy
post infectious GN
IgG and C3
Electron dense humps
hematuria and RBC casts
No hematuria
Nodular and diffuse glomeroscloeosis, leads to chronic renal failure
mild with prostate hyperplasia, high with carcinoma
paraneoplastic origin
polycyctic kidney dz
autosomal dominant
Renal cell carcinoma
Ischemic tubular necrosis
Rupture of tubular BM (tubulorrhexis)
1 day initiating phase, maintenance phase follows (rise in blood urea, w/ salt and water overload)
Steady rise in urine output with hypokalemia
Fibrinoid necrosis
malignant nephrosclerosis
Focal segmental
treatment is with corticosteroids, and ACE-inhibitors to control hypertension.
- hyalinosis, with a gradual progression
to diffuse global glomerulosclerosis. Immunofluorescence studies show IgM and C3 deposition.
- prognosis is poor, with fifty percent of patients progressing to end-stage
kidney disease in five years. The prognosis is better in children. The lesion recurs in 25-50% of transplanted kidneys, indicating an
extrarenal primarycause.
Kimmelstiel-Wilson lesion.
Diabetic Nephropathy. poor prognosis.
Nephrotic syndrome due to this case have chronic renal
failure. Treatment with ACE-inhibitors. Early detection of microalbuminuria
Elevated glycosylated Hgb A1c
Initially microalbinuria
Progressive loss of renal fn
ANCA positive
vasculitis (Wegener's)