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20 Cards in this Set

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  • Back
Addison's disease
inadequate amounts of corticosteriod hormones
Cushing's disease
overabundance of corticosteriod hormones
Histamine
chemical mediator of the inflammatory response released from mast cells.
What do histamines cause?
dilation and increased permeability of small blood vessels, constriction of small airways, increased secretion of mucus in airways and pain
Iatrogenic
caused by physician/vet
Nerve Block
loss of feeling or sensation produced by injfecting an anesthetic agent around a nerve to interfere with its ability to conduct impulses
Modulation
modification of nociceptive transmission
Prostoglandin
synthesized by cells from arachidonic acid that serves as a mediator of inflammation
Regional Anesthesia
loss of feeling or sensation in a large area of the body after injection of anesthetic agent into spinal canal or around peripheral nerves
Transduction
process that involves translation of noxious stimuli into electrical activity at sensory nerve endings
Nociceptors
"naked" nerve endings from which pain arises
Path of pain
nociceptors - A & C fibers - dorsal horn of spinal cord - thalamus - cerebral cortex
Hyperalgesia
area of tissue injury becomes more sensitive and threshold for subsequent stimuli decreases
NSAIDs mode of action
inhibit cyclooxygenase enzymes
cyclooxygenase 1 (cox 1)
maintains physiological functions like modulation of renal blood flow and synthesis of gastric mucosa
Cyclooxygenase 2 (cox 2)
promotes formlastion of prostoglandin from cell membrane arachidonic acid
Glucocorticoids mode of actions
block phospholipase
Phospholipase
enzyme necessary for production of prostroglandins and leukotrienes
NSAIDs side effects
gi ulceration & bleeding, nephrotoxicity, inhitibion of cartilage metabolism, bone marrow supression, bleeding tendencies
NSAID drugs (6)
Salicylates (aspirin)
Phenylbutazone
Flunixin
Dimethyl Sulfoxide (DMSO)Acetaminophen
Carprofen